Greater Trochanteric Pain Syndrome Case Type / Diagnosis
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Understanding Entheseal Changes: Definition and Life Course Changes Sébastien Villotte, Christopher J
Understanding Entheseal Changes: Definition and Life Course Changes Sébastien Villotte, Christopher J. Knüsel To cite this version: Sébastien Villotte, Christopher J. Knüsel. Understanding Entheseal Changes: Definition and Life Course Changes. International Journal of Osteoarchaeology, Wiley, 2013, Entheseal Changes and Occupation: Technical and Theoretical Advances and Their Applications, 23 (2), pp.135-146. 10.1002/oa.2289. hal-03147090 HAL Id: hal-03147090 https://hal.archives-ouvertes.fr/hal-03147090 Submitted on 19 Feb 2021 HAL is a multi-disciplinary open access L’archive ouverte pluridisciplinaire HAL, est archive for the deposit and dissemination of sci- destinée au dépôt et à la diffusion de documents entific research documents, whether they are pub- scientifiques de niveau recherche, publiés ou non, lished or not. The documents may come from émanant des établissements d’enseignement et de teaching and research institutions in France or recherche français ou étrangers, des laboratoires abroad, or from public or private research centers. publics ou privés. International Journal of Osteoarchaeology Understanding Entheseal Changes: Definition and Life Course Changes Journal: International Journal of Osteoarchaeology Manuscript ID: OA-12-0089.R1 Wiley - ManuscriptFor type: Commentary Peer Review Date Submitted by the Author: n/a Complete List of Authors: Villotte, Sébastien; University of Bradford, AGES Knusel, Chris; University of Exeter, Department of Archaeology entheses, enthesopathy, Musculoskeletal Stress Markers (MSM), Keywords: senescence, activity, hormones, animal models, clinical studies http://mc.manuscriptcentral.com/oa Page 1 of 27 International Journal of Osteoarchaeology 1 2 3 Title: 4 5 Understanding Entheseal Changes: Definition and Life Course Changes 6 7 8 Short title: 9 10 Understanding Entheseal Changes 11 12 13 Keywords: entheses; enthesopathy; Musculoskeletal Stress Markers (MSM); senescence; 14 15 activity; hormones; animal models; clinical studies 16 17 18 Authors: For Peer Review 19 20 Villotte S. -
Iliopsoas Tendonitis/Bursitis Exercises
ILIOPSOAS TENDONITIS / BURSITIS What is the Iliopsoas and Bursa? The iliopsoas is a muscle that runs from your lower back through the pelvis to attach to a small bump (the lesser trochanter) on the top portion of the thighbone near your groin. This muscle has the important job of helping to bend the hip—it helps you to lift your leg when going up and down stairs or to start getting out of a car. A fluid-filled sac (bursa) helps to protect and allow the tendon to glide during these movements. The iliopsoas tendon can become inflamed or overworked during repetitive activities. The tendon can also become irritated after hip replacement surgery. Signs and Symptoms Iliopsoas issues may feel like “a pulled groin muscle”. The main symptom is usually a catch during certain movements such as when trying to put on socks or rising from a seated position. You may find yourself leading with your other leg when going up the stairs to avoid lifting the painful leg. The pain may extend from the groin to the inside of the thigh area. Snapping or clicking within the front of the hip can also be experienced. Do not worry this is not your hip trying to pop out of socket but it is usually the iliopsoas tendon rubbing over the hip joint or pelvis. Treatment Conservative treatment in the form of stretching and strengthening usually helps with the majority of patients with iliopsoas bursitis. This issue is the result of soft tissue inflammation, therefore rest, ice, anti- inflammatory medications, physical therapy exercises, and/or injections are effective treatment options. -
Juvenile Spondyloarthropathies: Inflammation in Disguise
PP.qxd:06/15-2 Ped Perspectives 7/25/08 10:49 AM Page 2 APEDIATRIC Volume 17, Number 2 2008 Juvenile Spondyloarthropathieserspective Inflammation in DisguiseP by Evren Akin, M.D. The spondyloarthropathies are a group of inflammatory conditions that involve the spine (sacroiliitis and spondylitis), joints (asymmetric peripheral Case Study arthropathy) and tendons (enthesopathy). The clinical subsets of spondyloarthropathies constitute a wide spectrum, including: • Ankylosing spondylitis What does spondyloarthropathy • Psoriatic arthritis look like in a child? • Reactive arthritis • Inflammatory bowel disease associated with arthritis A 12-year-old boy is actively involved in sports. • Undifferentiated sacroiliitis When his right toe starts to hurt, overuse injury is Depending on the subtype, extra-articular manifestations might involve the eyes, thought to be the cause. The right toe eventually skin, lungs, gastrointestinal tract and heart. The most commonly accepted swells up, and he is referred to a rheumatologist to classification criteria for spondyloarthropathies are from the European evaluate for possible gout. Over the next few Spondyloarthropathy Study Group (ESSG). See Table 1. weeks, his right knee begins hurting as well. At the rheumatologist’s office, arthritis of the right second The juvenile spondyloarthropathies — which are the focus of this article — toe and the right knee is noted. Family history is might be defined as any spondyloarthropathy subtype that is diagnosed before remarkable for back stiffness in the father, which is age 17. It should be noted, however, that adult and juvenile spondyloar- reported as “due to sports participation.” thropathies exist on a continuum. In other words, many children diagnosed with a type of juvenile spondyloarthropathy will eventually fulfill criteria for Antinuclear antibody (ANA) and rheumatoid factor adult spondyloarthropathy. -
9 Impingement and Rotator Cuff Disease
Impingement and Rotator Cuff Disease 121 9 Impingement and Rotator Cuff Disease A. Stäbler CONTENTS Shoulder pain and chronic reduced function are fre- quently heard complaints in an orthopaedic outpa- 9.1 Defi nition of Impingement Syndrome 122 tient department. The symptoms are often related to 9.2 Stages of Impingement 123 the unique anatomic relationships present around the 9.3 Imaging of Impingement Syndrome: Uri Imaging Modalities 123 glenohumeral joint ( 1997). Impingement of the 9.3.1 Radiography 123 rotator cuff and adjacent bursa between the humeral 9.3.2 Ultrasound 126 head and the coracoacromial arch are among the most 9.3.3 Arthrography 126 common causes of shoulder pain. Neer noted that 9.3.4 Magnetic Resonance Imaging 127 elevation of the arm, particularly in internal rotation, 9.3.4.1 Sequences 127 9.3.4.2 Gadolinium 128 causes the critical area of the cuff to pass under the 9.3.4.3 MR Arthrography 128 coracoacromial arch. In cadaver dissections he found 9.4 Imaging Findings in Impingement Syndrome alterations attributable to mechanical impingement and Rotator Cuff Tears 130 including a ridge of proliferative spurs and excres- 9.4.1 Bursal Effusion 130 cences on the undersurface of the anterior margin 9.4.2 Imaging Following Impingement Test Injection 131 Neer Neer 9.4.3 Tendinosis 131 of the acromion ( 1972). Thus it was who 9.4.4 Partial Thickness Tears 133 introduced the concept of an impingement syndrome 9.4.5 Full-Thickness Tears 134 continuum ranging from chronic bursitis and partial 9.4.5.1 Subacromial Distance 136 tears to complete tears of the supraspinatus tendon, 9.4.5.2 Peribursal Fat Plane 137 which may extend to involve other parts of the cuff 9.4.5.3 Intramuscular Cysts 137 Neer Matsen 9.4.6 Massive Tears 137 ( 1972; 1990). -
Pediatrics-EOR-Outline.Pdf
DERMATOLOGY – 15% Acne Vulgaris Inflammatory skin condition assoc. with papules & pustules involving pilosebaceous units Pathophysiology: • 4 main factors – follicular hyperkeratinization with plugging of sebaceous ducts, increased sebum production, Propionibacterium acnes overgrowth within follicles, & inflammatory response • Hormonal activation of pilosebaceous glands which may cause cyclic flares that coincide with menstruation Clinical Manifestations: • In areas with increased sebaceous glands (face, back, chest, upper arms) • Stage I: Comedones: small, inflammatory bumps from clogged pores - Open comedones (blackheads): incomplete blockage - Closed comedones (whiteheads): complete blockage • Stage II: Inflammatory: papules or pustules surrounded by inflammation • Stage III: Nodular or cystic acne: heals with scarring Differential Diagnosis: • Differentiate from rosacea which has no comedones** • Perioral dermatitis based on perioral and periorbital location • CS-induced acne lacks comedones and pustules are in same stage of development Diagnosis: • Mild: comedones, small amounts of papules &/or pustules • Moderate: comedones, larger amounts of papules &/or pustules • Severe: nodular (>5mm) or cystic Management: • Mild: topical – azelaic acid, salicylic acid, benzoyl peroxide, retinoids, Tretinoin topical (Retin A) or topical antibiotics [Clindamycin or Erythromycin with Benzoyl peroxide] • Moderate: above + oral antibiotics [Minocycline 50mg PO qd or Doxycycline 100 mg PO qd], spironolactone • Severe (refractory nodular acne): oral -
Pes Anserine Bursitis
BRIGHAM AND WOMEN’S HOSPITAL Department of Rehabilitation Services Physical Therapy Standard of Care: Pes Anserine Bursitis ICD 9 Codes: 726.61 Case Type / Diagnosis: The pes anserine bursa lies behind the medial hamstring, which is composed of the tendons of the sartorius, gracilis and semitendinosus (SGT) muscles. Because these 3 tendons splay out on the anterior aspect of the tibia and give the appearance of the foot of a goose, pes anserine bursitis is also known as goosefoot bursitis.1 These muscles provide for medial stabilization of the knee by acting as a restraint to excessive valgus opening. They also provide a counter-rotary torque function to the knee joint. The pes anserine has an eccentric role during the screw-home mechanism that dampens the effect of excessively forceful lateral rotation that may accompany terminal knee extension.2 Pes anserine bursitis presents as pain, tenderness and swelling over the anteromedial aspect of the knee, 4 to 5 cm below the joint line.3 Pain increases with knee flexion, exercise and/or stair climbing. Inflammation of this bursa is common in overweight, middle-aged women, and may be associated with osteoarthritis of the knee. It also occurs in athletes engaged in activities such as running, basketball, and racquet sports.3 Other risk factors include: 1 • Incorrect training techniques, or changes in terrain and/or distanced run • Lack of flexibility in hamstring muscles • Lack of knee extension • Patellar malalignment Indications for Treatment: • Knee Pain • Knee edema • Decreased active and /or passive ROM of lower extremities • Biomechanical dysfunction lower extremities • Muscle imbalances • Impaired muscle performance (focal weakness or general conditioning) • Impaired function Contraindications: • Patients with active signs/symptoms of infection (fever, chills, prolonged and obvious redness or swelling at hip joint). -
Gluteal Tendinopathy
Gluteal Tendinopathy What is a Gluteal Tendinopathy? In lying Up until recently hip bursitis was diagnosed as the main Either on your bad hip or with bad cause of lateral hip pain but recent studies suggest that an hip hanging across body like so irritation of the gluteus muscle tendon is the likeliest cause. The tendon attaches onto a bony prominence (greater trochanter) and it is here that the tendon is subject to All these positions lead to increase friction of the tendon, compressive forces leading to irritation. can cause pain and slow the healing process. This can result in pain over the lateral hip which can refer down the outside For sleeping you might like to try these positions: of the thigh and into the knee. How common is it? Gluteal tendinopathy is relatively common affecting 10-25% of the population. It is 3 times more prevalent in women than men and is most common in women between the ages of 40 and 60. One of the reasons for this is women It is also important to modify your activity. Avoid or reduce tend to have a greater angle at their hip joint increasing things that flare up your pain, this could be climbing stairs compressive forces on the tendon. or hills or those longer walks/runs. Signs and Symptoms Exercise Therapy • Pain on the outside of your hip, can refer down outside of the thigh to the knee This is best administered by a Physiotherapist to suit the • Worse when going up and/or down stairs individual but below is a rough guide to exercises which • Worse lying on affected side (and sometimes on the can help a gluteal tendinopathy. -
Billing and Coding: Injections - Tendon, Ligament, Ganglion Cyst, Tunnel Syndromes and Morton's Neuroma (A57079)
Local Coverage Article: Billing and Coding: Injections - Tendon, Ligament, Ganglion Cyst, Tunnel Syndromes and Morton's Neuroma (A57079) Links in PDF documents are not guaranteed to work. To follow a web link, please use the MCD Website. Contractor Information CONTRACTOR NAME CONTRACT TYPE CONTRACT JURISDICTION STATE(S) NUMBER Noridian Healthcare Solutions, A and B MAC 01111 - MAC A J - E California - Entire State LLC Noridian Healthcare Solutions, A and B MAC 01112 - MAC B J - E California - Northern LLC Noridian Healthcare Solutions, A and B MAC 01182 - MAC B J - E California - Southern LLC Noridian Healthcare Solutions, A and B MAC 01211 - MAC A J - E American Samoa LLC Guam Hawaii Northern Mariana Islands Noridian Healthcare Solutions, A and B MAC 01212 - MAC B J - E American Samoa LLC Guam Hawaii Northern Mariana Islands Noridian Healthcare Solutions, A and B MAC 01311 - MAC A J - E Nevada LLC Noridian Healthcare Solutions, A and B MAC 01312 - MAC B J - E Nevada LLC Noridian Healthcare Solutions, A and B MAC 01911 - MAC A J - E American Samoa LLC California - Entire State Guam Hawaii Nevada Northern Mariana Created on 09/28/2019. Page 1 of 33 CONTRACTOR NAME CONTRACT TYPE CONTRACT JURISDICTION STATE(S) NUMBER Islands Article Information General Information Original Effective Date 10/01/2019 Article ID Revision Effective Date A57079 N/A Article Title Revision Ending Date Billing and Coding: Injections - Tendon, Ligament, N/A Ganglion Cyst, Tunnel Syndromes and Morton's Neuroma Retirement Date N/A Article Type Billing and Coding AMA CPT / ADA CDT / AHA NUBC Copyright Statement CPT codes, descriptions and other data only are copyright 2018 American Medical Association. -
A New Caenagnathid Dinosaur from the Upper Cretaceous Wangshi
www.nature.com/scientificreports OPEN A new caenagnathid dinosaur from the Upper Cretaceous Wangshi Group of Shandong, China, with Received: 12 October 2017 Accepted: 7 March 2018 comments on size variation among Published: xx xx xxxx oviraptorosaurs Yilun Yu1, Kebai Wang2, Shuqing Chen2, Corwin Sullivan3,4, Shuo Wang 5,6, Peiye Wang2 & Xing Xu7 The bone-beds of the Upper Cretaceous Wangshi Group in Zhucheng, Shandong, China are rich in fossil remains of the gigantic hadrosaurid Shantungosaurus. Here we report a new oviraptorosaur, Anomalipes zhaoi gen. et sp. nov., based on a recently collected specimen comprising a partial left hindlimb from the Kugou Locality in Zhucheng. This specimen’s systematic position was assessed by three numerical cladistic analyses based on recently published theropod phylogenetic datasets, with the inclusion of several new characters. Anomalipes zhaoi difers from other known caenagnathids in having a unique combination of features: femoral head anteroposteriorly narrow and with signifcant posterior orientation; accessory trochanter low and confuent with lesser trochanter; lateral ridge present on femoral lateral surface; weak fourth trochanter present; metatarsal III with triangular proximal articular surface, prominent anterior fange near proximal end, highly asymmetrical hemicondyles, and longitudinal groove on distal articular surface; and ungual of pedal digit II with lateral collateral groove deeper and more dorsally located than medial groove. The holotype of Anomalipes zhaoi is smaller than is typical for Caenagnathidae but larger than is typical for the other major oviraptorosaurian subclade, Oviraptoridae. Size comparisons among oviraptorisaurians show that the Caenagnathidae vary much more widely in size than the Oviraptoridae. Oviraptorosauria is a clade of maniraptoran theropod dinosaurs characterized by a short, high skull, long neck and short tail. -
The Anatomy of the Deep Infrapatellar Bursa of the Knee Robert F
0363-5465/98/2626-0129$02.00/0 THE AMERICAN JOURNAL OF SPORTS MEDICINE, Vol. 26, No. 1 © 1998 American Orthopaedic Society for Sports Medicine The Anatomy of the Deep Infrapatellar Bursa of the Knee Robert F. LaPrade,* MD Department of Orthopaedic Surgery, University of Minnesota, Minneapolis, Minnesota ABSTRACT knee joint, and to define a consistent surgical approach to the deep infrapatellar bursa. Disorders of the deep infrapatellar bursa are important to include in the differential diagnosis of anterior knee pain. Knowledge regarding its anatomic location can MATERIALS AND METHODS aid the clinician in establishing a proper diagnosis. Fifty cadaveric knees were dissected, and the deep infrapa- Thorough dissections of the anterior aspect of the knee of tellar bursa had a consistent anatomic location in all 50 nonpaired cadaveric knees were performed. There were specimens. The deep infrapatellar bursa was located 27 male and 23 female cadaveric knees with 25 right and directly posterior to the distal 38% of the patellar ten- 25 left knees. The average age of the specimens was 71.8 don, just proximal to its insertion on the tibial tubercle. years (range, 42 to 93). After the skin and subcutaneous There was no communication to the knee joint. Its tissues of the anterior aspect of the knee were carefully average width at the most proximal margin of the tibial dissected away, an approach to the deep infrapatellar tubercle was slightly wider than the average distal bursa of the knee was made through medial and lateral width of the patellar tendon. It was found to be partially arthrotomy incisions along the patella, followed by compartmentalized, with a fat pad apron extending transection of the quadriceps tendon from the patella. -
Percutaneous Reduction Techniques
AAOS 2016 Specialty Day Orthopaedic Trauma Associacion (OTA) Percutaneous Reduction Techniques Christian Krettek Trauma Department, Hannover Medical School, Germany [email protected] www.mhh-unfallchirurgie.de Short segment tibial fractures can be stabilized using different implants like plates, screws, external fixators and nails. This syllabus deals mainly with reduction techniques in the context of the use of intramedullary nails. 1.1 PREOPERATIVE PLANNING AND MANAGEMENT 1.1.1 Primary shortening and secondary overdistraction eases definitive nailing In the acute setting primary shortening of a shaft fracture is a helpful strategy to de- crease soft tissue tension and intra-compartmental pressure. However, reduction gets more difficult and more time consuming, the longer the fracture is kept in a shortened position. The concept of primary shortening (acute phase) and secondary (after 3 or 4 days) overdistraction eases definitive nailing. Using a electro-mechanical load cell, our group has compared the reduction forces in patients undergoing femoral nailing after Damage Control in shortend vs overdistracted fracture configuration. In the overdistrac- tion group, the reduction forces were lower (200 N +/-43.1 N vs. 336 N +/- 51.9 N, p = 0.007) and the reduction time was shorter (5.8 min +/-4.0 min vs. 28.3 min +/-21.8 min, p = 0.056). It was concluded, that DCO with the fracture shortened leads to higher restrai- ning forces & prolonged reduction time. Overdistraction should be performed as soon as possible under careful soft-tissue monitoring [1a, 2a]. Primary shortening and secondary overdistraction eases definitive nailing Example of a femoral shaft fracture stabilized in shortening first with an external fixator. -
Neurologic Outcomes in Friedreich Ataxia: Study of a Single-Site Cohort E415
Volume 6, Number 3, June 2020 Neurology.org/NG A peer-reviewed clinical and translational neurology open access journal ARTICLE Neurologic outcomes in Friedreich ataxia: Study of a single-site cohort e415 ARTICLE Prevalence of RFC1-mediated spinocerebellar ataxia in a North American ataxia cohort e440 ARTICLE Mutations in the m-AAA proteases AFG3L2 and SPG7 are causing isolated dominant optic atrophy e428 ARTICLE Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy revisited: Genotype-phenotype correlations of all published cases e434 Academy Officers Neurology® is a registered trademark of the American Academy of Neurology (registration valid in the United States). James C. Stevens, MD, FAAN, President Neurology® Genetics (eISSN 2376-7839) is an open access journal published Orly Avitzur, MD, MBA, FAAN, President Elect online for the American Academy of Neurology, 201 Chicago Avenue, Ann H. Tilton, MD, FAAN, Vice President Minneapolis, MN 55415, by Wolters Kluwer Health, Inc. at 14700 Citicorp Drive, Bldg. 3, Hagerstown, MD 21742. Business offices are located at Two Carlayne E. Jackson, MD, FAAN, Secretary Commerce Square, 2001 Market Street, Philadelphia, PA 19103. Production offices are located at 351 West Camden Street, Baltimore, MD 21201-2436. Janis M. Miyasaki, MD, MEd, FRCPC, FAAN, Treasurer © 2020 American Academy of Neurology. Ralph L. Sacco, MD, MS, FAAN, Past President Neurology® Genetics is an official journal of the American Academy of Neurology. Journal website: Neurology.org/ng, AAN website: AAN.com CEO, American Academy of Neurology Copyright and Permission Information: Please go to the journal website (www.neurology.org/ng) and click the Permissions tab for the relevant Mary E.