2021 NIH Virtual Postbac Poster
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COVID-19 Vaccine Weekly Update
Ramona Whittington From: Roxana Cruz Sent: Tuesday, February 23, 2021 10:02 PM To: [email protected]; [email protected]; Myrta Garcia; Lindsay Lanagan; catherine.threatt; [email protected]; Medical Directors; Dental Directors; [email protected]; [email protected] Cc: ClinicalTeam; Cassandra Caravello; Jana Eubank; Daniel Diaz; Shelby Tracy; Nancy Gilliam; Ramona Whittington Subject: COVID-19 Vaccine Weekly Update: Week 11 (02/22/2021) Attachments: COVIDVaccineAllocation-Week11_final.pdf; americanlungassociation_vaccineeducationtoolkit.pdf; ACP_Addressing Mistrust About COVID-19 Vaccines.pdf; COVIDVaccineAllocation-Week10.pdf Dear Fellow CMOs and Vaccine Coordinators, We hope you are well and that your communities have recovered since last week’s winter storm. As I am sure you know by now, COVID vaccine distribution was put on hold last week 2/15‐ 2/19 (Week 10) due to the inclement weather. Per the weekly DSHS Call today, the following updates were provided: • 100K of 1st dose vaccine not shipped/distributed • 300K of 2nd dose vaccine not shipped/distributed • 1 million vaccine doses will be distributed this week, to include last and this week’s allotments. • 600k 1st doses will go to 563 providers in 230 counties • Mass vaccination clinics this week: Dallas, Tarrant, Harris Counties COVID‐19 Vaccine Weekly Update: Week 11 (02/22/2021) During week 11, there were 61 health center sites that received allocations that total 18,550 doses this week. Vaccine Allocations for Week 11 (02/22/2021) attached w/orange highlighted health center sites. Vaccine Allocations for Week 10 (02/15/2022) although previously sent out, we have attached this list again for your reference. -
Coalition Communication: Healthcare
Updated 1/15/2021 Coalition Communication: Healthcare COVID-19 UPDATES We need your help in sharing information about the COVID-19 vaccine. KEY STATS Vaccine.coronavirus.ohio.gov is an online resource for Ohioans to learn which providers received a COVID-19 vaccine allotment and how to contact them. Data as of 1/14/2021 Tentative dates to start vaccinating these Phase 1B populations are: • Jan. 19, 2021—Ohioans 80 years of age and older. PUBLIC HEALTH • Jan. 25, 2021—Ohioans 75 years of age and older; those with severe ADVISORY SYSTEM congenital or developmental disorders. • Feb. 1, 2021—Ohioans 70 years of age and older; employees of K-12 schools that wish to remain or return to in-person or hybrid learning. • Feb. 8, 2021—Ohioans 65 years of age and older. When a new age group begins, vaccinations may not be complete for the previous age group. It will take a number of weeks to distribute all of the vaccines given the limited doses available. If you are older than 65, please connect with an Area Agencies on Aging about questions or if you need transportation assistance. For more information, visit aginig.ohio.gov or call 1-866-243-5678. More information can be found at coronavirus.ohio.gov. 21-DAY TRENDS INDUSTRY INFORMATION Case Average 7,316 ■ The Ad Council and the COVID Collaborative have released a series of Death Average 73 videos, available in a YouTube playlist, feature an introduction from Dr. Anthony Fauci and include experts leading healthcare organizations. Hospitalization 293 Average ■ BlackDoctor.org’s Making It Plain: What Black America Needs to Know ICU Admission 29 About COVID-19 and Vaccines aired on January 7 and is now available on- Average demand on YouTube. -
UMBC Alumnae Racing to Develop Coronavirus Vaccine
Newsletter SPRING 2020 To our UMBC/Meyerhoff families: We hope you and your families are all doing well during this strange and stressful time of Covid- 19. Although the world has changed quickly with so many things shut down and many of us sheltering at home, we hope this newsletter will represent a ray of sunshine during a dark and difficult time. Please enjoy this positive representation of our student and alumni community. MPA Board UMBC Alumnae Racing to Develop Coronavirus Vaccine Kizzmekia Corbett ’08, M16, biological sciences, says it feels like she’s “living in a constant adrenaline rush.” Maybe that’s because she and her team at the Vaccine Research Center at the National Insti- tute of Allergy and Infectious Diseases have been working around the clock for weeks. They’re racing to develop a vaccine for the coronavirus faster than it can race across the globe. “To be living in this moment where I have the opportunity to work on something that has imminent global importance…it’s just a surre- al moment for me,” Corbett says. Despite it feeling surreal, the advances Corbett and her team are making are very real, and they’re setting records. “We are making better progress than I could have ever hoped for,” she says. After three months of studies in test tubes and in animals, the vaccine her team developed is about to enter a phase I clinical trial, a crucial hur- dle on the way to FDA approval. Read the complete article about Kizzmekia and her team’s efforts to develop a Covid-19 vaccine in the latest UMBC magazine at https:// Kizzmekia Corbett, NIH magazine.umbc.edu/umbc-alumnae-racing-to-develop- coronavirus-vaccine/. -
Covid-19 Vaccination Disparities and Hesitancy
COVID-19 VACCINATION DISPARITIES AND HESITANCY + Stanley Thompson, MD, MHA, FACEP + David Hogan, DO, MPH, FACEP + Khadeja Haye, MD, MBA, FACOG teamhealth.com COVID-19 VACCINATION DISPARITIES AND HESITANCY Numerous studies have shown that the COVID-19 pandemic has disproportionately affected people of color in the United States. Black, Hispanic or Latinx, and Native American people are approximately four times more likely to be hospitalized and nearly three times more likely to die of COVID-19 than their majority counterparts. There are numerous reasons for these disparities, many of which are the same as other findings of disparity in other disease states (i.e. the social determinants of medicine). We know that vaccination is the key to preventing COVID-19 in all people. Vaccinated People – What We Know America’s vaccination effort is among the best in the world. However, where race is known, people of color are getting vaccinated at lower rates than white Americans. The Kaiser Family Foundation analysis reveals a significant disparity where race is known (about 54%) for those who have received at least one dose of vaccine as of April 13, 2021. It shows the racial/ethnic breakdown as White Americans 65%, Hispanic or Latinx 11%, Black 9%, Asian 5%, Indigenous American or Alaskan 2%, Native Hawaiian <1%, and Multiple race 9%. A YouGov poll further supports and illustrates this point as well. Among White adults polled, 26% have gotten the vaccine, compared to 17% of Black adults and 11% of Hispanic adults. Access to Vaccines A big part of what is occurring is disparate access to vaccination. -
CSE642 Final Version
Eindhoven University of Technology MASTER Dimensionality reduction of gene expression data Arts, S. Award date: 2018 Link to publication Disclaimer This document contains a student thesis (bachelor's or master's), as authored by a student at Eindhoven University of Technology. Student theses are made available in the TU/e repository upon obtaining the required degree. The grade received is not published on the document as presented in the repository. The required complexity or quality of research of student theses may vary by program, and the required minimum study period may vary in duration. General rights Copyright and moral rights for the publications made accessible in the public portal are retained by the authors and/or other copyright owners and it is a condition of accessing publications that users recognise and abide by the legal requirements associated with these rights. • Users may download and print one copy of any publication from the public portal for the purpose of private study or research. • You may not further distribute the material or use it for any profit-making activity or commercial gain Eindhoven University of Technology MASTER THESIS Dimensionality Reduction of Gene Expression Data Author: S. (Sako) Arts Daily Supervisor: dr. V. (Vlado) Menkovski Graduation Committee: dr. V. (Vlado) Menkovski dr. D.C. (Decebal) Mocanu dr. N. (Nikolay) Yakovets May 16, 2018 v1.0 Abstract The focus of this thesis is dimensionality reduction of gene expression data. I propose and test a framework that deploys linear prediction algorithms resulting in a reduced set of selected genes relevant to a specified case. Abstract In cancer research there is a large need to automate parts of the process of diagnosis, this is mainly to reduce cost, make it faster and more accurate. -
Virginia Commonwealth University Commencement Program Virginia Commonwealth University
Virginia Commonwealth University VCU Scholars Compass VCU Commencement Programs VCU University Archives 1983 Virginia Commonwealth University Commencement Program Virginia Commonwealth University Follow this and additional works at: http://scholarscompass.vcu.edu/vcucommence © Virginia Commonwealth University Downloaded from http://scholarscompass.vcu.edu/vcucommence/18 This Program is brought to you for free and open access by the VCU University Archives at VCU Scholars Compass. It has been accepted for inclusion in VCU Commencement Programs by an authorized administrator of VCU Scholars Compass. For more information, please contact [email protected]. RICHMOND The Coliseum May 14, 1983 10:00 A.M. BOARD OF VISITORS Virginia Commonwealth University Douglas H. Ludeman, Rector Daniel T. Balfour, Vice Rector Harold I. Nemuth, Secretary Mrs. FitzGerald Bemiss Thomas E. Butt Custis L. Coleman Benjamin W. Cotten F. Wilson Craigie, Jr. Sigsby W. Gayle Robert J. Grey Philip B. Morris William G. Reynolds W. Roy Smith Mrs. Charles G. Thalhimer Anne Marie Whittemore Jack H. Wyatt The audience is respectfully asked not to enter onto the floor of the Coliseum until after the ceremony has concluded and all graduates have left the Coliseum floor. PROGRAM The President of the University, Presiding Processional* Virginia Commonwealth University Pomp and Circumstance March No. I-Edward Elgar Symphony Orchestra Jack Jarrett, Conducting Invocation A. Patrick L. Prest Professor and Chairman of Program of Patient Counseling National Anthem Academic Festival Overture, Op. BO-Johannes Brahms VCU Symphony Orchestra Introduction of Guests Edmund F. Ackell President of the University Commencement Address Robert A. Wilson Third Rector of Virginia Commonwealth University Conferring of Honorary Degrees The President of the University Sydney Lewis Frances A. -
Photoperiodic Responses on Expression of Clock Genes, Synaptic Plasticity Markers, and Protein Translation Initiators the Impact of Blue-Enriched Light
Photoperiodic Responses on Expression of Clock Genes, Synaptic Plasticity Markers, and Protein Translation Initiators The Impact Of Blue-Enriched Light Master report Jorrit Waslander, s2401878 Behavioral Cognitive Neuroscience research master, N-track University of Groningen, the Netherlands Internship at: Bergen Stress and Sleep Group, University of Bergen, Norway Date: 13-7-2018 Internal supervisor, University of Groningen: P. (Peter) Meerlo External supervisor, University of Bergen: J. (Janne) Grønli Daily supervisor, University of Bergen: A. (Andrea) R. Marti Photoperiodic Responses in the PFC Table of Contents Summary ................................................................................................................................................. 3 Introduction ............................................................................................................................................. 5 Research Objective .............................................................................................................................. 8 Hypotheses .......................................................................................................................................... 8 Methods ................................................................................................................................................ 10 Experimental procedure .................................................................................................................... 10 Ethics ............................................................................................................................................ -
Novel Gene Discovery in Primary Ciliary Dyskinesia
Novel Gene Discovery in Primary Ciliary Dyskinesia Mahmoud Raafat Fassad Genetics and Genomic Medicine Programme Great Ormond Street Institute of Child Health University College London A thesis submitted in conformity with the requirements for the degree of Doctor of Philosophy University College London 1 Declaration I, Mahmoud Raafat Fassad, confirm that the work presented in this thesis is my own. Where information has been derived from other sources, I confirm that this has been indicated in the thesis. 2 Abstract Primary Ciliary Dyskinesia (PCD) is one of the ‘ciliopathies’, genetic disorders affecting either cilia structure or function. PCD is a rare recessive disease caused by defective motile cilia. Affected individuals manifest with neonatal respiratory distress, chronic wet cough, upper respiratory tract problems, progressive lung disease resulting in bronchiectasis, laterality problems including heart defects and adult infertility. Early diagnosis and management are essential for better respiratory disease prognosis. PCD is a highly genetically heterogeneous disorder with causal mutations identified in 36 genes that account for the disease in about 70% of PCD cases, suggesting that additional genes remain to be discovered. Targeted next generation sequencing was used for genetic screening of a cohort of patients with confirmed or suggestive PCD diagnosis. The use of multi-gene panel sequencing yielded a high diagnostic output (> 70%) with mutations identified in known PCD genes. Over half of these mutations were novel alleles, expanding the mutation spectrum in PCD genes. The inclusion of patients from various ethnic backgrounds revealed a striking impact of ethnicity on the composition of disease alleles uncovering a significant genetic stratification of PCD in different populations. -
Anatomical and Behavioral Investigation of C1ql3 in the Mouse Suprachiasmatic Nucleus David C
University of Connecticut OpenCommons@UConn UCHC Articles - Research University of Connecticut Health Center Research 6-2017 Anatomical and Behavioral Investigation of C1ql3 in the Mouse Suprachiasmatic Nucleus David C. Martinelli University of Connecticut School of Medicine and Dentistry Follow this and additional works at: https://opencommons.uconn.edu/uchcres_articles Part of the Life Sciences Commons, and the Medicine and Health Sciences Commons Recommended Citation Martinelli, David C., "Anatomical and Behavioral Investigation of C1ql3 in the Mouse Suprachiasmatic Nucleus" (2017). UCHC Articles - Research. 311. https://opencommons.uconn.edu/uchcres_articles/311 HHS Public Access Author manuscript Author ManuscriptAuthor Manuscript Author J Biol Rhythms Manuscript Author . Author Manuscript Author manuscript; available in PMC 2017 November 01. Published in final edited form as: J Biol Rhythms. 2017 June ; 32(3): 222–236. doi:10.1177/0748730417704766. Anatomical and Behavioral Investigation of C1ql3 in the Mouse Suprachiasmatic Nucleus Kylie S. Chew*,†, Diego C. Fernandez*,1, Samer Hattar*,‡,1, Thomas C. Südhof§,‖, and David C. Martinelli§,¶,2 *Department of Biology, The Johns Hopkins University, Baltimore, Maryland †Department of Biology, Stanford University School of Medicine, Stanford, California ‡The Solomon Snyder- Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, Maryland §Department of Molecular and Cellular Physiology, Stanford University School of Medicine, Stanford, California ‖Howard Hughes -
The Photoreceptors and Neural Circuits Driving the Pupillary Light Reflex
THE PHOTORECEPTORS AND NEURAL CIRCUITS DRIVING THE PUPILLARY LIGHT REFLEX by Alan C. Rupp A dissertation submitted to Johns Hopkins University in conformity with the requirements for the degree of Doctor of Philosophy Baltimore, Maryland January 28, 2016 This work is protected by a Creative Commons license: Attribution-NonCommercial CC BY-NC Abstract The visual system utilizes environmental light information to guide animal behavior. Regulation of the light entering the eye by the pupillary light reflex (PLR) is critical for normal vision, though its precise mechanisms are unclear. The PLR can be driven by two mechanisms: (1) an intrinsic photosensitivity of the iris muscle itself, and (2) a neural circuit originating with light detection in the retina and a multisynaptic neural circuit that activates the iris muscle. Even within the retina, multiple photoreceptive mechanisms— rods, cone, or melanopsin phototransduction—can contribute to the PLR, with uncertain relative importance. In this thesis, I provide evidence that the retina almost exclusively drives the mouse PLR using bilaterally asymmetric brain circuitry, with minimal role for the iris intrinsic photosensitivity. Intrinsically photosensitive retinal ganglion cells (ipRGCs) relay all rod, cone, and melanopsin light detection from the retina to brain for the PLR. I show that ipRGCs predominantly relay synaptic input originating from rod photoreceptors, with minimal input from cones or their endogenous melanopsin phototransduction. Finally, I provide evidence that rod signals reach ipRGCs using a non- conventional retinal circuit, potentially through direct synaptic connections between rod bipolar cells and ipRGCs. The results presented in this thesis identify the initial steps of the PLR and provide insight into the precise mechanisms of visual function. -
Case Report Persistent Eosinophilia: a Diagnostic Dilemma
Case Report DOI: 10.21276/APALM.2736 Persistent Eosinophilia: A Diagnostic Dilemma Hafsa Shabeer*, Chethana Mannem, Gayathri Bilagali Ramdas and Thejasvi Krishnamurthy Department of Pathology, Kempegowda Institute of Medical Sciences, Bangalore – 560004, Karnataka, India. ABSTRACT Chronic eosinophilic leukaemia-not otherwise specified (CEL-NOS) is a myeloproliferative neoplasm associated with an autonomous, clonal proliferation of eosinophilic precursors resulting in persistent eosinophilia. A 50-year-old female presented with easy fatiguability, cough and generalised swelling of the body. Investigations revealed anaemia with leucocytosis (56.150 x 103/ul) and 88% eosinophils (absolute eosinophil count was 49412/ul). Peripheral smear showed abnormal eosinophils exhibiting abnormal granulation and nuclear lobation. Reactive causes were ruled out and a bone marrow aspiration/biopsy revealed mildly hypercellular marrow with increased number of eosinophils and their precursors, 7% blasts along with dysplastic megakaryocytes - hypolobated and occasional segmented forms. Molecular studies including chromosomal and gene analysis were done. A combination of the clinical picture, laboratory and molecular studies led us to a diagnosis of CEL-NOS. The causes for eosinophilia are myriad and range from reactive causes like parasitic infestations to neoplasms in which eosinophils are a part of the neoplastic population/ are cytokine-mediated reactive component in the background of another neoplasm. The incidence of CEL-NOS is obscure due to significant overlap with Idiopathic Hypereosinophilic Syndrome (IHES). While CEL-NOS is a myeloproliferative neoplasm and its diagnosis can be made provided evidence of a clonality is present, IHES is a diagnosis of exclusion. It is important to differentiate the two entities as they carry different prognosis and modes of treatment. -
COSYNE 2005 Thursay, March 17Th — Sunday March 20Th Salt Lake City, Utah
COSYNE 2005 Thursay, March 17th — Sunday March 20th Salt Lake City, Utah Thursday, March 17th 6PM–8PM Reception 8PM Session 1 Keynote 8:00–8:10 Welcome and announcements 8:15–9:15 Keynote: Chuck Stevens (Salk Institute) Friday, March 18th 8AM–12PM Session 2 Talks 12–2PM Lunch break 2–6PM Session 3 Talks 6–8PM Dinner break 8PM–1AM Session 4 Posters Saturday, March 19th 8:30AM–12PM Session 5 Talks 12:15–2PM Lunch break 2–6PM Session 6 Talks 6–8PM Dinner break 8PM–1AM Session 7 Posters Sunday, March 20th 8:30AM–12:15PM Session 8 Talks 12:15–2PM Lunch break 2–3:50PM Session 9 Talks Main meeting ends / Travel to Snowbird for workshops 1 Detailed Schedule Thursday, March 17th 6PM–8PM Reception 8PM Session 1 Keynote Chair: Tony Zador (CSHL) 8:00–8:10 Welcome and announcements 8:15–9:15 Chuck Stevens (Salk Institute) A design principle for the scalable architecture of vertebrate brains Friday, March 18th 8AM–12PM Session 2 Talks Chair: Michael Shadlen (U Washington) 8:00–8:40 Eero Simoncelli (NYU) Testing and refining perceptual models with optimized stochastic stimuli 8:50–9:10 Nelson Spruston (Northwestern) Gating of dendritic spikes propagating toward the soma of CA1 pyramidal neurons 9:15–9:35 Andrea Benucci (Smith-Kettlewell) Imaging the dynamics of population responses in visual cortex 9:40–10:00 Daniel Butts (Harvard) The important aspects of visual encoding: relating receptive fields to mutual information rates in visual neurons 10:05–10:25 Coffee break 10:30–10:50 William Softky (RNI) Thalamic bursts as anti-spikes from a non-rectifying