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Mathias Jucker
Little Proteins, Big Clues After a Quarter of a Century, the Amyloid Hypothesis for Alzheimer’S Disease Is Reconnecting to Its Roots in Prion Research
CSF Protein Dynamics in Murine Models of A-Synucleinopathy and Cerebral B-Amyloidosis
Annual Report 2017
Dr. Melanie Meyer-Luehmann Curriculum Vitae
EXTRA VIEWS Ab Seeds and Prions
In Focus Neurology.Org/N
Modeling the Prion Aspect of Cerebral Β-‐Amyloidosis in Organotypic Slice
Aβ Seeds and Prions: How Close the Fit? Jay Rasmussen, University of Tübingen Mathias Jucker, University of Tübingen Lary Walker, Emory University
Endogenous Murine AΒ Increases Amyloid Deposition In
Corruptive Templating of Aβ in Alzheimer's Disease
Monkey, Mouse Or Zebrafish? Ethical and Scientific Considerations in Choosing Model Organisms for Animal Experiments
2005 Program
Conference Report: Dementia Research and Care and Its Impact in Switzerland
Β-Amyloid in Alzheimer's Disease Initiation and Phenotypic
Modeling Familial Danish Dementia in Mice Supports the Concept of the Amyloid Hypothesis of Alzheimerts Disease
Amyloidosis and Neurofibrillary Tau Pathology
Research and Perspectives in Alzheimer's Disease
Conversion of Synthetic Aβ to in Vivo Active Seeds and Amyloid Plaque
Top View
Self-Propagation of Pathogenic Protein Aggregates in Neurodegenerative Diseases Mathias Jucker, University of Tübingen Lary C Walker, Emory University
2017 Isn-Esn Meeting
Amyloid-Β Pathology Induced in Humans
PDF Hosted at the Radboud Repository of the Radboud University Nijmegen
Proteopathic Seeds and Neurodegenerative Diseases”
The Exceptional Vulnerability of Humans to Alzheimer’S Disease Lary C Walker, Emory University Mathias Jucker, University of Tübingen
Amyloid-Associated Neuron Loss and Gliogenesis in the Neocortex of Amyloid Precursor Protein Transgenic Mice
The Prion-Like Properties of Amyloid-Β Assemblies: Implications for Alzheimer's Disease