Hiatal Hernia
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Umbilical Hernia with Cholelithiasis and Hiatal Hernia
View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by Springer - Publisher Connector Yamanaka et al. Surgical Case Reports (2015) 1:65 DOI 10.1186/s40792-015-0067-8 CASE REPORT Open Access Umbilical hernia with cholelithiasis and hiatal hernia: a clinical entity similar to Saint’striad Takahiro Yamanaka*, Tatsuya Miyazaki, Yuji Kumakura, Hiroaki Honjo, Keigo Hara, Takehiko Yokobori, Makoto Sakai, Makoto Sohda and Hiroyuki Kuwano Abstract We experienced two cases involving the simultaneous presence of cholelithiasis, hiatal hernia, and umbilical hernia. Both patients were female and overweight (body mass index of 25.0–29.9 kg/m2) and had a history of pregnancy and surgical treatment of cholelithiasis. Additionally, both patients had two of the three conditions of Saint’s triad. Based on analysis of the pathogenesis of these two cases, we consider that these four diseases (Saint’s triad and umbilical hernia) are associated with one another. Obesity is a common risk factor for both umbilical hernia and Saint’s triad. Female sex, older age, and a history of pregnancy are common risk factors for umbilical hernia and two of the three conditions of Saint’s triad. Thus, umbilical hernia may readily develop with Saint’s triad. Knowledge of this coincidence is important in the clinical setting. The concomitant occurrence of Saint’s triad and umbilical hernia may be another clinical “tetralogy.” Keywords: Saint’s triad; Cholelithiasis; Hiatal hernia; Umbilical hernia Background of our knowledge, no previous reports have described the Saint’s triad is characterized by the concomitant occur- coexistence of umbilical hernia with any of the three con- rence of cholelithiasis, hiatal hernia, and colonic diverticu- ditions of Saint’s triad. -
PYLORIC STENOSIS of INFANCY-PERSPECTIVES Different Views from Different Bridges
Central Annals of Pediatrics & Child Health Perspective *Corresponding author IAN M. ROGERS, Department of Surgery, AIMST University, 46 Whitburn Road, Cleadon, Tyne and Wear, SR67QS, Kedah, Malaysia, Tel: 01915367944; PYLORIC STENOSIS OF INFANCY- 07772967160; Email: [email protected] Submitted: 18 March 2020 PERSPECTIVES Different views Accepted: 27 March 2020 Published: 30 March 2020 ISSN: 2373-9312 from different bridges Copyright © 2020 ROGERS IM IAN M ROGERS* OPEN ACCESS Department of Surgery, AIMST University, Malaysia Keywords Abstract • Neonatal hyperacidity; Neonatal hypergastrinaemia; Baby and parents perspectives of diagnosis Introduction: The differing experiences of the surgeon, he parent and the baby and treatment; Spectrum of presentation of are discussed in terms of what is presently known about the pathogenesis of pyloric pyloric stenosis of infancy; Primary hyperacidity stenosis of infancy (PS). pathogenesis of pyloric stenosis Methods: The experiences are culled from personal experience and from the comments made on online pressure and support groups for pyloric stenosis of infancy. These comments are reviewed from the standpoint of the Primary Hyperacidity pathogenesis of cause. Conclusion: The present perspectives of the PS surgeon and the difficulties experienced by the PS family fit well which the basic tenets of the Primary Hyperacidity theory. Reference to this theory allows a satisfactory explanation for difficulties in diagnosis and for the variability of presentation. INTRODUCTION In 1961 this theme was further developed by Dr Jacob. He again recognised the milder forms-those in which a long- term The Surgeon cure could similarly be achieved without surgery. A 1% mortality Everybody knows: For progressive pyloric stenosis of infancy in 100 patients was achieved with the usual medical measures (PS), pyloromyotomy reigns supreme! Impending catastrophe of which a reduced feeding regime was judged to be especially transformed safely, in a few minutes, into an enduring cure. -
Abdominal Pain - Gastroesophageal Reflux Disease
ACS/ASE Medical Student Core Curriculum Abdominal Pain - Gastroesophageal Reflux Disease ABDOMINAL PAIN - GASTROESOPHAGEAL REFLUX DISEASE Epidemiology and Pathophysiology Gastroesophageal reflux disease (GERD) is one of the most commonly encountered benign foregut disorders. Approximately 20-40% of adults in the United States experience chronic GERD symptoms, and these rates are rising rapidly. GERD is the most common gastrointestinal-related disorder that is managed in outpatient primary care clinics. GERD is defined as a condition which develops when stomach contents reflux into the esophagus causing bothersome symptoms and/or complications. Mechanical failure of the antireflux mechanism is considered the cause of GERD. Mechanical failure can be secondary to functional defects of the lower esophageal sphincter or anatomic defects that result from a hiatal or paraesophageal hernia. These defects can include widening of the diaphragmatic hiatus, disturbance of the angle of His, loss of the gastroesophageal flap valve, displacement of lower esophageal sphincter into the chest, and/or failure of the phrenoesophageal membrane. Symptoms, however, can be accentuated by a variety of factors including dietary habits, eating behaviors, obesity, pregnancy, medications, delayed gastric emptying, altered esophageal mucosal resistance, and/or impaired esophageal clearance. Signs and Symptoms Typical GERD symptoms include heartburn, regurgitation, dysphagia, excessive eructation, and epigastric pain. Patients can also present with extra-esophageal symptoms including cough, hoarse voice, sore throat, and/or globus. GERD can present with a wide spectrum of disease severity ranging from mild, intermittent symptoms to severe, daily symptoms with associated esophageal and/or airway damage. For example, severe GERD can contribute to shortness of breath, worsening asthma, and/or recurrent aspiration pneumonia. -
Massive Hiatal Hernia Involving Prolapse Of
Tomida et al. Surgical Case Reports (2020) 6:11 https://doi.org/10.1186/s40792-020-0773-8 CASE REPORT Open Access Massive hiatal hernia involving prolapse of the entire stomach and pancreas resulting in pancreatitis and bile duct dilatation: a case report Hidenori Tomida* , Masahiro Hayashi and Shinichi Hashimoto Abstract Background: Hiatal hernia is defined by the permanent or intermittent prolapse of any abdominal structure into the chest through the diaphragmatic esophageal hiatus. Prolapse of the stomach, intestine, transverse colon, and spleen is relatively common, but herniation of the pancreas is a rare condition. We describe a case of acute pancreatitis and bile duct dilatation secondary to a massive hiatal hernia of pancreatic body and tail. Case presentation: An 86-year-old woman with hiatal hernia who complained of epigastric pain and vomiting was admitted to our hospital. Blood tests revealed a hyperamylasemia and abnormal liver function test. Computed tomography revealed prolapse of the massive hiatal hernia, containing the stomach and pancreatic body and tail, with peripancreatic fluid in the posterior mediastinal space as a sequel to pancreatitis. In addition, intrahepatic and extrahepatic bile ducts were seen to be dilated and deformed. After conservative treatment for pancreatitis, an elective operation was performed. There was a strong adhesion between the hernial sac and the right diaphragmatic crus. After the stomach and pancreas were pulled into the abdominal cavity, the hiatal orifice was closed by silk thread sutures (primary repair), and the mesh was fixed in front of the hernial orifice. Toupet fundoplication and intraoperative endoscopy were performed. The patient had an uneventful postoperative course post-procedure. -
Eosinophilic Gastroenteritis Presenting with Severe Anemia and Near Syncope
J Am Board Fam Med: first published as 10.3122/jabfm.2012.06.110269 on 7 November 2012. Downloaded from BRIEF REPORT Eosinophilic Gastroenteritis Presenting with Severe Anemia and Near Syncope Nneka Ekunno, DO, MPH, Kirk Munsayac, DO, Allen Pelletier, MD, and Thad Wilkins, MD Eosinophilic gastrointestinal disorders or eosinophilic digestive disorders encompass a spectrum of rare gastrointestinal disorders that includes eosinophilic esophagitis, eosinophilic gastroenteritis, and eosinophilic colitis. Eosinophilic gastroenteritis is a rare inflammatory disease characterized by eosino- philic infiltration of the gastrointestinal tract. The clinical manifestations include anemia, dyspepsia, and diarrhea. Endoscopy with biopsy showing histologic evidence of eosinophilic infiltration is consid- ered definitive for diagnosis. Corticosteroid therapy, food allergen testing, elimination diets, and ele- mental diets are considered effective treatments for eosinophilic gastroenteritis. The treatment and prognosis of eosinophilic gastroenteritis is determined by the severity of the clinical manifestations. We describe a 24-year-old woman with eosinophilic gastroenteritis presenting as epigastric pain with a history of severe iron deficiency anemia, asthma, eczema, and allergic rhinitis, and we review the litera- ture regarding presentation, diagnostic testing, pathophysiology, predisposing factors, and treatment recommendations. (J Am Board Fam Med 2012;25:913–918.) Keywords: Case Reports, Eosinophilic Gastroenteritis, Gastrointestinal Disorders copyright. A 24-year-old nulliparous African-American woman During examination, her height was 62 inches, was admitted after an episode of near syncope asso- weight 117 lb, and body mass index 21.44 kg/m2. Her ciated with 2 days of fatigue and dizziness. She re- heart rate was 111 beats per minute, blood pressure ported gradual onset of dyspepsia over 2 to 3 121/57 mm Hg, respiratory rate 20 breaths per minute, months. -
CHAPTER 8 – DIGESTIVE SYSTEM OBJECTIVES on Completion of This Chapter, You Will Be Able To: • Describe the Digestive System
CHAPTER 8 – DIGESTIVE SYSTEM OBJECTIVES On completion of this chapter, you will be able to: • Describe the digestive system. • Describe the primary organs of the digestive system and state their functions. • Describe the two sets of teeth with which humans are provided. • Describe the three main portions of a tooth. • Describe the accessory organs of the digestive system and their functions. • Describe digestive differences of the child and older adult. • Analyze, build, spell, and pronounce medical words. • Comprehend drugs highlighted in this chapter • Describe diagnostic and laboratory tests related to the digestive system • Identify and define selected abbreviations. • Describe each of the conditions presented in the Pathology Spotlights. • Complete the Pathology Checkpoint. • Complete the Study and Review section and the Chart Note Analysis. OUTLINE I. Anatomy and Physiology Overview (Fig. 8–1, p. 189) Also known as the alimentary canal and/or gastrointestinal tract, this continuous tract begins with the mouth and ends with the anus. Consist of primary and accessory organs that convert food and fluids into a semiliquid that can be absorbed for use by the body. Three main functions of the digestive system are: 1. Digestion – the process by which food is changed in the mouth, stomach, and intestines by chemical, mechanical and physical action, so that it can be absorbed by the body. 2. Absorption –the process whereby nutrient material is taken into the bloodstream or lymph and travels to all cells of the body. 3. Elimination –the process whereby the solid products of digestion are excreted. A. Mouth (Fig. 8–2, p. 190) – a cavity formed by the palate, tongue, lips, and cheeks. -
Gastroesophageal Reflux Disease (GERD)
Guidelines for Clinical Care Quality Department Ambulatory GERD Gastroesophageal Reflux Disease (GERD) Guideline Team Team Leader Patient population: Adults Joel J Heidelbaugh, MD Objective: To implement a cost-effective and evidence-based strategy for the diagnosis and Family Medicine treatment of gastroesophageal reflux disease (GERD). Team Members Key Points: R Van Harrison, PhD Diagnosis Learning Health Sciences Mark A McQuillan, MD History. If classic symptoms of heartburn and acid regurgitation dominate a patient’s history, then General Medicine they can help establish the diagnosis of GERD with sufficiently high specificity, although sensitivity Timothy T Nostrant, MD remains low compared to 24-hour pH monitoring. The presence of atypical symptoms (Table 1), Gastroenterology although common, cannot sufficiently support the clinical diagnosis of GERD [B*]. Testing. No gold standard exists for the diagnosis of GERD [A*]. Although 24-hour pH monitoring Initial Release is accepted as the standard with a sensitivity of 85% and specificity of 95%, false positives and false March 2002 negatives still exist [II B*]. Endoscopy lacks sensitivity in determining pathologic reflux but can Most Recent Major Update identify complications (eg, strictures, erosive esophagitis, Barrett’s esophagus) [I A]. Barium May 2012 radiography has limited usefulness in the diagnosis of GERD and is not recommended [III B*]. Content Reviewed Therapeutic trial. An empiric trial of anti-secretory therapy can identify patients with GERD who March 2018 lack alarm or warning symptoms (Table 2) [I A*] and may be helpful in the evaluation of those with atypical manifestations of GERD, specifically non-cardiac chest pain [II B*]. Treatment Ambulatory Clinical Lifestyle modifications. -
SAS Journal of Surgery Rare Complication of a Hernia in The
SAS Journal of Surgery Abbreviated Key Title: SAS J Surg ISSN 2454-5104 Journal homepage: https://www.saspublishers.com/sasjs/ Rare Complication of a Hernia in the Linea Alba: Generalized Peritonitis Due to Perforation by a Chicken Bone Incarcerated in the Hernia Anas Belhaj*, Mohamed Fdil, Mourad Badri, Mohammed Lazrek, Younes Hamdouni Ahmed, Zerhouni, Tarik Souiki, Imane Toughraï, Karim Ibn Majdoub Hassani, Khalid Mazaz Visceral and Endocrinological Surgery Service II, Chu Hassan II, Fes, Morocco DOI: 10.36347/sasjs.2020.v06i03.016 | Received: 05.03.2020 | Accepted: 13.03.2020 | Published: 23.03.2020 *Corresponding author: Anas Belhaj Abstract Case Report Small intestine perforation by a foreign body is a rare cause of secondary peritonitis. We report the case of peritonitis due to an exceptional mechanism; a small perforation by incarceration of a bony flap in the small bowel due to the existence of a hernia of the white line. Keywords: Peritonitis, white line hernia, fragment of bone, incarceration. Copyright @ 2020: This is an open-access article distributed under the terms of the Creative Commons Attribution license which permits unrestricted use, distribution, and reproduction in any medium for non-commercial use (NonCommercial, or CC-BY-NC) provided the original author and source are credited. NTRODUCTION I Patient was conscious, with a temperature at Acute peritonitis is the acute inflammation of 38.8 ° C, a pulse at 120 bpm, accelerated breathing rate the peritoneal serosa. It can either be generalized to the at 22 cycles / min, and coldness of the extremities. The large peritoneal cavity, or localized, following a abdominal examination found a slight distension with bacterial or chemical peritoneal attack. -
A Rare Case of Pancreatitis from Pancreatic Herniation
Case Report J Med Cases. 2018;9(5):154-156 A Rare Case of Pancreatitis From Pancreatic Herniation David Doa, c, Steven Mudrocha, Patrick Chena, Rajan Prakashb, Padmini Krishnamurthyb Abstract nia sac, resulting in mediastinal abscess which was drained sur- gically. He had a prolonged post-operative recovery following Acute pancreatitis is a commonly encountered condition, and proper the surgery. In addition, he had a remote history of exploratory workup requires evaluation for underlying causes such as gallstones, laparotomy for a retroperitoneal bleed. He did not have history alcohol, hypertriglyceridemia, trauma, and medications. We present a of alcohol abuse. His past medical history was significant for case of pancreatitis due to a rare etiology: pancreatic herniation in the hypertension and osteoarthritis. context of a type IV hiatal hernia which involves displacement of the On examination, his vitals showed a heart rate of 106 stomach with other abdominal organs into the thoracic cavity. beats/min, blood pressure of 137/85 mm Hg, temperature of 37.2 °C, and respiratory rate of 20/min. Physical exam dem- Keywords: Pancreatitis; Herniation; Hiatal hernia onstrated left-upper quadrant tenderness without guarding or rebound tenderness, with normoactive bowel sounds. Lipase was elevated at 2,687 U/L (reference range: 73 - 383 U/L). His lipase with the first episode of abdominal pain had been 1,051 U/L. Hematocrit was 41.2% (reference range: 42-52%), Introduction white cell count was 7.1 t/mm (reference range 4.8 - 10.8 t/ mm) and creatinine was 1.0 mg/dL (references range: 0.5 - 1.4 Acute pancreatitis is a commonly encountered condition with mg/dL). -
Gastroesophageal Reflux Disease
The new england journal of medicine clinical practice Gastroesophageal Reflux Disease Peter J. Kahrilas, M.D. This Journal feature begins with a case vignette highlighting a common clinical problem. Evidence supporting various strategies is then presented, followed by a review of formal guidelines, when they exist. The article ends with the author’s clinical recommendations. A 53-year-old man, who is otherwise healthy and has a 20-year history of occasional heartburn, reports having had worsening heartburn for the past 12 months, with daily symptoms that disturb his sleep. He reports having had no dysphagia, gastroin- testinal bleeding, or weight loss and in fact has recently gained 20 lb (9 kg). What would you advise regarding his evaluation and treatment? The Clinical Problem From the Division of Gastroenterology, Gastroesophageal reflux disease is the most common gastrointestinal diagnosis re- Feinberg School of Medicine, Chicago. Ad- corded during visits to outpatient clinics.1 In the United States, it is estimated that dress reprint requests to Dr. Kahrilas at the Feinberg School of Medicine, 676 St. Clair 14 to 20% of adults are affected, although such percentages are at best approxima- St., Suite 1400, Chicago, IL 60611-2951, tions, given that the disease has a nebulous definition and that such estimates are or at [email protected]. based on the prevalence of self-reported chronic heartburn.2 A current definition of N Engl J Med 2008;359:1700-7. the disorder is “a condition which develops when the ref lux of stomach contents causes Copyright © 2008 Massachusetts Medical Society. troublesome symptoms (i.e., at least two heartburn episodes per week) and/or complications.”3 Several extraesophageal manifestations of the disease are well rec- ognized, including laryngitis and cough (Table 1). -
What Is a Paraesophageal Hernia?
JAMA PATIENT PAGE What Is a Paraesophageal Hernia? A paraesophageal hernia occurs when the lower part of the esophagus, the stomach, or other organs move up into the chest. The hiatus is an opening in the diaphragm (a muscle separating the chest from the abdomen) through which organs pass from the Paraesophageal or hiatal hernia The junction between the esophagus and the stomach (the gastroesophageal chest into the abdomen. The lower part of the esophagus and or GE junction) or other organs move from the abdomen into the chest. the stomach normally reside in the abdomen, just under the dia- phragm. The gastroesophageal (GE) junction is the area where Normal location of the esophagus, Type I hiatal hernia (sliding hernia) with the GE junction and stomach The GE junction slides through the the esophagus connects with the stomach and is usually located in the abdominal cavity diaphragmatic hiatus to an abnormal 1to2inchesbelowthediaphragm.Ahiatalorparaesophagealhernia position in the chest. occurs when the GE junction, the stomach, or other abdominal or- Esophagus gans such as the small intestine, colon, or spleen move up into the GE junction chest where they do not belong. There are several types of para- Hiatus esophagealhernias.TypeIisahiatalherniaorslidinghernia,inwhich the GE junction moves above the diaphragm, leaving the stomach in D I A P H R A the abdomen; this represents 95% of all paraesophageal hernias. G M Types II, III, and IV occur when part or all of the stomach and some- S T O M A C H times other organs move up into the chest. Common Symptoms of Paraesophageal Hernia More than half of the population has a hiatal or paraesophageal Less common types of paraesophageal hernias are classified based on the extent hernia. -
A Case Report
J Pediatr Adolesc Surg. 2020; 1:89-91 OPEN ACCESS DOI: https://doi.org/10.46831/jpas.v1i2.39 Case Report Postoperative gastric Outlet Obstruction following hiatal hernia repair in an infant: A case report Muhammad Jawad Afzal,1 Shabbir Ahmad,1 Farrakh Mehmood Satar,1 Sajid Iqbal Nayyer,2 Muhammad Bilal Mirza,2 Nabila Talat1 Department of Pediatric Surgery II, The Children’s Hospital and the Institute of Child Health, Lahore Cite as: Afzal MJ, Ahmad S, Satar FM, Nayyer SI, Mirza MB, Talat N. Postoperative gastric Outlet Obstruction following hiatal hernia repair in an infant: A case report J Pediatr Adolesc Surg. 2020; 1: 89-91. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited (https://creativecommons.org/ licenses/by/4.0/). ABSTRACT Background: Infantile hypertrophic pyloric stenosis (IHPS) is an exceedingly rare cause of postoperative emesis in a case of hiatal hernia. Occasionally it may simulate other etiology of gastric outlet obstruction. Case Presentation: A 32-day-old male baby presented with respiratory distress and vomiting since birth. Diagnosis of eventra- tion of left hemi diaphragm was made on CT Chest. At surgery, hiatal hernia with an intrathoracic stomach was found, which was repaired. On 5th postoperative day, the baby developed vomiting after feeding which gradually turned to be projectile in nature over a week. Contrast meal performed showed malpositioned stomach with delayed emptying. At re-operation, a well- formed olive of pylorus was encountered; Ramstedt pyloromyotomy was done.