Amputation of Penis Due to Electrocution- a Case Report
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Clinical Practice Guideline for Limb Salvage Or Early Amputation
Limb Salvage or Early Amputation Evidence-Based Clinical Practice Guideline Adopted by: The American Academy of Orthopaedic Surgeons Board of Directors December 6, 2019 Endorsed by: Please cite this guideline as: American Academy of Orthopaedic Surgeons. Limb Salvage or Early Amputation Evidence-Based Clinical Practice Guideline. https://www.aaos.org/globalassets/quality-and-practice-resources/dod/ lsa-cpg-final-draft-12-10-19.pdf Published December 6, 2019 View background material via the LSA CPG eAppendix Disclaimer This clinical practice guideline was developed by a physician volunteer clinical practice guideline development group based on a formal systematic review of the available scientific and clinical information and accepted approaches to treatment and/or diagnosis. This clinical practice guideline is not intended to be a fixed protocol, as some patients may require more or less treatment or different means of diagnosis. Clinical patients may not necessarily be the same as those found in a clinical trial. Patient care and treatment should always be based on a clinician’s independent medical judgment, given the individual patient’s specific clinical circumstances. Disclosure Requirement In accordance with AAOS policy, all individuals whose names appear as authors or contributors to this clinical practice guideline filed a disclosure statement as part of the submission process. All panel members provided full disclosure of potential conflicts of interest prior to voting on the recommendations contained within this clinical practice guideline. Funding Source This clinical practice guideline was funded exclusively through a research grant provided by the United States Department of Defense with no funding from outside commercial sources to support the development of this document. -
Neurological and Neurourological Complications of Electrical Injuries
REVIEW ARTICLE Neurologia i Neurochirurgia Polska Polish Journal of Neurology and Neurosurgery 2021, Volume 55, no. 1, pages: 12–23 DOI: 10.5603/PJNNS.a2020.0076 Copyright © 2021 Polish Neurological Society ISSN 0028–3843 Neurological and neurourological complications of electrical injuries Konstantina G. Yiannopoulou1, Georgios I. Papagiannis2, 3, Athanasios I. Triantafyllou2, 3, Panayiotis Koulouvaris3, Aikaterini I. Anastasiou4, Konstantinos Kontoangelos5, Ioannis P. Anastasiou6 1Neurological Department, Henry Dunant Hospital Centre, Athens, Greece 2Orthopaedic Research and Education Centre “P.N. Soukakos”, Biomechanics and Gait Analysis Laboratory “Sylvia Ioannou”, “Attikon” University Hospital, Athens, Greece 31st Department of Orthopaedic Surgery, Medical School, National and Kapodistrian University of Athens, Athens, Greece 4Medical School of Athens, National and Kapodistrian University of Athens, Athens, Greece 51st Department of Psychiatry, National and Kapodistrian University of Athens, Eginition Hospital, Athens, Greece 61st Urology Department, Laiko Hospital, National and Kapodistrian University of Athens, Athens, Greece ABSTRACT Electrical injury can affect any system and organ. Central nervous system (CNS) complications are especially well recognised, causing an increased risk of morbidity, while peripheral nervous system (PNS) complications, neurourological and cognitive and psychological abnormalities are less predictable after electrical injuries. PubMed was searched for English language clinical observational, retrospective, -
Case Report Myelopathy and Amnesia Following Accidental Electrical Injury
Spinal Cord (2002) 40, 253 ± 255 ã 2002 International Spinal Cord Society All rights reserved 1362 ± 4393/02 $25.00 www.nature.com/sc Case Report Myelopathy and amnesia following accidental electrical injury J Kalita*,1, M Jose1 and UK Misra1 1Department of Neurology, Sanjay Gandhi Postgraduate Institute of Medical Sciences Lucknow, India Objective: Documentation of MRI and neurophysiological changes following accidental electrical injury. Setting: Tertiary care referral teaching hospital at Lucknow, India. Results: A 30-year-old lady developed amnesia and spastic paraparesis with loss of pin prick sensation below the second thoracic spinal segment following electrocution. Her spinal MRI was normal and cranial MRI revealed T2 hyperintensity in the right putamen. Peroneal, sural and electromyography were normal. Tibial central sensory conduction time was normal but central motor conduction time to lower limbs and right upper limb was prolonged. Conclusion: Neurophysiological study and MRI may help in understanding the pathophy- siological basis of neurological sequelae following electrical injury. Spinal Cord (2002) 40, 253 ± 255. DOI: 10.1038/sj/sc/3101275 Keywords: electrical injury; MRI; evoked potential; myelopathy; amnesia Introduction Rural electri®cation has received great attention from across the road. Her hands were wet, the road was the government for improving agricultural and small ¯ooded with water and the wire was conducting AC of scale industry development in India. This has inherent 11 000 V. Immediately, she had fallen down and the hazards because of the ignorance of villagers and poor wire stuck to her chest. The current ¯ow was maintenance of electrical cables. This results in several discontinued after about 5 min and she was discovered electrical accidents caused by the touching of live wires. -
Scalp Eczema Factsheet the Scalp Is an Area of the Body That Can Be Affected by Several Types of Eczema
12 Scalp eczema factsheet The scalp is an area of the body that can be affected by several types of eczema. The scalp may be dry, itchy and scaly in a chronic phase and inflamed (red), weepy and painful in an acute (eczema flare) phase. Aside from eczema, there are a number of reasons why the scalp can become dry and itchy (e.g. psoriasis, fungal infection, ringworm, head lice etc.), so it is wise to get a firm diagnosis if there is uncertainty. Types of eczema • Hair clips and headgear – especially those containing that affect the scalp rubber or nickel. Seborrhoeic eczema (dermatitis) is one of the most See the NES booklet on Contact Dermatitis for more common types of eczema seen on the scalp and hairline. details. It can affect babies (cradle cap), children and adults. The Irritant contact dermatitis is a type of eczema that skin appears red and scaly and there is often dandruff as occurs when the skin’s surface is irritated by a substance well, which can vary in severity. There may also be a rash that causes the skin to become dry, red and itchy. on other parts of the face, such as around the eyebrows, For example, shampoos, mousses, hair gels, hair spray, eyelids and sides of the nose. Seborrhoeic eczema can perm solution and fragrance can all cause irritant contact become infected. See the NES factsheets on Adult dermatitis. See the NES booklet on Contact Dermatitis for Seborrhoeic Dermatitis and Infantile Seborrhoeic more details. Dermatitis and Cradle Cap for more details. -
Study Guide Medical Terminology by Thea Liza Batan About the Author
Study Guide Medical Terminology By Thea Liza Batan About the Author Thea Liza Batan earned a Master of Science in Nursing Administration in 2007 from Xavier University in Cincinnati, Ohio. She has worked as a staff nurse, nurse instructor, and level department head. She currently works as a simulation coordinator and a free- lance writer specializing in nursing and healthcare. All terms mentioned in this text that are known to be trademarks or service marks have been appropriately capitalized. Use of a term in this text shouldn’t be regarded as affecting the validity of any trademark or service mark. Copyright © 2017 by Penn Foster, Inc. All rights reserved. No part of the material protected by this copyright may be reproduced or utilized in any form or by any means, electronic or mechanical, including photocopying, recording, or by any information storage and retrieval system, without permission in writing from the copyright owner. Requests for permission to make copies of any part of the work should be mailed to Copyright Permissions, Penn Foster, 925 Oak Street, Scranton, Pennsylvania 18515. Printed in the United States of America CONTENTS INSTRUCTIONS 1 READING ASSIGNMENTS 3 LESSON 1: THE FUNDAMENTALS OF MEDICAL TERMINOLOGY 5 LESSON 2: DIAGNOSIS, INTERVENTION, AND HUMAN BODY TERMS 28 LESSON 3: MUSCULOSKELETAL, CIRCULATORY, AND RESPIRATORY SYSTEM TERMS 44 LESSON 4: DIGESTIVE, URINARY, AND REPRODUCTIVE SYSTEM TERMS 69 LESSON 5: INTEGUMENTARY, NERVOUS, AND ENDOCRINE S YSTEM TERMS 96 SELF-CHECK ANSWERS 134 © PENN FOSTER, INC. 2017 MEDICAL TERMINOLOGY PAGE III Contents INSTRUCTIONS INTRODUCTION Welcome to your course on medical terminology. You’re taking this course because you’re most likely interested in pursuing a health and science career, which entails proficiencyincommunicatingwithhealthcareprofessionalssuchasphysicians,nurses, or dentists. -
318 Reports the Scalp Topography of the Human Visually Evoked Subcortical Potential. G. F. A
Invest. Ophthalmol. Vis. Sci. 318 Reports March 1980 We are indebted to Dr. G. van Lith, Oogziekenhuis, gested the optic nerve as its origin. Cracco and Rotterdam, and to Dr. D. van Norren, Ooglijdersgast- Cracco3 described early oscillatory potentials at huis, Utrecht, for testing our lens design and for sug- 100 cy/sec recorded from a wide scalp distribution gestions for improvement in the manuscript. of electrodes, referred to earlobe electrodes. Early From the Kliniek voor Oogheelkunde, Rijks-Universi- in 1979 we identified a triphasic positive- teit Groningen, Groningen, The Netherlands. Submit- negative-positive component (msec) in some sub- ted for publication July 2, 1979. Reprint requests: Aart jects at latencies of positive 22 (P22)> negative 27 C. Kooijman, Kliniek voor Oogheelkunde, Rijks-Uni- (N27), and positive 35 (Pas).4 Since it appeared im- versiteit Groningen, Oostersingel 59, 9713 EZ Gronin- portant to delineate this component from both the gen, The Netherlands. scalp-recorded ERG and the VECP, we have car- Key words: ERG, Ganzfeld stimulator, LED light ried out a topographic study of the scalp dis- tribution. Materials and method. Observations were made REFERENCES on 14 normal volunteer subjects (eight male and 1. Thijssen JM, Braakhuis W, Pinckers A, and van Lith six female) ages between 19 and 38 years (mean 26 G: Standardized electro-ophthalmography. In Pro- years). All had visual acuities of 6/6 or better. For ceedings of the 170th meeting of the Netherlands this topographical study, electrodes were placed Ophthalmological Society. Junk, The Hague, 1976, according to the International 10/20 system.5 In p. -
Reducing Amputation Rates After Severe Frostbite JENNIFER TAVES, MD, and THOMAS SATRE, MD, University of Minnesota/St
FPIN’s Help Desk Answers Reducing Amputation Rates After Severe Frostbite JENNIFER TAVES, MD, and THOMAS SATRE, MD, University of Minnesota/St. Cloud Hospital Family Medicine Resi- dency Program, St. Cloud, Minnesota Help Desk Answers pro- Clinical Question in the tPA plus iloprost group with severe vides answers to questions submitted by practicing Is tissue plasminogen activator (tPA) effec- disease and only three and nine digits in the family physicians to the tive in reducing digital amputation rates in other treatment arms. Thus, no conclusions Family Physicians Inquiries patients with severe frostbite? can be reached about the effect of tPA plus Network (FPIN). Members iloprost compared with iloprost alone. of the network select Evidence-Based Answer questions based on their A 2007 retrospective cohort trial evalu- relevance to family medi- In patients with severe frostbite, tPA plus a ated the use of tPA in six patients with severe cine. Answers are drawn prostacyclin may be used to decrease the risk of frostbite.2 After rapid rewarming, patients from an approved set of digital amputation. (Strength of Recommen- underwent digital angiography, and those evidence-based resources and undergo peer review. dation [SOR]: B, based on a single randomized with significant perfusion defects received controlled trial [RCT].) tPA can be used alone intraarterial tPA (0.5 to 1 mg per hour IV The complete database of evidence-based ques- and is associated with lower amputation rates infusion) and heparin (500 units per hour tions and answers is compared with local wound care. (SOR: C, IV infusion) for up to 48 hours. Patients who copyrighted by FPIN. -
Department of Veterans Affairs 8320-01
This document is scheduled to be published in the Federal Register on 02/22/2013 and available online at http://federalregister.gov/a/2013-04134, and on FDsys.gov DEPARTMENT OF VETERANS AFFAIRS 8320-01 38 CFR Part 17 RIN 2900-AO21 Criteria for a Catastrophically Disabled Determination for Purposes of Enrollment AGENCY: Department of Veterans Affairs. ACTION: Proposed rule. SUMMARY: The Department of Veterans Affairs (VA) proposes to amend its regulation concerning the manner in which VA determines that a veteran is catastrophically disabled for purposes of enrollment in priority group 4 for VA health care. The current regulation relies on specific codes from the International Classification of Diseases, Ninth Revision, Clinical Modification (ICD-9-CM) and Current Procedural Terminology (CPT®). We propose to state the descriptions that would identify an individual as catastrophically disabled, instead of using the corresponding ICD-9-CM and CPT® codes. The revisions would ensure that our regulation is not out of date when new versions of those codes are published. The revisions would also broaden some of the descriptions for a finding of catastrophic disability. Additionally, we would eliminate the Folstein Mini Mental State Examination (MMSE) as a criterion for determining whether a veteran meets the definition of catastrophically disabled, because we have determined that the MMSE is no longer a necessary clinical assessment tool. DATES: Comments on the proposed rule must be received by VA on or before [Insert date 60 days after date of publication in the FEDERAL REGISTER]. ADDRESSES: Written comments may be submitted through http://www.regulations.gov; by mail or hand-delivery to the Director, Regulations Management (02REG), Department of Veterans Affairs, 810 Vermont Avenue, NW, Room 1068, Washington, DC 20420; or by fax to (202) 273-9026. -
Lower Limb Amputation Booklet
A Resource Guide For Lower Limb Amputation Table of Contents Reasons for Amputation . 3 Emotional Adjustments to Amputation . 4 Caring for your Residual Limb . 6 Skin Care Shaping Contracture Management Pressure Relief Sound Limb Preservation Skin Problems Associated with Amputation . 8 Phantom Limb Sensation/Pain . 10 Physical Therapy Following Amputation . 12 Stretches Exercises Preparing for Prosthetic Evaluation . 18 Prosthetic Options . 19 Socket Suspension Knee Feet Caring for your Prosthetic . .. 23 Resources . 24 Websites . 26 Reasons for Amputation There are many reasons for amputations; these are some of the more common causes: Poor Circulation The most common reason for amputation is peripheral artery disease, which leads to poor circulation due to narrowing of the arteries . Without blood flowing throughout the entire limb, the tissues are deprived of oxygen and nutrients . Without oxygen and nutrients, the tissue may begin to die, and this may lead to infection . If the infection becomes too severe, there may be need for amputation . Non-healing wounds or infection People with decreased sensation in the lower extremities may develop wounds and be unaware of these wounds until the wound site has become severe and even infected . If the wound is not responding to antibiotics, the wound may become too severe and there may be need for amputation . Trauma Many types of trauma may result in limb loss, these include, but are not limited to: » Motor vehicle accident » Serious burn » Machinery Accidents » Severe fractures due to falls » Frostbite The most important aspect of many health concerns is prevention . If you have lost a limb due to complications with impaired sensation or circulation, you are at a higher risk for further injury, or new injury to your sound, or intact, limb . -
Instructor Guide for Tactical Field Care 3C Communication, Evacuation Prioroties and Cpr 180801 1
INSTRUCTOR GUIDE FOR TACTICAL FIELD CARE 3C COMMUNICATION, EVACUATION PRIOROTIES AND CPR 180801 1 Tactical Combat Casualty Care for Medical Personnel August 2017 Next, we will discuss communication, evacuation priorities, and 1. (Based on TCCC-MP Guidelines 170131) CPR in TFC. Tactical Field Care 3c Communication, Evacuation Priorities and CPR Disclaimer “The opinions or assertions contained herein are the private views of the authors and are not to be construed 2. as official or as reflecting the views of the Departments Read the text. of the Army, Air Force, Navy or the Department of Defense.” - There are no conflict of interest disclosures. LEARNING OBJECTIVES Terminal Learning Objective • Communicate combat casualty care items effectively in Tactical Field Care. Enabling Learning Objectives 3. Read the text. • Identify the importance and techniques of communication with a casualty in Tactical Field Care. • Identify the importance and techniques of communicating casualty information with unit tactical leadership. INSTRUCTOR GUIDE FOR TACTICAL FIELD CARE 3C COMMUNICATION, EVACUATION PRIOROTIES AND CPR 180801 2 LEARNING OBJECTIVES Enabling Learning Objectives • Identify the importance and techniques of communicating casualty information with evacuation assets or receiving facilities. 4. Read the text. • Identify the relevant tactical and casualty data involved in communicating casualty information. • Identify the evacuation urgencies recommended in the TCCC TACEVAC “Nine Rules of Thumb” and the JTS evacuation guidelines • Identify the information requirements and format of the 9-Line MEDEVAC Request. LEARNING OBJECTIVES Terminal Learning Objective • Describe cardiopulmonary resuscitation (CPR) considerations in Tactical Field Care. Enabling Learning Objectives 5. Read the text. • Identify considerations for cardiopulmonary resuscitation in tactical field care. -
Curry-Assisted Diagnosis in the Rheumatology Clinic Sarah L
Oxford Medical Case Reports, 2015; 6, 297–299 doi: 10.1093/omcr/omv040 Case Report CASE REPORT Curry-assisted diagnosis in the rheumatology clinic Sarah L. Donaldson1,*, Maura Cobine-Davies1, Ann W. Morgan2, Andrew Gough3, and Sarah L. Mackie2 1Leeds Teaching Hospitals NHS Trust, Leeds, UK, 2Leeds Institute of Rheumatic and Musculoskeletal Medicine, University of Leeds, Leeds, UK, and 3Rheumatology Department, Harrogate and District Foundation NHS Trust, Harrogate, UK *Correspondence address. 25 Oakdale Glen, Harrogate, North Yorkshire HG1 2JY, UK. Tel: +44-7745700247; E-mail: [email protected] Abstract We report five cases of glucocorticoid-responsive mouth symptoms in polymyalgia rheumatica/giant cell arteritis (GCA); three cases of tongue pain exacerbated by hot/spicy food, a case of scalp pain made worse by eating hot/spicy food and a case of sore tongue as a presenting feature of GCA. These cases emphasize the importance of asking about mouth symptoms and changes in taste when evaluating patients with suspected GCA. INTRODUCTION pain on eating [8]. The author mentions that burning or painful tongue has been reported in three previous cases of GCA [8]. Giant cell arteritis (GCA) is a systemic large-vessel vasculitis We report five cases of glucocorticoid-responsive mouth (LVV) affecting people older than 50 years. It classically causes symptoms in PMR/GCA; three cases of tongue pain exacerbated headache and ischaemia of cranial structures, resulting in jaw by spicy food, a case of scalp pain made worse by eating spicy claudication and visual disturbance. GCA may be accompanied food and a case of sore tongue as a presenting feature of GCA. -
Guidelines for BLS/ALS Medical Providers Current As of March 2019
Tactical Emergency Casualty Care (TECC) Guidelines for BLS/ALS Medical Providers Current as of March 2019 DIRECT THREAT CARE (DTC) / HOT ZONE Guidelines: 1. Mitigate any immediate threat and move to a safer position (e.g. initiate fire attack, coordinated ventilation, move to safe haven, evacuate from an impending structural collapse, etc). Recognize that threats are dynamic and may be ongoing, requiring continuous threat assessments. 2. Direct the injured first responder to stay engaged in the operation if able and appropriate. 3. Move patient to a safer position: a. Instruct the alert, capable patient to move to a safer position and apply self-aid. b. If the patient is responsive but is injured to the point that he/she cannot move, a rescue plan should be devised. c. If a patient is unresponsive, weigh the risks and benefits of an immediate rescue attempt in terms of manpower and likelihood of success. Remote medical assessment techniques should be considered to identify patients who are dead or have non-survivable wounds. 4. Stop life threatening external hemorrhage if present and reasonable depending on the immediate threat, severity of the bleeding and the evacuation distance to safety. Consider moving to safety prior to application of the tourniquet if the situation warrants. a. Apply direct pressure to wound, or direct capable patient to apply direct pressure to own wound and/or own effective tourniquet. b. Tourniquet application: i. Apply the tourniquet as high on the limb as possible, including over the clothing if present. ii. Tighten until cessation of bleeding and move to safety.