OPCS4 Classification of Surgical Operations and Procedures (Heart)
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Guidelines on the Diagnosis and Management of Pericardial
European Heart Journal (2004) Ã, 1–28 ESC Guidelines Guidelines on the Diagnosis and Management of Pericardial Diseases Full Text The Task Force on the Diagnosis and Management of Pericardial Diseases of the European Society of Cardiology Task Force members, Bernhard Maisch, Chairperson* (Germany), Petar M. Seferovic (Serbia and Montenegro), Arsen D. Ristic (Serbia and Montenegro), Raimund Erbel (Germany), Reiner Rienmuller€ (Austria), Yehuda Adler (Israel), Witold Z. Tomkowski (Poland), Gaetano Thiene (Italy), Magdi H. Yacoub (UK) ESC Committee for Practice Guidelines (CPG), Silvia G. Priori (Chairperson) (Italy), Maria Angeles Alonso Garcia (Spain), Jean-Jacques Blanc (France), Andrzej Budaj (Poland), Martin Cowie (UK), Veronica Dean (France), Jaap Deckers (The Netherlands), Enrique Fernandez Burgos (Spain), John Lekakis (Greece), Bertil Lindahl (Sweden), Gianfranco Mazzotta (Italy), Joa~o Morais (Portugal), Ali Oto (Turkey), Otto A. Smiseth (Norway) Document Reviewers, Gianfranco Mazzotta, CPG Review Coordinator (Italy), Jean Acar (France), Eloisa Arbustini (Italy), Anton E. Becker (The Netherlands), Giacomo Chiaranda (Italy), Yonathan Hasin (Israel), Rolf Jenni (Switzerland), Werner Klein (Austria), Irene Lang (Austria), Thomas F. Luscher€ (Switzerland), Fausto J. Pinto (Portugal), Ralph Shabetai (USA), Maarten L. Simoons (The Netherlands), Jordi Soler Soler (Spain), David H. Spodick (USA) Table of contents Constrictive pericarditis . 9 Pericardial cysts . 13 Preamble . 2 Specific forms of pericarditis . 13 Introduction. 2 Viral pericarditis . 13 Aetiology and classification of pericardial disease. 2 Bacterial pericarditis . 14 Pericardial syndromes . ..................... 2 Tuberculous pericarditis . 14 Congenital defects of the pericardium . 2 Pericarditis in renal failure . 16 Acute pericarditis . 2 Autoreactive pericarditis and pericardial Chronic pericarditis . 6 involvement in systemic autoimmune Recurrent pericarditis . 6 diseases . 16 Pericardial effusion and cardiac tamponade . -
Cardiology Today Jan-Feb 2019.Pdf
VOLUME XXIII No. 1 JANUARY-FEBRUARY 2019 PAGES 1-40 Rs. 1700/- ISSN 0971-9172 RNI No. 66903/97 www.cimsasia .com Cardiology MANAGING DIRECTOR & PUBLISHER Dr. Monica Bhatia TODAY EDITOR IN CHIEF OP Yadava SECTION EDITORS SR Mittal (ECG, CPC), David Colquhou n (Reader’s Choice) EDITORIAL NATIONAL EDITORIAL ADVISORY BOARD Circadian Rhythm of the Body - Is it the Holy Arun K Purohit, Arun Malhotra, Ashok Seth, Grail ? 3 Ashwin B Mehta, CN Manjunath, DS Gambhir, OP YADAVA GS Sainani, Harshad R Gandhi, I Sathyamurthy, Jagdish Hiremath, JPS Sawhney, KK Talwar, K Srinath Reddy, KP Misra, ML Bhatia, Mohan Bhargava, MR Girinath, Mukul Misra, Nakul Sinha, PC Manoria, Peeyush Jain, Praveen Jain, Ramesh Arora, Ravi R Kasliwal, S Jalal, S Padmavati, Satyavan Sharma, SS Ramesh, Sunil Kumar Modi, Yatin Mehta, Yogesh Varma, R Aggarwala. INTERNATIONAL EDITORIAL ADVISORY BOARD REVIEW ARTICLE Andrew M Tonkin, Bhagwan Koirala, Carlos A Mestres, Chuen N Lee, David M Colquhoun, Davendra Mehta, Contrast Induced Nephropathy: How to Enas A Enas, Gerald M Pohost, Glen Van Arsdell, Indranill Basu Ray, James B Peter, James F Benenati, Predict and Prevent? 5 Kanu Chatterjee, Noe A Babilonia, Pascal R Vouhe, RAGHAV BANSAL, VIVEKA KUMAR Paul A Levine, Paul Simon, P K Shah, Prakash Deedwania, Salim Yusuf, Samin K Sharma, Sanjeev Saxena, Sanjiv Kaul, Yutaka Imoto. DESK EDITOR Gandhali DESIGNER A run Kharkwal REVIEW ARTICLE OFFICES CIMS Medica India Pvt Ltd How do I Manage My Patients with Heart (Previously known as UBM Medica India Pvt Ltd.) Failure with Preserved Ejection Fraction? 10 Registered Office MOHAMMED SADIQ AZAM, DAYASAGAR RAO V Margosa Building, No. -
Cardiac Arrhythmias Following the Creation Ofan Atrial Septal Defect in Patients with Transposition of the Great Arteries
Thorax: first published as 10.1136/thx.28.2.147 on 1 March 1973. Downloaded from Thorax (1973), 28, 147. Cardiac arrhythmias following the creation of an atrial septal defect in patients with transposition of the great arteries R. J. MOENE, J. P. ROOS, and A. EYGELAAR Departments of Paediatric Cardiology and Cardiology, Free University Hospital, Amsterdam, and the Department of Thoracic Surgery, University Hospital, Groningen, The Netherlands In 64 children with transposition of the great arteries who underwent a Blalock-Hanlon pro- cedure, pre- and postoperative electrocardiograms were studied regarding the incidence and nature of rhythm disturbances. In another group of 19 patients with transposition of the great arteries, the atrial septal defect was created by a different surgical technique (fossa ovalis resection); this group was studied in the same way and the results were compared. After the Blalock-Hanlon procedure seven patients developed arrhythmias including atrio- ventricular (A-V) dissociation, wandering pacemaker, bradycardia, atrial flutter, supraventricular tachycardia, supraventricular premature beats, and ectopic atrial rhythm. After fossa ovalis resection rhythm disturbances were present in three patients. Despite their relatively high incidence it seems unlikely that arrhythmias are a major factor copyright. contributing to death irrespective of the technique used; some arrhythmias are transient and serious disturbances of long duration are rare. In complete transposition of the great arteries the tion) using temporary -
Dye Dilution Curves After the Artificial Atrial Septostomy in Three Infants with the Transposition of the Great Vessels
Pohoku J. exp. Med., 1970, 100, 39-46 Dye Dilution Curves after the Artificial Atrial Septostomy in Three Infants with the Transposition of the Great Vessels Hiroshi Onoki, Tetsuo Sato, Ichiki Kano and Keiko Mochizuki Department of Pediatrics (Prof. Ts. Arakawa), Faculty of Medicine, Tohoku University, Sendai Hemodynamic consequences after the Rashkind and Miller balloon atrial septostomy were successfully evaluated by means of the dye dilution technic in three infants with the complete transposition of the great vessels. The complete transposition of the great vessels has been the most common cause of death in infants born with congenital malformations of the heart; accord ing to Boesen1 forty-two per cent of the patients with this anomaly succumbed within one month of life and seventy-three per cent within the first three months of life. In 1964 Mustard2 reported a case of the transposition of the great vessels in which the successful result of the radical operation was obtained by adopting a two-stage correction technic; that is, creation of an atrial septal defect was done by Blalock-Hanlon's procedure with thoracotomy when 20 days of life, then successful radical operation was carried out at the age of 18 months. In 1966 Rashkind and Miller3 devised a method for the creation of an atrial septal defect without thoracotomy as a palliative approach to the complete transposi tion of the great vessels. Then in 1968, Rashkind and Miller4 reported thirty-one infants with the transposition of the great vessels who were subjected to the balloon atrial septostomy of theirs with a marked improvement in the interatrial blood commu nication. -
Balloon Aortic Valvuloplasty
Original Research Article Journal of Structural Heart Disease, May 2015, Received: December 8, 2014 Volume 1, Issue 1: 20-32 Accepted: December 15, 2014 Published online: May 2015 DOI: http://dx.doi.org/10.12945/j.jshd.2015.00009-14 Balloon Aortic Valvuloplasty Patient Selection and Technical Considerations Ted Feldman, MD, FESC, FACC, MSCAI*, Mohammad Sarraf, MD, Wes Pedersen, MD, FACC, FSCAI Evanston Hospital, NorthShore University Health System, Evanston, Illinois NOTE: This manuscript includes videos. Not all PDF readers support video. For desktop computers we recommend using Adobe Acrobat Reader. To view videos within a PDF on an iPad we recommend viewing in ezPDF Reader or PDF Expert. On desktop computers you may view videos full screen by clicking on a video, right click on the video, then choose Full Screen Multimedia. Abstract BAV occurs in the vast majority of patients. While in BAV has had resurgence in association with the dissem- many this clinical improvement is short-lived, a ma- ination of TAVR. The lack of clear mortality benefit from jority of patients feel improved symptoms for as long BAV does not translate to lack of efficacy as a palliative as 1 year [1]. The utility of this therapy as a palliative therapy. BAV remains a useful bridge to surgical AVR or TAVR, and for symptom relief in patients who are not treatment is seen best among patients, who truly candidates for either AVR approach. It is also useful as a have no other option [2]. For example, the extreme diagnostic test for patients with low gradient-low out- risk patient, who is a candidate for neither surgical put AS, and for those with mixed pulmonary and aortic nor transcatheter AVR may undergo BAV periodically valvular disease. -
Coronary Artery Disease and Transcatheter Aortic Valve Replacement JACC State-Of-The-Art Review
JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY VOL. 74, NO. 3, 2019 ª 2019 BY THE AMERICAN COLLEGE OF CARDIOLOGY FOUNDATION PUBLISHED BY ELSEVIER THE PRESENT AND FUTURE JACC STATE-OF-THE-ART REVIEW Coronary Artery Disease and Transcatheter Aortic Valve Replacement JACC State-of-the-Art Review Laurent Faroux, MD, MSC, Leonardo Guimaraes, MD, Jérôme Wintzer-Wehekind, MD, Lucia Junquera, MD, Alfredo Nunes Ferreira-Neto, MD, David del Val, MD, Guillem Muntané-Carol, MD, Siamak Mohammadi, MD, Jean-Michel Paradis, MD, Josep Rodés-Cabau, MD ABSTRACT About one-half of transcatheter aortic valve replacement (TAVR) candidates have coronary artery disease (CAD), and controversial results have been reported regarding the effect of the presence and severity of CAD on clinical outcomes post-TAVR. In addition to coronary angiography, promising data has been recently reported on both the use of computed tomography angiography and the functional invasive assessment of coronary lesions in the work-up pre-TAVR. While waiting for the results of ongoing randomized trials, percutaneous revascularization of significant coronary lesions has been the routine strategy in TAVR candidates with CAD. Also, scarce data exists on the incidence, characteristics, and management of coronary events post-TAVR, and increasing interest exist on potential coronary access challenges in patients requiring coronary angiography/intervention post-TAVR. This review provides an updated overview of the current landscape of CAD in TAVR recipients, focusing on its prevalence, clinical impact, pre- and post-procedural evaluation and management, unresolved issues and future perspectives. (J Am Coll Cardiol 2019;74:362–72) © 2019 by the American College of Cardiology Foundation. -
Aortic Valve Disease and Associated Complex CAD: the Interventional Approach
Journal of Clinical Medicine Review Aortic Valve Disease and Associated Complex CAD: The Interventional Approach Federico Marin 1 , Roberto Scarsini 2, Rafail A. Kotronias 1 , Dimitrios Terentes-Printzios 1, Matthew K. Burrage 1 , Jonathan J. H. Bray 3, Jonathan L. Ciofani 4 , Gabriele Venturi 2, Michele Pighi 2, Giovanni L. De Maria 1 and Adrian P. Banning 1,* 1 Oxford Heart Centre, Oxford University Hospitals, NHS Trust, Oxford OX3 9DU, UK; [email protected] (F.M.); [email protected] (R.A.K.); [email protected] (D.T.-P.); [email protected] (M.K.B.); [email protected] (G.L.D.M.) 2 Department of Cardiology, University of Verona, 37129 Verona, Italy; [email protected] (R.S.); [email protected] (G.V.); [email protected] (M.P.) 3 Institute of Life Sciences 2, Swansea Bay University Health Board and Swansea University Medical School, Swansea SA2 8QA, UK; [email protected] 4 Department of Cardiology, Royal North Shore Hospital, Sydney 2065, Australia; [email protected] * Correspondence: [email protected] Abstract: Coronary artery disease (CAD) is highly prevalent in patients with severe aortic stenosis (AS). The management of CAD is a central aspect of the work-up of patients undergoing transcatheter aortic valve implantation (TAVI), but few data are available on this field and the best percutaneous coronary intervention (PCI) practice is yet to be determined. A major challenge is the ability to Citation: Marin, F.; Scarsini, R.; elucidate the severity of bystander coronary stenosis independently of the severity of aortic valve Kotronias, R.A.; Terentes-Printzios, stenosis and subsequent impact on blood flow. -
Atrial Septal Stenting to Increase Interatrial Shunting in Cyanotic Congenital Heart Diseases: a Report of Two Cases
422 Türk Kardiyol Dern Arş - Arch Turk Soc Cardiol 2011;39(5):422-426 doi: 10.5543/tkda.2011.01368 Atrial septal stenting to increase interatrial shunting in cyanotic congenital heart diseases: a report of two cases Siyanotik doğuştan kalp hastalıklarında interatriyal şantı artırmak amacıyla atriyal septuma stent uygulaması: İki olgu sunumu Yalım Yalçın, M.D., Cenap Zeybek, M.D.,§ İbrahim Özgür Önsel, M.D.,# Mehmet Salih Bilal, M.D.† Departments of Pediatric Cardiology, #Anesthesiology and Reanimation, and †Cardiovascular Surgery, Medicana International Hospital; §Department of Pediatric Cardiology, Şişli Florence Nightingale Hospital, İstanbul Summary – Aiming to increase mixing at the atrial level, Özet – Siyanotik doğuştan kalp hastalığı tanısıyla izle- atrial septal stenting was performed in two pediatric nen iki bebekte, atriyal düzeyde karışımı artırmak ama- cases with cyanotic congenital cardiac diseases. The cıyla atriyal septuma stent yerleştirme işlemi uygulandı. first case was a 3-month-old male infant with transpo- Birinci olgu, büyük arterlerin transpozisyonu tanısıyla sition of the great arteries. The second case was an izlenen üç aylık bir erkek bebekti. Diğer olgu, ameliyat 18-month-old male infant with increased central venous sonrası dönemde sağ ventrikül çıkım yolu tıkanıklığına pressure due to postoperative right ventricular outflow bağlı olarak santral venöz basınç yüksekliği gelişen 18 tract obstruction. Premounted bare stents of 8 mm in aylık bir erkek bebekti. Her iki olguda da 8 mm çapında, diameter were used in both cases. The length of the balona monte edilmiş çıplak stent kullanıldı. Stent uzun- stent was 20 mm in the first case and 30 mm in the lat- luğu ilk olguda 20 mm, ikinci olguda 30 mm idi. -
Appendix A: Surgical Procedure Terms and Definitions
Appendix A: Surgical Procedure Terms and Definitions Anomalous Systemic Venous Connection Anomalous Systemic Venous Connection Repair Repair includes a range of surgical approaches, including, among others: ligation of anomalous vessels, reimplantation of anomalous vessels (with or without use of a conduit), or redirection of anomalous systemic venous flow through directly to the pulmonary circulation (bidirectional Glenn to redirect LSVC or RSVC to left or right pulmonary artery, respectively). Aortic Aneurysm Aortic aneurysm repair Aortic aneurysm repair by any technique. Aortic Dissection Aortic Dissection repair Aortic dissection repair by any technique. Aortic Root Replacement Aortic Root Replacement, Bioprosthetic Replacement of the aortic root (that portion of the aorta attached to the heart; it gives rise to the coronary arteries) with a bioprosthesis (e.g., porcine) in a conduit, often composite. Aortic Root Replacement, Mechanical Replacement of the aortic root (that portion of the aorta attached to the heart; it gives rise to the coronary arteries) with a mechanical prosthesis in a composite conduit. Aortic Root Replacement, Homograft Replacement of the aortic root (that portion of the aorta attached to the heart; it gives rise to the coronary arteries) with a homograft Aortic Root Replacement, Valve sparing Replacement of the aortic root (that portion of the aorta attached to the heart; it gives rise to the coronary arteries) without replacing the aortic valve (using a tube graft). Aortic Valve Disease Ross Procedure Replacement of the aortic valve with a pulmonary autograft and replacement of the pulmonary valve with a homograft conduit. Konno Procedure (with and without aortic valve replacement) Relief of left ventricular outflow tract obstruction associated with aortic annular hypoplasia, aortic valvar stenosis and/or aortic valvar insufficiency via Konno aortoventriculoplasty. -
Balloon Atrial Septostomy in Complete Transposition of Great Arteries in Infancy
Br Heart J: first published as 10.1136/hrt.32.1.61 on 1 January 1970. Downloaded from British HeartJournal, I970, 32, X6i. Balloon atrial septostomy in complete transposition of great arteries in infancy A. W. Venables From Royal Children's Hospital, Melbourne, Australia The results of 26 completed balloon atrial septostomies in complete arterial transposition in infancy are described, and complications discussed. Of 7 deaths following this procedure, 3 were clearly or probably related to it or to its failure to produce an adequate septal defect, while 4 were unrelated. Atrial perforations occurred on 4 occasions. Necropsy information regarding the defects produced by the procedure is given. Anatomical features of the fossa ovalis appear to determine the size of the defect created. The effect of the procedure is illustrated by photographs of representative necropsy specimens. Apparently adequate initial defects do not guarantee satisfactory long-term palliation, and 4 of iI infants followed for more than 6 months after effective initial palliation have required surgical procedures to provide more adequate atrial mixing of blood. Despite this, the procedure appears to offer considerable advantage over initial surgical procedures to create atrial defects. The value of palliative procedures in com- Subjects and methods plete arterial transposition is indisputable. From to mid-May I23 cases of trans- ig60 i969, http://heart.bmj.com/ Most commonly, atrial septal defects arz cre- position of the great arteries in infancy were diag- ated in order to increase effective pulmonary nosed in the Cardiac Unit of the Royal Children's flow. Since I966 the balloon catheter tech- Hospital, Melbourne. -
42 Pericardiocentesis (Perform) 341
PROCEDURE Pericardiocentesis (Perform) 42 Kathleen M. Cox PURPOSE: Pericardiocentesis is the removal of excess fl uid from the pericardial sac for identifi cation of the etiology of pericardial effusion by fl uid analysis (diagnostic pericardiocentesis) and/or prevention or treatment of cardiac tamponade (therapeutic pericardiocentesis). result of trauma, myocardial infarction, or iatrogenic PREREQUISITE NURSING injury, whereas chronic effusions can result from condi- KNOWLEDGE tions such as bacterial or viral pericarditis, cancer, autoim- mune disorders, uremia, etc. 2 With a decrease in cardiac • Advanced cardiac life support (ACLS) knowledge and output, the patient often develops chest pain, dyspnea, skills are required. tachycardia, tachypnea, pallor, cyanosis, impaired cere- • Knowledge and skills related to sterile technique are bral and renal function, diaphoresis, hypotension, neck needed. vein distention, distant or faint heart sounds, and pulsus • Clinical and technical competence in the performance of paradoxus. 4 pericardiocentesis is required. • The amount of fl uid in the pericardium is evaluated • Knowledge of cardiovascular anatomy and physiology is through chest radiograph, two-dimensional echocardio- needed. gram, electrocardiography (ECG), and clinical fi ndings. • The pericardial space normally contains 20–50 mL of Chest x-rays may not be diagnostically signifi cant in fl uid. patients with acute traumatic tamponade. 6 • Pericardial fl uid has electrolyte and protein profi les similar • Pericardiocentesis to remove fl uid from the pericardial to plasma. sac is performed therapeutically to relieve tamponade or • Pericardial effusion is generally defi ned as the accumula- to diagnose the etiology of the effusion. An acute tampon- tion of fl uid within the pericardial sac that exceeds the ade resulting in hemodynamic instability necessitates an stretch capacity of the pericardium, generally more than emergency procedure. -
Icd-9-Cm (2010)
ICD-9-CM (2010) PROCEDURE CODE LONG DESCRIPTION SHORT DESCRIPTION 0001 Therapeutic ultrasound of vessels of head and neck Ther ult head & neck ves 0002 Therapeutic ultrasound of heart Ther ultrasound of heart 0003 Therapeutic ultrasound of peripheral vascular vessels Ther ult peripheral ves 0009 Other therapeutic ultrasound Other therapeutic ultsnd 0010 Implantation of chemotherapeutic agent Implant chemothera agent 0011 Infusion of drotrecogin alfa (activated) Infus drotrecogin alfa 0012 Administration of inhaled nitric oxide Adm inhal nitric oxide 0013 Injection or infusion of nesiritide Inject/infus nesiritide 0014 Injection or infusion of oxazolidinone class of antibiotics Injection oxazolidinone 0015 High-dose infusion interleukin-2 [IL-2] High-dose infusion IL-2 0016 Pressurized treatment of venous bypass graft [conduit] with pharmaceutical substance Pressurized treat graft 0017 Infusion of vasopressor agent Infusion of vasopressor 0018 Infusion of immunosuppressive antibody therapy Infus immunosup antibody 0019 Disruption of blood brain barrier via infusion [BBBD] BBBD via infusion 0021 Intravascular imaging of extracranial cerebral vessels IVUS extracran cereb ves 0022 Intravascular imaging of intrathoracic vessels IVUS intrathoracic ves 0023 Intravascular imaging of peripheral vessels IVUS peripheral vessels 0024 Intravascular imaging of coronary vessels IVUS coronary vessels 0025 Intravascular imaging of renal vessels IVUS renal vessels 0028 Intravascular imaging, other specified vessel(s) Intravascul imaging NEC 0029 Intravascular