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FNIP1
Fnip1 Regulates Skeletal Muscle Fiber Type Specification, Fatigue Resistance, and Susceptibility to Muscular Dystrophy
Analysis of the Indacaterol-Regulated Transcriptome in Human Airway
Loss of C9orf72 Enhances Autophagic Activity Via Deregulated Mtor and TFEB Signaling
WDR41 Supports Lysosomal Response to Changes in Amino Acid Availability
Genome-Wide Expression Profiling Establishes Novel Modulatory Roles
NRF1) Coordinates Changes in the Transcriptional and Chromatin Landscape Affecting Development and Progression of Invasive Breast Cancer
C9orf72 Binds SMCR8, Localizes to Lysosomes and Regulates Mtorc1 Signaling
Integrative Analysis of Disease Signatures Shows Inflammation Disrupts Juvenile Experience-Dependent Cortical Plasticity
Folliculin Variants Linked to Birt-Hogg-Dubé Syndrome Are Targeted for Proteasomal Degradation
Variation in Protein Coding Genes Identifies Information Flow
Defining the Role of Folliculin and Its Interacting Partners
FLCN Antibody (C-Term) Affinity Purified Rabbit Polyclonal Antibody (Pab) Catalog # Ap8658b
Genomic Analyses Implicate Noncoding De Novo Variants in Congenital Heart Disease
57612 FNIP2 (D3T8Z) Rabbit Mab
The Mitf/TFE Family of Transcription Factors: Master Regulators of Organelle Signaling, Metabolism, and Stress Adaptation Logan Slade and Thomas Pulinilkunnil
What Is BHD? Contents 1
Loss of Fnip1 Results in Renal Cyst Formation and Synergizes with TSC1 Loss to Promote Mtorc1 Activation
Atlas Journal
Top View
Lncrna DRAIR Is Downregulated in Diabetic Monocytes and Modulates Inflammatory Phenotype Via Epigenetic Mechanisms
Cryo-EM Structure of C9ORF72-SMCR8-WDR41 Reveals
Anti-FNIP1 Antibody (ARG22626)
Ubiquitin-Dependent Control of Myogenic Development: Mechanistic Insights Into Getting Huge, and Staying Huge
Cryo-EM Structure of C9ORF72–SMCR8–WDR41 Reveals the Role As a GAP for Rab8a and Rab11a
Developmental Biology Mutation of Fnip1 Is Associated
Hasumi Et Al., 2008
Ontology-Based Validation and Identification of Regulatory
Fnip1 Regulates Skeletal Muscle Fiber Type Specification, Fatigue Resistance, and Susceptibility to Muscular Dystrophy
Folliculin Haploinsufficiency Causes Cellular Dysfunction of Pleural Mesothelial Cells
Glomerular Expression Pattern of Long Non-Coding Rnas in the Type 2
Loss of FLCN-FNIP1/2 Induces a Non- Canonical Interferon Response In
Alternative Splicing Regulates Vesicular Trafficking Genes in Cardiomyocytes
Fnip1 Regulates Skeletal Muscle Fiber Type Specification, Fatigue Resistance, and Susceptibility to Muscular Dystrophy
Recruitment of Folliculin to Lysosomes Supports the Amino Acid–Dependent Activation of Rag Gtpases
PRODUCT SPECIFICATION Product Datasheet
Supplementary Table 1. a Full List of Cancer Genes
Baba Et Al., 2006