Intracellular Zinc Release-Activated ERK-Dependent GSK-3Β–P53 and Noxa–Mcl-1 Signaling Are Both Involved in Cardiac Ischemi
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Cell Death and Differentiation (2011) 18, 1678 & 2011 Macmillan Publishers Limited All rights reserved 1350-9047/11 www.nature.com/cdd Corrigendum Intracellular zinc release-activated ERK-dependent GSK-3b–p53 and Noxa–Mcl-1 signaling are both involved in cardiac ischemic-reperfusion injury C-L Lin1,2,5, H-C Tseng1,5, R-F Chen2,5, W-P Chen3,5, M-J Su3, K-M Fang*,4 and M-L Wu*,1 Cell Death and Differentiation (2011) 18, 1678; doi:10.1038/cdd.2011.106 Correction to: Cell Death and Differentiation (2011) 18, This error has now been rectified, and the correct article 1651–1663; doi:10.1038/cdd.2011.80; published online 10 appears in this issue. The html and online pdf versions have June 2011 also been rectified, and now carry the correct paper. Since the publication of this article, the authors of the above The authors apologize for any inconvenience caused. paper have noticed that K-M Fang was omitted as one of the corresponding authors in its online version. 1Institute of Physiology, College of Medicine, National Taiwan University, Taipei, Taiwan; 2Department of Life Science and Institute of Zoology, National Taiwan University, Taipei, Taiwan; 3Institute of Pharmacology, College of Medicine, National Taiwan University, Taipei, Taiwan and 4Department of Dental Technology and Material Science, Central Taiwan University of Science and Technology, Taichung, Taiwan *Corresponding authors: M-L Wu, Institute of Physiology, College of Medicine, National Taiwan University, No. 1, section 1, Jen-Ai Road, Taipei 001, Taiwan. Tel: þ 88 622 312 3456 extn 88242; Fax: þ 88 622 322 4330; E-mail: [email protected] or K-M Fang, Department of Dental Technology and Material Sciences, Central Taiwan University of Science and Technology, No. 666, Buzih Road, Beitun District, Taichung City 40601, Taiwan. Tel: þ 88 642 239 1647 extn 7406; Fax: þ 88 642 277 5625; E-mail: [email protected] 5These authors contributed equally to this work..