DOCSLIB.ORG

Angina Pectoris with Syncope Due to Paroxysmal Atrioventricular Block: Role of Ischaemia Report of Two Cases

Total Page：16

File Type：pdf, Size：1020Kb

Download full-text PDF Read full-text
Angina Pectoris with Syncope Due to Paroxysmal Atrioventricular Block: Role of Ischaemia Report of Two Cases Br Heart J: first published as 10.1136/hrt.36.6.577 on 1 June 1974. Downloaded from British Heart Journal, 1974, 36, 577-58I. Angina pectoris with syncope due to paroxysmal atrioventricular block: role of ischaemia Report of two cases Paul Chiche, Robert Haiat, and Pierre Steff From Service de Cardiologie et Urgences Circulatoires, H6pital Tenon, Paris, France Two patients who experienced angina pectoris resulting in syncope, caused by paroxysmal atrioventricular block, are reported. In both cases circulatory arrest occurred at the peak of the anginal attack. It was related in one case to ventricular standstill and in the other to runs of ventricular tachycardia (wave bursts 'torsades de pointe'). In both cases abolition of the syncopal attacks was achieved by insertion of a permanent demand pacemaker. These observations illustrate one of the possible mechanisms of sudden death in patients with coronary disease. They stress the primary role of acute ischaemia in the genesis of arrhythmias both during an attack of angina and the course of asymptomatic atherosclerotic heart disease. The occurrence of syncope caused by paroxysmal then lost consciousness for 45 seconds. The electro- atrioventricular (AV) block during attacks of angina cardiogram recorded during syncope (Fig. 2) showed pectoris in patients with ischaemic heart disease is complete AV block with prolonged ventricular standstill rare. To our knowledge only 5 documented reports (3 5 sec). A permanent demand pacemaker (Stanium http://heart.bmj.com/ We Monopolar) was implanted using the jugular vein. have been published so far. thought it relevant During a nine-month follow-up period the patient is to describe two new cases, which demonstrate one doing well: he is free of any syncope though he does of the possible mechanisms of sudden death in experience episodic anginal pain. patients with atherosclerotic heart disease. Summary A 78-year-old man with coronary insuffi- Case reports ciency and bifascicular block (right bundle-branch block Case I and left anterosuperior hemiblock) experienced, at the A 78-year-old man presented with a past history of peak of anginal pain, syncope caused by paroxysmal AV on October 2, 2021 by guest. Protected copyright. hypertension and mild renal insufficiency. In I969 he block and ventricular standstill. Relief from syncopal experienced for the first time an attack of angina pec- attacks was obtained after insertion of a permanent toris. Since I972 syncope frequently occurred at the demand pacemaker. peak of anginal pain, at rest or on exercise. The patient complained of typical constrictive pain, then fainted. Case 2 Syncope lasted less than one minute, and was associated An 8o-year-old woman presented with a past history of with urinary incontinence. Neurological emiation hypertension and mild cardiac failure. Since I97I she and two encephalograms revealed no abnormality. The repeatedly experienced short attacks of thoracic con- patient was referred to us in January I973 because of the strictive pain immediately followed by dizziness or recent recurrence of syncopal attacks. Physical exam- syncope. The patient was referred to us in May 1973 ination was non-contributory except for a grade 2/6 for cardiac evaluation. Physical examination was non- systolic murmur at the apex and a moderate cardio- contributory except for a grade if6 midsystolic murmur megaly on the chest x-ray film. The electrocardiogram at the apex. The basic electrocardiogram showed sinus showed sinus rhythm with right bundle-branch block rhythm with normal PR interval and left bundle-branch and left anterosuperior hemiblock (Fig. i). Paroxysmal block. The patient was placed under continuous electro- AV block was suspected and the patient was placed cardiographic monitoring. On several occasions the under continuous electrocardiographic monitoring. The tracing showed asymptomatic episodes of high degree next day at 5.45 a.m. he was awakened by a severe retro- AV block (Fig. 3). The day after referral the patient had sternal constrictive pain similar to the previous ones, thoracic constrictive pain identical with the previous Received 13 November 1973. ones and then lost consciousness. The electrocardiogram Br Heart J: first published as 10.1136/hrt.36.6.577 on 1 June 1974. Downloaded from 578 Chiche, Haiat, and Steff II II l11111 RLLF V1 2 3 V456 FIG. I Case 2. 9 January 1973. Sinus rhythm is present with normal PR interval and right bundle-branch block, with left anterosuperior hemiblock. http://heart.bmj.com/ FIG. 2 Case . 10 anuary 1973. 5 a.m. During a syncopal episode which occurred at the height of an anginal attach complete atrioventricular bloch with prolonged ventricular asystoleI7(.a5 sec) is recorded fromn the electrocardiogram. H111Igig~~~~~~~iR F on October 2, 2021 by guest. Protected copyright. -r-:t FIG. 3 Case 2. II May 1973. In the absence of anginal pain an asymptomatic high grade atrioventricular block with occasional capture is recorded. Br Heart J: first published as 10.1136/hrt.36.6.577 on 1 June 1974. Downloaded from Angina pectoris with syncope due to paroxysmal atrioventricular block: role of ischaemia 579 I A~~~~~~~~~~~~~~~~e I-t-v FIG. 4 Case 2. 12 May 1973. During an attack of angina pectoris followed by syncope, wave bursts ('torsades de pointe') appear. recorded during the syncopal attack (Fig. 4) showed a The occurrence of a syncopal attack during an high grade AV block, and, for the first time, runs of episode of angina is rare (Gallavardin, I922; ventricular tachycardia (wave bursts: 'torsades de Golden, I944). Pathogenesis of the syncope include pointe' (Dessertenne, I966)). The next day a permanent (Chiche, I972): increase in vagal tone; cerebral demand pacemaker (Stanium Monopolar) was implanted using the jugular vein. During a six-month follow-up ischaemia resulting from the reduction in myo- the patient is doing well: she is free of any syncope cardial performance due to acute ischaemia (Chiche, though does experience a few attacks of angina pectoris. I972); arrhythmias as especially observed in Prinz- metal's angina: sinoatrial block (Dorra et al., Summary An 8o-year-old woman with coronary in- I968); ventricular fibrillation (Raynaud et al., I969; sufficiency and left bundle-branch block experienced Schwartz, Schwedel, and Schwartz, I966), and syncopal attacks at the peak of anginal pain. Between the paroxysmal AV block which is discussed here. attacks of angina continuous electrocardiographic moni- Syncopal attacks caused by paroxysmal AV block toring showed episodes of high degree AV block which during angina, to our knowledge, so far have been remained asymptomatic. During an anginal attack syn- http://heart.bmj.com/ cope occurred and simultaneously wave bursts ('torsades mentioned in only 5 documented reports (Galla- de pointe') were recorded on the electrocardiogram. vardin, 1922; Botti, I966; Schwartz et al., I966; Relief from syncopal attacks was obtained after inser- Raynaud et al., I969; Faivre et al., 1972). Galla- tion of a permanent demand pacemaker. vardin (I922) was the first to report these in what is now a classical description: 'Every time the patients have violent attacks of angina they are subject to Discussion fits of dizziness or actual syncope. Slowing of the Asymptomatic atrioventricular (AV) conduction pulse cannot always be recorded because once the disturbances are known to occur in about 20 per attack ends everything returns to normal. Un- on October 2, 2021 by guest. Protected copyright. cent of cases in the acute phase of myocardial in- doubtedly it is only during an attack that one can farction. They are more frequent in posteroinferior notice the slowing or maybe the disappearance of (I5%) than anterior (5%) infarctions (Himbert the pulse ... The linking of the fits of dizziness or and Lenegre, I967). As a rule, they are due to rever- syncope to conduction disturbances is still more sible ischaemia of the AV node and are transient. likely to be true with the finding of an unusually Asymptomatic AV conduction disturbances are prolonged a-c interval in the venous pulse tracing'. probably caused by a similar mechanism, but are More recent cases (Botti, I966; Schwartz et al., quite unusual during an attack of angina. Auzepy I966; Raynaud et al., I969) have made it possible to et al. (I962) reported one case out of I95. A few state precisely the mechanism of the circulatory other reports (Prinzmetal et al., 1959; Bouvrain, arrest: ventricular pause (Case i), or 'wave bursts' Fortin, and Coumel, I963; Nadal-Ginard and ('torsades de pointe') (Case 2). This last term refers Cardenas, I973; Oliva, Potts, and Pluss, I973) in- to runs of ventricular tachycardia which have been clude cases seen during variant angina pectoris individualized according to their particular mor- described by Prinzmetal et al. (I959); in these cases, phology (Dessertenne, I966). When recorded on the degree of block varied from a second-degree three simultaneous leads the electrocardiogram block (Mobitz I (Nadal-Ginard and Cardenas, evidences remarkable periodical changes in QRS I973) or 2: I (Bouvrain et al., I963) to a complete amplitude (gradual decrease then increase) and heart block (Nadal-Ginard and Cardenas, I973). orientation (the complexes seem to 'twist' them- Br Heart J: first published as 10.1136/hrt.36.6.577 on 1 June 1974. Downloaded from 58o Chiche, Haiat, and Steff selves around the baseline). In American reports out angina (as anginal pain is a late and/or incon- 'torsades de pointes' are usually referred as 'tran- stant sign of myocardial ischaemia). As clearly sient ventricular fibrillation' (Schwartz, Orloff, and shown, ischaemia may lead to severe arrhythmias Fox, 1949) or 'chaotic ventricular tachycardia' and must be treated so in an endeavour to prevent (Scherf, Cohen, and Shafiiha, I967). sudden death (Chiche, I973). In our patients, syncope caused by paroxysmal AV block occurred at the peak of typical anginal References attacks which did not have the features of Prinz- attacks were to Amsterdam, E.
Load more

Recommended publications
  • First Degree Atrioventricular Block Patrick Loftis Marquette University, [email protected]
    Marquette University e-Publications@Marquette Physician Assistant Studies Faculty Research and Physician Assistant Studies, Department Publications 1-21-2011 First Degree Atrioventricular Block Patrick Loftis Marquette University, [email protected] James F. Ginter Aurora Cardiovascular Services Published version. Journal of the American Academy of Physician Assistants, Vol. 24, No. 1 (January 2011). Permalink. © 2011, American Academy of Physician Assistants and Haymarket Media Inc. Used with permission. First-degree atrioventricular blocks - Print Article - JAAPA http://www.jaapa.com/first-degree-atrioventricular-blocks/printarticle/1... << Return to First-degree atrioventricular blocks Patrick Loftis, PA-C, MPAS, RN, James F. Ginter, MPAS, PA-C January 21 2011 An atrioventricular (AV) block is a common cardiac abnormality. It involves a slowing or blockage of the electrical impulse coming from the sinoatrial (SA) node at or around the AV node (Figure 1). AV blocks are characterized by the slowing or partial or complete blocking of the impulse. This discussion will focus on first- degree atrioventricular block, which is the slowing or partial blocking of the impulse; complete blockade will be discussed in a future segment of this department. Despite the name, no impulse is actually blocked in first-degree AV block. Instead, each impulse is simply slowed at or near the atrioventricular node. On an ECG, AV block is manifested by a prolonged PR interval, which is measured from the beginning of the P wave to the beginning of the QRS complex. The ECG criterion for a first-degree atrioventricular block is a PR interval greater than 200 milliseconds. Symptoms First-degree AV block by itself does not result in symptoms.
    [Show full text]
  • Atrioventricular Block in Children with Multisystem Inflammatory Syndrome Audrey Dionne, Douglas Y
    Atrioventricular Block in Children With Multisystem Inflammatory Syndrome Audrey Dionne, MD,a,b Douglas Y. Mah, MD,a,b Mary Beth F. Son, MD,b,c Pui Y. Lee, MD, PhD,b,c Lauren Henderson, MD, MMSc,b,c Annette L. Baker, MSC, PNP,a,b Sarah D. de Ferranti, MD,a,b David R. Fulton, MD,a,b Jane W. Newburger, MD, MPH,a,b Kevin G. Friedman, MDa,b BACKGROUND: Children are at risk for multisystem inflammatory syndrome in children (MIS-C) abstract after infection with severe acute respiratory syndrome coronavirus 2. Cardiovascular complications, including ventricular dysfunction and coronary dilation, are frequent, but there are limited data on arrhythmic complications. METHODS: Retrospective cohort study of children and young adults aged #21 years admitted with MIS-C. Demographic characteristics, electrocardiogram (ECG) and echocardiogram findings, and hospital course were described. RESULTS: Among 25 patients admitted with MIS-C (60% male; median age 9.7 [interquartile range 2.7–15.0] years), ECG anomalies were found in 14 (56%). First-degree atrioventricular block (AVB) was seen in 5 (20%) patients a median of 6 (interquartile range 5–8) days after onset of fever and progressed to second- or third-degree AVB in 4 patients. No patient required intervention for AVB. All patients with AVB were admitted to the ICU (before onset of AVB) and had ventricular dysfunction on echocardiograms. All patients with second- or third- degree AVB had elevated brain natriuretic peptide levels, whereas the patient with first- degree AVB had a normal brain natriuretic peptide level. No patient with AVB had an elevated troponin level.
    [Show full text]
  • An Extremely Rare Cause of Wolff-Parkinson
    108 Erciyes Med J 2019; 41(1): 108–10 • DOI: 10.14744/etd.2018.18165 An Extremely Rare Cause of Wolff-Parkinson-White Syndrome: Rhabdomyoma in Association With Tuberous Sclerosis CASE REPORT Özlem Elkıran , Cemşit Karakurt , Damla İnce ABSTRACT Rhabdomyomas are the most common primary cardiac tumors in infants and children. They are usually associated with tuberous sclerosis (TS). As the tumors tend to regress spontaneously, surgical intervention is not usually performed unless they become obstructive or cause incessant arrhythmias. We report an extremely rare case of rhabdomyoma serving as a substrate for Wolff-Parkinson-White (WPW) syndrome and intractable supraventricular tachycardia accompanied by TS. Our case is particularly interesting because it was diagnosed prenatally. The signs of WPW syndrome disappeared from the elec- trocardiogram with the regression of the tumor. Keywords: Wolff-Parkinson-White Syndrome, child, rhabdomyoma INTRODUCTION Rhabdomyomas are the most common cardiac tumors in infants and children, and they are closely related with tuberous sclerosis (TS). A significant part of rhabdomyomas is asymptomatic, and they regress on follow-up. However, symptoms of cardiac failure, arrhythmias, and obstruction can be observed depending on their location in the heart. They require urgent medical or surgical treatment (1, 2). Cite this article as: Elkıran Ö, Karakurt C, İnce D. An Extremely Rhabdomyoma-related arrhythmias are reported as premature atrial and ventricular contractions, supraventricular Rare Cause of and ventricular tachycardia, sinus node dysfunction, atrioventricular block, and Wolff-Parkinson-White (WPW) Wolff-Parkinson-White syndrome (1, 3, 4). There are only a few studies of WPW syndrome occurring in association with TS, with and Syndrome: Rhabdomyoma in Association With without rhabdomyoma.
    [Show full text]
  • Cardiac Pacing in Incomplete Atrioventricular Block with Atrial Fibrillation
    Br Heart J: first published as 10.1136/hrt.35.11.1154 on 1 November 1973. Downloaded from British Heart journal, I973, 35, I154-1I60. Cardiac pacing in incomplete atrioventricular block with atrial fibrillation D. S. Reid, S. J. Jachuck, and C. B. Henderson From the Department of Cardiology, Newcastle General Hospital, Newcastle upon Tyne Three cases with a slow irregular ventricular response to atrialfibrillation, who benefitedfrom cardiac pacing, are described; two had ischaemic heart disease, and one had cardiomyopathy. In thefirst case the slow ventricu- lar response to atrialfibrillation was a result of incomplete atrioventricular nodal block, and in the other two His bundle electrograms demonstrated that the slow ventricular response was due to bilateral bundle-branch block. The association of atrial fibrillation and conduction delays in the atrioventricular node and bundle- branches is discussed. The value of His bundle recordings in the investigation of these cases is shown and the importance of cardiac pacing in treatment is stressed. Cardiac pacing is now a generally accepted method hypotension. Intravenous atropine given before transfer of treatment in patients with complete heart block or had resulted in a paroxysm of ventricular tachycardia. bilateral bundle-branch block who have Adam- On admission there was no evidence of cardiac failure Stokes attacks or a low output to and the blood pressure was I05/50 mmHg. An electro- syndrome leading cardiogram showed atrial fibrillation with an irregular angina or cardiac failure. cardiac is However, pacing ventricular response of 40 to 44 beats a minute and acute now also becoming more widely used in the treat- inferolateral myocardial infarction (Fig.
    [Show full text]
  • Neurological Associations of Chronic Heart Block
    J Neurol Neurosurg Psychiatry: first published as 10.1136/jnnp.39.6.571 on 1 June 1976. Downloaded from Journal ofNeurology, Neurosurgery, andPsychiatry, 1976, 39, 571-575 Neurological associations of chronic heart block C. D. LAMBERT AND A. J. FAIRFAX From the Departments of Neurology and Cardiology, St. George's Hospital, London SYNOPSIS A large group of patients with chronic heart block was studied for evidence of neurological disorder. Six out of 892 patients were found who had neuromuscular disease related to the conduction disturbance. In four patients, cardiac involvement appeared selectively to affect the conducting tissues. Three of these patients had a scapuloperoneal syndrome, the fourth, the oculocraniosomatic syndrome. In the remaining two patients, one with limb girdle dystrophy and the other with dystrophia myotonica, cardiomyopathy was present in addition to the conduction disturbance. This paper reports the first comprehensive with incomplete heart block or transient heart study of the frequency and nature of neuro- block secondary to myocardial infarction were not Protected by copyright. logical disorders occurring in association with included. complete heart block. The most common Those cases with evidence of neuromuscular disease were examined in detail, a family history pathological cause of chronic heart block is taken, serum creatine phosphokinase levels meas- idiopathic bundle branch fibrosis (Davies, ured and, when possible, electromyography per- 1971) thought by Lenegre, who described it, to formed. Muscle biopsy specimens, obtained under be a selective myopathy of the cardiac con- local anaesthetic, were examined using conven- ducting 'tissues (Lenegre, 1964). His original tional morphological and histochemical techniques series of 66 cases contained two patients with (Dubowitz and Brooke, 1973).
    [Show full text]
  • Wolff-Parkinson-White Syndrome Type B with Tachycardia-Dependent
    Br Heart J: first published as 10.1136/hrt.43.4.481 on 1 April 1980. Downloaded from Case reports Br HeartJ 1980; 43: 481-6 Wolff-Parkinson-White syndrome type B with tachycardia-dependent (phase 3) block in the accessory pathway and in left bundle-branch coexisting with rate-unrelated right bundle-branch block IVAN J MENDOZA, AGUSTIN CASTELLANOS, RUEY J SUNG From the Division of Cardiology, Department of Medicine, University of Miami School of Medicine, Miami, Florida, USA suMMARY A patient with Wolff-Parkinson-White syndrome type B developed 2:1 atrioventricular block resulting from the association of persistent right bundle-branch block with tachycardia-dependent (phase 3) left bundle-branch block. Electrophysiological studies disclosed the coexistence of a tachy- cardia-dependent (phase 3) block in the accessory pathway. This conduction disturbance was exposed, not by carotid sinus massage as in previous studies, but by pacing-induced prolongation of the interval between two consecutively conducted atrial impulses. Furthermore, the surface electrocardiogram showed, at different times, ventricular complexes resulting from: (1) exclusive atrioventricular conduc- tion through the normal pathway without bundle-branch block; (2) predominant, or exclusive, atrio- ventricular conduction through a right-sided accessory pathway; (3) exclusive atrioventricular conduction http://heart.bmj.com/ through the normal pathway with right bundle-branch block; (4) exclusive conduction through the normal pathway, with left bundle-branch block; (5) fusion between (1) and (2); and finally, (6) fusion between (2) and (3) However, QRS complexes resulting from simultaneously occurring Wolff-Parkinson-White syndrome type B and left bundle-branch block could not be identified.
    [Show full text]
  • Lyme Carditis Airley E
    Infect Dis Clin N Am 22 (2008) 275–288 Lyme Carditis a b Airley E. Fish, MD, MPH , Yuri B. Pride, MD , a, Duane S. Pinto, MD * aDivision of Cardiology, Department of Medicine, Beth Israel Deaconess Medical Center, Harvard Medical School, 1 Deaconess Road, Palmer 415, Boston, MA 02215, USA bDepartment of Medicine, Beth Israel Deaconess Medical Center, Harvard Medical School, 330 Brookline Avenue, Deaconess 311, Boston, MA 02215, USA Lyme borreliosis, or Lyme disease, is a globally occurring, systemic disease caused by the spirochete Borrelia burgdorferi and transmitted by the Ixodes tick. The disease classically is divided into three stages. Stage 1, the early localized stage, generally occurs several days or up to 1 month after the initial tick bite. It is notable for an influenza-like illness and often is accompanied by the erythema migrans (EM) rash. Stage 2, the early dissem­ inated stage, occurs weeks to months after EM. Neurologic symptoms and musculoskeletal complaints are the hallmarks of this stage. Cardiac abnor­ malities, predominantly involving the conduction system and myocardium, also may manifest at this time. Stage 3 occurs several months to years after EM and is characterized by a monoarthritis or oligoarthritis affecting the large joints, and the development and progression of neurologic sequelae. Steere and colleagues first described the cardiovascular complications of Lyme disease nearly 30 years ago in a retrospective report of 20 North American cases. Australian and European cases were reported in the early to mid-1980s. The principal manifestation of Lyme carditis is self-limited conduction derangement, most commonly involving the atrioventricular node.
    [Show full text]
  • Firstdegree AV Blockan Entirely Benign Finding Or a Potentially
    REVIEW ARTICLE First-Degree AV Block—An Entirely Benign Finding or a Potentially Curable Cause of Cardiac Disease? ∗ ∗ Fredrik Holmqvist, M.D., Ph.D. † and James P. Daubert, M.D. ∗ From the Clinical Cardiac Electrophysiology, Duke University Medical Center, Durham, NC; and †Department of Cardiology, Lund University, Lund, Sweden First-degree atrioventricular (AV) block is a delay within the AV conduction system and is defined as a prolongation of the PR interval beyond the upper limit of what is considered normal (generally 0.20 s). Up until recently, first-degree AV block was considered an entirely benign condition. In fact, some complain that it is a misnomer since there is only delay and no actual block in the AV conduction system (usually within the AV node). However, it has long been acknowledged that extreme forms of first-degree AV block (typically a PR interval exceeding 0.30 s) can cause symptoms due to inadequate timing of atrial and ventricular contractions, similar to the so-called pacemaker syndrome. Consequently, the current guidelines state that permanent pacemaker implantation is reasonable for first-degree AV block with symptoms similar to those of pacemaker syndrome or with hemodynamic compromise, but also stresses that there is little evidence to suggest that pacemakers improve survival in patients with isolated first-degree AV block. Recent reports suggest that it may be time to revisit the impact of first-degree AV block. Also, several findings in post hoc analyses of randomized device trials give important insights in possible treatment options. The present review aims to provide an update on the current knowledge concerning the impact of first-degree AV block and also to address the issue of pacing in patients with this condition.
    [Show full text]
  • Transient First-Degree Atrioventricular Block in a Young Patient
    Letter to the Editor 50 Editöre Mektup Transient First-Degree Atrioventricular Block in a Young Patient Genç Hastada Geçici 1. Derece Atriyoventriküler Blok Vakası Murat Ayan1, Enes Elvin Gül2, Osman Sönmez2, Gökhan Altunbaş2 1Deparment of Emergency Medicine, Meram School of Medicine, Selcuk University, Konya, Turkey 2Department of Cardiology, Meram School of Medicine, Selcuk University, Konya, Turkey Introduction Case Report Atrioventricular block (AVB), is characterized as a conduction A 20-year-old man presented at our ED with breathlessness and delay from the atrium to the ventricle or a result of conduction chest pain. He had no previous history of diabetes mellitus, hyper- abnormality in the atrium, atrioventricular node (AVN) or His- tension, or coronary heart disease. On admission, his blood pressure Purkinje system. Classically AVB is divided into three classes: first, was 120/80 mmHg and pulse was 73 bpm. He had no history of second, and third (complete) AVB (1). First degree AVB is diagnosed smoking, alcohol, or drug usage. Twelve-lead electrocardiogram when the PR interval exceeds 200 msec in the electrocardiogram. (ECG) revealed first-degree AVB with the PR interval of 352 msec The etiology of the first-degree AVB is numerous and generally (Figure 1a and 1b). Cardiovascular examination revealed normal physiologic. Nevertheless, first-degree AVB may be associated with a findings. His laboratory profile revealed a white blood cell count of higher risk of coronary heart disease (2). The prognosis of first- 8900/mm3 (4-10000/mm3), hemoglobin of 13.3 g/dl (12.1-17.2 g/dL), degree AVB in patients admitted to the emergency department (ED) hematocrit of 40.2% (36.1-50.3%), platelet count of 236.000/mm3 is unknown.
    [Show full text]
  • Pseudo-Atrioventricular Block Due to Premature Systoles with Concealed Conduction Boguslaw Godlewski
    Henry Ford Hospital Medical Journal Volume 23 | Number 2 Article 4 6-1975 Pseudo-Atrioventricular Block due to Premature Systoles with Concealed Conduction Boguslaw Godlewski Wolf F. C. Duvernoy Remigio Garcia Follow this and additional works at: https://scholarlycommons.henryford.com/hfhmedjournal Part of the Life Sciences Commons, Medical Specialties Commons, and the Public Health Commons Recommended Citation Godlewski, Boguslaw; Duvernoy, Wolf F. C.; and Garcia, Remigio (1975) "Pseudo-Atrioventricular Block due to Premature Systoles with Concealed Conduction," Henry Ford Hospital Medical Journal : Vol. 23 : No. 2 , 65-70. Available at: https://scholarlycommons.henryford.com/hfhmedjournal/vol23/iss2/4 This Article is brought to you for free and open access by Henry Ford Health System Scholarly Commons. It has been accepted for inclusion in Henry Ford Hospital Medical Journal by an authorized editor of Henry Ford Health System Scholarly Commons. Henry Ford Hosp. Med. Journal Vol. 23, No. 2, 1975 Pseudo-Atrioventricular Block due to Premature Systoles with Concealed Conduction Boguslaw Godlewski, MD; Wolf F. C. Duvernoy, MD; and Remigio Garcia, MD* fremature junctional beats are usually de­ tected in the surface electrocardiogram Two patients are presented with EKC (EKC) by the presence of antegrade and/or findings suggesting pseudo AV block retrograde conduction.^ - ^ Concealed junc­ Mobitz type II. Pseudo AV block was tional or ventricular premature beats may related to the presence of premature junc­ be solely manifested by their influence on tional or ventricular beats with concealed the conduction of the subsequent beats conduction. Response to therapy confirmed giving rise to varying prolongation ofthe AV the initial diagnosis of pseudo AV block.
    [Show full text]
  • Third Degree Atrioventricular Block
    COMPLETE HEART BLOCK (THIRD-DEGREE ATRIOVENTRICULAR BLOCK) BASICS OVERVIEW The heart of the dog or cat is composed of four chambers; the top two chambers are the right and left atria and the bottom two chambers are the right and left ventricles In order to pump blood to the lungs and body, the heart must work in a coordinated fashion; the normal control or “pacemaker” of the heart is the sinoatrial (SA) node, which starts the electrical impulse to begin the coordinated contraction of the heart muscles—the electrical impulse causes the atria to contract, pumping blood into the ventricles; the electrical impulse moves through the atrioventricular (AV) node and into the ventricles, causing the ventricles to contract and to pump blood to the lungs (right ventricle) and the body (left ventricle) The normal heart rate for dogs varies based on the size of the dog; however, the general range is 60 to 180 beats per minute (with smaller dogs have faster normal heart rates) The general range for normal heart rate in cats is 120 to 240 beats per minute An electrocardiogram (“ECG”) is a recording of the electrical impulse activity of the heart; the normal ECG is a tracing with P, QRS, and T waves; the P waves are the first upward deflection of the ECG tracing that look like a “bump” in the tracing; the P waves are a measure of the electrical activity of the atria; the QRS looks like an exaggerated “W” with the Q wave being a short, downward deflection, the R being a tall, spiked upward deflection, and the S being another short, downward deflection;
    [Show full text]
  • Atrio-Ventricular Block in Children with Multisystem Inflammatory Syndrome
    Prepublication Release Atrio-Ventricular Block in Children With Multisystem Inflammatory Syndrome Audrey Dionne, MD, Douglas Y. Mah, MD, Mary Beth F. Son, MD, Pui Y. Lee MD, PhD, Lauren Henderson, MD, MMSc, Annette L. Baker, MSC, PNP, Sarah D. de Ferranti, MD, David R. Fulton, MD, Jane W. Newburger, MD, MPH, Kevin G. Friedman, MD DOI: 10.1542/peds.2020-009704 Journal: Pediatrics Article Type: Regular Article Citation: Dionne A, Mah DY, Son MBF, et al. Atrio-ventricular block in children with multisystem inflammatory syndrome. Pediatrics. 2020; doi: 10.1542/peds.2020-009704 This is a prepublication version of an article that has undergone peer review and been accepted for publication but is not the final version of record. This paper may be cited using the DOI and date of access. This paper may contain information that has errors in facts, figures, and statements, and will be corrected in the final published version. The journal is providing an early version of this article to expedite access to this information. The American Academy of Pediatrics, the editors, and authors are not responsible for inaccurate information and data described in this version. ©2020 American Academy of Pediatrics Downloaded from www.aappublications.org/news by guest on September 29, 2021 Prepublication Release Atrio-Ventricular Block in Children With Multisystem Inflammatory Syndrome Audrey Dionne, MDa,c, Douglas Y. Mah, MDa,c, Mary Beth F. Son, MDb,c, Pui Y. Lee, MD, PhDb,c, Lauren Henderson, MD, MMScb,c, Annette L. Baker, MSC, PNPa,c, Sarah D. de Ferranti, MDa,c, David R. Fulton, MDa,c, Jane W.
    [Show full text]