A Abatacept, 1491 ABCDE Acronym, 1185 ABCDE Rule, 771 Abdominal
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Glossary for Narrative Writing
Periodontal Assessment and Treatment Planning Gingival description Color: o pink o erythematous o cyanotic o racial pigmentation o metallic pigmentation o uniformity Contour: o recession o clefts o enlarged papillae o cratered papillae o blunted papillae o highly rolled o bulbous o knife-edged o scalloped o stippled Consistency: o firm o edematous o hyperplastic o fibrotic Band of gingiva: o amount o quality o location o treatability Bleeding tendency: o sulcus base, lining o gingival margins Suppuration Sinus tract formation Pocket depths Pseudopockets Frena Pain Other pathology Dental Description Defective restorations: o overhangs o open contacts o poor contours Fractured cusps 1 ww.links2success.biz [email protected] 914-303-6464 Caries Deposits: o Type . plaque . calculus . stain . matera alba o Location . supragingival . subgingival o Severity . mild . moderate . severe Wear facets Percussion sensitivity Tooth vitality Attrition, erosion, abrasion Occlusal plane level Occlusion findings Furcations Mobility Fremitus Radiographic findings Film dates Crown:root ratio Amount of bone loss o horizontal; vertical o localized; generalized Root length and shape Overhangs Bulbous crowns Fenestrations Dehiscences Tooth resorption Retained root tips Impacted teeth Root proximities Tilted teeth Radiolucencies/opacities Etiologic factors Local: o plaque o calculus o overhangs 2 ww.links2success.biz [email protected] 914-303-6464 o orthodontic apparatus o open margins o open contacts o improper -
Pediatrics-EOR-Outline.Pdf
DERMATOLOGY – 15% Acne Vulgaris Inflammatory skin condition assoc. with papules & pustules involving pilosebaceous units Pathophysiology: • 4 main factors – follicular hyperkeratinization with plugging of sebaceous ducts, increased sebum production, Propionibacterium acnes overgrowth within follicles, & inflammatory response • Hormonal activation of pilosebaceous glands which may cause cyclic flares that coincide with menstruation Clinical Manifestations: • In areas with increased sebaceous glands (face, back, chest, upper arms) • Stage I: Comedones: small, inflammatory bumps from clogged pores - Open comedones (blackheads): incomplete blockage - Closed comedones (whiteheads): complete blockage • Stage II: Inflammatory: papules or pustules surrounded by inflammation • Stage III: Nodular or cystic acne: heals with scarring Differential Diagnosis: • Differentiate from rosacea which has no comedones** • Perioral dermatitis based on perioral and periorbital location • CS-induced acne lacks comedones and pustules are in same stage of development Diagnosis: • Mild: comedones, small amounts of papules &/or pustules • Moderate: comedones, larger amounts of papules &/or pustules • Severe: nodular (>5mm) or cystic Management: • Mild: topical – azelaic acid, salicylic acid, benzoyl peroxide, retinoids, Tretinoin topical (Retin A) or topical antibiotics [Clindamycin or Erythromycin with Benzoyl peroxide] • Moderate: above + oral antibiotics [Minocycline 50mg PO qd or Doxycycline 100 mg PO qd], spironolactone • Severe (refractory nodular acne): oral -
Ankylosed Primary Molars, Andlaw (1974) Described Surface Defects from Bicuspids Preceded by Non-Ankylosed 11 Molars
PEDIATRICDENTISTRY/Copyright (~) 1980 The AmericanAcademy of Pedodontics/Vol. 2, No, 1 Ankylosedprimary mola.rs: Results and treatment recommendat,onsfrom an eight-year longitudinal study Louise Brearley Messer,B.D.Sc., L.D.S., M.D.Sc. Jay T. Cline, D.D.S., M.A. Abstract continues concomitantly with vertical alveolar bone growth,~,3 and the tooth is immobile to manual rock- A total of 263 ankyloscd primarymolars in 107 ing.4,5 children aged three to 12 years was studied for four years. Forty-six children remainedin the study for eight years. The etiology of the condition remains unknown. Extrinsic causative factors implicated are local me- Observationof affected dentitions showedthat the con- ~ ~ dition waslikely to recur. Threeclinical pa~ternsfor the chanical trauma, disturbed local metabolism, local- condition are described. Typically, maxillary molars be- ized infection, 6 chemical or thermal irritation 7 and came ankylosed earlier and demonstrated more severe tooth reimplantation, s Intrinsic factors cited include a in[raocclusion than mandibularmolars. Mandibularfirst genetic or congenital gap in the periodontal liga- molars usually remainedslightly or moderatelyin#a- ment. Since both erupting and exfoliating teeth show occluded; mandibularsecond molars and maxillary first alternating periods of resorption and deposition of and second molars showedprogressively severe infra- bone and cementum,° aberrant deposition of these tis- occlusion. Followingeither extraction or ex~oliation of the suesI° may produce an area of ankylosing tissue.Z, affected molars, the succedaneousbicuspids did not differ In a summaryof studies reporting the prevalence of in either coronal morphologyor in distribution of enamel ankylosed primary molars, Andlaw (1974) described surface defects from bicuspids preceded by non-ankylosed 11 molars. -
Oral Allergy Syndrome (OAS)
Oral Allergy Syndrome (OAS) The itchy, watery eyes, or that sudden tingling, itching or burning sensation in your mouth is all too familiar: it must be ragweed season again! After your soccer game, the juicy watermelon you share with a friend makes your mouth itchy, and you decide that maybe next time you will have to pass on the watermelon. But this is strange: you knew you were allergic to ragweed, but your reaction to watermelon is brand new. Although there is still so much we do not know about allergies, we do know that certain types of foods, or pollen like ragweed, are common culprits when it comes to giving the body an allergic reaction. Allergic reactions happen when a person’s immune system recognizes certain proteins called allergens, as foreign or unsafe. The body’s immune system then triggers an allergic response, like the swelling in your tongue and lips, to fight off the allergen. Oral Allergy Syndrome Some allergies can be much more complex, even downright sneaky. Oral Allergy Syndrome (OAS) is one such allergy. Certain types of fruits, vegetables, and nuts can trigger OAS, but you can also develop OAS even if you were not previously allergic to any of these foods. OAS only occurs in people who have pollen allergies. It is caused by allergens in fruit, vegetables and nuts that are very similar to allergens in pollen. Most only experience oral symptoms, but about 10% can experience nausea or stomach upset, and less than 5% will develop more serious whole-body allergic reactions, such as generalized hives, trouble breathing, or loss of consciousness. -
Nutrition Perspectives
Volume 44 Issue 2, March/April 2019 NutritionUniversity of California, Davis, Department ofPerspectives Nutrition and the Center for Nutrition in Schools Magnesium Helps Keep Vitamin D Levels Table of From Being Too Low or Too High Contents If some is good, more is better, right? Not always, Magnesium Helps especially when it comes to Keep Vitamin D 1 vitamin D. Vitamin D plays Levels From Being an integral role in calcium Too Low or Too High absorption and in bone health. Vitamin D deficiency has been linked to variety of Letter from diseases, including certain 2 types of cancer, multiple the Editors sclerosis cardiovascular disease, arthritis, osteoporosis, diabetes, and rickets. On the other hand, too much vitamin D can cause What is Oral toxicity, with symptoms such as GI discomfort, diarrhea, irregular 3 heartbeat, drowsiness, headaches, and muscle and joint pain. Allergy Syndome? Past studies suggest that magnesium supplementation may help maintain levels of vitamin D in the blood in the sweet spot of not too high or too low. Spicy Food May Help In order to understand how magnesium affects vitamin D in Preventing High 5 regulation, researchers at the Vanderbilt-Ingram Cancer Center Blood Pressure conducted a study to determine how magnesium supplements impact vitamin D levels in the blood. Participants (n=180) that were considered high risk Compound in Pomegranates May of developing colon cancer 7 were randomly assigned to Help Prevent Damage receive either a magnesium from IBD in Mice supplement or a placebo. Over 12 weeks, participants visited the clinic three times Fat Around the Middle May Be to provide blood samples and 8 have their height and weight Influenced by the Types of Food We Eat Magnesium continued on page 3 Volume 44 Letter from the Editors Welcome to a special UC Davis student edition of Nutrition Perspectives. -
Pharmacology of Local Anesthesia
By Mohammad Hussein Zaki Lecturer Oral & Maxillofacial Surgery Faculty of Dentistry – Minia University . Impacted tooth. Failure of the tooth to fully erupt into the oral cavity within its expected developmental time period and can no longer reasonably be expected to do so. A tooth that can not, or will not, erupt into its normal functioning positions. Impacted tooth. An impacted tooth is one that is erupted, partially erupted or unerupted and will not eventually assume a normal arch relationship with the other teeth and tissues. Unerupted tooth. Includes impacted teeth and teeth that are in the process of erupting. Impacted teeth seen in the following order of frequency: 1. Mandibular third molars. 2. Maxillary third molars. 3. Maxillary canine. 4. Mandibular premolar. 5. Maxillary premolar. 6. Mandibular canine. 7. Maxillary central incisors. 8. Maxillary lateral incisors. Systemic factors: • Syndromes. Cleidocranial dysplasia. • Endocrine deficiencies. Hypothyroidism. • Irradiation. Local factors: • Obstruction for eruption. Irregularity in position of an adjacent tooth. Density of the overlying soft tissue or bone. Prolonged deciduous tooth retention. Supernumerary teeth. • Tooth ankylosis. • Dilaceration of roots. • Malposed tooth germs. Local factors: • Pathological lesions. • Cleft lip and palate. • Arch-length deficiency. Phylogenic theory. Mendelian theory. Clinical. Radiographic. • Periapical. Radiographic. • Periapical. Radiographic. • Occlusal. Radiographic. • Panorama. Radiographic. • Panorama. Radiographic. • CT. Radiographic. • CBCT. Impacted teeth other than third molars. Exposure. • A procedure that allows natural eruption of impacted teeth. • It should be done as soon as it is determined that the tooth is not going to erupt spontaneously. • Spontaneous eruption versus orthodontic assistance. Impacted teeth other than third molars. Exposure. Impacted teeth other than third molars. -
Eruption Abnormalities in Permanent Molars: Differential Diagnosis and Radiographic Exploration
DOI: 10.1051/odfen/2014054 J Dentofacial Anom Orthod 2015;18:403 © The authors Eruption abnormalities in permanent molars: differential diagnosis and radiographic exploration J. Cohen-Lévy1, N. Cohen2 1 Dental surgeon, DFO specialist 2 Dental surgeon ABSTRACT Abnormalities of permanent molar eruption are relatively rare, and particularly difficult to deal with,. Diagnosis is founded mainly on radiographs, the systematic analysis of which is detailed here. Necessary terms such as non-eruption, impaction, embedding, primary failure of eruption and ankylosis are defined and situated in their clinical context, illustrated by typical cases. KEY WORDS Molars, impaction, primary failure of eruption (PFE), dilaceration, ankylosis INTRODUCTION Dental eruption is a complex developmen- at 0.08% for second maxillary molars and tal process during which the dental germ 0.01% for first mandibular molars. More re- moves in a coordinated fashion through cently, considerably higher prevalence rates time and space as it continues the edifica- were reported in retrospective studies based tion of the root; its 3-dimensional pathway on orthodontic consultation records: 2.3% crosses the alveolar bone up to the oral for second molar eruption abnormalities as epithelium to reach its final position in the a whole, comprising 1.5% ectopic eruption, occlusion plane. This local process is regu- 0.2% impaction and 0.6% primary failure of lated by genes expressing in the dental fol- eruption (PFE) (Bondemark and Tsiopa4), and licle, at critical periods following a precise up to 1.36% permanent second molar iim- chronology, bilaterally coordinated with fa- paction according to Cassetta et al.6. cial growth. -
Temporo-Mandibular Joint Disorders and Homoeopathy
SUBJECTIVE Temporo-mandibular joint disorders and homoeopathy By Dr Priya Singh Abstract: TMDs are the second most common musculoskeletal problem resulting in pain and disability.It can be really painful and disturbing in day today activities. People usually ignore the symptoms and opt for treatments only in severe cases.The homoeopathic literature is filled with lots of drugs that can help to manage signs and symptoms of this disorder and produce effective cures based on simillimum. This article deals with homoeopathic approach to management of TMDs. Keywords: temporo-mandibular joint, temporo-mandibular joint disorders,treatment,homoeopathy Abbreviations: TMJ- temporo-mandibular joint, TMD- temporo-mandibular disease, DC/TMD- diagnostic criteria for temporomandibular disorders Introduction Depending on the practitioner and in the female population, compared the diagnostic methodology, the to males. Scientists relate to the he temporo-mandibular joint is term TMD has been used to charac- female jaw structure, vitamin Tone of the most heavily utilised terise a wide range of conditions di- deficiencies, varying hormones, and underappreciated joints in the versely presented as pain in the face pain gene variant and internal stress human body. Mechanically, the or the jaw joint area, limited mouth management. Young females less TMJ is what allows you to open and opening, closed or open lock of the than 30 years old are at increased TMJ, abnormal occlusal wear, click- close your mouth,and extend and risk of temporo-mandibular joint ing or popping sounds in the jaw move your jaw from side to side. disorder.In contrast to the previous joints, and other complaints.[1] Functionally, it facilitates chewing, reports, some recent studies have talking, and facial expressions. -
Avoidable Complication and Patient Care During Orthodontic Treatment
Suhashini Ramanathan et al /J. Pharm. Sci. & Res. Vol. 7(12), 2015, 1096-1098 Avoidable Complication and Patient Care during Orthodontic Treatment 1 2 Dr. Suhashini Ramanathan BDS , Dr. Navaneethan Ramasamy MDS(ORTHODONTICS) Saveetha Dental College and Hospitals Chennai Abstract Aim: Orthodontic treatment helps in improving facial and dental aesthetics. Orthodontic treatment involves the usage brackets,bands,wires inside the oral cavity. During the course of treatment, proper care of the appliances by the patient and the Orthodontist is essential. Objective: Helps in better treatment and to avoid any complication during the course of the treatment. Background: The brackets and bands provide for a rough surface which leads to increased plaque and calculus accumulation. Arch wires, brackets and bands can also lead to ulcerations in the oral mucosa. The Orthodontic tooth movement also leads to certain complications like root resorption, gingival enlargement, loss of tooth vitality etc. This is further complicated by the allergic tendencies of the patient to certain materials used in Orthodontic therapy. Reason for the study: Hence it is imperative that the patient as well as the dentist is made aware of the various complications that can occur with Orthodontic treatment and how to deal with them. This review would serve to do the same. INTRODUCTION 2. root- root resortion Every treatment in the dental specialty has its own set of ankylosis complications orthodontic therapy being no exception. 3. pulp-ischemia Dental aesthetics are a key factor in overall physical pulpitis attractiveness, which also contributes to self-esteem.1This necrosis is one of the main reasons for patients to undergo 4. -
Gingival Diseases in Children and Adolescents
8932 Indian Journal of Forensic Medicine & Toxicology, October-December 2020, Vol. 14, No. 4 Gingival Diseases in Children and Adolescents Sulagna Pradhan1, Sushant Mohanty2, Sonu Acharya3, Mrinali Shukla1, Sonali Bhuyan1 1Post Graduate Trainee, 2Professor & Head, 3Professor, Department of Paediatric and Preventive Dentistry, Institute of Dental Sciences, Siksha O Anusandhan (Deemed to be University), Bhubaneswar 751003, Odisha, India Abstract Gingival diseases are prevalent in both children and adolescents. These diseases may or may not be associated with plaques, maybe familial in some cases, or may coexist with systemic illness. However, gingiva and periodontium receive scant attention as the primary dentition does not last for a considerable duration. As gingival diseases result in the marked breakdown of periodontal tissue, and premature tooth loss affecting the nutrition and global development of a child/adolescent, precise identification and management of gingival diseases is of paramount importance. This article comprehensively discusses the nature, spectrum, and management of gingival diseases. Keywords: Gingival diseases; children and adolescents; spectrum, and management. Introduction reddish epithelium with mild keratinization may be misdiagnosed as inflammation. Lesser variability in the Children are more susceptible to several gingival width of the attached gingiva in the primary dentition diseases, paralleling to those observed in adults, though results in fewer mucogingival problems. The interdental vary in numerous aspects. Occasionally, natural variations papilla is broad buccolingual, and narrow mesiodistally. in the gingiva can masquerade as genuine pathology.1 The junctional epithelium associated with the deciduous On the contrary, a manifestation of a life-threatening dentition is thicker than the permanent dentition. underlying condition is misdiagnosed as normal gingiva. -
Pathogenic Viruses Commonly Present in the Oral Cavity and Relevant Antiviral Compounds Derived from Natural Products
medicines Review Pathogenic Viruses Commonly Present in the Oral Cavity and Relevant Antiviral Compounds Derived from Natural Products Daisuke Asai and Hideki Nakashima * Department of Microbiology, St. Marianna University School of Medicine, Kawasaki 216-8511, Japan * Correspondence: [email protected]; Tel.: +81-44-977-8111 Received: 24 October 2018; Accepted: 7 November 2018; Published: 12 November 2018 Abstract: Many viruses, such as human herpesviruses, may be present in the human oral cavity, but most are usually asymptomatic. However, if individuals become immunocompromised by age, illness, or as a side effect of therapy, these dormant viruses can be activated and produce a variety of pathological changes in the oral mucosa. Unfortunately, available treatments for viral infectious diseases are limited, because (1) there are diseases for which no treatment is available; (2) drug-resistant strains of virus may appear; (3) incomplete eradication of virus may lead to recurrence. Rational design strategies are widely used to optimize the potency and selectivity of drug candidates, but discovery of leads for new antiviral agents, especially leads with novel structures, still relies mostly on large-scale screening programs, and many hits are found among natural products, such as extracts of marine sponges, sea algae, plants, and arthropods. Here, we review representative viruses found in the human oral cavity and their effects, together with relevant antiviral compounds derived from natural products. We also highlight some recent emerging pharmaceutical technologies with potential to deliver antivirals more effectively for disease prevention and therapy. Keywords: anti-human immunodeficiency virus (HIV); antiviral; natural product; human virus 1. Introduction The human oral cavity is home to a rich microbial flora, including bacteria, fungi, and viruses. -
Complications in Pediatric Facial Fractures
Complications in Pediatric Facial Fractures Mimi T. Chao, M.D.,1 and Joseph E. Losee, M.D.1 ABSTRACT Despite recent advances in the diagnosis, treatment, and prevention of pediatric facial fractures, little has been published on the complications of these fractures. The existing literature is highly variable regarding both the definition and the reporting of adverse events. Although the incidence of pediatric facial fractures is relative low, they are strongly associated with other serious injuries. Both the fractures and their treatment may have long-term consequence on growth and development of the immature face. This article is a selective review of the literature on facial fracture complications with special emphasis on the complications unique to pediatric patients. We also present our classification system to evaluate adverse outcomes associated with pediatric facial fractures. Prospective, long- term studies are needed to fully understand and appreciate the complexity of treating children with facial fractures and determining the true incidence, subsequent growth, and nature of their complications. KEYWORDS: Pediatric facial fracture, complications The treatment of pediatric facial fractures is mandibular nerve palsy after open reduction and internal constantly evolving, and recent advances in prevention, fixation of a mandible fracture); and type 3—those diagnosis, and management were reviewed by Zimmer- resulting from subsequent growth and development mann et al in 2006.1 This article is a selective review of (i.e., asymmetric mandibular growth after condylar frac- the literature, expanding upon the adverse outcomes or ture). A patient may have any or all of these types of complications commonly seen during the management adverse outcome (i.e., malocclusion following mandibu- of pediatric facial trauma patients.