Sports Related Skin Infections Position Statement and Guidelines
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Specific Disease Exclusion for Schools
SPECIFIC DISEASE EXCLUSION FOR SCHOOLS See individual fact sheets for more information on the diseases listed below. Bed Bugs None. Acute Bronchitis (Chest Until fever is gone (without the use of a fever reducing medication) and Cold)/Bronchiolitis the child is well enough to participate in routine activities. Campylobacteriosis None, unless the child is not feeling well and/or has diarrhea and needs to use the bathroom frequently. Exclusion may be necessary during outbreaks. Anyone with Campylobacter should not go in lakes, pools, splash pads, water parks, or hot tubs until after diarrhea has stopped. Staff with Campylobacter may be restricted from working in food service. Call your local health department to see if these restrictions apply. Chickenpox Until all blisters have dried into scabs; usually by day 6 after the rash began. Chickenpox can occur even if someone has had the varicella vaccine. These are referred to as breakthrough infections. Breakthrough infections develop more than 42 days after vaccination, are usually less severe, have an atypical presentation (low or no fever, less than 50 skin lesions), and are shorter in duration (4 to 6 days). Bumps, rather than blisters, may develop; therefore, scabs may not present. Breakthrough cases should be considered infectious. These cases should be excluded until all sores (bumps/blisters/scabs) have faded or no new sores have occurred within a 24-hour period, whichever is later. Sores do not need to be completely resolved before the case is allowed to return. Conjunctivitis (Pinkeye) No exclusion, unless the child has a fever or is not healthy enough to participate in routine activities. -
Coexistence of Antibodies to Tick-Borne
Mem Inst Oswaldo Cruz, Rio de Janeiro, Vol. 98(3): 311-318, April 2003 311 Coexistence of Antibodies to Tick-borne Agents of Babesiosis and Lyme Borreliosis in Patients from Cotia County, State of São Paulo, Brazil Natalino Hajime Yoshinari/+, Milena Garcia Abrão, Virginia Lúcia Nazário Bonoldi, Cleber Oliveira Soares*, Claudio Roberto Madruga*, Alessandra Scofield**, Carlos Luis Massard**, Adivaldo Henrique da Fonseca** Laboratório de Investigação em Reumatologia (LIM-17), Hospital das Clínicas, Faculdade de Medicina, Universidade de São Paulo, Av. Dr. Arnaldo 455, 3º andar, 01246-903 São Paulo, SP, Brasil *Embrapa Gado de Corte, Campo Grande, MS, Brasil **Universidade Federal Rural do Rio de Janeiro, Seropédica, RJ, Brasil This paper reports a case of coinfection caused by pathogens of Lyme disease and babesiosis in brothers. This was the first case of borreliosis in Brazil, acquired in Cotia County, State of São Paulo, Brazil. Both children had tick bite history, presented erythema migrans, fever, arthralgia, mialgia, and developed positive serology (ELISA and Western-blotting) directed to Borrelia burgdorferi G 39/40 and Babesia bovis antigens, mainly of IgM class antibodies, suggestive of acute disease. Also, high frequencies of antibodies to B. bovis was observed in a group of 59 Brazilian patients with Lyme borreliosis (25.4%), when compared with that obtained in a normal control group (10.2%) (chi-square = 5.6; p < 0.05). Interestingly, both children presented the highest titers for IgM antibodies directed to both infective diseases, among all patients with Lyme borreliosis. Key words: lyme borreliosis - lyme disease - spirochetosis - borreliosis - babesiosis - coinfection - tick-borne disease - Brazil Babesiosis is a tick-borne disease distributed world- The first case of babesiosis in a healthy person, with wide, caused by hemoprotozoans of the genus Babesia, intact spleen, was reported in 1969 in a woman from Nan- which infects wild and domestic animals, promoting eco- tucket Island (Massachusetts, USA)(Wester et al. -
Bacterial Infections Diseases Picture Cause Basic Lesion
page: 117 Chapter 6: alphabetical Bacterial infections diseases picture cause basic lesion search contents print last screen viewed back next Bacterial infections diseases Impetigo page: 118 6.1 Impetigo alphabetical Bullous impetigo Bullae with cloudy contents, often surrounded by an erythematous halo. These bullae rupture easily picture and are rapidly replaced by extensive crusty patches. Bullous impetigo is classically caused by Staphylococcus aureus. cause basic lesion Basic Lesions: Bullae; Crusts Causes: Infection search contents print last screen viewed back next Bacterial infections diseases Impetigo page: 119 alphabetical Non-bullous impetigo Erythematous patches covered by a yellowish crust. Lesions are most frequently around the mouth. picture Lesions around the nose are very characteristic and require prolonged treatment. ß-Haemolytic streptococcus is cause most frequently found in this type of impetigo. basic lesion Basic Lesions: Erythematous Macule; Crusts Causes: Infection search contents print last screen viewed back next Bacterial infections diseases Ecthyma page: 120 6.2 Ecthyma alphabetical Slow and gradually deepening ulceration surmounted by a thick crust. The usual site of ecthyma are the legs. After healing there is a permanent scar. The pathogen is picture often a streptococcus. Ecthyma is very common in tropical countries. cause basic lesion Basic Lesions: Crusts; Ulcers Causes: Infection search contents print last screen viewed back next Bacterial infections diseases Folliculitis page: 121 6.3 Folliculitis -
Herpes Gladiatorum (HG)? - HG Is a Skin Infection Caused by the Herpes Simplex Type 1 Virus
Herpes Gladitorum Fact Sheet 1. What is herpes gladiatorum (HG)? - HG is a skin infection caused by the Herpes simplex type 1 virus. 2. How do you get HG? - This skin infection is spread by direct skin-to-skin contact. Wrestling with HG lesions will spread this infection to other wrestlers. 3. What is HG illness like? a. Generally, lesions appear within eight days after exposure to an infected person, but in some cases the lesions take longer to appear. Good personal hygiene and thorough cleaning and disinfecting of all equipment are essential to helping prevent the spread of this and other skin infections. b. All wrestlers with skin sores or lesions should be referred to a physician for evaluation and possible treatment. These individuals should not participate in practice or competition until their lesions have healed. c. Before skin lesions appear, some people have a sore throat, swollen lymph nodes, fever or tingling on the skin. HG lesions appear as a cluster of blisters and may be on the face, extremities or trunk. Seek medical care immediately for lesions in or around the eye. d. Every wrestler should be evaluated by a knowledgeable, unbiased adult for infectious rashes and excluded from practice and competition if suspicious rashes are present until evaluation and clearance by a competent professional. 4. What are the serious complications from HG? - The virus can “hide out” in the nerves and reactivate later, causing another infection. Generally, recurrent infections are less severe and don’t last as long. However, a recurring infection is just as contagious as the original infection, so the same steps need to be taken to prevent infecting others. -
Herpes: a Patient's Guide
Herpes: A Patient’s Guide Herpes: A Patient’s Guide Introduction Herpes is a very common infection that is passed through HSV-1 and HSV-2: what’s in a name? ....................................................................3 skin-to-skin contact. Canadian studies have estimated that up to 89% of Canadians have been exposed to herpes simplex Herpes symptoms .........................................................................................................4 type 1 (HSV-1), which usually shows up as cold sores on the Herpes transmission: how do you get herpes? ................................................6 mouth. In a British Columbia study, about 15% of people tested positive for herpes simplex type 2 (HSV-2), which Herpes testing: when is it useful? ..........................................................................8 is the type of herpes most commonly thought of as genital herpes. Recently, HSV-1 has been showing up more and Herpes treatment: managing your symptoms ...................................................10 more on the genitals. Some people can have both types of What does herpes mean to you: receiving a new diagnosis ......................12 herpes. Most people have such minor symptoms that they don’t even know they have herpes. What does herpes mean to you: accepting your diagnosis ........................14 While herpes is very common, it also carries a lot of stigma. What does herpes mean to you: dating with herpes ....................................16 This stigma can lead to anxiety, fear and misinformation -
Skin Disease and Disorders
Sports Dermatology Robert Kiningham, MD, FACSM Department of Family Medicine University of Michigan Health System Disclosures/Conflicts of Interest ◼ None Goals and Objectives ◼ Review skin infections common in athletes ◼ Establish a logical treatment approach to skin infections ◼ Discuss ways to decrease the risk of athlete’s acquiring and spreading skin infections ◼ Discuss disqualification and return-to-play criteria for athletes with skin infections ◼ Recognize and treat non-infectious skin conditions in athletes Skin Infections in Athletes ◼ Bacterial ◼ Herpetic ◼ Fungal Skin Infections in Athletes ◼ Very common – most common cause of practice-loss time in wrestlers ◼ Athletes are susceptible because: – Prone to skin breakdown (abrasions, cuts) – Warm, moist environment – Close contacts Cases 1 -3 ◼ 21 year old male football player with 4 day h/o left axillary pain and tenderness. Two days ago he noticed a tender “bump” that is getting bigger and more tender. ◼ 16 year old football player with 3 day h/o mildly tender lesions on chin. Started as a single lesion, but now has “spread”. Over the past day the lesions have developed a dark yellowish crust. ◼ 19 year old wrestler with a 3 day h/o lesions on right side of face. Noticed “tingling” 4 days ago, small fluid filled lesions then appeared that have now started to crust over. Skin Infections Bacterial Skin Infections ◼ Cellulitis ◼ Erysipelas ◼ Impetigo ◼ Furunculosis ◼ Folliculitis ◼ Paronychea Cellulitis Cellulitis ◼ Diffuse infection of connective tissue with severe inflammation of dermal and subcutaneous layers of the skin – Triad of erythema, edema, and warmth in the absence of underlying foci ◼ S. aureus or S. pyogenes Erysipelas Erysipelas ◼ Superficial infection of the dermis ◼ Distinguished from cellulitis by the intracutaneous edema that produces palpable margins of the skin. -
Herpes Gladiatorum Fact Sheet
WHAT IS HERPES GLADIATORUM OR “MAT HERPES”? HERPES GLADIATORUM IS A SKIN INFECTION CAUSED BY THE HERPES SIMPLEX VIRUS TYPE I. IT IS SOMETIMES CALLED “MAT HERPES” BECAUSE ATHLETES WHO PARTICIPATE IN HIGH- CONTACT SPORTS, LIKE WRESTLING, MAY HAVE GREATER CHANCES OF CONTRACTING THE VIRUS. HOW IS IT SPREAD? Herpes gladiatorum is spread by direct skin–to–skin contact with infected lesions or by contact with contaminated items (e.g., mats, towels, cell phones). WHO IS AT RISK FOR HERPES GLADIATORUM? Anyone can become infected, but athletes and their families are at greatest risk for infection. WHAT ARE THE SYMPTOMS? Herpes gladiatorum typically begins as a skin rash with blisters or fluid–filled lesions. The rash is commonly seen on the head, face, neck, shoulders, arms, or trunk of the body. If the eyes or surrounding areas become infected, seek medical treatment immediately. A sore throat, swollen lymph nodes, fever, and skin tingling may precede or accompany the rash. HOW LONG DOES IT TAKE FOR SYMPTOMS TO APPEAR? In most cases, symptoms will develop approximately 8 days after exposure to the virus, “Mat Herpes”but may present anywhere from 2 days to 2 weeks later. CAN A PERSON HAVE REPEATED EPISODES OF SYMPTOMS? Yes. Once a person has contracted the virus, it will remain in their body for life. They may have periodic bouts of symptoms. Recurrent episodes of rash and lesions are typically milder than the first episode. HOW LONG IS A PERSON ABLE TO SPREAD THE VIRUS? Herpes gladiatorum can be spread until the blisters or lesions are no longer present or until a doctor determines the person is no longer infectious. -
Pediatric Cutaneous Bacterial Infections Dr
PEDIATRIC CUTANEOUS BACTERIAL INFECTIONS DR. PEARL C. KWONG MD PHD BOARD CERTIFIED PEDIATRIC DERMATOLOGIST JACKSONVILLE, FLORIDA DISCLOSURE • No relevant relationships PRETEST QUESTIONS • In Staph scalded skin syndrome: • A. The staph bacteria can be isolated from the nares , conjunctiva or the perianal area • B. The patients always have associated multiple system involvement including GI hepatic MSK renal and CNS • C. common in adults and adolescents • D. can also be caused by Pseudomonas aeruginosa • E. None of the above PRETEST QUESTIONS • Scarlet fever • A. should be treated with penicillins • B. should be treated with sulfa drugs • C. can lead to toxic shock syndrome • D. can be associated with pharyngitis or circumoral pallor • E. Both A and D are correct PRETEST QUESTIONS • Strep can be treated with the following antibiotics • A. Penicillin • B. First generation cephalosporin • C. clindamycin • D. Septra • E. A B or C • F. A and D only PRETEST QUESTIONS • MRSA • A. is only acquired via hospital • B. can be acquired in the community • C. is more aggressive than OSSA • D. needs treatment with first generation cephalosporin • E. A and C • F. B and C CUTANEOUS BACTERIAL PATHOGENS • Staphylococcus aureus: OSSA and MRSA • Gp A Streptococcus GABHS • Pseudomonas aeruginosa CUTANEOUS BACTERIAL INFECTIONS • Folliculitis • Non bullous Impetigo/Bullous Impetigo • Furuncle/Carbuncle/Abscess • Cellulitis • Acute Paronychia • Dactylitis • Erysipelas • Impetiginization of dermatoses BACTERIAL INFECTION • Important to diagnose early • Almost always -
Building Blocks of Clinical Practice Helping Athletic Trainers Build a Strong Foundation
Building Blocks of Clinical Practice Helping Athletic Trainers Build a Strong Foundation Issue #2: Bacterial Infections of the Skin Folliculitis Carbuncles Definition: Definition: • Inflammation of a hair follicle • A complication of folliculitis, a carbuncle is several • Can progress down hair follicle or into multiple furuncles that have merged.merged follicles and result in a furuncle or carbuncle • Carbuncles are readily transmitted by skin-to-skin contact. Often, the direct cause of a carbuncle cannot Causes: be determined.determined • Bacterial or viral infection, chemical irritation • Secondary to skin injury, which introduces bacteria to Causes: the area • Most carbuncles are caused by the bacteria • Shaving hairy areas may facilitate infection staphylococcus aureus. The infection is contagious and • Occlusion of hair-bearing areas may facilitate growth may spread to other areas of the body or other people.people of microbes • Friction Symptoms: • Hyperhidrosis • A carbuncle is a swollen lump or mass under the skin which may be the size of a pea or as larlargege as a golf ball.ball Symptoms: • The carbuncle may be red and irritated and might hurt • Areas are usually non-tender or slightly tender when you touch it.it • Area may itch • Pain gets worse as it fills with pus and dead tissue.tissue • Erythematous perifollicular papules or pustules may • Other signs and symptoms include itching at the site of develop infection, skin inflammation around the wound, general • Grouped lesions ill feeling, fever, or fatigue. Pain improves -
Human Herpes Viruses 10/06/2012
Version 2.0 Human Herpes Viruses 10/06/2012 Name comes from the Greek 'Herpein' - 'to creep' = chronic/latent/recurrent infections. Types • HHV-1: Herpes simplex type I • HHV-2: Herpes simplex type II • HHV-3: Varicella-zoster virus (VZV) • HHV-4: Epstein-Barr virus (EBV) • HHV-5: Cytomegalovirus (CMV) • HHV-6: Human herpesvirus type 6 (HBLV) • HHV-7: Human herpesvirus type 7 • HHV-8: Kaposi's sarcoma herpesvirus (KSHV) They belong to the following three families: • Alphaherpesviruses: HSV I & II; VZV • Betaherpesviruses: CMV, HHV-6 and HHV-7 • Gammaherpesviruses: EBV and HHV-8 Herpes simplex virus types I and II (HHV1 & 2) Primary infection usually by 2yr of age through mucosal break in mouth, eye or genitals or via minor abrasions in the skin. Asymptomatic or minor local vesicular lesions. Local multiplication → viraemia and systemic infection → migration along peripheral sensory axons to ganglia in the CNS → subsequent life-long latent infection with periodic reactivation → virus travels back down sensory nerves to surface of body and replicates, causing tissue damage: Manifestations of primary HSV infection • Systemic infection , e.g. fever, sore throat, and lymphadenopathy may pass unnoticed. If immunocompromised it may be life-threatening pneumonitis, and hepatitis. • Gingivostomatitis: Ulcers filled with yellow slough appear in the mouth. • Herpetic whitlow: Finger vesicles. Often affects childrens' nurses. • Traumatic herpes (herpes gladiatorum): Vesicles develop at any site where HSV is ground into the skin by brute force. E.g. wrestlers. • Eczema herpeticum: HSV infection of eczematous skin; usually children. • Herpes simplex meningitis: This is uncommon and usually self-limiting (typically HSV II in women during a primary attack) • Genital herpes: Usually HSV type II • HSV keratitis: Corneal dendritic ulcers. -
Herpes Gladiatorum (Mat Herpes): a Fact Sheet
Herpes Gladiatorum (Mat Herpes): A Fact Sheet Herpes gladiatorum is a skin infection caused by the herpes simplex virus. This skin infection is spread by direct skin-to-skin contact. Sports that involve close contact with herpes gladiatorum lesions may spread this infection to other athletes. Generally, lesions (sores) appear within 8 days after exposure to an infected person, but in some cases it may take longer to appear. Good personal hygiene and thorough cleansing and disinfecting of all equipment are essential to helping prevent the spread of this and other skin infections. All athletes with skin sores or lesions should be referred to a physician or primary care provider for evaluation and possible treatment. These individuals should not participate in practice or competition until their lesions have healed. Before skin lesions appear, some people have a sore throat, swollen lymph nodes, fever or tingling on the skin. Herpes gladiatorum lesions appear as a cluster of blisters and may be on the face, arms, legs or trunk. Seek medical care immediately for lesions in or around the eye. Herpes gladiatorum infections can recur. The virus can "hide out" in the nerves and reactivate later, causing another infection. Generally, recurrent infections are less severe and don't last as long. However, a recurring infection is just as contagious as the original infection, so the same steps need to be taken to prevent infecting others. • Personal hygiene for athletes is essential. • Shower at school immediately after practice, using soap and water. • Always use your own plastic bottle of liquid soap. Use your own soap and towel. -
Modern Surgery, 4Th Edition, by John Chalmers Da Costa Rare Medical Books
Thomas Jefferson University Jefferson Digital Commons Modern Surgery, 4th edition, by John Chalmers Da Costa Rare Medical Books 1903 Modern Surgery - Chapter 31. Diseases of the Skin and Nails John Chalmers Da Costa Jefferson Medical College Follow this and additional works at: https://jdc.jefferson.edu/dacosta_modernsurgery Part of the History of Science, Technology, and Medicine Commons Let us know how access to this document benefits ouy Recommended Citation Da Costa, John Chalmers, "Modern Surgery - Chapter 31. Diseases of the Skin and Nails" (1903). Modern Surgery, 4th edition, by John Chalmers Da Costa. Paper 14. https://jdc.jefferson.edu/dacosta_modernsurgery/14 This Article is brought to you for free and open access by the Jefferson Digital Commons. The Jefferson Digital Commons is a service of Thomas Jefferson University's Center for Teaching and Learning (CTL). The Commons is a showcase for Jefferson books and journals, peer-reviewed scholarly publications, unique historical collections from the University archives, and teaching tools. The Jefferson Digital Commons allows researchers and interested readers anywhere in the world to learn about and keep up to date with Jefferson scholarship. This article has been accepted for inclusion in Modern Surgery, 4th edition, by John Chalmers Da Costa by an authorized administrator of the Jefferson Digital Commons. For more information, please contact: [email protected]. 896 Diseases of the Skin and Nails XXXI. DISEASES OF THE SKIN AND NAILS. Dermatitis venenata results from irritants and from garments con- taining arsenic, but is generally due to rhus-poisoning. Rhus-poisoning arises from the poison-oak, the poison-ash, the poison-ivy, and other species of sumach.