DOCSLIB.ORG

Reference List Concerning Plantar Fasciitis

Total Page：16

File Type：pdf, Size：1020Kb

Download full-text PDF Read full-text
Reference List Concerning Plantar Fasciitis PLANTAR FASCIITIS RMA ID Reference List for RMA240-2 as at February 2015 Number Abate M, Schiavone C, Di Carlo L, et al (2012). Achilles tendon and plantar 72228 fascia in recently diagnosed type II diabetes: role of body mass index. Clin Rheumatol, 31(7): 1109-13. Acevedo JI, Beskin JL (1998). Complications of plantar fascia rupture 14526 associated with corticosteroid injection. Foot & Ankle International, 19(2): 91-7. Agunwa WCR (1989). Laterality and incidence of adhesive capsulitis of the 14476 shoulder and plantar fasciitis among rural Saudis in the Jeddah zone. Annals of the Rheumatic Diseases, 48(4): 351. Akfirat M, Sen C, Gunes T (2003). Ultrasonographic appearance of the plantar 39688 fasciitis. Journal of Clinical Imaging, Vol 27 pp 353-357. Aldridge T (2004). Diagnosing heel pain in adults. American Family Physician, 39716 70(2) pp 332-338. Almeida SA, Williams KM, Shaffer RA, & Brodine SK (1999). Epidemiological 17605 patterns of musculoskeletal injuries and physical training. Medicine & Science in Sports & Exercise, Vol 31 (8) pp 1176-82. Amaravati RS (2005). Massive calcaneal enthesopathy in a non-healing leg 39883 ulcer: a case report. J Orthop Surg 13(2): 211-213. American Academy of Family Physicians (2005). Plantar fasciitis: a common 40016 cause of heel pain. http://familydoctor.org/140.xml Updated 03/06 Ames DL (1986). Overuse Syndrome. The Journal of the Florida Medical 4289 Association, 73(8): 607-8. Andrews JR (1983). Overuse Syndromes of the Lower Extremity. Clinics in 4280 Sports Medicine, 2(1): 137-48. Anonymous (2001). Plantar fasciitis. 39941 http://orthoinfo.aaos.org/fact/printer_page.cfm?topcategory=Foot&Thread_ID= 144. Downloaded 28 August, 2006. Anonymous (2005). Plantar fasciitis. 40015 http://www.intellihealth.com/IH/ihtIH/WSIHW000/9339/10550.html Last updated October 6, 2005. Anonymous (2006). Five most common pathomechanical foot dysfunctions. 39944 http://www.footmaxx.com/clinicians/five.html#equinus Downloaded 23 August 2006 Apley AG, Solomon L (1993). The ankle and foot. AG Aspley, L Solomon L 4214 (Eds). Apley's system of orthopaedics and fractures, 7th Edition, 486-98. Butterworth Heinemann, Oxford. Arnett FC (2005). Seronegative spondyloarthritis. ACP Medicine. Obtained 39712 from http://acp.medicine.com/acpmedicine/pdf/med1503.pdf Ayub A, Yale SH, Bibbo C (2005). Common foot disorders. Clinical Medicine & 39714 Research, 3(2) pp 116-119. Page 1 of 15 Badlissi F, Dunn JE, Link CL, Keysor JJ, McKinlay JB, Felson DT (2005). Foot 39689 musculoskeletal disorders, pain, and foot-related functional limitation in older persons. J Am Geriatr Soc, Vol 53 pp 1029-1033. Baer A, Patel VP, McCormack R (2005). The approach to the painful joint. 40017 http://www.emedicine.com/med/topic3562.htm. Last updated April 8, 2005. Balint PV, Kane D, Wilson H, et al (2002). Ultrasonography of entheseal 39751 insertions in the lower limb in spondyloarthropathy. Ann Rheum Dis, 61(10) pp 905-910. Barrett SL, O'Malley R (1999). Plantar fasciitis and other causes of heel pain. 17590 American Family Physician, Vol 59(8) pp 2200-2206. Barry NN & McGuire JL (1996). Acute injuries and specific problems in adult 17592 athletes. Rheumatic Disease Clinics of North America, Vol 22(3) pp 531-549. Bassiouni M (1965). Incidence of Calcaneal Spurs in Osteoarthritis and 4259 Rheumatoid Arthritis, and in Control Patients. Annals of the Rheumatic Diseases, 24: 490-3. 4281 Baxter DE (1994). The Heel In Sport. Clinics in Sports Medicine, 13(4): 683-93. Ben Kibler W, et al (1991). Functional biomechanical deficits in running 4293 athletes with plantar fasciitis. The American Journal of Sports Medicine, 19(1): 66-71. Benjamin M, Toumi H, Ralphs JR, Bydder G, Best TM, Milz S (2006). Where 39727 tendons and ligaments meet bone: attachment sites ('entheses') in relation to exercise and/or mechanical load. J Anat, Vol 208 pp 471-490. Blair SN & Caspersen CJ (1985). Risk factors & running injuries. Med Sci 17666 Sports Exercise, Vol 17(2) p xii. Blake RL, Ferguson HJ (1993). Walking and hiking injuries. A one year follow- 17615 up study. Journal of the American Podiatric Medical Association, Vol 83(9) pp 499-503. Bolgla LA, Malone TR (2004). Plantar fasciitis and the windlass mechanism: a 39720 biomechanical link to clinical practice. Journal of Athletic Training, 39(1) pp 77- 82. Bolivar YA, Munuera PV, Padillo JP (2013). Relationship between tightness of 72686 the posterior muscles of the lower limb and plantar fasciitis. Foot & Ankle International, 34(1): 42-8. Bordelon RL (1993). Heel pain. RA Mann and MJ Coughlin (Eds). Surgery of 4215 the Foot and Ankle, 6th Edition: 837-57. Mosby, St Louis. Brubaker CE & James SL (1974). Injuries to runners. Journal of Sports 17672 Medicine, Vol 2(4) pp 189-198. 39711 Buchbinder R (2004). Plantar fasciitis. N Engl J Med, Vol 350 pp 2159-2166. Buchbinder R (2014). Plantar fasciitis. Retrieved 15 October 2014, from 73211 http://www.uptodate.com/contents/plantar-fasciitis Butts JF, Gebke KB (2011). Getting injured runners back on track. The Journal 73184 of Family Practice, 60(11): 646-52. Bylak J & Hutchinson MR (1998). Common sports injuries in young tennis 17661 players. Sports Medicine, Vol 26(2) pp 119-32. Campbell-Giovaniello KJ (1997). Clinical snapshot. Plantar fasciitis. Strategies 14482 to relieve a common orthopedic syndrome. Am J Nurs, 97(9): 38-9. Caselli MA & Sobel E (2001). Sports medicine: what are the best orthotics for 40304 plantar fasciitis. Podiatry Today, Vol 14, Issue 12, obtained from http://www.podiatrytoday.com/article/43 Chandler TJ, Kibler WB (1993). A Biomechanical Approach to the Prevention, 4278 Treatment and Rehabilitation of Plantar Fasciitis. Sports Medicine, 15(5): 344 - 52. Chandler TJ, Kibler WB (1993). A biomechanical approach to the prevention, 14548 treatment and rehabilitation of plantar fasciitis. Sports Medicine, 15(5): 344-52. Page 2 of 15 Chang HK, Lee DH, Jung SM & Choi SJ, et al (2002). The comparison 40446 between Behcet's disease and spondyloarthritides: does Behcet's disease belong to the spondyloarthropathy complex? J Korean Med Sci 17: 524-529. Chen L, Greisberg J (2009). Achilles lengthening procedures. Foot Ankle Clin 71792 N Am, 14: 627-37. Cheung JT-M, Zhang M, An K-N (2006). Effect of Achilles tendon loading on 39695 plantar fascia tension in the standing foot. Clinical Biomechanics, Vol 21 pp 194-203. Chigwanda PC (1997). A prospective study of plantar fasciitis in Harare. 14468 Central African Journal of Medicine, 43(1): 23-5. Clancy WG (1980). Runners' Injuries Part two. Evaluation and treatment of 4277 specific injuries. The American Journal of Sports Medicine, 8(4): 287-9. Clement DB, Taunton JE, Smart GW, McNicol KL (1981). A survey of overuse 9934 running injuries. The Physician and Sports Medicine, 9(5): 47-58. Cohen MR & Cohen EP (1995). Enthesopathy and atypical gouty arthritis 40210 following renal transplantation: a case control study. Rev Rhum Engl Ed 62(2):86-90. Cole C, Seto C, Gazewood J (2005). Plantar fasciitis: evidence-based review 39717 of diagnosis and therapy. American Family Physician, 72(11) pp 2237-2242. Collins K, et al (1989). Overuse injuries in triathletes - A study of the 1986 4275 Seafair Triathlon. The American Journal of Sports Medicine, 17(5): 675-80. 4242 Condemi JJ (1992). The Autoimmune Diseases. JAMA, 268(20): 2882-92. Cook SD, Brinker MR, Poche M (1990). Running Shoes Their Relationship to 4269 Running Injuries. Sports Medicine, 10(1): 1-8. Cornwall MW, McPoil TG (1999). Plantar fasciitis: etiology and treatment. 39734 Journal of Orthopaedic & Sports Physical Therapy, 29(12) pp 756-760. Cowan DN, Jones BH, Frykman PN, Polly DW Jr, Harman EA, Rosenstein RM, & Rosenstein MT (1996). Lower limb morphology and risk of overuse injury 17609 among male infantry trainees. Medicine & Science in Sports & Exercise, Vol 28(8) pp 945-52. Craig ME, Duffin AC, Gallego PH et al (2008). Plantar fascia thickness, a 73189 measure of tissue glycation, predicts the development of complications in adolescents with type 1 diabetes. Diabetes Care, 31(6): 1201-6. Crystal RG (1993). Sarcoidosis. Harrison's Principles of Internal Medicine, 13th 4216 Edition, Chapter 292: 1679-84. Cuellar ML, Espinoza LR (1995). Psoriatic Arthritis - Current Developments. 4287 The Journal of the Florida Medical Association, 82(5): 338-42. Cuellar ML, et al (1994). Recent developments in psoriatic arthritis. Current 4252 Opinion in. Rheumatology, 6(6): 378-84. Cullen NP, Singh D (2006). Plantar fasciitis: a review. British Journal of 39743 Hospital Medicine, 67(2) pp 72-76. Cutts S, Obi N, Pasapula C et al (2012). Plantar fasciitis. Annals of the Royal 73190 College of Surgeons of England, 94: 539-42. Dailey JM (1991). Differential diagnosis and treatment of heel pain. Clinics. 4217 Podiatric Medicine and Surgery, 8(1): l53- 66. D'Ambrogi E, Giacomozzi C, Macellari V & Uccioli L (2005)[Abstract only]. 40209 Abnormal foot function in diabetic patients: the altered onset of Windlass mechanism. Diabet Med;22(12):1713-1719. D'Ambrogi E, Macellari V, Giurato L, Caselli A, et al (2003). Contribution of 39723 plantar fascia to the increased forefoot pressures in diabetic patients. Diabetes Care, 26(5) pp 1525-1529. de Miguel E, Cobo T, Munoz-Fernandez S, et al (2009). Validity of enthesis 71768 ultrasound assessment in spondyloarthropathy. Ann Rheum Dis, 68(2): 169-74. DeMaio M, Paine R, Mangine R, Drez D (1993). Plantar fasciitis. Sports Med 4218 Rehab Series, 16(10): 1153-62. Page 3 of 15 DeMaio M, Paine R, Mangine RE et al (1993). Plantar fasciitis. Orthopedics, 14528 16(10): 1153-63. Dorland I, Newman WA (1994). Spondylitis. DM Anderson, J Keith, PD Novak, 4219 MA Elliot (Eds). Dorland's Illustrated Medical Dictionary, 28th Edition,: 1563. WB Saunders, Philadelphia. Dougados M (1995). Diagnostic features of ankylosing spondylitis.
Load more

Recommended publications
  • Rotator Cuff Tendon Ruptures and Degeneration As the First Manifestation of Polymyalgia Rheumatica Disease - a Case Report
    Open Access Austin Journal of Clinical Case Reports Case Report Rotator Cuff Tendon Ruptures and Degeneration as the First Manifestation of Polymyalgia Rheumatica Disease - A Case Report Bazoukis G1*, Michelongona P2, Papadatos SS1, Pagkalidou E1, Grigoropoulou P1, Fragkou A1 and Abstract Yalouris A1 Polymyalgia Rheumatica (PMR) is a common rheumatic disease of the 1Department of Internal Medicine, General Hospital of elderly. Although it is a well-established disease, its causes and pathophysiology Athens “Elpis”, Greece remain unclear. In our case report we present an 83-year-old female presented 2Department of Internal Medicine, General Hospital of at the emergency department because of fever and diarrhea. Her medical Korinthos, Greece history included a recent orthopedic surgery because of tendons rupture of the *Corresponding author: George Bazoukis, rotator cuff. Her blood exams showed increased inflammatory markers and a Department of Internal Medicine, General Hospital of three-digit ESR. The diagnosis of PMR was set after the exclusion of infectious Athens “Elpis”, Greece and other diseases that mimic PMR symptoms. To the best of our knowledge, it is the first time that rotator cuff tendons rupture and degeneration is the first Received: June 05, 2016; Accepted: August 02, 2016; manifestation of PMR disease. Clinicians should be aware of the degeneration Published: September 08, 2016 of the shoulder and hip extra-articular structures in PMR and they should keep in mind that it can be the first manifestation of the disease. Keywords: Polymyalgia rheumatica; Rotator cuff denegeration; Tendon rupture Introduction and infraspinatus muscles as well as significant tendinopathy of the subscapularis and long head of biceps muscles.
    [Show full text]
  • Single Injection May Quell Chronic Plantar Fasciitis
    March 2006 • www.rheumatologynews.com Lupus/CT Diseases 27 Single Injection May Quell Chronic Plantar Fasciitis BY BRUCE JANCIN lief in nine such patients treated in open- try botulinum toxin A because of pub- greater than 4 on a 0-10 visual analog Denver Bureau label fashion. Based upon these highly en- lished reports citing its general analgesic scale. At week 2 this score was halved. At couraging results, a randomized, double- effect and inhibition of inflammatory pain. week 6 it was quartered. The same pattern V IENNA — Botulinum toxin A injection blind, and placebo-controlled clinical trial The nine patients selected for botu- of improvement was noted for maximum shows promise as a novel therapeutic op- is now planned, according to Dr. Placzek linum toxin A injection averaged age 55 pain during the previous 48 hours. tion for chronic plantar fasciitis patients of Charité Hospital, Berlin. years, with a 14-month history of plantar No muscle weakness or other adverse unresponsive to conventional treatments, Most patients with chronic plantar fasci- fasciitis refractory to all standard mea- events were observed, he noted at the Dr. Richard Placzek reported at the annual itis respond to physical therapy, cortico- sures. Follow-up evaluations were con- meeting sponsored by the European European Congress of Rheumatology. steroid injections, orthotics, acupuncture, ducted 2 weeks postinjection and every 1- League Against Rheumatism. Patients in- A single 200-unit injection into the and/or high-energy ultrasound extracor- 3 months thereafter up to 52 weeks. dicated they were satisfied with the pain painful region at the origin of the plantar poreal shock wave therapy.
    [Show full text]
  • The Painful Heel Comparative Study in Rheumatoid Arthritis, Ankylosing Spondylitis, Reiter's Syndrome, and Generalized Osteoarthrosis
    Ann Rheum Dis: first published as 10.1136/ard.36.4.343 on 1 August 1977. Downloaded from Annals of the Rheumatic Diseases, 1977, 36, 343-348 The painful heel Comparative study in rheumatoid arthritis, ankylosing spondylitis, Reiter's syndrome, and generalized osteoarthrosis J. C. GERSTER, T. L. VISCHER, A. BENNANI, AND G. H. FALLET From the Department of Medicine, Division of Rheumatology, University Hospital, Geneva, Switzerland SUMMARY This study presents the frequency of severe and mild talalgias in unselected, consecutive patients with rheumatoid arthritis, ankylosing spondylitis, Reiter's syndrome, and generalized osteoarthosis. Achilles tendinitis and plantar fasciitis caused a severe talalgia and they were observed mainly in males with Reiter's syndrome or ankylosing spondylitis. On the other hand, sub-Achilles bursitis more frequently affected women with rheumatoid arthritis and rarely gave rise to severe talalgias. The simple calcaneal spur was associated with generalized osteoarthrosis and its frequency increased with age. This condition was not related to talalgias. Finally, clinical and radiological involvement of the subtalar and midtarsal joints were observed mainly in rheumatoid arthritis and occasionally caused apes valgoplanus. copyright. A 'painful heel' syndrome occurs at times in patients psoriasis, urethritis, conjunctivitis, or enterocolitis. with inflammatory rheumatic disease or osteo- The antigen HLA B27 was present in 29 patients arthrosis, causing significant clinical problems. Very (80%O). few studies have investigated the frequency and characteristics of this syndrome. Therefore we have RS 16 PATIENTS studied unselected groups of patients with rheuma- All of our patients had the complete triad (non- toid arthritis (RA), ankylosing spondylitis (AS), gonococcal urethritis, arthritis, and conjunctivitis).
    [Show full text]
  • REPETITIVE STRAIN INJURY AWARENESS Tenosynovitis
    REPETITIVE STRAIN INJURY RSIA AWARENESS Tenosynovitis RSI Conditions The term Repetitive Strain Injury is an umbrella term used to describe a number of specific musculoskeletal conditions, including tenosynovitis, as well as ‘diffuse RSI’, which is more difficult to define but which recent research attributes to nerve damage. These conditions are often occupational in origin. Lack of adequate diagnosis or access to appropriate treatment can exacerbate the condition and sometimes leads to job loss and economic hardship. What is Tenosynovitis? Tenosynovitis is the tender swelling of the rope or cord like structures (tendons) which connect muscles to the bones in order to work the joints of the body, and inflammation of the lining of the protective synovial sheath that covers these tendons. Areas most frequently affected are the hand, wrist or arms, although it may occur at any tendon site. De Quervain’s or Stenosing Tenosynovitis results from inflammation or constriction of the tendons on the thumb side of the wrist. A localised swelling affecting the flexor tendons of the hand is known as Trigger Finger. The Symptoms When the gliding surfaces of the tendon and sheath become roughened and inflamed from overuse, tenosynovitis will present as aching, tenderness and swelling of the affected area. There may also be also stiffness of the joint, shooting pains up the arm and creaking tendons (crepitus). The ability to grip can be lost. A localised swelling at the base of the thumb may indicate De Quervain’s. Tenosynovitis can just last a few days, but in some cases may go on for many weeks or even months.
    [Show full text]
  • Palindromic Rheumatism Or When Do You Decide to Treat an Asymptomatic Seropositive RA Patient? What Is This?
    Palindromic Rheumatism or When do you decide to treat an asymptomatic seropositive RA patient? What Is This? • 11.15.15 • 56 yo man comes for 2nd opinion for bouts of severe large joint monoarthritis lasting 24 hours or longer. • Vague about duration “10-15 years.” Had wrist synovectomy 2005 after “trauma.” • Saw rheumatologist 2012: ACPA>500, RF 60. • Loss of shoulder motion in all planes. • At the conclusion of this presentation, the participant should be able to: – Appreciate the relationship of Palindromic Rheumatism (PR) and progression to RA – Understand the biology of intercritical PR – Define the utility of prevention strategies – Comprehend the yield of imaging in PR and how it informs PR pathophysiology • Should we try to prevent? How? Annual transition to RA is greater than 15% in which of the following ACPA+ pts? A. Arthralgia B. Arthralgia + Imaging + CRP C. Palindromic Rheumatism D. Asymptomatic Twin E. Interstitial Lung Disease Rheumatoid Arthritis Pathogenesis Tolerance broken-AutoAb appear Adaptive Immune Response Locus and Trigger? Systemic Nature? “Amplification” Synovial Targeting with variable kinetics? Innate vs Adaptive Immunity? Joint Targeting ACPA-IC deposit or are formed de novo in joint? Or something else? Tissue Injury Rheumatoid Arthritis Persistence of the Systemic Trigger? Systemic autoimmunity & inflammation T cells/B Cells Immune Complexes TNF, IL-6, GM-CSF No treatment shown to eliminate systemic process Where does MTX work? Joint Inflammation MF, FLS, Cartilage, Bone RA Centers in Synovium, Destroying All Around It? Why Is Palindromic Rheumatism Palindromic? Systemic inflammation Followed by resolution? e.g. like gout? Why does it resolve? Why does it stop resolving? Single Joint Inflammation Palindromic Rheumatism (PR) • How frequent is PR as an initial presentation of RA? • What is the mechanism of PR? • Is synovitis present during its intercritical phase? • What is the frequency of progression to RA in 5 years? Treatment? Is Palindromic Rheumatism a Common Presentation? Frequency relative to new onset RA is: A.
    [Show full text]
  • Eosinophilic Fasciitis (Shulman Syndrome)
    CONTINUING MEDICAL EDUCATION Eosinophilic Fasciitis (Shulman Syndrome) Sueli Carneiro, MD, PhD; Arles Brotas, MD; Fabrício Lamy, MD; Flávia Lisboa, MD; Eduardo Lago, MD; David Azulay, MD; Tulia Cuzzi, MD, PhD; Marcia Ramos-e-Silva, MD, PhD GOAL To understand eosinophilic fasciitis to better manage patients with the condition OBJECTIVES Upon completion of this activity, dermatologists and general practitioners should be able to: 1. Recognize the clinical presentation of eosinophilic fasciitis. 2. Discuss the histologic findings in patients with eosinophilic fasciitis. 3. Differentiate eosinophilic fasciitis from similarly presenting conditions. CME Test on page 215. This article has been peer reviewed and is accredited by the ACCME to provide continuing approved by Michael Fisher, MD, Professor of medical education for physicians. Medicine, Albert Einstein College of Medicine. Albert Einstein College of Medicine designates Review date: March 2005. this educational activity for a maximum of 1 This activity has been planned and implemented category 1 credit toward the AMA Physician’s in accordance with the Essential Areas and Policies Recognition Award. Each physician should of the Accreditation Council for Continuing Medical claim only that credit that he/she actually spent Education through the joint sponsorship of Albert in the activity. Einstein College of Medicine and Quadrant This activity has been planned and produced in HealthCom, Inc. Albert Einstein College of Medicine accordance with ACCME Essentials. Drs. Carneiro, Brotas, Lamy, Lisboa, Lago, Azulay, Cuzzi, and Ramos-e-Silva report no conflict of interest. The authors report no discussion of off-label use. Dr. Fisher reports no conflict of interest. We present a case of eosinophilic fasciitis, or Shulman syndrome apart from all other sclero- Shulman syndrome, in a 35-year-old man and dermiform states.
    [Show full text]
  • Tendon Lesions and Soft Tissue Rheumatism-Great Outback Or Great Opportunity?
    Annals ofthe Rheumatic Diseases 1996; 55: 1-3 ARD Ann Rheum Dis: first published as 10.1136/ard.55.1.1 on 1 January 1996. Downloaded from Annals of the Rheumatic Diseases Leader Tendon lesions and soft tissue rheumatism-great outback or great opportunity? In 1979, Dixon described soft tissue rheumatism as 'the This group of diseases make up a high proportion of great outback ofRheumatology, a vastfrontier land, iUl-defined rheumatological practice in the UK, but traditionally little and little explained, itsfeatures poorly categorised andfarfrom effort has been made to understand their underlying internationally agreed.' Has much changed in the past 15 pathology. There are a number of reasons for this. Soft years? tissue lesions are not life threatening, unlike the much rarer immunological and inflammatory rheumatological diseases. Soft tissues are rarely biopsied, so samples are difficult Soft tissue rheumatism in 1995 to obtain and current animal models do not necessarily Musculoskeletal symptoms without frank arthritis are very reflect the chronic degenerative lesions found in aging common. Most of us suffer such symptoms each year. patients. However, while there is lack of pathological They usually occur in one region, in an individual who is information on many of these lesions, there are usually otherwise well. These regional musculoskeletal disorders, adequate clinical features to allow identification of which have commonly been overlooked in the planning individual conditions. and provision of health care, are disorders of major and Perhaps the biggest hindrance to progress in our increasing importance. understanding of soft tissue rheumatism has been a Soft tissue rheumatism accounts for up to 25% of negative attitude to the affected tissues, which are still http://ard.bmj.com/ all hospital consultations for the rheumatic disorders.' thought of as inert, homogeneous structures.
    [Show full text]
  • Foot Pain & Psoriatic Arthritis
    WHEATON • ROCKVILLE • CHEVY CHASE • WASHINGTON, DC Foot Pain & Psoriatic Arthritis Daniel El-Bogdadi, MD, FACR Arthritis and Rheumatism Associates, P.C. Do you feel heel pain in the Plantar fasciitis ARTHRITIS morning or after a period of can be caused by inactivity? This might be an a number AND indication of plantar fasciitis that of factors, RHEUMATISM could be part of an underlying including aerobic ASSOCIATES, P.C. psoriatic arthritis condition. dance exercise, running, and Board Certified Rheumatologists Psoriatic arthritis is an inflammatory ballet. arthritis that typically causes pain, Herbert S.B. Baraf MD FACP MACR swelling and stiffness in the Robert L. Rosenberg peripheral joints or spine. However, MD FACR CCD if you have psoriatic arthritis, you Evan L. Siegel may notice that not only are the MD FACR joints and spine involved, but Emma DiIorio symptoms may also occur in the soft MD FACR tissue such as tendons or ligaments. David G. Borenstein MD MACP MACR This usually happens in a place Alan K. Matsumoto where tendons and ligaments attach There are several treatment options MD FACP FACR to bone, known as the enthesis. for plantar fasciitis: David P. Wolfe When this attachment gets inflamed MD FACR it is called enthesitis. • The first immediate intervention is Paul J. DeMarco MD FACP FACR to decrease or stop any Shari B. Diamond One of the more common areas to repetitive activities, such as MD FACP FACR get enthesitis is in the Achilles running or dancing, which may Ashley D. Beall tendon. Another common area for aggravate the condition. MD FACR inflammation is in thick band of (over) Angus B.
    [Show full text]
  • PALINDROMIC RHEUMATISM by STEPHEN MATTINGLY Department of Rheumatology and Physical Medicine, the Middlesex Hospital
    Ann Rheum Dis: first published as 10.1136/ard.25.4.307 on 1 July 1966. Downloaded from Ann. rheum. Dis. (1966), 25, 307. PALINDROMIC RHEUMATISM BY STEPHEN MATTINGLY Department of Rheumatology and Physical Medicine, The Middlesex Hospital Palindromic rheumatism was first described by Painful non-pitting tender swellings an inch or more Hench and Rosenberg in 1941, and three years later a in diameter, and occasionally much larger, appeared detailed account of their 34 cases appeared in the over the forearms, back of wrist, or heel. Some- Archives of Internal Medicine (Hench and Rosen- times the finger tips became swollen and transient berg, 1944). A number of individual case reports intra-cutaneous or subcutaneous nodules were subsequently appeared in the literature (Ameen, observed on the hands, but usually disappeared 1954; Cain, 1944; Ferry, 1943; Ginsburg, 1948; within a few days. Grego and Harkins, 1944; Gryboski, 1948; Hopkins Patients suffering from palindromic rheumatism and Richmond, 1947; Lewitus, 1954; Mazer, 1942; remained well and did not lose weight. Attacks were Neligan, 1946; Paul and Logan, 1944; Paul and usually afebrile. Radiographs were normal in most Carr, 1945; Perl, 1947; Rotes Querol, 1956; Salo- cases or showed coincidental degenerative changes. mon, 1946; Scheinberg, 1947; Thompson, 1942; Laboratory investigations usually gave normal Parkes Weber, 1946; Wingfield, 1945; Wirtschafter, results, although there was sometimes a transient Williams, and Gaulden, 1955; Wolfson and Alter, rise in the erythrocyte sedimentation rate during an 1948; Wassmann, 1950; Zentner, 1953). However, attack, the white cell count might show a relative there have been few reports of series of patients with lymphocytosis, and the serum fatty acids were in- this syndrome (Ansell and Bywaters, 1959; Dames creased in some patients.
    [Show full text]
  • Palindromic Rheumatism Information Booklet (PDF)
    Palindromic rheumatism Palindromic rheumatism information booklet Contents Zoe’s story 4 What is palindromic rheumatism? 6 Symptoms 6 Causes 8 Diagnosis 10 How will palindromic rheumatism affect me? 11 Treatment 13 Managing symptoms 16 Living with palindromic rheumatism 20 Research and new developments 22 Glossary 22 We’re the 10 million people living with arthritis. We’re the carers, researchers, health professionals, friends and parents all united Useful addresses 25 in our ambition to ensure that one day, no one will have to live Where can I find out more? 26 with the pain, fatigue and isolation that arthritis causes. Talk to us 27 We understand that every day is different. We know that what works for one person may not help someone else. Our information is a collaboration of experiences, research and facts. We aim to give you everything you need to know about your condition, the treatments available and the many options you can try, so you can make the best and most informed choices for your lifestyle. We’re always happy to hear from you whether it’s with feedback on our information, to share your story, or just to find out more about the work of Versus Arthritis. Contact us at [email protected] Registered office: Versus Arthritis, Copeman House, St Mary’s Gate, Chesterfield S41 7TD Words shown in bold are explained in the glossary on p.22. Registered Charity England and Wales No. 207711, Scotland No. SC041156. Page 2 of 28 Page 3 of 28 When I went back after being on naproxen and hydroxychloroquine for four weeks, I felt the best I’d ever felt, and I thought perhaps he was going to discharge me.
    [Show full text]
  • Hypermobility, the Ehlers-Danlos Syndromes and Chronic Pain D
    Hypermobility, the Ehlers-Danlos syndromes and chronic pain D. Syx, I. De Wandele, L. Rombaut, F. Malfait Center for Medical Genetics, ABSTRACT the nature and mediators of chronic Ghent University and Ghent Chronic widespread pain is a common pain are needed in order to potentially University Hospital, Ghent, Belgium. complaint among individuals affected identify novel targets for therapeutic in- Delfien Syx, PhD by generalised joint hypermobility. In tervention and optimise treatment. Inge De Wandele, PT, PhD the absence of other conditions that Lies Rombaut, PT, PhD cause chronic pain, these individuals Hypermobility, joint hypermobility Fransiska Malfait, MD, PhD are usually diagnosed with joint hyper- syndrome and Ehlers-Danlos Please address correspondence to: mobility syndrome (JHS). JHS is a mul- syndromes Dr Fransiska Malfait, Center for Medical Genetics, tifactorial trait with a strong genetic Joint hypermobility (JH) implies a Ghent University Hospital, basis, but no specific genetic markers. range of joint movement that exceeds De Pintelaan 185, Clinical overlap of JHS is seen with what is considered to be normal for that 9000 Ghent, Belgium. heritable connective tissue disorders, joint, taking into account the individu- E-mail: [email protected] particularly with the Ehlers-Danlos al’s age, gender and ethnicity. JH can be Received and accepted on September 8, syndrome, hypermobile type (hEDS). limited to one or a few joints (localised 2017. The Ehlers-Danlos syndromes (EDS) JH), but when present at multiple sites Clin Exp Rheumatol 2017; 35 (Suppl. 107): comprise a heterogeneous group of rare the term generalised JH (GJH) is pre- S116-S122. monogenic conditions that are charac- ferred (1).
    [Show full text]
  • 2010 Rheumatoid Arthritis Classification Criteria
    ARTHRITIS & RHEUMATISM Vol. 62, No. 9, September 2010, pp 2569–2581 DOI 10.1002/art.27584 © 2010, American College of Rheumatology Arthritis & Rheumatism An Official Journal of the American College of Rheumatology www.arthritisrheum.org and www.interscience.wiley.com 2010 Rheumatoid Arthritis Classification Criteria An American College of Rheumatology/European League Against Rheumatism Collaborative Initiative Daniel Aletaha,1 Tuhina Neogi,2 Alan J. Silman,3 Julia Funovits,1 David T. Felson,2 Clifton O. Bingham, III,4 Neal S. Birnbaum,5 Gerd R. Burmester,6 Vivian P. Bykerk,7 Marc D. Cohen,8 Bernard Combe,9 Karen H. Costenbader,10 Maxime Dougados,11 Paul Emery,12 Gianfranco Ferraccioli,13 Johanna M. W. Hazes,14 Kathryn Hobbs,15 Tom W. J. Huizinga,16 Arthur Kavanaugh,17 Jonathan Kay,18 Tore K. Kvien,19 Timothy Laing,20 Philip Mease,21 Henri A. Ménard,22 Larry W. Moreland,23 Raymond L. Naden,24 Theodore Pincus,25 Josef S. Smolen,1 Ewa Stanislawska-Biernat,26 Deborah Symmons,27 Paul P. Tak,28 Katherine S. Upchurch,18 Jirˇí Vencovsky´,29 Frederick Wolfe,30 and Gillian Hawker31 This criteria set has been approved by the American College of Rheumatology (ACR) Board of Directors and the Euro- pean League Against Rheumatism (EULAR) Executive Committee. This signifies that the criteria set has been quantita- tively validated using patient data, and it has undergone validation based on an external data set. All ACR/EULAR- approved criteria sets are expected to undergo intermittent updates. The American College of Rheumatology is an independent, professional, medical and scientific society which does not guarantee, warrant, or endorse any commercial product or service.
    [Show full text]