DOCSLIB.ORG
Explore
Sign Up
Log In
Upload
Search
Home
» Tags
» CBLB (gene)
CBLB (gene)
CBL Mutations in Myeloproliferative Neoplasms Are Also Found in the Gene’S Proline-Rich Domain and in Patients with the V617FJAK2
Supplementary Information Method CLEAR-CLIP. Mouse Keratinocytes
Anti-CBLB Antibody (ARG57312)
Upregulation of E3 Ubiquitin Ligase CBLC Enhances EGFR
Brief Genetics Report
Polymorphic Variation in the CBLB Gene in Human Type 1 Diabetes
Brief Genetics Report
Quantitative Interactomics in Primary T Cells Unveils TCR Signal
Oncogenic Signaling by Leukemia-Associated Mutant Cbl Proteins Scott Nadeau1,2, Wei An1,2, Nick Palermo1, Dan Feng1, Gulzar Ahmad1, Lin Dong, Gloria E
Mutant Cbl Proteins As Oncogenic Drivers in Myeloproliferative Disorders
Cellular Immunology Regulation of Immune Responses by E3 Ubiquitin
Short Loop Motif Profiling of Protein Interaction Networks in Acute Myeloid
Tumor Rejection in Cblb−/− Mice Depends on IL-9 and Th9 Cells
Original Article
Cblb) Is a Critical Failsafe Against Autoimmune Disease Due to Autoimmune Regulator (Aire)Deficiency
Co-Recruitment Analysis of the CBL and CBLB Signalosomes in Primary
Rewiring of B Cell Receptor Signaling by Epstein–Barr Virus LMP2A
Intestinal Mucosa-Derived DNA Methylation Signatures in The
Top View
Ndfip1 Mediates Peripheral Tolerance to Self and Exogenous Antigen by Inducing Cell Cycle Exit in Responding CD4 T Cells
Gain of CBL-Interacting Protein, a Possible Alternative to CBL Mutations in Myeloid Malignancies
E1321-Catching the Driver Mutations in Ewing Sarcoma Tumours: an In
Ubiquitin Ligase Cbl-B and Obesity-Induced Insulin Resistance
Function CBLB Links Genetic Risk with Type I IFN Associated Variant Of
Recruitment Analysis of the CBL and CBLB Signalosomes in Primary T Cells Identifies CD5 As a Key Regulator of TCR-Induced Ubiquitylation
Analysis of Mirna Expression Profiles in the Liver of Clock∆ 19 Mutant Mice
Decoding the Role of CBLB for Innate Immune Responses Regulating 2 Systemic Dissemination During Non-Tuberculous Mycobacteria 3 Infection
Autoinhibition and Phosphorylation-Induced Activation Mechanisms of Human Cancer and Autoimmune Disease-Related E3 Protein Cbl-B
Negative Regulation of Nk Cell Activation by Cbl-B and Tam Receptors
T Cell Receptor Genotype and Ubash3a Determine Susceptibility to Rat Autoimmune Diabetes
Table of Contents List of Investigators
Genome-Wide CRISPR Screens in Primary Human T Cells Reveal Key Regulators of Immune Function
To Type 1 Diabetes Islet-Reactive CD4 T Cells and Susceptibility
Cbl-B Degradation Modulates T Cell Responses by Controlling Protein
CD5 Signalosome Coordinates Antagonist TCR Signals to Control the Generation of Treg Cells Induced by Foreign Antigens
Targeting CBLB As a Potential Therapeutic Approach For
Integrative Phosphoproteome and Interactome Analysis of the Role of Ubash3b in BCR-ABL Signaling