RESPIRATORY DISORDERS: ASTHMA and COPD Self-Study Module 15.0 Contact Hours
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Section 8 Pulmonary Medicine
SECTION 8 PULMONARY MEDICINE 336425_ST08_286-311.indd6425_ST08_286-311.indd 228686 111/7/121/7/12 111:411:41 AAMM CHAPTER 66 EVALUATION OF CHRONIC COUGH 1. EPIDEMIOLOGY • Nearly all adult cases of chronic cough in nonsmokers who are not taking an ACEI can be attributed to the “Pathologic Triad of Chronic Cough” (asthma, GERD, upper airway cough syndrome [UACS; previously known as postnasal drip syndrome]). • ACEI cough is idiosyncratic, occurrence is higher in female than males 2. PATHOPHYSIOLOGY • Afferent (sensory) limb: chemical or mechanical stimulation of receptors on pharynx, larynx, airways, external auditory meatus, esophagus stimulates vagus and superior laryngeal nerves • Receptors upregulated in chronic cough • CNS: cough center in nucleus tractus solitarius • Efferent (motor) limb: expiratory and bronchial muscle contraction against adducted vocal cords increases positive intrathoracic pressure 3. DEFINITION • Subacute cough lasts between 3 and 8 weeks • Chronic cough duration is at least 8 weeks 4. DIFFERENTIAL DIAGNOSIS • Respiratory tract infection (viral or bacterial) • Asthma • Upper airway cough syndrome (postnasal drip syndrome) • CHF • Pertussis • COPD • GERD • Bronchiectasis • Eosinophilic bronchitis • Pulmonary tuberculosis • Interstitial lung disease • Bronchogenic carcinoma • Medication-induced cough 5. EVALUATION AND TREATMENT OF THE COMMON CAUSES OF CHRONIC COUGH • Upper airway cough syndrome: rhinitis, sinusitis, or postnasal drip syndrome • Presentation: symptoms of rhinitis, frequent throat clearing, itchy -
Understanding Asthma
Understanding Asthma The Mount Sinai − National Jewish Health Respiratory Institute was formed by the nation’s leading respiratory hospital National Jewish Health, based in Denver, and top ranked academic medical center the Icahn School of Medicine at Mount Sinai in New York City. Combining the strengths of both organizations into an integrated Respiratory Institute brings together leading expertise in diagnosing and treating all forms of respiratory illness and lung disease, including asthma, chronic obstructive pulmonary disease (COPD), interstitial lung disease (ILD) and bronchiectasis. The Respiratory Institute is based in New York City on the campus of Mount Sinai. njhealth.org Understanding Asthma An educational health series from National Jewish Health IN THIS ISSUE What Is Asthma? 2 How Does Asthma Develop? 4 How Is Asthma Diagnosed? 5 What Are the Goals of Treatment? 7 How Is Asthma Managed? 7 What Things Make Asthma Worse and How Can You Control Them? 8 Nocturnal Asthma 18 Occupational Asthma 19 Medication Therapy 20 Monitoring Your Asthma 29 Using an Action Plan 33 Living with Asthma 34 Note: This information is provided to you as an educational service of National Jewish Health. It is not meant as a substitute for your own doctor. © Copyright 1998, revised 2014, 2018 National Jewish Health What Is Asthma? This booklet, prepared by National Jewish Health in Denver, is intended to provide information to people with asthma. Asthma is a chronic respiratory disease — sometimes worrisome and inconvenient — but a manageable condition. With proper understanding, good medical care and monitoring, you can keep asthma well controlled. That’s our treatment goal at National Jewish Health: to teach patients and families how to manage asthma, so that they can lead full and productive lives. -
Problems in Family Practice
problems in Family Practice Coughing in Childhood Hyman Sh ran d , M D Cambridge, M assachusetts Coughing in childhood is a common complaint involving a wide spectrum of underlying causes which require a thorough and rational approach by the physician. Most children who cough have relatively simple self-limiting viral infections, but some may have serious disease. A dry environment, allergic factors, cystic fibrosis, and other major illnesses must always be excluded. A simple clinical approach, and the sensible use of appropriate investigations, is most likely to succeed in finding the cause, which can allow precise management. The cough reflex as part of the defense mechanism of the respiratory tract is initiated by mucosal changes, secretions or foreign material in the pharynx, larynx, tracheobronchial Table 1. Persistent Cough — Causes in Childhood* tree, pleura, or ear. Acting as the “watchdog of the lungs,” the “good” cough prevents harmful agents from Common Uncommon Rare entering the respiratory tract; it also helps bring up irritant material from Environmental Overheating with low humidity the airway. The “bad” cough, on the Allergens other hand, serves no useful purpose Pollution Tobacco smoke and, if persistent, causes fatigue, keeps Upper Respiratory Tract the child (and parents) awake, inter Recurrent viral URI Pertussis Laryngeal stridor feres with feeding, and induces vomit Rhinitis, Pharyngitis Echo 12 Vocal cord palsy Allergic rhinitis Nasal polyp Vascular ring ing. It is best suppressed. Coughs and Prolonged use of nose drops Wax in ear colds constitute almost three quarters Sinusitis of all illness in young children. The Lower Respiratory Tract Asthma Cystic fibrosis Rt. -
Lesson 1 ELECTROMYOGRAPHY 1 Motor Unit Recruitment
Physiology Lessons for use with the Biopac Science Lab MP40 Lesson 12 Respiration 1 Apnea PC running Windows® XP or Mac® OS X 10.3-10.4 Lesson Revision 3.15.2006 BIOPAC Systems, Inc. 42 Aero Camino, Goleta, CA 93117 (805) 685-0066, Fax (805) 685-0067 [email protected] www.biopac.com © BIOPAC Systems, Inc. 2006 Page 2 Biopac Science Lab Lesson 12 The Respiratory Cycle I. SCIENTIFIC PRINCIPLES All body cells require oxygen for metabolism and produce carbon dioxide as a metabolic waste product. The respiratory system supplies oxygen to the blood for delivery to cells, and removes carbon dioxide added to the blood by the cells. Cyclically breathing in and out while simultaneously circulating blood between the lungs and other body tissues facilitates the exchange of oxygen and carbon dioxide between the body and the external environment. This process serves cells by maintaining rates of oxygen delivery and carbon dioxide removal adequate to meet the cells’ metabolic needs. The breathing cycle, or respiratory cycle, consists of inspiration during which new air containing oxygen is inhaled, followed by expiration during which old air containing carbon dioxide is exhaled. Average adult people at rest breathe at a frequency of 12 to 15 breaths per minute (BPM), and with each cycle, move an equal volume of air, called tidal volume (TV), into and back out of the lungs. The actual value of tidal volume varies in direct proportion to the depth of inspiration. During normal, quiet, unlabored breathing (eupnea) at rest, adult tidal volume is about 450 ml to 500 ml. -
Exercise-Induced Dyspnea in College-Aged Athletes
A Peer Reviewed Publication of the College of Health Care Sciences at Nova Southeastern University Dedicated to allied health professional practice and education http://ijahsp.nova.edu Vol. 11 No. 3 ISSN 1540-580X Exercise-Induced Dyspnea in College-Aged Athletes Katherine R. Newsham, PhD, ATC 1 Ethel M. Frese, PT, DPT, CCS2 Richard A. McGuire, PhD, CCC-SLP 3 Dennis P. Fuller, PhD, CCC-SLP 4 Blakeslee E. Noyes, MD 5 1. Assistant Professor, Department of Physical Therapy and Athletic Training, Saint Louis University, St. Louis, MO 2. Associate Professor, Department of Physical Therapy and Athletic Training, Saint Louis University, St. Louis, MO 3. Professor, Department of Communication Sciences & Disorders, Saint Louis University, St. Louis, MO 4. Associate Professor, Department of Communication Sciences & Disorders, Saint Louis University, St. Louis, MO 5. Professor, Saint Louis University School of Medicine, St. Louis, MO, Director of Pulmonary Medicine, Cardinal Glennon Children’s Medical Center, St. Louis, MO United States CITATION: Newsham K, Frese E, McGuire R, Fuller D, Noyes B. Exercise-Induced Dyspnea in College-Aged Athletes. The Internet Journal of Allied Health Sciences and Practice. July 2013. Volume 11 Number 3. ABSTRACT Purpose: Shortness of breath or difficulty breathing during exercise is referred to as exercise-induced dyspnea (EID), and is a common complaint from athletes. The purpose of this study was to assess the prevalence of EID among college aged athletes and to explore the medical encounters, including diagnostic testing, arising from this complaint. Method: We surveyed intercollegiate (n=122) and club sport (n=103) athletes regarding their experience with EID, including medical diagnoses, diagnostic procedures, environmental factors, and treatment effectiveness. -
Asthma Exacerbation Management
CLINICAL PATHWAY ASTHMA EXACERBATION MANAGEMENT TABLE OF CONTENTS Figure 1. Algorithm for Asthma Exacerbation Management – Outpatient Clinic Figure 2. Algorithm for Asthma Management – Emergency Department Figure 3. Algorithm for Asthma Management – Inpatient Figure 4. Progression through the Bronchodilator Weaning Protocol Table 1. Pediatric Asthma Severity (PAS) Score Table 2. Bronchodilator Weaning Protocol Target Population Clinical Management Clinical Assessment Treatment Clinical Care Guidelines for Treatment of Asthma Exacerbations Children’s Hospital Colorado High Risk Asthma Program Table 3. Dosage of Daily Controller Medication for Asthma Control Table 4. Dosage of Medications for Asthma Exacerbations Table 5. Dexamethasone Dosing Guide for Asthma Figure 5. Algorithm for Dexamethasone Dosing – Inpatient Asthma Patient | Caregiver Education Materials Appendix A. Asthma Management – Outpatient Appendix B. Asthma Stepwise Approach (aka STEPs) Appendix C. Asthma Education Handout References Clinical Improvement Team Page 1 of 24 CLINICAL PATHWAY FIGURE 1. ALGORITHM FOR ASTHMA EXACERBATION MANAGEMENT – OUTPATIENT CLINIC Triage RN/MA: • Check HR, RR, temp, pulse ox. Triage level as appropriate • Notify attending physician if patient in severe distress (RR greater than 35, oxygen saturation less than 90%, speaks in single words/trouble breathing at rest) Primary RN: • Give oxygen to keep pulse oximetry greater than 90% Treatment Inclusion Criteria 1. Give nebulized or MDI3 albuterol up to 3 doses. Albuterol dosing is 0.15 to 0.3mg/kg per 2007 • 2 years or older NHLBI guidelines. • Treated for asthma or asthma • Less than 20 kg: 2.5 mg neb x 3 or 2 to 4 puffs MDI albuterol x 3 exacerbation • 20 kg or greater: 5 mg neb x 3 or 4 to 8 puffs MDI albuterol x 3 • First time wheeze with history consistent Note: For moderate (dyspnea interferes with activities)/severe (dyspnea at rest) exacerbations you with asthma can add atrovent to nebulized albuterol at 0.5mg/neb x 3. -
Design of Respiration Rate Meter Using Flexible Sensor
JEEMI, Vol. 2, No. 1, January 2020, pp: 13-18 DOI: 10.35882/jeeemi.v2i1.3 ISSN:2656-8632 Design of Respiration Rate Meter Using Flexible Sensor Sarah Aghnia Miyagi#,1, Muhammad Ridha Mak’ruf1, Endang Dian Setyoningsih1, Tarak Das2 1Department of Electromedical Engineering Poltekkes Kemenkes, Surabaya Jl. Pucang Jajar Timur No. 10, Surabaya, 60245, Indonesia 2Department of Biomedical Engineering Netaji Subhash, Engineering College Kolkata, India #[email protected], [email protected], [email protected], [email protected] Abstract— Respiration rate is an important physiological parameter that helps to provide important information about the patient's health status, especially from the human respiratory system. So it is necessary to measure the human respiratory rate by calculating the number of respiratory frequencies within 1 minute. The respiratory rate meter is a tool used to calculate the respiratory rate by counting the number of breaths for 1 minute. The author makes a tool to detect human respiratory rate by using a sensor that detects the ascend and descend of the chest cavity based on a microcontroller so that the operator can measure the breathing rate more practically and accurately. Component tool contains analog signal conditioning circuit and microcontroller circuit accompanied by display in the form of LCD TFT. The results of measurement data on 10 respondents obtained an average error value, namely the position of the right chest cavity 6.6%, middle chest cavity 7.92%, and left chest cavity 6.85%. This value is still below the error tolerance limit of 10%. It can be concluded that to obtain the best measurement results, the sensor is placed in the position of the right chest cavity. -
Chapter 22 *Lecture Powerpoint
Chapter 22 *Lecture PowerPoint The Respiratory System *See separate FlexArt PowerPoint slides for all figures and tables preinserted into PowerPoint without notes. Copyright © The McGraw-Hill Companies, Inc. Permission required for reproduction or display. Introduction • Breathing represents life! – First breath of a newborn baby – Last gasp of a dying person • All body processes directly or indirectly require ATP – ATP synthesis requires oxygen and produces carbon dioxide – Drives the need to breathe to take in oxygen, and eliminate carbon dioxide 22-2 Anatomy of the Respiratory System • Expected Learning Outcomes – State the functions of the respiratory system – Name and describe the organs of this system – Trace the flow of air from the nose to the pulmonary alveoli – Relate the function of any portion of the respiratory tract to its gross and microscopic anatomy 22-3 Anatomy of the Respiratory System • The respiratory system consists of a system of tubes that delivers air to the lung – Oxygen diffuses into the blood, and carbon dioxide diffuses out • Respiratory and cardiovascular systems work together to deliver oxygen to the tissues and remove carbon dioxide – Considered jointly as cardiopulmonary system – Disorders of lungs directly effect the heart and vice versa • Respiratory system and the urinary system collaborate to regulate the body’s acid–base balance 22-4 Anatomy of the Respiratory System • Respiration has three meanings – Ventilation of the lungs (breathing) – The exchange of gases between the air and blood, and between blood and the tissue fluid – The use of oxygen in cellular metabolism 22-5 Anatomy of the Respiratory System • Functions – Provides O2 and CO2 exchange between blood and air – Serves for speech and other vocalizations – Provides the sense of smell – Affects pH of body fluids by eliminating CO2 22-6 Anatomy of the Respiratory System Cont. -
08-0205: N.M. and DEPARTMENT of the NAVY, PUGET S
United States Department of Labor Employees’ Compensation Appeals Board __________________________________________ ) N.M., Appellant ) ) and ) Docket No. 08-205 ) Issued: September 2, 2008 DEPARTMENT OF THE NAVY, PUGET ) SOUND NAVAL SHIPYARD, Bremerton, WA, ) Employer ) __________________________________________ ) Appearances: Oral Argument July 16, 2008 John Eiler Goodwin, Esq., for the appellant No appearance, for the Director DECISION AND ORDER Before: DAVID S. GERSON, Judge COLLEEN DUFFY KIKO, Judge JAMES A. HAYNES, Alternate Judge JURISDICTION On October 30, 2007 appellant filed a timely appeal from a November 17, 2006 decision of the Office of Workers’ Compensation Programs denying his occupational disease claim. Pursuant to 20 C.F.R. §§ 501.2(c) and 501.3, the Board has jurisdiction over the merits of the claim. ISSUE The issue is whether appellant has established that he sustained occupational asthma in the performance of duty due to accepted workplace exposures. On appeal, he, through his attorney, asserts that the Office did not provide Dr. William C. Stewart, the impartial medical examiner, with a complete, accurate statement of accepted facts. FACTUAL HISTORY On December 8, 2004 appellant, then a 57-year-old insulator, filed an occupational disease claim (Form CA-2) asserting that he sustained occupational asthma and increasing shortness of breath due to workplace exposures to fiberglass, silicates, welding smoke, polychlorobenzenes, rubber, dusts, gases, fumes and smoke from “burning out” submarines from 1991 through January -
Clinical Perspectives on the Association Between Respiratory Syncytial Virus and Reactive Airway Disease Nele Sigurs
Respiratory Research Vol 3 Suppl 1 Sigurs Clinical perspectives on the association between respiratory syncytial virus and reactive airway disease Nele Sigurs Department of Pediatrics, Borås Central Hospital, Borås, Sweden Corresponding author: Nele Sigurs (e-mail: [email protected]) Received: 24 May 2002 Accepted: 30 May 2002 Published: 24 June 2002 Respir Res 2002, 3 (suppl 1):S8-S14 © 2002 BioMed Central Ltd (Print ISSN 1465-9921; Online ISSN 1465-993X) Abstract Asthma is a leading cause of morbidity and mortality among children worldwide, as is respiratory syncytial virus (RSV). This report reviews controlled retrospective and prospective studies conducted to investigate whether there is an association between RSV bronchiolitis in infancy and subsequent development of reactive airway disease or allergic sensitization. Findings indicate that such a link to bronchial obstructive symptoms does exist and is strongest for children who experienced severe RSV illness that requires hospitalization. However, it is not yet clear what roles genetic predisposition and environmental or other risk factors may play in the interaction between RSV bronchiolitis and reactive airway disease or allergic sensitization. Randomized, prospective studies utilizing an intervention against RSV, such as a passive immunoprophylactic agent, may determine whether preventing RSV bronchiolitis reduces the incidence of asthma. Keywords: allergy, asthma, bronchiolitis, reactive airway disease, respiratory syncytial virus Introduction nesses are at even greater risk for serious infection and Childhood asthma is a serious global public health hospitalization from RSV [3]. problem. According to the World Health Organization [1], asthma is the most common chronic disease in children. In RSV bronchiolitis is characterized by expiratory wheezing some areas of the world the incidence in children is over and respiratory distress [4]. -
Management of Acute Exacerbation of Asthma and Chronic Obstructive Pulmonary Disease in the Emergency Department
Management of Acute Exacerbation of Asthma and Chronic Obstructive Pulmonary Disease in the Emergency Department Salvador J. Suau, MD*, Peter M.C. DeBlieux, MD KEYWORDS Asthma Asthmatic crisis COPD AECOPD KEY POINTS Management of severe asthma and chronic obstructive pulmonary disease (COPD) exac- erbations require similar medical interventions in the acute care setting. Capnography, electrocardiography, chest x-ray, and ultrasonography are important diag- nostic tools in patients with undifferentiated shortness of breath. Bronchodilators and corticosteroids are first-line therapies for both asthma and COPD exacerbations. Noninvasive ventilation, magnesium, and ketamine should be considered in patients with severe symptoms and in those not responding to first-line therapy. A detailed plan reviewed with the patient before discharge can decrease the number of future exacerbations. INTRODUCTION Acute asthma and chronic obstructive pulmonary disease (COPD) exacerbations are the most common respiratory diseases requiring emergent medical evaluation and treatment. Asthma accounts for more than 2 million visits to emergency departments (EDs), and approximately 4000 annual deaths in the United States.1 In a similar fashion, COPD is a major cause of morbidity and mortality. It affects more than 14.2 million Americans (Æ9.8 million who may be undiagnosed).2 COPD accounts for more than 1.5 million yearly ED visits and is the fourth leading cause of death Disclosures: None. Louisiana State University, University Medical Center of New Orleans, 2000 Canal Street, D&T 2nd Floor - Suite 2720, New Orleans, LA 70112, USA * Corresponding author. E-mail address: [email protected] Emerg Med Clin N Am 34 (2016) 15–37 http://dx.doi.org/10.1016/j.emc.2015.08.002 emed.theclinics.com 0733-8627/16/$ – see front matter Ó 2016 Elsevier Inc. -
Defective Regulation of Immune Responses in Croup Due to Parainfluenza Virus
716 WELLIVER ET AL. Science 221: 1067-1070 20. Mawhinney TP, Feather MS, Martinez JR, Barbero GJ 1979 The chronically 17. Quissell DO 1980 Secretory response of dispersed rat submandibular cells: I. reserpinized rat as an animal model for cystic fibrosis: acute effect of Potassium release. Am J Physiol 238:C90-C98 isoproterenol and pilocarpine upon pulmonary lavage fluid. Pediatr Res 18. Lowry OH, Rosebrough NF, Farrar AL, Randall RJ 195 1 Protein measurement 13:760-763 with the Folin phenol reagent. J Biol Chem 193:265-268 21. Frizzell RA, Fields M, Schultz SG 1979 Sodium-coupled chloride transport by 19. Perlmutter J, Martinez JR 1978 The chornically reserpinized rat as a possible epithelial tissues. Am J Physiol 236:FI-F8 model for cystic fibrosis: VII. Alterations in the secretory response to secretin 22. Welsh M 1983 Inhibition of chloride secretion by furosemide in canine tracheal and to cholecystokinin from the pancreas in vivo. Pediatr Res 12: 188- 194 epithelium. J Memb Biol71:219-226 003 1-3998/85/1907-07 16$02.00/0 PEDIATRIC RESEARCH Vol. 19, No. 7, 1985 Copyright O 1985 International Pediatric Research Foundation, Inc. Printed in U.S.A. Defective Regulation of Immune Responses in Croup Due to Parainfluenza Virus ROBERT C. WELLIVER, MARTHA SUN, AND DEBORAH RINALDO Department ofPediatrics, State University of New York at Buffalo, and Division of Infectious Diseases, Children S Hospital, Buffalo, New York 14222 ABSTRACT. In order to determine if defects in regulation Croup is a common respiratory illness of childhood, yet fairly of immune responses play a role in the pathogenesis of little is known about its pathogenesis.