Cardiovascular Examination
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Practical Cardiac Auscultation
LWW/CCNQ LWWJ306-08 March 7, 2007 23:32 Char Count= Crit Care Nurs Q Vol. 30, No. 2, pp. 166–180 Copyright c 2007 Wolters Kluwer Health | Lippincott Williams & Wilkins Practical Cardiac Auscultation Daniel M. Shindler, MD, FACC This article focuses on the practical use of the stethoscope. The art of the cardiac physical exam- ination includes skillful auscultation. The article provides the author’s personal approach to the patient for the purpose of best hearing, recognizing, and interpreting heart sounds and murmurs. It should be used as a brief introduction to the art of auscultation. This article also attempts to illustrate heart sounds and murmurs by using words and letters to phonate the sounds, and by presenting practical clinical examples where auscultation clearly influences cardiac diagnosis and treatment. The clinical sections attempt to go beyond what is available in standard textbooks by providing information and stethoscope techniques that are valuable and useful at the bedside. Key words: auscultation, murmur, stethoscope HIS article focuses on the practical use mastered at the bedside. This article also at- T of the stethoscope. The art of the cardiac tempts to illustrate heart sounds and mur- physical examination includes skillful auscul- murs by using words and letters to phonate tation. Even in an era of advanced easily avail- the sounds, and by presenting practical clin- able technological bedside diagnostic tech- ical examples where auscultation clearly in- niques such as echocardiography, there is still fluences cardiac diagnosis and treatment. We an important role for the hands-on approach begin by discussing proper stethoscope selec- to the patient for the purpose of evaluat- tion and use. -
Mosby: Mosby's Nursing Video Skills
Mosby: Mosby's Nursing Video Skills Procedural Guideline for Assessing Apical Pulse Procedure Steps 1. Verify the health care provider’s orders. 2. Gather the necessary equipment and supplies. 3. Perform hand hygiene. 4. Provide for the patient’s privacy. 5. Introduce yourself to the patient and family if present. 6. Identify the patient using two identifiers. 7. Assess for factors that can affect the apical pulse rate and rhythm, such as medical history, disease processes, age, exercise, position changes, medications, temperature, or sympathetic stimulation. 8. Gloves are only worn if nurse will be in contact with bodily fluids or the patient is in protective precautions. 9. Help the patient into a supine or sitting position, and expose the sternum and the left side of the chest. 10. Locate the point of maximal impulse (PMI, or apical impulse). To do this, find the angle of Louis, which feels like a bony prominence just below the suprasternal notch. 11. Slide your fingers down each side of the angle to find the second intercostal space (ICS). Carefully move your fingers down the left side of the sternum to the fifth intercostal space and over to the left midclavicular line. 12. Feel the PMI as a light tap about 1 to 2 centimeters in diameter, reflecting the apex of the heart. 13. If the PMI is not where you would expect, as in a patient whose left ventricle is enlarged, inch your fingers along the fifth intercostal space until you feel the PMI. 14. Remember where you felt the PMI: over the apex of the heart in the fifth intercostal space at the left midclavicular line. -
Auscultation of Abdominal Arterial Murmurs
Auscultation of abdominal arterial murmurs C. ARTHUR MYERS, D.O.,° Flint, Michigan publications. Goldblatt's4 work on renal hyperten- sion has stimulated examiners to begin performing The current interest in the diagnostic value of ab- auscultation for renal artery bruits in their hyper- dominal arterial bruits is evidenced by the number tensive patients. of papers and references to the subject appearing in Stenosis, either congenital or acquired, and aneu- the recent literature. When Vaughan and Thoreki rysms are responsible for the vast majority of audi- published an excellent paper on abdominal auscul- ble renal artery bruits (Fig. 2). One should be tation in 1939, the only reference they made to highly suspicious of a renal artery defect in a hy- arterial murmurs was that of the bruit of abdominal pertensive patient with an epigastric murmur. Moser aortic aneurysm. In more recent literature, however, and Caldwell5 have produced the most comprehen- there is evidence of increased interest in auscultat- sive work to date on auscultation of the abdomen ing the abdomen for murmurs arising in the celiac, in renal artery disease. In their highly selective superior mesenteric, splenic, and renal arteries. series of 50 cases of abdominal murmurs in which The purpose of this paper is to review some of aortography was performed, renal artery disease the literature referable to the subject of abdominal was diagnosed in 66 per cent of cases. Their con- murmurs, to present some cases, and to stimulate clusions were that when an abdominal murmur of interest in performing auscultation for abdominal high pitch is found in a patient with hypertension, bruits as a part of all physical examinations. -
Intra-Operative Auscultation of Heart and Lungs Sounds: the Importance of Sound Transmission
Intra-Operative Auscultation of more readily when stethoscopes are used. Loeb Heart and Lungs Sounds: (2) has reported that the response time to detect an abnormal value on an intraoperative The Importance of Sound monitor display and it was 61 seconds with 16% Transmission of the abnormal values not being recognized in 5 minutes. Whereas, Copper et al, (3) found the Anthony V. Beran, PhD* meantime between an event and detection with a stethoscope was 34 seconds. This Introduction suggests that changes in cardio-pulmonary function may be detected more readily with a Sometimes we put so much emphasis on stethoscope (1). Auscultation of heart and lung electronic monitoring devices we forget that sounds during perioperative period is useful our own senses often detect things before a only if the Esophageal Stethoscope provides machine can. Seeing condensation in airway strong, clear transmission of the sounds to the device or clear mask can serve to indicate the anesthesia provider. This study evaluates the presence of ventilation before the signal has sound transmission properties of several even reached the equipment. Sometimes the Esophageal Stethoscopes currently available in sense of smell can be the first thing to aid in the the market. detection of a disconnected airway device or circuit. Similarly, in some situations listening for Methods the presence of abnormal heart or airway sounds can help detect the onset of critical To evaluate the sound transmission properties incidents quicker than electronic monitors. But of the Esophageal Stethoscopes in vitro study in recent years the art of listening has changed was performed. A system that simulates the in the practice of Anesthesia. -
Bradycardia; Pulse Present
Bradycardia; Pulse Present History Signs and Symptoms Differential • Past medical history • HR < 60/min with hypotension, acute • Acute myocardial infarction • Medications altered mental status, chest pain, • Hypoxia / Hypothermia • Beta-Blockers acute CHF, seizures, syncope, or • Pacemaker failure • Calcium channel blockers shock secondary to bradycardia • Sinus bradycardia • Clonidine • Chest pain • Head injury (elevated ICP) or Stroke • Digoxin • Respiratory distress • Spinal cord lesion • Pacemaker • Hypotension or Shock • Sick sinus syndrome • Altered mental status • AV blocks (1°, 2°, or 3°) • Syncope • Overdose Heart Rate < 60 / min and Symptomatic: Exit to Hypotension, Acute AMS, Ischemic Chest Pain, Appropriate NO Acute CHF, Seizures, Syncope, or Shock Protocol(s) secondary to bradycardia Typically HR < 50 / min YES Airway Protocol(s) AR 1, 2, 3 if indicated Respiratory Distress Reversible Causes Protocol AR 4 if indicated Hypovolemia Hypoxia Chest Pain: Cardiac and STEMI Section Cardiac Protocol Adult Protocol AC 4 Hydrogen ion (acidosis) if indicated Hypothermia Hypo / Hyperkalemia Search for Reversible Causes B Tension pneumothorax 12 Lead ECG Procedure Tamponade; cardiac Toxins Suspected Beta- IV / IO Protocol UP 6 Thrombosis; pulmonary Blocker or Calcium P Cardiac Monitor (PE) Channel Blocker Thrombosis; coronary (MI) A Follow Overdose/ Toxic Ingestion Protocol TE 7 P If No Improvement Transcutaneous Pacing Procedure P (Consider earlier in 2nd or 3rd AVB) Notify Destination or Contact Medical Control Revised AC 2 01/01/2021 Any local EMS System changes to this document must follow the NC OEMS Protocol Change Policy and be approved by OEMS 1 Bradycardia; Pulse Present Adult Cardiac Adult Section Protocol Pearls • Recommended Exam: Mental Status, HEENT, Skin, Heart, Lungs, Abdomen, Back, Extremities, Neuro • Identifying signs and symptoms of poor perfusion caused by bradycardia are paramount. -
1. Intermittent Chest Pain: Angina: • Stable: (Caused By
CVS: 1. Intermittent chest pain: Angina: • Stable: (caused by chronic narrowing in one or more coronary arteries), episodes of pain are precipitated by exertion and may occur more readily when walking in cold or windy weather, after a large meal or while carrying a heavy load; the pain is promptly relieved by rest and/or sublingual glyceryl nitrate (GTN) spray, and typically lasts for less than 10 minutes. • unstable angina (caused by a sudden severe narrowing in a coronary artery), there is usually an abrupt onset or worsening of chest pain episodes that may occur on minimal exertion or at rest. • Retrosternal/ Progressive onset/ increase in intensity over 1–2 minutes/ Constricting, heavy/ Sometimes arm(s), neck, epigastrium/ Associated with breathlessness/ Intermittent, with episodes lasting 2–10 minutes/ Triggered by emotion, exertion, especially if cold, windy/ Relieved by rest, nitrates Mild to moderate. • Aggravated by thyroxine or drug-induced anemia, e.g. aspirin or NSAIDs Esophageal: • Retrosternal or epigastric/ Over 1–2 minutes; can be sudden (spasm)/ C: Gripping, tight or burning/ R: Often to back, sometimes to arms/ A: Heartburn, acid reflux/ T: Intermittent, often at night-time; variable duration/ Lying flat/some foods may trigger/ Not relieved by rest; nitrates sometimes relieve/ Usually mild but esophageal spasm can mimic myocardial infarction. 2. Acute chest pain: MI: • SOCRATES: Retrosternal/ Rapid over a few minutes/ Constricting, heavy/ Often to arm(s), neck, jaw, sometimes epigastrium/ Sweating, nausea, vomiting, breathlessness, feeling of impending death (angor animi)/ Acute presentation; prolonged duration/ ’Stress’ and exercise rare triggers, usually spontaneous/ Not relieved by rest or nitrates/ Usually severe. -
Scoliosis, Alters the Position of the Beat
Br Heart J: first published as 10.1136/hrt.8.3.162 on 1 July 1946. Downloaded from THE HEART IN STERNAL DEPRESSION BY WILLIAM EVANS From the Cardiac Department ofthe London Hospital Received June 25, 1946 The place where the apex beat appears on the chest wall depends as much on the symmetry of the thorax as on the size of the heart. A change in the alignment of the spine, the posterior fulcrum of the thoracic cage, in the form of scoliosis, alters the position of the beat. Local deformity of the ribs which form the walls of the cage will do the same thing. Deformity of the sternum, the anterior fulcrum of the thorax, as a cause of displacement of the apex beat has received less attention. The effects of depression of the sternum (pectus excavatum) on the shape and position of the heart have been studied in sixteen adults examined during the past year. DESCRIPTION OF CASES All sixteen patients had been referred for an explanation of certain signs connected with the heart, with the knowledge that deformity of the chest was present, but without appreciating that the two conditions might be related. In many of them suspicion of heart disease had led to restriction of their physical activities and to a change of design for their future livelihood. The symptoms that had caused the patients to seek medical advice in the first place were http://heart.bmj.com/ TABLE I SUMMARY OF FINDNGS IN 16 HEALTHY SUBJECTS WITH DEPRESSED STERNUM Antero-posterior Radiological findings in anterior view chest measurement _ _ Case Age Sternal in inches No. -
Arrhythmia What Is It?
Arrhythmia What is it? Most of us have felt our heart race or skip a beat. It’s fairly normal every once and a while. But for some people, it’s a sign of arrhythmia – a disorder of your heart rate or rhythm – that needs to be checked out by a specialist. If you have an arrhythmia (there are multiple types), your heart either beats: • too fast • too slow or • with an irregular pattern Did You Know? This change in your heart rhythm is usually caused by a “glitch” Our heart beats an average of in your heart’s electrical activity, which tells the heart when to 70 to 80 times a minute and contract and pump blood to the body. Your heart doesn’t beat over 100,000 times a day! It’s with the regularity of a Swiss watch, and many factors can cause no wonder millions of people an irregularity. notice palpitations such as skipping a beat, fluttering or a Some of these factors include: racing heart. • having had a heart attack • having heart failure • blood chemistry imbalances • abnormal hormone levels • alcohol, caffeine and other substances or medicines • a variety of inherited abnormalities 8 Tips for Staying Heart Healthy with Arrhythmias Living with an arrhythmia varies tremendously from one person to the next. It will depend on the type of arrhythmia you have, how serious it is and the recommended treatment. Some people can take a single medication to correct their heart’s rhythm; others undergo electrophysiology studies or require a pacemaker or implantable defibrillator. No matter what kind of arrhythmia you have, there are things you can do to keep your heart healthy and ticking as it should. -
5 Precordial Pulsations
Chapter 5 / Precordial Pulsations 113 5 Precordial Pulsations CONTENTS MECHANICS AND PHYSIOLOGY OF THE NORMAL APICAL IMPULSE PHYSICAL PRINCIPLES GOVERNING THE FORMATION OF THE APICAL IMPULSE NORMAL APICAL IMPULSE AND ITS DETERMINANTS ASSESSMENT OF THE APICAL IMPULSE LEFT PARASTERNAL AND STERNAL MOVEMENTS RIGHT PARASTERNAL MOVEMENT PULSATIONS OVER THE CLAVICULAR HEADS PULSATIONS OVER THE SECOND AND/OR THIRD LEFT INTERCOSTAL SPACES SUBXIPHOID IMPULSE PRACTICAL POINTS IN THE CLINICAL ASSESSMENT OF PRECORDIAL PULSATIONS REFERENCES In this chapter the pulsations of the precordium will be discussed in relation to their identification, the mechanisms of their origin, and their pathophysiological and clinical significance. Precordial pulsations include the “apical impulse,” left parasternal movement, right parasternal movement, pulsations of the clavicular heads, pulsations over the second left intercostal space, and subxiphoid impulses. MECHANICS AND PHYSIOLOGY OF THE NORMAL APICAL IMPULSE Since during systole the heart contracts, becoming smaller and therefore moving away from the chest wall, why should one feel a systolic outward movement (the apical impulse) at all? Logically speaking there should not be an apical impulse. Several different methods of recording the precordial motion have been used to study the apical impulse going back to the late 19th century (1,2). Among the more modern methods, the notable ones are the recordings of the apexcardiogram (3–17), the impulse cardiogram (18), and the kinetocardiogram (19–21). While apexcardiography records the relative displacement of the chest wall under the transducer pickup device, which is often held by the examiner’s hands, the proponents of the impulse cardiography and kinetocardiography point out that these methods allow the recording of the absolute movement of the chest wall because the pickup device is anchored to a fixed point held 113 114 Cardiac Physical Examination in space away from the chest. -
Jugular Venous Pressure
NURSING Jugular Venous Pressure: Measuring PRACTICE & SKILL What is Measuring Jugular Venous Pressure? Measuring jugular venous pressure (JVP) is a noninvasive physical examination technique used to indirectly measure central venous pressure(i.e., the pressure of the blood in the superior and inferior vena cava close to the right atrium). It is a part of a complete cardiovascular assessment. (For more information on cardiovascular assessment in adults, see Nursing Practice & Skill ... Physical Assessment: Performing a Cardiovascular Assessment in Adults ) › What: Measuring JVP is a screening mechanism to identify abnormalities in venous return, blood volume, and right heart hemodynamics › How: JVP is determined by measuring the vertical distance between the sternal angle and the highest point of the visible venous pulsation in the internal jugular vein orthe height of the column of blood in the external jugular vein › Where: JVP can be measured in inpatient, outpatient, and residential settings › Who: Nurses, nurse practitioners, physician assistants, and treating clinicians can measure JVP as part of a complete cardiovascular assessment What is the Desired Outcome of Measuring Jugular Venous Pressure? › The desired outcome of measuring JVP is to establish the patient’s JVP within the normal range or for abnormal JVP to be identified so that appropriate treatment may be initiated. Patients’ level of activity should not be affected by having had the JVP measured ICD-9 Why is Measuring Jugular Venous Pressure Important? 89.62 › The JVP is -
The Carotid Bruit on September 25, 2021 by Guest
AUGUST 2002 221 Pract Neurol: first published as 10.1046/j.1474-7766.2002.00078.x on 1 August 2002. Downloaded from INTRODUCTION When faced with a patient who may have had a NEUROLOGICAL SIGN stroke or transient ischaemic attack (TIA), one needs to ask oneself some simple questions: was the event vascular?; where was the brain lesion, and hence its vascular territory?; what was the cause? A careful history and focused physical examination are essential steps in getting the right answers. Although one can learn a great deal about the state of a patient’s arteries from expensive vascular imaging techniques, this does not make simple auscultation of the neck for carotid bruits redundant. In this brief review, we will therefore defi ne the place of the bruit in the diagnosis and management of patients with suspected TIA or stroke. WHY ARE CAROTID BRUITS IMPORTANT? A bruit over the carotid region is important because it may indicate the presence of athero- sclerotic plaque in the carotid arteries. Throm- boembolism from atherosclerotic plaque at the carotid artery bifurcation is a major cause of TIA and ischaemic stroke. Plaques occur preferentially at the carotid bifurcation, usually fi rst on the posterior wall of the internal carotid artery origin. The growth of these plaques and their subsequent disintegration, surface ulcera- tion, and capacity to throw off emboli into the Figure 1 Where to listen for a brain and eye determines the pattern of subse- bifurcation/internal carotid quent symptoms. The presence of an arterial http://pn.bmj.com/ artery origin bruit – high up bruit arising from stenosis at the origin of the under the angle of the jaw. -
CARDIOLOGY Section Editors: Dr
2 CARDIOLOGY Section Editors: Dr. Mustafa Toma and Dr. Jason Andrade Aortic Dissection DIFFERENTIAL DIAGNOSIS PATHOPHYSIOLOGY (CONT’D) CARDIAC DEBAKEY—I ¼ ascending and at least aortic arch, MYOCARDIAL—myocardial infarction, angina II ¼ ascending only, III ¼ originates in descending VALVULAR—aortic stenosis, aortic regurgitation and extends proximally or distally PERICARDIAL—pericarditis RISK FACTORS VASCULAR—aortic dissection COMMON—hypertension, age, male RESPIRATORY VASCULITIS—Takayasu arteritis, giant cell arteritis, PARENCHYMAL—pneumonia, cancer rheumatoid arthritis, syphilitic aortitis PLEURAL—pneumothorax, pneumomediasti- COLLAGEN DISORDERS—Marfan syndrome, Ehlers– num, pleural effusion, pleuritis Danlos syndrome, cystic medial necrosis VASCULAR—pulmonary embolism, pulmonary VALVULAR—bicuspid aortic valve, aortic coarcta- hypertension tion, Turner syndrome, aortic valve replacement GI—esophagitis, esophageal cancer, GERD, peptic OTHERS—cocaine, trauma ulcer disease, Boerhaave’s, cholecystitis, pancreatitis CLINICAL FEATURES OTHERS—musculoskeletal, shingles, anxiety RATIONAL CLINICAL EXAMINATION SERIES: DOES THIS PATIENT HAVE AN ACUTE THORACIC PATHOPHYSIOLOGY AORTIC DISSECTION? ANATOMY—layers of aorta include intima, media, LR+ LRÀ and adventitia. Majority of tears found in ascending History aorta right lateral wall where the greatest shear force Hypertension 1.6 0.5 upon the artery wall is produced Sudden chest pain 1.6 0.3 AORTIC TEAR AND EXTENSION—aortic tear may Tearing or ripping pain 1.2–10.8 0.4–0.99 produce