Postpartum Hemorrhage.Hemorrhage
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Placental Abruption
Placental Abruption Definition: Placental separation, either partial or complete prior to the birth of the fetus Incidence 0.5 – 1% (4). Risk factors include hypertension, smoking, preterm premature rupture of membranes, cocaine abuse, uterine myomas, and previous abruption (5). Diagnosis: Symptoms (may present with any or all of these) o Vaginal bleeding (usually dark and non-clotting). o Abdominal pain and/or back pain varying from intermittent to severe. o Uterine contractions are usually present and may vary from low amplitude/high frequency to hypertonus. o Fetal distress or fetal death. Ultrasound o Adherent retroplacental clot OR may just appear to be a thick placenta o Resolving hematomas become hypoechoic within one week and sonolucent within 2 weeks May be a diagnosis of exclusion if vaginal bleeding and no other identified etiology Consider in differential with uterine irritability on toco and cat II-III tracing, as small proportion present without bleeding Classification: Grade I: Slight vaginal bleeding and some uterine irritability are usually present. Maternal blood pressure, and fibrinogen levels are unaffected. FHR remains normal. Grade II: Mild to moderate vaginal bleeding seen; tetanic contractions may be present. Blood pressure usually normal, but tachycardia may be present. May be postural hypotension. Decreased fibrinogen; with levels below 250 mg percent; may be evidence of fetal distress. Reviewed 01/16/2020 1 Updated 01/16/2020 Grade III: Bleeding is moderate to severe, but may be concealed. Uterus tetanic and painful. Maternal hypotension usually present. Fetal death has occurred. Fibrinogen levels are less then 150 mg percent with thrombocytopenia and coagulation abnormalities. -
Case 1: Postpartum Hemorrhage Secondary to Uterine Atony
Case 1: Postpartum Hemorrhage Secondary to Uterine Atony Learning Objectives By the end of this scenario, each care team member should be able to successfully do the following: ▪ Recognize risk factors for postpartum hemorrhage. ▪ Identify postpartum hemorrhage due to uterine atony and be able to treat with appropriate medical management. ▪ Demonstrate teamwork and communication skills during a simulated postpartum hemorrhage. Planned Completion Points To successfully complete this scenario, the care team should successfully do the following: ▪ Recognize uterine atony as the etiology for postpartum hemorrhage. ▪ Perform uterine massage. ▪ Administer two different uterotonic medications. ▪ Call for blood (e.g. 2 units of PRBCs). OR Page | 1 © 2019 American College of Obstetricians and Gynecologists ▪ If 10 minutes has elapsed after recognition of hemorrhage and the team has not corrected the hemorrhage or called for blood. Expected Duration Approximately 60 minutes (30 minutes for simulation / 30 minutes for debriefing). Case Scenario Patient: Marla Smith Mrs. Marla Smith is a 38-year-old G3P2012 who was admitted in active labor at 39+3 weeks and had a spontaneous vaginal delivery 30 minutes ago. Her delivery was uncomplicated. She had a first-degree laceration that did not require repair. She is approximately 30 minutes postpartum and has just called out because she feels dizzy and has more bleeding. Patient Information ▪ She has no significant past medical history. ▪ She has no known drug allergies. ▪ Her pregnancy was uncomplicated except for an elevated 1-hour glucose screen with a normal 3- hour glucose tolerance test. Laboratory Data (On Admission): ▪ Hemoglobin: 12.2 ▪ Hematocrit: 36.6 ▪ WBC: 12,000 ▪ Platelets: 218,000 Delivery Information ▪ Measurement of cumulative blood loss (as quantitative as possible) from the delivery was 300cc. -
Uterine Rupture After Misoprostol Use for Termination of Pregnancy in Second Trimester with Previous Caesarean Section: a Case Report Marjan Ghaemi*
Case Report iMedPub Journals Critical Care Obstetrics and Gynecology 2018 http://www.imedpub.com/ Vol.4 No.3:14 ISSN 2471-9803 DOI: 10.21767/2471-9803.1000167 Uterine Rupture after Misoprostol Use for Termination of Pregnancy in Second Trimester with Previous Caesarean Section: A Case Report Marjan Ghaemi* Yas Hospital, Tehran University of Medical Sciences, Tehran, Iran *Corresponding author: Marjan Ghaemi, Yas Hospital, Tehran University of Medical Sciences, Tehran, Iran, Tel: 0098-912-1967735; E-mail: [email protected] Received date: September 25, 2018; Accepted date: October 16, 2018; Published date: October 22, 2018 Copyright: © 2018 Ghaemi M. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Citation: Ghaemi M (2018) Uterine Rupture after Misoprostol Use for Termination of Pregnancy in Second Trimester with Previous Caesarean Section: A Case Report. Crit Care Obst Gyne Vol.4 No.3:14. cesarean section, hospitalized for severe oligohydramnios with unknown etiology and no history of fluid leakage. In her Abstract previous surgical history, she mentioned laparoscopic cholecystectomy 10 years ago. Considering her medical history Modalities for termination of pregnancy may result in some and normal kidney and bladder in fetal ultrasound, we did not adverse effects especially uterine rupture in women with have proved data for her severe oligohydramnios. Amnio- the previous cesarean section. Here we report uterine infusion was performed to prevent foetal cord compression and rupture in the 23rd week of pregnancy after Misoprostol uses for abortion induction in second pregnancy with the to assess foetal structures. -
Management of Prolonged Decelerations ▲
OBG_1106_Dildy.finalREV 10/24/06 10:05 AM Page 30 OBGMANAGEMENT Gary A. Dildy III, MD OBSTETRIC EMERGENCIES Clinical Professor, Department of Obstetrics and Gynecology, Management of Louisiana State University Health Sciences Center New Orleans prolonged decelerations Director of Site Analysis HCA Perinatal Quality Assurance Some are benign, some are pathologic but reversible, Nashville, Tenn and others are the most feared complications in obstetrics Staff Perinatologist Maternal-Fetal Medicine St. Mark’s Hospital prolonged deceleration may signal ed prolonged decelerations is based on bed- Salt Lake City, Utah danger—or reflect a perfectly nor- side clinical judgment, which inevitably will A mal fetal response to maternal sometimes be imperfect given the unpre- pelvic examination.® BecauseDowden of the Healthwide dictability Media of these decelerations.” range of possibilities, this fetal heart rate pattern justifies close attention. For exam- “Fetal bradycardia” and “prolonged ple,Copyright repetitive Forprolonged personal decelerations use may onlydeceleration” are distinct entities indicate cord compression from oligohy- In general parlance, we often use the terms dramnios. Even more troubling, a pro- “fetal bradycardia” and “prolonged decel- longed deceleration may occur for the first eration” loosely. In practice, we must dif- IN THIS ARTICLE time during the evolution of a profound ferentiate these entities because underlying catastrophe, such as amniotic fluid pathophysiologic mechanisms and clinical 3 FHR patterns: embolism or uterine rupture during vagi- management may differ substantially. What would nal birth after cesarean delivery (VBAC). The problem: Since the introduction In some circumstances, a prolonged decel- of electronic fetal monitoring (EFM) in you do? eration may be the terminus of a progres- the 1960s, numerous descriptions of FHR ❙ Complete heart sion of nonreassuring fetal heart rate patterns have been published, each slight- block (FHR) changes, and becomes the immedi- ly different from the others. -
A Guide to Obstetrical Coding Production of This Document Is Made Possible by Financial Contributions from Health Canada and Provincial and Territorial Governments
ICD-10-CA | CCI A Guide to Obstetrical Coding Production of this document is made possible by financial contributions from Health Canada and provincial and territorial governments. The views expressed herein do not necessarily represent the views of Health Canada or any provincial or territorial government. Unless otherwise indicated, this product uses data provided by Canada’s provinces and territories. All rights reserved. The contents of this publication may be reproduced unaltered, in whole or in part and by any means, solely for non-commercial purposes, provided that the Canadian Institute for Health Information is properly and fully acknowledged as the copyright owner. Any reproduction or use of this publication or its contents for any commercial purpose requires the prior written authorization of the Canadian Institute for Health Information. Reproduction or use that suggests endorsement by, or affiliation with, the Canadian Institute for Health Information is prohibited. For permission or information, please contact CIHI: Canadian Institute for Health Information 495 Richmond Road, Suite 600 Ottawa, Ontario K2A 4H6 Phone: 613-241-7860 Fax: 613-241-8120 www.cihi.ca [email protected] © 2018 Canadian Institute for Health Information Cette publication est aussi disponible en français sous le titre Guide de codification des données en obstétrique. Table of contents About CIHI ................................................................................................................................. 6 Chapter 1: Introduction .............................................................................................................. -
Cord Prolapse
CLINICAL PRACTICE GUIDELINE CORD PROLAPSE CLINICAL PRACTICE GUIDELINE CORD PROLAPSE Institute of Obstetricians and Gynaecologists, Royal College of Physicians of Ireland and the Clinical Strategy and Programmes Division, Health Service Executive Version: 1.0 Publication date: March 2015 Guideline No: 35 Revision date: March 2017 1 CLINICAL PRACTICE GUIDELINE CORD PROLAPSE Table of Contents 1. Revision History ................................................................................ 3 2. Key Recommendations ....................................................................... 3 3. Purpose and Scope ............................................................................ 3 4. Background and Introduction .............................................................. 4 5. Methodology ..................................................................................... 4 6. Clinical Guidelines on Cord Prolapse…… ................................................ 5 7. Hospital Equipment and Facilities ....................................................... 11 8. References ...................................................................................... 11 9. Implementation Strategy .................................................................. 14 10. Qualifying Statement ....................................................................... 14 11. Appendices ..................................................................................... 15 2 CLINICAL PRACTICE GUIDELINE CORD PROLAPSE 1. Revision History Version No. -
Concurrent Intraoperative Uterine Rupture and Placenta Accreta. Do
Concurrent intraoperative uterine rupture and placenta accreta Romanian Journal of Anaesthesia and Intensive Care 2018 Vol 25 No 1, 83-85 CASE REPORT DOI: http://dx.doi.org/10.21454/rjaic.7518.251.acc Concurrent intraoperative uterine rupture and placenta accreta. Do preoperative chronic hypertension, preterm premature rupture of membranes, chorioamnionitis, and placental abruption provide warning to this rare occurrence? M. Anthony Cometa, Scott M. Wasilko, Adam L. Wendling Department of Anesthesiology, Division of Obstetric Anesthesiology, University of Florida College of Medicine, Gainesville, FL, USA Abstract Uterine and placental pathology can be a major cause of morbidity and mortality in the parturient and infant. When presenting alone, placental abruption, uterine rupture, or placenta accreta can result in significant peripartum hemorrhage, requiring aggressive surgical and anesthetic management; however, the presence of multiple concurrent uterine and placental pathologies can result in significant morbidity and mortality. We present the anesthetic management of a parturient who underwent an urgent cesarean delivery for non- reassuring fetal tracing in the setting of chronic hypertension, preterm premature rupture of membranes, and chorioamnionitis who was subsequently found to have placental abruption, uterine rupture, and placenta accreta. Keywords: preterm premature rupture of membranes (PPROM), chorioamnionitis, placental abruption, uterine rupture, placenta accrete, cesarean-hysterectomy Received: 14 March 2018 / Accepted: 30 March 2018 Rom J Anaesth Intensive Care 2018; 25: 83-85 operative bleeding that requires aggressive fluid and blood product management and resuscitation [5]. At present, the literature does not reference the Introduction presentation and anesthetic management of these aforementioned conditions being concurrently present Various placental and uterine pathologies can result in one parturient. -
OBGYN-Study-Guide-1.Pdf
OBSTETRICS PREGNANCY Physiology of Pregnancy: • CO input increases 30-50% (max 20-24 weeks) (mostly due to increase in stroke volume) • SVR anD arterial bp Decreases (likely due to increase in progesterone) o decrease in systolic blood pressure of 5 to 10 mm Hg and in diastolic blood pressure of 10 to 15 mm Hg that nadirs at week 24. • Increase tiDal volume 30-40% and total lung capacity decrease by 5% due to diaphragm • IncreaseD reD blooD cell mass • GI: nausea – due to elevations in estrogen, progesterone, hCG (resolve by 14-16 weeks) • Stomach – prolonged gastric emptying times and decreased GE sphincter tone à reflux • Kidneys increase in size anD ureters dilate during pregnancy à increaseD pyelonephritis • GFR increases by 50% in early pregnancy anD is maintaineD, RAAS increases = increase alDosterone, but no increaseD soDium bc GFR is also increaseD • RBC volume increases by 20-30%, plasma volume increases by 50% à decreased crit (dilutional anemia) • Labor can cause WBC to rise over 20 million • Pregnancy = hypercoagulable state (increase in fibrinogen anD factors VII-X); clotting and bleeding times do not change • Pregnancy = hyperestrogenic state • hCG double 48 hours during early pregnancy and reach peak at 10-12 weeks, decline to reach stead stage after week 15 • placenta produces hCG which maintains corpus luteum in early pregnancy • corpus luteum produces progesterone which maintains enDometrium • increaseD prolactin during pregnancy • elevation in T3 and T4, slight Decrease in TSH early on, but overall euthyroiD state • linea nigra, perineum, anD face skin (melasma) changes • increase carpal tunnel (median nerve compression) • increased caloric need 300cal/day during pregnancy and 500 during breastfeeding • shoulD gain 20-30 lb • increaseD caloric requirements: protein, iron, folate, calcium, other vitamins anD minerals Testing: In a patient with irregular menstrual cycles or unknown date of last menstruation, the last Date of intercourse shoulD be useD as the marker for repeating a urine pregnancy test. -
A Rare Case of Placenta Percreta Causing Uterine Rupture and Massive Haemoperitoneum in 2Nd Trimester: an Obvious Near Miss
International Journal of Reproduction, Contraception, Obstetrics and Gynecology Parmar C et al. Int J Reprod Contracept Obstet Gynecol. 2021 Jun;10(6):2495-2497 www.ijrcog.org pISSN 2320-1770 | eISSN 2320-1789 DOI: https://dx.doi.org/10.18203/2320-1770.ijrcog20212201 Case Report A rare case of placenta percreta causing uterine rupture and massive haemoperitoneum in 2nd trimester: an obvious near miss Chirayu Parmar*, Mittal Parmar, Gayatri Desai Department of Gynecology, Kasturba Hospital, SEWA Rural, Jhagadi, Gujarat, India Received: 30 December 2020 Accepted: 05 May 2021 *Correspondence: Dr. Chirayu Parmar, E-mail: [email protected] Copyright: © the author(s), publisher and licensee Medip Academy. This is an open-access article distributed under the terms of the Creative Commons Attribution Non-Commercial License, which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. ABSTRACT Placenta accreta spectrum is very rarely encountered with ruptured uterus and is commonly seen in third trimester of pregnancy. Hereby, a case of placenta percreta with uterine rupture in second trimester of pregnancy is presented. 40 year old women with previous 2 LSCS presented in emergency department with ninteen weeks pregnancy and massive haemoperitoneum. Emergency laprotomy revealed uterine rupture alnong with placenta percreta for which obstetric hysterectomy was done. Although, a rare occurrence, obstetricians should consider patients placenta accreta spectrum in -
Rational Use of Uterotonic Drugs During Labour and Childbirth
Prevention and initial management of postpartum haemorrhage Rational use of uterotonic drugs during labour and childbirth Prevention and treatment of postpartum haemorrhage December 2008 Editors This manual is made possible through sup- Prevention of postpartum hemorrhage port provided to the POPPHI project by the initiative (POPPHI) Office of Health, Infectious Diseases and Nu- trition, Bureau for Global Health, US Agency for International Development, under the POPPHI Contacts terms of Subcontract No. 4-31-U-8954, under Contract No. GHS-I-00-03-00028. POPPHI is For more information or additional copies of implemented by a collaborative effort be- this brochure, please contact: tween PATH, RTI International, and Engen- Deborah Armbruster, Project Director derHealth. PATH 1800 K St., NW, Suite 800 Washington, DC 20006 Tel: 202.822.0033 Susheela M. Engelbrecht Senior Program Officer, PATH PO Box 70241 Overport Durban 4067 Tel: 27.31.2087579, Fax: 27.31.2087549 [email protected] Copyright © 2009, Program for Appropriate Tech- www.pphprevention.org nology in Health (PATH). All rights reserved. The material in this document may be freely used for educational or noncommercial purposes, provided that the material is accompanied by an acknowl- edgement line. Table of contents Preface………………………………………………………………………………………………………………………………………………….3 Supportive care during labour and childbirth…………………………………………………………………………………….4 Rational use of uterotonic drugs during labour………………………………………………………………………………...5 Indications and precautions for augmentation -
Early Accreta and Uterine Rupture in the Second Trimester
Open Access Case Report DOI: 10.7759/cureus.2904 Early Accreta and Uterine Rupture in the Second Trimester Joshua A. Ronen 1 , Krystal Castaneda 2 , Sara Y. Sadre 3 1. Internal Medicine, Texas Tech University Health Sciences Center of the Permian Basin, Odessa, USA 2. MS3/Ross University School of Medicine, California Hospital Medical Center, Los Angeles, USA 3. MS4/Ross University School of Medicine, California Hospital Medical Center, Los Angeles, USA Corresponding author: Joshua A. Ronen, [email protected] Abstract The differential diagnosis of third trimester bleeding can range from placenta abruptia to placenta previa to uterine rupture and the placenta accreta spectrum (PAS). However, patients with risk factors such as multiple cesarean sections (c-sections), advanced maternal age (AMA), grand multiparity, and single-layer uterine closure are at greater risk of developing these complications earlier than we would traditionally expect. This case recounts a 38-year-old gravida 6 preterm 3 term 1 abortus 1 live 4 (G6P3114) at 23 weeks and five days gestational age (GA) with a past medical history of preterm pregnancy, pre-eclampsia, chronic abruptia, three previous c-sections, and low-lying placenta who presented to the emergency department (ED) with vaginal bleeding. Initial workup revealed placenta accreta and possible percreta. The patient was placed on intramuscular (IM) corticosteroids in anticipation of preterm delivery. As soon as the patient was stable, she was discharged home. She presented to a different hospital the next day with the same complaints. Imaging was consistent with accreta and her presentation with abruption. During the hospital stay, the patient went into threatened preterm labor (PTL). -
The Early Diagnosis and Treatment Strategy of Maternal Near Miss
Central Archives of Emergency Medicine and Critical Care Bringing Excellence in Open Access Short Communication *Corresponding author Youguo Chen, Center of Studies for Psychology and Social Development, Southwest University, 188 Shizi The Early Diagnosis and Street, Suzhou, Jiangsu, PR of China, 215006, Email: Submitted: 17 May 2016 Treatment Strategy of Maternal Accepted: 13 June 2016 Published: 14 June 2016 near Miss Copyright © 2016 Chen et al. Fangrong Shen1, Rong Jiang2, and Youguo Chen3* 1Department of Obstetrics and Gynecology, Soochow University, China OPEN ACCESS 2Laboratory of Stem Cell and Tissue Engineering, Chongqing Medical University, China 3Center of Studies for Psychology and Social Development, Southwest University, China Keywords • Maternal near miss • Early diagnosis Abstract • Management The WHO criteria for maternal near miss (MNM), defined as “a woman who nearly • Socioeconomic factors died but survived a complication that occurred during pregnancy, childbirth or within 42 days of termination of pregnancy”. This review mainly analyses the amniotic fluid embolism (AFE), acute fatty liver of pregnancy (AFLP), HELLP syndrome and severe preeclampsia which may lead most cases of MNM. Finally, summarize the factors and managements associated with maternal near-miss morbidity. INTRODUCTION Amniotic fluid embolism (AFE) Maternal near miss refers to someone who survived a severe Introduction: complication in pregnancy, childbirth, or the postpartum period. catastrophic obstetrics complication occurring during labor Amniotic fluid embolism (AFE) is a With the developing of our society, more and more people are and delivery or immediately postpartum, and is characterized concerned about the maternal near-miss morbidity and mortality. by sudden cardiovascular collapse, respiratory distress, altered measures to decrease the mortality of MNM-most happened They also pay attention to the scientific and effective treatment mental status and disseminated intravascular coagulation (DIC).