Nursing Care of the Child with a Respiratory Disorder
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Apnea of Prematurity Eric C
CLINICAL REPORT Guidance for the Clinician in Rendering Pediatric Care Apnea of Prematurity Eric C. Eichenwald, MD, FAAP, COMMITTEE ON FETUS AND NEWBORN Apnea of prematurity is one of the most common diagnoses in the NICU. abstract Despite the frequency of apnea of prematurity, it is unknown whether recurrent apnea, bradycardia, and hypoxemia in preterm infants are harmful. Research into the development of respiratory control in immature animals and preterm infants has facilitated our understanding of the pathogenesis and treatment of apnea of prematurity. However, the lack of consistent defi nitions, monitoring practices, and consensus about clinical signifi cance leads to signifi cant variation in practice. The purpose of this clinical report is to review the evidence basis for the defi nition, epidemiology, and treatment of apnea of prematurity as well as discharge recommendations for preterm infants diagnosed with recurrent apneic events. BACKGROUND This document is copyrighted and is property of the American Academy of Pediatrics and its Board of Directors. All authors have Apnea of prematurity is one of the most common diagnoses in the NICU. fi led confl ict of interest statements with the American Academy of Pediatrics. Any confl icts have been resolved through a process Despite the frequency of apnea of prematurity, it is unknown whether approved by the Board of Directors. The American Academy of recurrent apnea, bradycardia, and hypoxemia in preterm infants are Pediatrics has neither solicited nor accepted any commercial involvement in the development of the content of this publication. harmful. Limited data suggest that the total number of days with apnea and resolution of episodes at more than 36 weeks’ postmenstrual age Clinical reports from the American Academy of Pediatrics benefi t from expertise and resources of liaisons and internal (AAP) and external (PMA) are associated with worse neurodevelopmental outcome in reviewers. -
Bronchopulmonary Hygiene Protocol
BRONCHOPULMONARY HYGIENE PROTOCOL MD order for Bronchopulmonary Hygiene Protocol Evaluate Indications: 9 Difficulty with secretion clearence with sputum production > 25 ml/day 9 Evidence of retained secretions 9 Mucus plug induced atelectasis 9 Foreign body in airway 9 Diagnosis of cystic fibrosis, bronchiectasis, or cavitating lung disease Yes Does contraindication or potential hazard exist? No Address any immediate need and contact MD/RN Select method based on: 9 Patient preference/comfort/pain avoidance 9 Observation of effectiveness with trial 9 History with documented effectiveness Method may include: 9 Manual chest percussion and positioning 9 External chest wall vibration 9 Intrapulmonary percussion Adminster therapy no less than QID and PRN, supplemented by suctioning for all patients with artificial airways Re-evaluate pt every 24 hours, and 24 hours after discontinued Assess Outcomes: Goals achieved? 9 Optimal hydration with sputum production < 25 ml/day 9 Breath sounds from diminished to adventitious with ronchi cleared by cough 9 Patient subjective impression of less retention and improved clearance 9 Resolution/Improvement in chest X-ray 9 Improvement in vital signs and measures of gas exchange 9 If on ventilator, reduced resistance and improved compliance Care Plan Considerations: Discontinue therapy if improvement is observed and sustained over a 24-hour period. Patients with chronic pulmonary disease who maintain secretion clearance in their home environment should remain on treatment no less than their home frequency. Hyperinflation Protocol should be considered for patients who are at high risk for pulmonary complications as listed in the indications for Hyperinflation Protocol. 5/5/03 (Jan Phillips-Clar, Rick Ford, Judy Tietsort, Jay Peters, David Vines) AARC References for Bronchopulmonary Algorithm 1. -
Teaching Cases of the Month
Teaching Cases of the Month A 60-Year Old Man Presenting With Yellow Nail Syndrome Ariel Modrykamien MD and Omar Minai MD Introduction Blood total protein was 5.2 g/dL, and lactate dehydroge- nase was 350 U/L. His FEV1 was 64% of predicted, forced Yellow nail syndrome is a rare disorder of unclear eti- vital capacity was 60% of predicted, the ratio of FEV1 to ology. Since its original description by Samman and White,1 forced vital capacity was 0.81, and his diffusion capacity many other reports and associations have been described. for carbon monoxide was 97% of predicted. Current evidence suggests that its pathologic mechanism Echocardiogram revealed normal cavities and valve is based on lymphatic dysfunction, which is thought to be function. Left-side thoracentesis obtained a cloudy pleural acquired rather than congenital. However, our understand- fluid with total protein 3.9 g/dL, lactate dehydrogenase ing of its pathogenesis is still speculative, as it relies on 144 U/L, glucose 100 mg/dL, and pH 7.6, with 79% lym- anecdotal observations. We report a case of a man who phocytes. Pleural fluid cultures for bacterial, fungal, and presented with dry cough, shortness of breath, bilateral acid-fast bacilli were negative. Cytology was negative for lower-extremity edema, and pleural effusion. Yellow nail malignant cells. Flow cytometry was negative for chronic syndrome was diagnosed. lymphoproliferative disorders. Tuberculosis skin test was also negative. Case Summary Radiology Findings A 60-year-old man presented with 2 months of persis- tent dry cough. He noticed dyspnea with moderate effort, Chest radiograph revealed a left-side pleural effusion such as walking 2–3 blocks, and bilateral lower-extremity (Fig. -
Spontaneous Pneumothorax in COVID-19 Patients Treated with High-Flow Nasal Cannula Outside the ICU: a Case Series
International Journal of Environmental Research and Public Health Case Report Spontaneous Pneumothorax in COVID-19 Patients Treated with High-Flow Nasal Cannula outside the ICU: A Case Series Magdalena Nalewajska 1, Wiktoria Feret 1 , Łukasz Wojczy ´nski 1, Wojciech Witkiewicz 2 , Magda Wi´sniewska 1 and Katarzyna Kotfis 3,* 1 Department of Nephrology, Transplantology and Internal Medicine, Pomeranian Medical University, 70–111 Szczecin, Poland; [email protected] (M.N.); [email protected] (W.F.); [email protected] (Ł.W.); [email protected] (M.W.) 2 Department of Cardiology, Pomeranian Medical University, 70–111 Szczecin, Poland; [email protected] 3 Department of Anesthesiology, Intensive Therapy and Acute Intoxications, Pomeranian Medical University in Szczecin, 70–111 Szczecin, Poland * Correspondence: katarzyna.kotfi[email protected] Abstract: The coronavirus disease 2019 (COVID-19) caused by the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) has become a global pandemic and a burden to global health at the turn of 2019 and 2020. No targeted treatment for COVID-19 infection has been identified so far, thus supportive treatment, invasive and non-invasive oxygen support, and corticosteroids remain a common therapy. High-flow nasal cannula (HFNC), a non-invasive oxygen support method, has become a prominent treatment option for respiratory failure during the SARS-CoV-2 pandemic. Citation: Nalewajska, M.; Feret, W.; HFNC reduces the anatomic dead space and increases positive end-expiratory pressure (PEEP), Wojczy´nski,Ł.; Witkiewicz, W.; allowing higher concentrations and higher flow of oxygen. Some studies suggest positive effects of Wi´sniewska,M.; Kotfis, K. HFNC on mortality and avoidance of intubation. -
Gas Exchange
Gas exchange Gas exchange occurs as a result of respiration, when carbon dioxide is excreted and oxygen taken up, and photosynthesis, when oxygen is excreted and carbon dioxide is taken up. The rate of gas exchange is affected by: • the area available for diffusion • the distance over which diffusion occurs • the concentration gradient across the gas exchange surface • the speed with which molecules diffuse through membranes. Efficient gas exchange systems must: • have a large surface area to volume ratio • be thin • have mechanisms for maintaining steep concentration gradients across themselves • be permeable to gases. Single-celled organisms are aquatic and their cell surface membrane has a sufficiently large surface area to volume ratio to act as an efficient gas exchange surface. In larger organisms, permeable, thin, flat structures have all the properties of efficient gas exchange surfaces but need water to prevent their dehydration and give them mechanical support. Since the solubility of oxygen in water is low, organisms that obtain their oxygen from water can maintain only a low metabolic rate. In small and thin organisms, the distance from gas exchange surface to the inside of the organism is short enough for diffusion of gases to be efficient. Diffusion gradients are maintained because gases are continually used up or produced. In larger organisms, simple diffusion is not an efficient way of transporting gases between cells in the body and the gas exchange surface. In many animals a blood circulatory system carries gases to and from the gas exchange surface. The gas-carrying capacity of the blood is increased by respiratory pigments, such as haemoglobin. -
Respiratory Examination Cardiac Examination Is an Essential Part of the Respiratory Assessment and Vice Versa
Respiratory examination Cardiac examination is an essential part of the respiratory assessment and vice versa. # Subject steps Pictures Notes Preparation: Pre-exam Checklist: A Very important. WIPE Be the one. 1 Wash your hands. Wash your hands in Introduce yourself to the patient, confirm front of the examiner or bring a sanitizer with 2 patient’s ID, explain the examination & you. take consent. Positioning of the patient and his/her (Position the patient in a 3 1 2 Privacy. 90 degree sitting position) and uncover Exposure. full exposure of the trunk. his/her upper body. 4 (if you could not, tell the examiner from the beginning). 3 4 Examination: General appearance: B (ABC2DEVs) Appearance: young, middle aged, or old, Begin by observing the and looks generally ill or well. patient's general health from the end of the bed. Observe the patient's general appearance (age, Around the bed I can't state of health, nutritional status and any other see any medications, obvious signs e.g. jaundice, cyanosis, O2 mask, or chest dyspnea). 1 tube(look at the lateral sides of chest wall), metered dose inhalers, and the presence of a sputum mug. 2 Body built: normal, thin, or obese The patient looks comfortable and he doesn't appear short of breath and he doesn't obviously use accessory muscles or any heard Connections: such as nasal cannula wheezes. To determine this, check for: (mention the medications), nasogastric Dyspnea: Assess the rate, depth, and regularity of the patient's 3 tube, oxygen mask, canals or nebulizer, breathing by counting the respiratory rate, range (16–25 breaths Holter monitor, I.V. -
Mechanisms of Pulmonary Gas Exchange Abnormalities During Experimental Group B Streptococcal Infusion
003 I -3998/85/1909-0922$02.00/0 PEDIATRIC RESEARCH Vol. 19, No. 9, I985 Copyright 0 1985 International Pediatric Research Foundation, Inc. Printed in (I.S. A. Mechanisms of Pulmonary Gas Exchange Abnormalities during Experimental Group B Streptococcal Infusion GREGORY K. SORENSEN, GREGORY J. REDDING, AND WILLIAM E. TRUOG ABSTRACT. Group B streptococcal sepsis in newborns obtained from GBS (5, 6). Arterial Poz fell by 9 torr in association produces pulmonary arterial hypertension and hypoxemia. with the increase in pulmonary arterial pressure (4). In contrast, The purpose of this study was to investigate the mecha- the neonatal piglet infused with GBS demonstrated both pul- nisms by which hypoxemia occurs. Ten anesthetized, ven- monary arterial hypertension and profound arterial hypoxemia tilated piglets were infused with 2 x lo9 colony forming (7). These results suggest that the neonatal pulmonary vascula- unitstkg of Group B streptococci over a 30-min period. ture may respond to bacteremia differently from that of adults. Pulmonary arterial pressure rose from 14 ? 2.8 to 38 ? The relationship between Ppa and the matching of alveolar 6.7 torr after 20 min of the bacterial infusion (p< 0.01). ventilation and pulmonary perfusion, a major determinant of During the same period, cardiac output fell from 295 to arterial oxygenation during room air breathing (8), has not been 184 ml/kg/min (p< 0.02). Arterial Po2 declined from 97 studied in newborns. The predictable rise in Ppa with an infusion 2 7 to 56 2 11 torr (p< 0.02) and mixed venous Po2 fell of group B streptococcus offers an opportunity to delineate the from 39.6 2 5 to 28 2 8 torr (p< 0.05). -
The Child with Altered Respiratory Status
Path: K:/LWW-BOWDEN-09-0101/Application/LWW-BOWDEN-09-0101-016.3d Date: 3rd July 2009 Time: 16:31 User ID: muralir 1BlackLining Disabled CHAPTER 16 The Child With Altered Respiratory Status Do you remember the Diaz fam- Case History leave Lela, the baby sister, ily from Chapter 9, in which with Claudia’s mother, Selma, Jose has trouble taking his asthma medication, and head to the hospital. and in Chapter 4, in which Jose’s little sister, At the hospital the emergency department Lela, expresses her personality even as a new- nurse observes Jose sitting cross-legged and born? Jose is 4 years old and was diagnosed leaning forward on his hands. His mouth is with asthma this past fall, about 6 months ago. open and he is breathing hard with an easily Since that time Claudia, his mother, has audible inspiratory wheeze. He is using his noticed several factors that trigger his asthma subclavicular accessory muscles with each including getting sick, pollen, and cold air. breath. His respiratory rate is 32 breaths per One evening following a warm early-spring minute, his pulse is 112 beats per minutes, day, Jose is outside playing as the sun sets and and he is afebrile. Claudia explains that he the air cools. When he comes inside he was fine; he was outside running and playing, begins to cough. Claudia sets up his nebulizer and then came in and began coughing and and gives him a treatment of albuterol, which wheezing, and she gave him an albuterol lessens his coughing. Within an hour, Jose is treatment. -
Apnea and Control of Breathing Christa Matrone, M.D., M.Ed
APNEA AND CONTROL OF BREATHING CHRISTA MATRONE, M.D., M.ED. DIOMEL DE LA CRUZ, M.D. OBJECTIVES ¢ Define Apnea ¢ Review Causes and Appropriate Evaluation of Apnea in Neonates ¢ Review the Pathophysiology of Breathing Control and Apnea of Prematurity ¢ Review Management Options for Apnea of Prematurity ¢ The Clinical Evidence for Caffeine ¢ The Role of Gastroesophageal Reflux DEFINITION OF APNEA ¢ Cessation of breathing for greater than 15 (or 20) seconds ¢ Or if accompanied by desaturations or bradycardia ¢ Differentiate from periodic breathing ¢ Regular cycles of respirations with intermittent pauses of >3 S ¢ Not associated with other physiologic derangements ¢ Benign and self-limiting TYPES OF APNEA CENTRAL ¢ Total cessation of inspiratory effort ¢ Absence of central respiratory drive OBSTRUCTIVE ¢ Breathing against an obstructed airway ¢ Chest wall motion without nasal airflow MIXED ¢ Obstructed respiratory effort after a central pause ¢ Accounts for majority of apnea in premature infants APNEA IS A SYMPTOM, NOT A DIAGNOSIS Martin RJ et al. Pathogenesis of apnea in preterm infants. J Pediatr. 1986; 109:733. APNEA IN THE NEONATE: DIFFERENTIAL Central Nervous System Respiratory • Intraventricular Hemorrhage • Airway Obstruction • Seizure • Inadequate Ventilation / Fatigue • Cerebral Infarct • Hypoxia Infection Gastrointestinal • Sepsis • Necrotizing Enterocolitis • Meningitis • Gastroesophageal Reflux Hematologic Drug Exposure • Anemia • Perinatal (Ex: Magnesium, Opioids) • Polycythemia • Postnatal (Ex: Sedatives, PGE) Cardiovascular Other • Patent Ductus Arteriosus • Temperature instability • Metabolic derangements APNEA IN THE NEONATE: EVALUATION ¢ Detailed History and Physical Examination ¢ Gestational age, post-natal age, and birth history ¢ Other new signs or symptoms ¢ Careful attention to neurologic, cardiorespiratory, and abdominal exam APNEA IN THE NEONATE: EVALUATION ¢ Laboratory Studies ¢ CBC/diff and CRP ¢ Cultures, consideration of LP ¢ Electrolytes including magnesium ¢ Blood gas and lactate ¢ Radiologic Studies ¢ Head ultrasound vs. -
Msc Nursing SYLLABUS.Pdf
Syllabus for Courses affiliated to the Kerala University of Health Sciences Thrissur 680596 2016 MASTER OF SCIENCE IN NURSING Medical Surgical Nursing Course Code 271 (2016‐17 admission onwards) 2016 REGULATIONS 2. COURSE CONTENT 2.1 Title of course: MASTER OF SCIENCE IN NURSING – MSc Medical Surgical Nursing Sub specialties :‐ a. Cardio Vascular & Thoracic Nursing b. Critical care Nursing, Oncology Nursing c. Neurosciences Nursing d. Nephro‐Urology Nursing e. Orthopedic Nursing f. GastroEnterology Nursing. 2.2 AIM & OBJECTIVES OF THE COURSE AIM Aim of the postgraduate programme in nursing is to prepare graduates to assume responsibilities as Clinical Nurse Specialists, Nurse Practitioners, Consultants, Educators, Researchers and Administrators in a wide variety of professional settings. OBJECTIVES On completion of the 2 year M.Sc Nursing programme, the graduates will be able to:‐ 1. Apply the concepts, theories and principles of nursing science. 2. Demonstrate competencies in nursing practice. 3. Practice as Clinical Nurse Specialist. 4. Establish collaborative relationship with the members of other disciplines. 5. Assume leadership in various care settings. 6. Participate in health planning, implementation and evaluation at different levels of health care system. 7. Function as effective nurse educators and nurse managers. 8. Conduct independent nursing research and utilize the research findings in nursing practice and education. 1 9. Critically evaluate various educational programmes in nursing. 10. Demonstrate interest and positive attitude in continuing education for personal and professional growth. 11. Demonstrate advanced skills and competence in the nursing management of patients with various medical and surgical conditions. 12. Incorporate evidence based nursing practice and identify the areas of research in the field of medical and surgical nursing. -
1 British Thoracic Society Guidelines Recommendations for The
Thorax Online First, published on September 28, 2007 as 10.1136/thx.2007.077370 Thorax: first published as 10.1136/thx.2007.077370 on 28 September 2007. Downloaded from British Thoracic Society Guidelines Recommendations for the assessment and management of cough in children MD Shields, A Bush, ML Everard, S McKenzie and R Primhak on behalf of the British Thoracic Society Cough Guideline Group Michael D Shields Dept. of Child Health, Queen’s University of Belfast, Clinical Institute, Grosvenor Road, Belfast, BT12 6BJ Email: [email protected] Andrew Bush Royal Brompton Hospital, Sydney Street, London, SW3 6NP Email: [email protected] Mark L Everard Dept of Paediatrics, Sheffield Children’s Hospital, Western Bank, Sheffield, S. Yorkshire, S10 2TH. Email: [email protected] Sheila McKenzie Queen Elizabeth Children’s Services, http://thorax.bmj.com/ The Royal London Hospital, Whitechapel, London, E1 1BB Email: [email protected] Robert Primhak Dept. of Paediatrics, Sheffield Children’s Hospital, on September 29, 2021 by guest. Protected copyright. Western Bank, Sheffield, S. Yorkshire, S10 2TH. Email: [email protected] “the Corresponding Author (Michael D Shields) has the right to grant on behalf of all authors and does grant on behalf of all authors, an exclusive licence (or non exclusive for government employees) on a worldwide basis to the BMJ Publishing Group Ltd and its Licensees to permit this article (if accepted) to be published in [THORAX] editions and any other BMJPG Ltd products to exploit all subsidiary rights, as set out in our licence 1 Copyright Article author (or their employer) 2007. -
Effects of Caffeine in Lung Mechanics of Extremely Low Birth Weight Infants
Journal of Clinical Anesthesia and Pain Medicine Research Article Effects of Caffeine in Lung Mechanics of Extremely Low Birth Weight Infants This article was published in the following Scient Open Access Journal: Journal of Clinical Anethesia and Pain Medicine Received March 09, 2017; Accepted April 12, 2017; Published April 20, 2017 George Hatzakis*, Dominic Fitzgerald, Abstract G. Michael Davis, Christopher Newth, Philippe Jouvet and Larry Lands Objective: To investigate the effect of caffeine citrate (methyxanthine) on the pattern of From the Departments of Anesthesia, Surgery breathing and lung mechanics in extremely low birth weight (ELBW) infants with apnea of and Pediatrics, Children’s Hospital Los Angeles, prematurity (AOP), during mechanical ventilation and following extubation while breathing Keck School of Medicine, University of Southern spontaneously. California, Los Angeles CA; Pediatric Respiratory Medicine, McGill University, Montreal, Canada; Methods: In this pilot prospective observational study 39 ELBW infants were monitored: Sainte-Justine Hospital, University of Montreal, Twenty AOP - diagnosed with respiratory distress syndrome (RDS) - and 19 controls. Montreal, Canada; and Pediatric Respiratory and Infants with AOP were assessed on mechanical ventilation before caffeine administration Sleep Medicine, University of Sydney, Sydney, and immediately after extubation which occurred at 11-14 days post- caffeine citrate Australia. commencement. Control infants were compared to the post- caffeine group. Breathing pattern parameters, lung mechanics and work of breathing were assessed. Results: Caffeine citrate seemed to markedly increase Tidal Volume (VT) in the post caffeine group when compared to the control group (7.3 ± 2.0 ml/kg and 5.7 ± 1.5 ml/kg respectively) and slightly decreased breathing rate (64 ± 17 and 70 ± 19 breaths/min), respectively.