Spinal and Radicular Pain Syndromes
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Peroneal Neuropathy Misdiagnosed As L5 Radiculopathy: a Case Report Michael D Reife1,2* and Christopher M Coulis3,4,5
Reife and Coulis Chiropractic & Manual Therapies 2013, 21:12 http://www.chiromt.com/content/21/1/12 CHIROPRACTIC & MANUAL THERAPIES CASE REPORT Open Access Peroneal neuropathy misdiagnosed as L5 radiculopathy: a case report Michael D Reife1,2* and Christopher M Coulis3,4,5 Abstract Objective: The purpose of this case report is to describe a patient who presented with a case of peroneal neuropathy that was originally diagnosed and treated as a L5 radiculopathy. Clinical features: A 53-year old female registered nurse presented to a private chiropractic practice with complaints of left lateral leg pain. Three months earlier she underwent elective left L5 decompression surgery without relief of symptoms. Intervention and outcome: Lumbar spine MRI seven months prior to lumbar decompression surgery revealed left neural foraminal stenosis at L5-S1. The patient symptoms resolved after she stopped crossing her legs. Conclusion: This report discusses a case of undiagnosed peroneal neuropathy that underwent lumbar decompression surgery for a L5 radiculopathy. This case study demonstrates the importance of a thorough clinical examination and decision making that ensures proper patient diagnosis and management. Keywords: Peroneal neuropathy, Lumbar radiculopathy, Chiropractic Background Case presentation The most common entrapment neuropathy in the lo- The patient is a 53-year-old registered nurse who was wer extremity is common peroneal mononeuropathy, ac- referred to the author’s office in August 2003 by her pri- counting for approximately 15% of all mononeuropathies mary care physician with a chief complaint of left leg in adults [1]. Most injuries occur at the fibular head and pain. Her symptoms began in October 2002 after she fell can be the result of many factors including chronic low off an ambulance landing on her left hip. -
A Simplified Fascial Model of Pelvic Anatomical Surgery: Going Beyond
Anatomical Science International https://doi.org/10.1007/s12565-020-00553-z ORIGINAL ARTICLE A simplifed fascial model of pelvic anatomical surgery: going beyond parametrium‑centered surgical anatomy Stefano Cosma1 · Domenico Ferraioli2 · Marco Mitidieri3 · Marcello Ceccaroni4 · Paolo Zola5 · Leonardo Micheletti1 · Chiara Benedetto1 Received: 13 March 2020 / Accepted: 5 June 2020 © The Author(s) 2020 Abstract The classical surgical anatomy of the female pelvis is limited by its gynecological oncological focus on the parametrium and burdened by its modeling based on personal techniques of diferent surgeons. However, surgical treatment of pelvic diseases, spreading beyond the anatomical area of origin, requires extra-regional procedures and a thorough pelvic anatomical knowl- edge. This study evaluated the feasibility of a comprehensive and simplifed model of pelvic retroperitoneal compartmen- talization, based on anatomical rather than surgical anatomical structures. Such a model aims at providing an easier, holistic approach useful for clinical, surgical and educational purposes. Six fresh-frozen female pelves were macroscopically and systematically dissected. Three superfcial structures, i.e., the obliterated umbilical artery, the ureter and the sacrouterine ligament, were identifed as the landmarks of 3 deeper fascial-ligamentous structures, i.e., the umbilicovesical fascia, the urogenital-hypogastric fascia and the sacropubic ligament. The retroperitoneal areolar tissue was then gently teased away, exposing the compartments delimited by these deep fascial structures. Four compartments were identifed as a result of the intrapelvic development of the umbilicovesical fascia along the obliterated umbilical artery, the urogenital-hypogastric fascia along the mesoureter and the sacropubic ligaments. The retroperitoneal compartments were named: parietal, laterally to the umbilicovesical fascia; vascular, between the two fasciae; neural, medially to the urogenital-hypogastric fascia and visceral between the sacropubic ligaments. -
Brachial-Plexopathy.Pdf
Brachial Plexopathy, an overview Learning Objectives: The brachial plexus is the network of nerves that originate from cervical and upper thoracic nerve roots and eventually terminate as the named nerves that innervate the muscles and skin of the arm. Brachial plexopathies are not common in most practices, but a detailed knowledge of this plexus is important for distinguishing between brachial plexopathies, radiculopathies and mononeuropathies. It is impossible to write a paper on brachial plexopathies without addressing cervical radiculopathies and root avulsions as well. In this paper will review brachial plexus anatomy, clinical features of brachial plexopathies, differential diagnosis, specific nerve conduction techniques, appropriate protocols and case studies. The reader will gain insight to this uncommon nerve problem as well as the importance of the nerve conduction studies used to confirm the diagnosis of plexopathies. Anatomy of the Brachial Plexus: To assess the brachial plexus by localizing the lesion at the correct level, as well as the severity of the injury requires knowledge of the anatomy. An injury involves any condition that impairs the function of the brachial plexus. The plexus is derived of five roots, three trunks, two divisions, three cords, and five branches/nerves. Spinal roots join to form the spinal nerve. There are dorsal and ventral roots that emerge and carry motor and sensory fibers. Motor (efferent) carries messages from the brain and spinal cord to the peripheral nerves. This Dorsal Root Sensory (afferent) carries messages from the peripheral to the Ganglion is why spinal cord or both. A small ganglion containing cell bodies of sensory NCS’s sensory fibers lies on each posterior root. -
Shoulder Pain – Complexity to Simplicity
Shoulder Pain – Complexity to Simplicity A Regional Approach to Shoulder Pain The best way to approach a problem of shoulder pain is to look at the pain from a regional point of view. This allows for easy identification of specific pathological problems that occur at each region. In this talk, we look at the patterns of pain that occur with the different regions around the shoulder. From this, we will focus on the pathological problems that can be expected in each region and then look at specific physical examination findings that can be used to confirm a diagnosis or help further clarify the problem. Although this talk is focused on the diagnosis of causes of shoulder pain, we also take a look at some updates and salient features of treatment. In general, pain in the region of the shoulder can be grouped into 4 main types. These relate to specific patterns of pain that allow history taking to help us focus on the likely cause(s) before moving on to examination. A fairly clear-cut diagnosis is almost always achievable without the need to resort to special tests to help make the diagnosis. Of course, this does not include all possible causes of pain. The uncommon problem will still need a thorough and complete approach to determining cause, often with the requirement for special investigations. Remember that this is a good guide. Don't neglect other causes of referred pain that may cause pain in and around the shoulder. Included here is cardiac pain, diaphragmatic pain, apical lung disease and malignant bone pain. -
Neuropathy, Radiculopathy & Myelopathy
Neuropathy, Radiculopathy & Myelopathy Jean D. Francois, MD Neurology & Neurophysiology Purpose and Objectives PURPOSE Avoid Confusing Certain Key Neurologic Concepts OBJECTIVES • Objective 1: Define & Identify certain types of Neuropathies • Objective 2: Define & Identify Radiculopathy & its causes • Objective 3: Define & Identify Myelopathy FINANCIAL NONE DISCLOSURE Basics What is Neuropathy? • The term 'neuropathy' is used to describe a problem with the nerves, usually the 'peripheral nerves' as opposed to the 'central nervous system' (the brain and spinal cord). It refers to Peripheral neuropathy • It covers a wide area and many nerves, but the problem it causes depends on the type of nerves that are affected: • Sensory nerves (the nerves that control sensation>skin) causing cause tingling, pain, numbness, or weakness in the feet and hands • Motor nerves (the nerves that allow power and movement>muscles) causing weakness in the feet and hands • Autonomic nerves (the nerves that control the systems of the body eg gut, bladder>internal organs) causing changes in the heart rate and blood pressure or sweating • It May produce Numbness, tingling,(loss of sensation) along with weakness. It can also cause pain. • It can affect a single nerve (mononeuropathy) or multiple nerves (polyneuropathy) Neuropathy • Symptoms usually start in the longest nerves in the body: Feet & later on the hands (“Stocking-glove” pattern) • Symptoms usually spread slowly and evenly up the legs and arms. Other body parts may also be affected. • Peripheral Neuropathy can affect people of any age. But mostly people over age 55 • CAUSES: Neuropathy has a variety of forms and causes. (an injury systemic illness, an infection, an inherited disorder) some of the causes are still unknown. -
Sciatica and Chronic Pain
Sciatica and Chronic Pain Past, Present and Future Robert W. Baloh 123 Sciatica and Chronic Pain Robert W. Baloh Sciatica and Chronic Pain Past, Present and Future Robert W. Baloh, MD Department of Neurology University of California, Los Angeles Los Angeles, CA, USA ISBN 978-3-319-93903-2 ISBN 978-3-319-93904-9 (eBook) https://doi.org/10.1007/978-3-319-93904-9 Library of Congress Control Number: 2018952076 © Springer International Publishing AG, part of Springer Nature 2019 This work is subject to copyright. All rights are reserved by the Publisher, whether the whole or part of the material is concerned, specifically the rights of translation, reprinting, reuse of illustrations, recitation, broadcasting, reproduction on microfilms or in any other physical way, and transmission or information storage and retrieval, electronic adaptation, computer software, or by similar or dissimilar methodology now known or hereafter developed. The use of general descriptive names, registered names, trademarks, service marks, etc. in this publication does not imply, even in the absence of a specific statement, that such names are exempt from the relevant protective laws and regulations and therefore free for general use. The publisher, the authors, and the editors are safe to assume that the advice and information in this book are believed to be true and accurate at the date of publication. Neither the publisher nor the authors or the editors give a warranty, express or implied, with respect to the material contained herein or for any errors or omissions that may have been made. The publisher remains neutral with regard to jurisdictional claims in published maps and institutional affiliations. -
Interventional Chronic Pain Treatment in Mature Theaters of Operation
28. INTERVENTIONAL CHRONIC PAIN pain, nonradicular arm pain, groin pain, noncardiac spinal and myofascial pain); and anticonvulsants TREATMENT IN MATURE THEATERS chest pain, and neck pain. The most common diag- and tricyclic antidepressants (usually prescribed for OF OPERATION noses conferred on these patients were lumbosacral radicular and other forms of neuropathic pain). The radiculopathy, recurrence of postsurgical pain, large majority of patients received at least one inter- IMPACT OF NONBATTLE-RELATED INJURIES lumbar facetogenic pain, myofascial pain, neuro- ventional procedure. The most frequently employed AND TREATMENT pathic pain, and lumbar degenerative disc disease. nerve blocks were lumbar transforaminal epidural The most common noninterventional treatments steroid injections (ESIs), trigger point injections, Acute nonbattle injuries (NBIs) and chronic pain have been nonsteroidal antiinflammatory drugs cervical ESIs, lumbar facet blocks, various groin conditions that recur during war have been termed (NSAIDs; > 90%); physical therapy referral (for back blocks, and plantar fascia injections. Table 28-1 lists the “hidden epidemic” by the former surgeon pain, neck pain, and leg pain); muscle relaxants (for procedures for common nerve blocks conducted in general of the US Army, James Peake. Since statistics have been kept, the impact of NBIs on unit readiness TABLE 28-1 has increased. In World War I, NBI was the fourth leading cause of soldier attrition. In World War II PROCEDURES FOR COMMON NERVE BLOCKS CONDUCTED IN THEATER and the Korean conflict, NBIs were the third leading cause of morbidity. By the Vietnam War, NBIs had Injection Injectate Need for Comments become the leading cause of hospital admissions, Volume* (mL) Fluoroscopy? where they have remained ever since. -
Surgery for Lumbar Radiculopathy/ Sciatica Final Evidence Report
Surgery for Lumbar Radiculopathy/ Sciatica Final evidence report April 13, 2018 Health Technology Assessment Program (HTA) Washington State Health Care Authority PO Box 42712 Olympia, WA 98504-2712 (360) 725-5126 www.hca.wa.gov/hta [email protected] Prepared by: RTI International–University of North Carolina Evidence-based Practice Center Research Triangle Park, NC 27709 www.rti.org This evidence report is based on research conducted by the RTI-UNC Evidence-based Practice Center through a contract between RTI International and the State of Washington Health Care Authority (HCA). The findings and conclusions in this document are those of the authors, who are responsible for its contents. The findings and conclusions do not represent the views of the Washington HCA and no statement in this report should be construed as an official position of Washington HCA. The information in this report is intended to help the State of Washington’s independent Health Technology Clinical Committee make well-informed coverage determinations. This report is not intended to be a substitute for the application of clinical judgment. Anyone who makes decisions concerning the provision of clinical care should consider this report in the same way as any medical reference and in conjunction with all other pertinent information (i.e., in the context of available resources and circumstances presented by individual patients). This document is in the public domain and may be used and reprinted without permission except those copyrighted materials that are clearly noted in the document. Further reproduction of those copyrighted materials is prohibited without the specific permission of copyright holders. -
Management of Radicular Pain
Management of Radicular Pain Mel Cusi MBBS, FACSP, FFSEM (UK) Sport & Exercise Medicine Physician Dr Mel Cusi Sport & Exercise Medicine Physician Management of Radicular Pain A. Background B. Epidemiology C. Diagnosis D. Treatment Dr Mel Cusi Sport & Exercise Medicine Physician A. Background • Names and concepts – Radicular pain – Radiculopathy • Structures that can produce radicular Sx – Sinu‐vertebral nerve – Nerve root • Mechanisms of pain – Direct toxic effect of disc material – Chemical substances Dr Mel Cusi Sport & Exercise Medicine Physician B. Epidemiology • Occurs in 3‐5% of the population – More frequent in males in their 40’s – More frequent in females in their 50’s • In sporting population – More frequent in sports that combine spinal flexion/extension with rotation – Fast bowlers, gymnasts, dancers, RU backrowers, golfers, weightlifters, baseball pitchers Dr Mel Cusi Sport & Exercise Medicine Physician C. Diagnosis • Radicular pain is only a descriptive symptom • Diagnosis is made on the usual basis of – History – Clinical examination – Appropriate investigations (when required) Dr Mel Cusi Sport & Exercise Medicine Physician History • Acute LBP radiating to buttock / lower limb • Worse with flexion, sneezing, coughing. Sitting worse than standing • Some pointers – Referred pain from L1‐3 does not reach the knee – Unusual Symptoms (weight loss, fever, chills) point to something else – Beware of cauda equina: surgical emergency Dr Mel Cusi Sport & Exercise Medicine Physician Neurological Examination • Sensation – Subjective -
Studies on Brain and Spinal Cord Tumors
Studies on Brain and Spinal Cord Tumors Chapter 1 Osteochondroma of the Spine Iraj Lotfinia Professor of Neurosurgery, Tabriz Universsity of medical science, Tabriz, Iran. Fax: 00984113340830; Email: [email protected] Abstract Osteochondroma (OC) is the most common benign tumor of the bones, and it remains the most common precursor for secondary chondrosarcoma, which often occurs in the long bones’ metaphyseal areas. Rarely, it is also found in the spine. This tumor comprises a cartilage capped bone projection and is observed in both solitary and multiple forms. In many cases, the lesion can be definitively diagnosed according to radiological characteristics, but the rarity of these lesions in the spine, gradual onset of symptoms, and the frequent lack of observation of lesions in plain radiography may delay the diagnosis or cause misdiagnosis. These lesions are be- nign and do not risk the patient’s life; however, they rarely may be found to be a malignant degeneration that transformed into chondrosarcoma. When the lesion has led to clinical symptoms or has faced the patient with cosmetic challenges, or when definitive diagnosis is unknown, treatment is required. The primary treatment is the surgical removal of the lesion. Timely diagnosis and complete resection of the le- sion using surgery lead to complete recovery and prevent recurrence. 1. Introduction According to the World Health Organization’s (WHO’s) definition in 2002, osteocar- tilaginous exostosis are benign bone neoplasms covered by a cartilaginous cap created at the outer surface of the bone by endochondral ossification [1]. Osteochondroma (OC) is the most common benign primary tumor of the bone. -
Anatomical Study of the Superior Cluneal Nerve and Its Estimation of Prevalence As a Cause of Lower Back Pain in a South African Population
Anatomical study of the superior cluneal nerve and its estimation of prevalence as a cause of lower back pain in a South African population by Leigh-Anne Loubser (10150804) Dissertation to be submitted in full fulfilment of the requirements for the degree Master of Science in Anatomy In the Faculty of Health Science University of Pretoria Supervisor: Prof AN Van Schoor1 Co-supervisor: Dr RP Raath2 1 Department of Anatomy, University of Pretoria 2 Netcare Jakaranda Hospital, Pretoria 2017 DECLARATION OF ORIGINALITY UNIVERSITY OF PRETORIA The Department of Anatomy places great emphasis upon integrity and ethical conduct in the preparation of all written work submitted for academic evaluation. While academic staff teach you about referencing techniques and how to avoid plagiarism, you too have a responsibility in this regard. If you are at any stage uncertain as to what is required, you should speak to your lecturer before any written work is submitted. You are guilty of plagiarism if you copy something from another author’s work (e.g. a book, an article, or a website) without acknowledging the source and pass it off as your own. In effect, you are stealing something that belongs to someone else. This is not only the case when you copy work word-for-word (verbatim), but also when you submit someone else’s work in a slightly altered form (paraphrase) or use a line of argument without acknowledging it. You are not allowed to use work previously produced by another student. You are also not allowed to let anybody copy your work with the intention of passing if off as his/her work. -
ICD9 & ICD10 Neuromuscular Codes
ICD-9-CM and ICD-10-CM NEUROMUSCULAR DIAGNOSIS CODES ICD-9-CM ICD-10-CM Focal Neuropathy Mononeuropathy G56.00 Carpal tunnel syndrome, unspecified Carpal tunnel syndrome 354.00 G56.00 upper limb Other lesions of median nerve, Other median nerve lesion 354.10 G56.10 unspecified upper limb Lesion of ulnar nerve, unspecified Lesion of ulnar nerve 354.20 G56.20 upper limb Lesion of radial nerve, unspecified Lesion of radial nerve 354.30 G56.30 upper limb Lesion of sciatic nerve, unspecified Sciatic nerve lesion (Piriformis syndrome) 355.00 G57.00 lower limb Meralgia paresthetica, unspecified Meralgia paresthetica 355.10 G57.10 lower limb Lesion of lateral popiteal nerve, Peroneal nerve (lesion of lateral popiteal nerve) 355.30 G57.30 unspecified lower limb Tarsal tunnel syndrome, unspecified Tarsal tunnel syndrome 355.50 G57.50 lower limb Plexus Brachial plexus lesion 353.00 Brachial plexus disorders G54.0 Brachial neuralgia (or radiculitis NOS) 723.40 Radiculopathy, cervical region M54.12 Radiculopathy, cervicothoracic region M54.13 Thoracic outlet syndrome (Thoracic root Thoracic root disorders, not elsewhere 353.00 G54.3 lesions, not elsewhere classified) classified Lumbosacral plexus lesion 353.10 Lumbosacral plexus disorders G54.1 Neuralgic amyotrophy 353.50 Neuralgic amyotrophy G54.5 Root Cervical radiculopathy (Intervertebral disc Cervical disc disorder with myelopathy, 722.71 M50.00 disorder with myelopathy, cervical region) unspecified cervical region Lumbosacral root lesions (Degeneration of Other intervertebral disc degeneration,