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ASXL1
From Clonal Hematopoiesis to Therapy-Related Myeloid Neoplasms: the Silent Way of Cancer Progression
The Landscape of Somatic Mutations in Epigenetic Regulators Across 1,000 Paediatric Cancer Genomes
Familial and Somatic BAP1 Mutations Inactivate ASXL1/2-Mediated Allosteric
Frequent Mutation of the Polycomb-Associated Gene ASXL1 in the Myelodysplastic Syndromes and in Acute Myeloid Leukemia
Role of the Bap1/Asxl1 Complex in Malignant Transformation and Therapeutic Response
The Distinct Biological Implications of Asxl1 Mutation and Its Roles In
Oncoscore: a Novel, Internet-Based Tool to Assess the Oncogenic Potential of Genes
Chromatin Regulator Asxl1 Loss and Nf1 Haploinsufficiency Cooperate to Accelerate Myeloid Malignancy
ASXL1 Mutations in Younger Adult Patients with Acute Myeloid Leukemia: a Study by the German-Austrian Acute Myeloid Leukemia Study Group
The Impact of Epigenetic Modifications in Myeloid Malignancies
The BAP1 Deubiquitinase Complex Is a General Transcriptional Co-Activator
BAP1/ASXL1 Recruitment and Activation for H2A Deubiquitination
Bohring-Opitz Syndrome (BOS)
Expression of Cell–Cell Interacting Genes Distinguishes HLXB9/TEL from MLL-Positive Childhood Acute Myeloid Leukemia
Pathogenic ASXL1 Somatic Variants in Reference Databases Complicate Germline Variant
ASXL1 Mutation Analysis Test Description 2016 03 03.Indd
ASXL1 and SETBP1 Mutations and Their Prognostic Contribution in Chronic Myelomonocytic Leukemia: a Two-Center Study of 466 Patients
Promoterless Transposon Mutagenesis Drives Solid Cancers Via Tumor Suppressor Inactivation
Top View
RAS Mutations Drive Proliferative Chronic Myelomonocytic Leukemia Via Activation of a Novel KMT2A-PLK1 Axis
Mayo Prognostic Model for WHO-Defined Chronic
Circasxl1-1 Regulates BAP1 Deubiquitinase Activity in Leukemia
Dynamics of ASXL1 Mutation and Other Associated Genetic Alterations During Disease Progression in Patients with Primary Myelodysplastic Syndrome
PR-DUB Maintains Expression of Critical Genes Through FOXK1/2 and ASXL1/2/3
Mutated ASXL1 and Number of Somatic Mutations As Possible
Inherited Causes of Clonal Haematopoiesis in 97,691 Whole Genomes
ASXL1 (NM 015338) Human Untagged Clone – SC309230 | Origene
BAP1 Axis: New Factors in Myelopoiesis, Cancer and Epigenetics
ASXL1 Gene ASXL Transcriptional Regulator 1
Mutations of ASXL1 Gene in Myeloproliferative Neoplasms
Chronic Myelomonocytic Leukemia Gold Jubilee
Identification of Bap1 As a Predisposing Gene for Malignant Mesothelioma
Mutations in ASXL1 Are Associated with Poor Prognosis Across The
ASXL1 Exon 12 Mutations Are Frequent in AML with Intermediate Risk Karyotype and Are Independently Associated with an Adverse Outcome
Familial and Somatic BAP1 Mutations Inactivate ASXL1/2-Mediated
PR-DUB Maintains the Expression of Critical Genes Through FOXK1/2- and ASXL1/2/3-Dependent Recruitment to Chromatin and H2ak119ub1 Deubiquitination
The ASXL1-G643W Variant Accelerates the Development Of