Update on the Evaluation of Transient Vision Loss
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Summary Benchmarks for Preferred Practice Pattern® Guidelines
SUMMARY BENCHMARKS FOR PREFERRED PRACTICE PATTERN® GUIDELINES TABLE OF CONTENTS Summary Benchmarks for Preferred Practice Pattern Guidelines Introduction . 1 Glaucoma Primary Open-Angle Glaucoma (Initial Evaluation) . 3 Primary Open-Angle Glaucoma (Follow-up Evaluation) . 5 Primary Open-Angle Glaucoma Suspect (Initial and Follow-up Evaluation) . 6 Primary Angle-Closure Disease (Initial Evaluation and Therapy) . 8 Retina Age-Related Macular Degeneration (Initial and Follow-up Evaluation) . 10 Age-Related Macular Degeneration (Management Recommendations) . 11 Diabetic Retinopathy (Initial and Follow-up Evaluation) . 12 Diabetic Retinopathy (Management Recommendations) . 13 Idiopathic Epiretinal Membrane and Vitreomacular Traction (Initial Evaluation and Therapy) . 14 Idiopathic Macular Hole (Initial Evaluation and Therapy) . 15 Posterior Vitreous Detachment, Retinal Breaks, and Lattice Degeneration (Initial and Follow-up Evaluation) . 17 Retinal and Ophthalmic Artery Occlusions (Initial Evaluation and Therapy) . 18 Retinal Vein Occlusions (Initial Evaluation and Therapy) . 19 Cataract/Anterior Segment Cataract (Initial and Follow-up Evaluation) . 20 Cornea/External Disease Bacterial Keratitis (Initial Evaluation) . 22 Bacterial Keratitis (Management Recommendations) . 23 Blepharitis (Initial and Follow-up Evaluation) . 24 Conjunctivitis (Initial Evaluation) . 25 Conjunctivitis (Management Recommendations) . 26 Corneal Ectasia (Initial Evaluation and Follow-up) . 27 Corneal Edema and Opacification (Initial Evaluation) . 28 Corneal Edema -
Repair of Open Globe Injury
Advances in Ophthalmology & Visual System Case Report Open Access Case report: repair of open globe injury Abstract Volume 3 Issue 1 - 2015 Globe rupture is a common sight-threatening ophthalmic emergency. We present a twelve-year-old girl with ocular trauma by sharp object resulting in corneal laceration, Nader Hussein Fouad Hassan Department of Ophthalmology, Benha University Hospital, hyphema, traumatic cataract and iris prolapse. This case illustrates the importance of early Egypt diagnosis, proper wound repair of ruptured globe as well as management of post-operative complications and value of early post-operative visual rehabilitation. Correspondence: Nader Hussein Fouad Hassan, Ophthalmology department, Benha University Hospital, Keywords: globe rupture, open globe injury, corneal laceration Kaliyobeya, Egypt, Tel +201224727982, Email Received: October 16, 2015 | Published: October 19, 2015 Abbreviations: FB, foreign body; VA, visual acuity; CT scan, patient then was given tetanus immunization, antiemetics, analgesics computerized tomography; RD, retinal detachment; INR, international and intravenous broad spectrum antibiotics and prepared for globe normalized ratio; GA, general anesthesia; D, diopter rupture repair under GA. Laboratory tests included a full blood count, liver function tests, INR, prothrombin time, partial thromboplastin Introduction time, kidney function tests, all were within normal. Ruptured Globe is a common ophthalmic emergency. It is a leading cause of blindness. Early examination with high level of suspicion is recommended in any patient with ocular trauma. Many signs may obscure detection of globe rupture, so the ophthalmologist should examine the patient thoroughly and treat the patient as early as impossible including early visual rehabilitation for young patients to prevent amblyopia. Globe rupture may be associated with traumatic hyphema, traumatic cataract, lens dislocation, vitreous hemorrhage, retinal detachment, uveal prolapse, scleral wound, orbital wall fractures, or optic nerve damage. -
URGENT/EMERGENT When to Refer Financial Disclosure
URGENT/EMERGENT When to Refer Financial Disclosure Speaker, Amy Eston, M.D. has a financial interest/agreement or affiliation with Lansing Ophthalmology, where she is employed as a ophthalmologist. 58 yr old WF with 6 month history of decreased vision left eye. Ache behind the left eye for 2-3 months. Using husband’s contact lens solution made it feel better. Seen by two eye care professionals. Given glasses & told eye exam was normal. No past ocular history Medical history of depression Takes only aspirin and vitamins 20/20 OD 20/30 OS Eye Pressure 15 OD 16 OS – normal Dilated fundus exam & slit lamp were normal Pupillary exam was normal Extraocular movements were full Confrontation visual fields were full No red desaturation Color vision was slightly decreased but the same in both eyes Amsler grid testing was normal OCT disc – OD normal OS slight decreased RNFL OCT of the macula was normal Most common diagnoses: Dry Eye Optic Neuritis Treatment - copious amount of artificial tears. Return to recheck refraction Visual field testing Visual Field testing - Small defect in the right eye Large nasal defect in the left eye Visual Field - Right Hemianopsia. MRI which showed a subacute parietal and occipital lobe infarct. ANISOCORIA Size of the Pupil Constrictor muscles innervated by the Parasympathetic system & Dilating muscles innervated by the Sympathetic system The Sympathetic System Begins in the hypothalamus, travels through the brainstem. Then through the upper chest, up through the neck and to the eye. The Sympathetic System innervates Mueller’s muscle which helps to elevate the upper eyelid. -
Differentiate Red Eye Disorders
Introduction DIFFERENTIATE RED EYE DISORDERS • Needs immediate treatment • Needs treatment within a few days • Does not require treatment Introduction SUBJECTIVE EYE COMPLAINTS • Decreased vision • Pain • Redness Characterize the complaint through history and exam. Introduction TYPES OF RED EYE DISORDERS • Mechanical trauma • Chemical trauma • Inflammation/infection Introduction ETIOLOGIES OF RED EYE 1. Chemical injury 2. Angle-closure glaucoma 3. Ocular foreign body 4. Corneal abrasion 5. Uveitis 6. Conjunctivitis 7. Ocular surface disease 8. Subconjunctival hemorrhage Evaluation RED EYE: POSSIBLE CAUSES • Trauma • Chemicals • Infection • Allergy • Systemic conditions Evaluation RED EYE: CAUSE AND EFFECT Symptom Cause Itching Allergy Burning Lid disorders, dry eye Foreign body sensation Foreign body, corneal abrasion Localized lid tenderness Hordeolum, chalazion Evaluation RED EYE: CAUSE AND EFFECT (Continued) Symptom Cause Deep, intense pain Corneal abrasions, scleritis, iritis, acute glaucoma, sinusitis, etc. Photophobia Corneal abrasions, iritis, acute glaucoma Halo vision Corneal edema (acute glaucoma, uveitis) Evaluation Equipment needed to evaluate red eye Evaluation Refer red eye with vision loss to ophthalmologist for evaluation Evaluation RED EYE DISORDERS: AN ANATOMIC APPROACH • Face • Adnexa – Orbital area – Lids – Ocular movements • Globe – Conjunctiva, sclera – Anterior chamber (using slit lamp if possible) – Intraocular pressure Disorders of the Ocular Adnexa Disorders of the Ocular Adnexa Hordeolum Disorders of the Ocular -
A Case of Uveitis-Hyphema-Glaucoma Syndrome Due to Express Miniature Implantation
Henry Ford Health System Henry Ford Health System Scholarly Commons Case Reports Medical Education Research Forum 2020 5-2020 A Case of Uveitis-Hyphema-Glaucoma Syndrome due to ExPRESS Miniature Implantation Andrew Hou Henry Ford Health System, [email protected] Madeline Hasbrook Henry Ford Health System David A. Crandall Henry Ford Health System, [email protected] Follow this and additional works at: https://scholarlycommons.henryford.com/merf2020caserpt Recommended Citation Hou, Andrew; Hasbrook, Madeline; and Crandall, David A., "A Case of Uveitis-Hyphema-Glaucoma Syndrome due to ExPRESS Miniature Implantation" (2020). Case Reports. 135. https://scholarlycommons.henryford.com/merf2020caserpt/135 This Poster is brought to you for free and open access by the Medical Education Research Forum 2020 at Henry Ford Health System Scholarly Commons. It has been accepted for inclusion in Case Reports by an authorized administrator of Henry Ford Health System Scholarly Commons. A Case of Uveitis-Hyphema-Glaucoma Syndrome due to ExPRESS Miniature Implantation Andrew Hou MD, Madeline Hasbrook MD, David Crandall MD Department of Ophthalmology, Henry Ford Health System, Detroit, Michigan Purpose Results Discussion To report a case of a 69-year-old patient We believe etiology for the UGH who developed uveitis-glaucoma- syndrome the significant iridodonesis seen hyphema (UGH) syndrome after an on gonioscopy. In the interim between uneventful ExPRESS (Alcon, Fort Worth, surgery and presentation, the patient had TX) mini shunt surgery for advanced been prescribed tamsulosin for his benign primary open-angle glaucoma. To our prostatic hyperplasia. knowledge, this is the first case report Tamsulosin has been well established as describing glaucoma device induced UGH the cause of intraoperative floppy iris syndrome and its successful treatment syndrome secondary to iris dilator atrophy. -
Two Cases of Endogenous Endophthalmitis That Progressed To
rine to more distal vessels may have led to vasoconstric- References tion and subsequent vasospasm. In conclusion, epinephrine can lead to OAO following 1. Niemi G. Advantages and disadvantages of adrenaline in accidental intra-arterial injection of subcutaneously ad- regional anaesthesia. Best Pract Res Clin Anaesthesiol ministered local anesthetics. Hence, physicians should 2005;19:229-45. carefully administer local anesthesia while considering the 2. Park KH, Kim YK, Woo SJ, et al. Iatrogenic occlusion of possibility that such a complication may occur. the ophthalmic artery after cosmetic facial filler injections: a national survey by the Korean Retina Society. JAMA Byung Gil Moon Ophthalmol 2014;132:714-23. Retina Center, Department of Ophthalmology, HanGil Eye 3. Lazzeri D, Agostini T, Figus M, et al. Blindness following Hospital, Incheon, Korea cosmetic injections of the face. Plast Reconstr Surg 2012;129:995-1012. June-Gone Kim 4. Savino PJ, Burde RM, Mills RP. Visual loss following intra- Department of Ophthalmology, Asan Medical Center, University of Ulsan College of Medicine, Seoul, Korea nasal anesthetic injection. J Clin Neuroophthalmol E-mail: [email protected] 1990;10:140-4. 5. Webber B, Orlansky H, Lipton C, Stevens M. Complica- tions of an intra-arterial injection from an inferior alveolar Conflict of Interest nerve block. J Am Dent Assoc 2001;132:1702- 4. No potential conflict of interest relevant to this article was reported. Korean J Ophthalmol 2017;31(3):279-281 litus presented with blurred vision of the right eye for 10 https://doi.org/10.3341/kjo.2017.0002 days. Abdominal and chest computed tomography showed an emphysematous prostatic abscess with multiple pulmo- nary lesions. -
Transient Monocular Visual Loss
Transient Monocular Visual Loss VALÉRIE BIOUSSE, MD AND JONATHAN D. TROBE, MD ● PURPOSE: To provide a practical update on the diagno- when most episodes of TMVL do not cause total loss of sis and management of transient monocular visual loss vision.2 (TMVL). The most important step in evaluating a patient with ● DESIGN: Perspective. visual loss is to establish whether the visual loss is ● METHODS: Review of the literature. monocular or binocular.2,5 Monocular visual loss always ● RESULTS: TMVL is an important clinical symptom. It results from lesions anterior to the chiasm (the eye or the has numerous causes but most often results from tran- optic nerve), whereas binocular visual loss results from sient retinal ischemia. It may herald permanent visual lesions of both eyes or optic nerves, or, more likely, of the loss or a devastating stroke, and patients with TMVL chiasm or retrochiasmal visual pathway. should be evaluated urgently. A practical approach to the Deciding whether an episode of abrupt visual loss evaluation of the patient with TMVL must be based on occurred in one eye or both is not always easy. Very few the patient’s age and the suspected underlying etiology. patients realize that binocular hemifield (homonymous) In the older patient, tests should be performed to inves- visual field loss affects the fields of both eyes. They will tigate giant cell arteritis, atherosclerotic large vessel usually localize it to the eye that lost its temporal field. The disease, and cardiac abnormalities. In the younger pa- best clues to the fact that visual loss was actually binocular tient, TMVL is usually benign and the evaluation should are reading impairment (monocular visual loss does not be tailored to the particular clinical setting. -
Acute Keratoconus-Like Corneal Hydrops Secondary to Ocular
perim Ex en l & ta a l ic O p in l h Journal of Clinical & Experimental t C h f a o l m l a o Ke et al., J Clin Exp Ophthalmol 2017, 8:6 n l o r g u y o Ophthalmology J DOI: 10.4172/2155-9570.1000694 ISSN: 2155-9570 Case Report Open Access Acute Keratoconus-Like Corneal Hydrops Secondary to Ocular Massage Following Trabeculectomy Hongmin Ke, Chengguo Zuo and Mingkai Lin* State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen University, 54 Xianlie Nan Road, Guangzhou, China *Corresponding author: Mingkai Lin, State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, 54 Xianlie Nan Road, Guangzhou, China, 510060, E- mail: [email protected] Received date: November 13, 2017; Accepted date: November 21, 2017; Published date: November 23, 2017 Copyright: © 2017 Ke H, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Abstract Purpose: To report a case of acute keratoconus-like corneal hydrops in a patient with long-term ocular massage following trabeculectomy. Methods: Case report and review of medical literature. Results: A rare complication of acute keratoconus-like corneal hydrops occurred in a patient following the use of ocular massage to maintain satisfactory aqueous humor filtration after trabeculectomy. The patient had a history of high myopia but denied previous ocular trauma, allergic disease and a family history of keratoconus. Slit-lamp examination demonstrated keratoconus-like corneal hydrops with formation of epithelial microcystic, and intrastromal cleft. -
Ocular Ischemic Syndrome As the Initial Presenting Feature of Cyto
Korean J Ophthalmol Vol.32, No.5, 2018 Korean J Ophthalmol 2018;32(5):428-429 tests for human immunodeficiency virus, varicella zoster https://doi.org/10.3341/kjo.2018.0017 virus and herpes simplex virus were negative. An anterior chamber paracentesis was performed for polymerase chain reaction. The polymerase chain reaction results were posi- Ocular Ischemic Syndrome as the tive for CMV, and negative for varicella-zoster virus, herpes Initial Presenting Feature of Cyto- simplex virus, and Epstein-Barr virus. megalovirus Retinitis An intravitreal injection of ganciclovir was administered. The patient was also treated with oral valganciclovir. The Dear Editor, retinitis and vitritis improved gradually over the following Cytomegalovirus (CMV) retinitis is an opportunistic in- week; however, the retinal vessels appeared slightly attenu- fection that usually affects immunocompromised patients. ated. Two months later, surgery for neovascular glaucoma Severe intraocular CMV infections in immunocompro- was recommended. However, the patient refused further mised patients are typically asymptomatic, and can involve therapeutic interventions given poor visual prognosis and mild anterior uveitis [1]. One recent paper reported retinal financial concerns. arterial occlusion due to CMV infection in elderly people In immunocompromised patients, CMV retinitis typical- [1]. To the best of our knowledge, we report the first case of ly causes cotton wool spots along the retinal vessels, ac- ocular ischemic syndrome (OIS) that initially presented as companied by many retinal hemorrhages [1]. In contrast, CMV retinitis. CMV infection of the eye in immunocompetent patients is A 71-year-old man with diabetes presented with a one- atypical, and usually limited to the anterior segment [2]. -
Retinal Emergencies
When to Refer to RETINA Joseph M. Coney, MD February 17, 2017 Memphis, TN Financial Disclosure Commercial Interest What was received For what role Aerpio Grant Support Contracted Research Alcon Laboratories Grant Support Contracted Research Alimera Consulting Fee Consultant/Advisor Allergan Consulting Fee Consultant/Advisor Allergan Grant Support Contracted Research Apellis Grant Support Contracted Research Genentech Grant Support Contracted Research Genentech Consulting Fee Consultant/Advisor Hoffman La Roche Grant Support Contracted Research Lowy Medical Research Institute Grant Support Contracted Research Notal Vision Consulting Fee Consultant/Advisor Ohr Grant Support Contracted Research Ophthotech Grant Support Contracted Research Regeneron Equity Ownership Interest Regeneron Consulting Fee Consultant/Advisor Tyrogenex Grant Support Contracted Research Overview – Alphabet Soup . Endophthalmitis/IOI . VH . CRAO . CRVO/BRVO . PVD . NPDR/PDR . RD . ERM . NVI/NVA . MH . CNVM . VMT Endophthalmitis Hypopyon Hypopyon Bacterial Endophthalmitis . Types . Acute post-operative . Chronic post-operative . Bleb-associated . Endogenous . Intravitreal injection-related . Post-traumatic Bacterial Endophthalmitis . Course/Prognosis . Depends on . type of endophthalmitis . duration of time to presentation & Rx . virulence of organism . Bleb-associated, post-traumatic, endogenous endophthalmitis have poorest prognosis Bacterial Endophthalmitis . Presenting Symptoms . Decreased vision . Pain . Red eye . Swollen lid . Hypopyon . Most common organisms – Acute Post-op . Staphylococcus epidermidis (70%) . Other gram positives (24.2%) . Staph aureus (10%) . Streptococcus (9%) . Enterococcus (2.2%) Bacterial Endophthalmitis . Treatment . Prompt intervention critical to restore vision/salvage globe . Vitreous tap & intravitreal antibiotic injections can be done in office with no delays . In very severe or resistant cases, pars plana vitrectomy in operating room Central Retinal Artery Occlusion CRAO . Incidence . About 1:10,000 general patient visits . Most patients over 60 years . -
Eleventh Edition
SUPPLEMENT TO April 15, 2009 A JOBSON PUBLICATION www.revoptom.com Eleventh Edition Joseph W. Sowka, O.D., FAAO, Dipl. Andrew S. Gurwood, O.D., FAAO, Dipl. Alan G. Kabat, O.D., FAAO Supported by an unrestricted grant from Alcon, Inc. 001_ro0409_handbook 4/2/09 9:42 AM Page 4 TABLE OF CONTENTS Eyelids & Adnexa Conjunctiva & Sclera Cornea Uvea & Glaucoma Viitreous & Retiina Neuro-Ophthalmic Disease Oculosystemic Disease EYELIDS & ADNEXA VITREOUS & RETINA Blow-Out Fracture................................................ 6 Asteroid Hyalosis ................................................33 Acquired Ptosis ................................................... 7 Retinal Arterial Macroaneurysm............................34 Acquired Entropion ............................................. 9 Retinal Emboli.....................................................36 Verruca & Papilloma............................................11 Hypertensive Retinopathy.....................................37 Idiopathic Juxtafoveal Retinal Telangiectasia...........39 CONJUNCTIVA & SCLERA Ocular Ischemic Syndrome...................................40 Scleral Melt ........................................................13 Retinal Artery Occlusion ......................................42 Giant Papillary Conjunctivitis................................14 Conjunctival Lymphoma .......................................15 NEURO-OPHTHALMIC DISEASE Blue Sclera .........................................................17 Dorsal Midbrain Syndrome ..................................45 -
Internuclear Ophthalmoplegia and Horner's Syndrome Due To
362 Journal of the Royal Society of Medicine Volume 86 June 1993 Internuclear ophthalmoplegia and Her erythrocyte sedimentation rate (ESR) was 105 in the Horner's syndrome due to presumed first hour, the white blood cell count was 11.1 x109/l, giant cell arteritis platelets 475x 109/1, the blood film showed some target cells and ++ rouleaux. Her alkaline phosphatase was 226 and her 7y-glutamine transferase 102. A computerized tomography scan reported atrophic changes in the cerebral hemispheres, and an old infarct was noted in the left temporal lobe. Within Ayman Askari MRCP1 0 M P Jolobe FRCP2 6 days of starting treatment the headache improved remarkably, and in the following 2 months the patient D I Shepherd FRCP2 'Princess Royal Hospital; maintained her progress with no change in the neurological Telford, Shropshire and 2Tameside General Hospital signs. The ESR was 47 within 8 days of admission and 3 by Ashton-under-Lyne, Lancashire day 38 of starting treatment. Keywords: giant cell arteritis; ophthalmoplegia; Horner's syndrome; Discussion ataxic nystagmus Our aim is to draw attention to the association between neuro-ophthalmic signs and presumed GCA. Leukocytosis, abnormal latex fixation test and fever are in keeping with cell arteritis is an inflammatory, the diagnosis. It is likely that many patients with GCA have Giant (GCA) occlusive ophthalmoplegia in the course of their illness and failure arteriopathy of unknown aetiology. It is common in the to detect it may be due to the transient nature of this elderly, with an incidence of 17.4 per 100 000 ofthe popula- manifestation3.