DOCSLIB.ORG

Hoogleraar Neuropsychologie

Total Page：16

File Type：pdf, Size：1020Kb

Download full-text PDF Read full-text
Hoogleraar Neuropsychologie Curriculum vitae Prof. dr. Jelle Jolles Hoogleraar Neuropsychologie NeuroPsy Amsterdam [email protected] Vrije Universiteit Amsterdam [email protected] 5 oktober 2018 2 Curriculum vitae Prof. dr. Jelle Jolles, Hoogleraar Neuropsychologie NeuroPsy Vossiusstraat 30-H 1071 AG Amsterdam Faculteit Gedrags- en Bewegingswetenschappen, Vrije Universiteit Amsterdam Van der Boechorststraat 7-9 1081 BT Amsterdam E-mail: [email protected], [email protected] Telefoon: +31 629596171 Twitter: @jellejolles Websites: www.jellejolles.nl, www.neuropsy-academie.nl, www.neuropsych-publishers, www.hersenenenleren.nl Datum: 5 oktober 2018 3 © J. Jolles / Neuropsych Publishers, Amsterdam 2018 Alle rechten voorbehouden. Niets uit deze uitgave mag worden verveelvoudigd, opgeslagen in een geautomatiseerd gegevensbestand, of openbaar gemaakt, in enige vorm of op enige wijze, hetzij elektronisch, mechanisch, door fotokopieën, opnamen of enige andere manier, zonder voorafgaande schriftelijke toestemming van de uitgever. Voor zover het maken van kopieën uit deze uitgave is toegestaan op grond van artikel 16B Auteurswet 1912 jº het Besluit van 20 juni 1974, Stb. 351, zoals gewijzigd bij het Besluit van 23 augustus 1985, Stb. 471 en artikel 17 Auteurswet 1912, dient men de daarvoor wettelijk verschuldigde vergoedingen te voldoen aan de Stichting Reprorecht (Postbus 3051, 2130 KB Hoofddorp). Voor het overnemen van gedeelte(n) uit deze uitgave in bloemlezingen, readers en andere compilatiewerken (artikel 16 Auteurswet 1912) dient men zich tot de uitgever te wenden. 4 Samenvatting Jelle Jolles is hoogleraar Neuropsychologie aan de Vrije Universiteit Amsterdam. Hij is oprichter van het Centrum Brein & Leren en verantwoordelijk voor de adviespraktijk NeuroPsy. Het is zijn doel om onderwijs en opvoeding te laten profiteren van de inzichten die de laatste jaren zijn verkregen over de hersenen, en over het cognitief en neuropsychologisch functioneren. Zijn huidige activiteiten richten zich vooral op de relatie tussen leren, brein en ontplooiing bij kinderen, tieners en jong-volwassenen en de factoren die daar bepalend voor zijn. Jolles pleit voor een actieve dialoog tussen wetenschap en de praktijk met als doel het verbeteren van de ontplooiingsmogelijkheden van jeugdigen en op levenlang leren.In 2011 verscheen zijn boek ‘Ellis en het verbreinen’ en in 2016 zijn bestseller ‘Het tienerbrein: Over de adolescent tussen biologie en omgeving’. Jolles is opgeleid in twee richtingen: in de gedragswetenschappen (psychologie/neuropsychologie) en in de hersenwetenschappen (neurochemie/neurobiologie). Hij is sinds 1982 klinisch neuropsycholoog. Sedertdien is hij werkzaam geweest op het grensvlak van wetenschap en toepassing in de praktijk. Als wetenschapper en praktijkprofessional werkte hij vanaf zijn promotie in 1980in de gezondheidszorg voor patiënten met een hersenstoornis en voor ouderen met cognitieve problemen. Vanaf 1995 richtte hij zich ook op kinderen en jeugdigen met functiestoornissen (leerproblemen en ADHD) en op het brede gebied van het onderwijs. Hij was verantwoordelijk voor het opzetten van de Maastrichtse Geheugenpolikliniek, voor de Leerpoli voor kinderen in Maastricht en voor het Centrum Brein & Leren in Maastricht en in Amsterdam. In de periode tussen 1980 en 2018 heeft Jolles veel functies en taken bekleed in wetenschappelijke organisaties en in de gezondheidszorg. Hij heeft veel functies gehad als bestuurslid of bestuursvoorzitter van wetenschappelijke verenigingen en vakverenigingen. Zo was hij voorzitter van de landelijke stuurgroep ‘Hersenen en leren’ van NWO en OCW. Tussen 2010 en 2017 was hij landelijk coördinator van het grote ontwikkelingsprogramma ‘Leren’ onder de koepel van het Nationaal Initiatief Hersenen en cognitie (NIHC) van NWO. Biografie: http://www.jellejolles.nl/korte-biografie/ Nadere informatie: websites jellejolles.nl, neuropsy-academie.nl, neuropsych-publishers.nl Overzicht van wetenschappelijke output Aantal promoties/proefschriften: 81 Hirsch factor: 101 (Scolar.Google 5/10/2018) Aantal internationale wetenschappelijke artikelen: 600 Aantal wetenschappelijke en vakpublicaties in het nederlands: 194 Aantal interviews gepubliceerd in vakbladen en publieksmedia: 271 (v.a. 2000) Bijdragen aan kranten/tijdschriften: 104 (v.a. 2000) Output in sociale media: 150 (v.a. 2003) Bijdragen aan radio/TV: 24 (v.a. 2010) Rapporten en diversen: 89 (v.a. 2003) Lezingen: 263 (v.a. 2008) 5 6 Inhoudsopgave 1. Het curriculum vitae in grote lijnen 9 Het curriculum vitae in grote lijnen 11 Aanstellingen 12 2. Bestuur en management 14 Bestuur en science management 15 Bestuur en management gezondheidszorginstellingen 18 Bestuur wetenschappelijke en maatschappelijke organisties en boards 19 Lidmaatschappen 20 Symposium- en congresorganisatie 21 3. Verworven subsidies 23 4. Onderwijs 29 5. Wetenschappelijke proefschriften 33 6. Wetenschappelijke publicaties (SCI/SSCI) en boeken/hoofdstukken 41 7. Nederlandstalige publicaties 81 8. Interviews in vaktijdschriften en publieksmedia 95 9. Andere producten 109 Bijdragen aan artikelen in vaktijdschriften en publieksmedia 111 Bijdragen aan radio- en televisieprogramma’s 117 Columns, weblogs en andere bijdragen aan sociale media 119 Rapporten en diversen 131 10. Keynote lezingen en workshops 137 7 8 Hoofdstuk 1 Het curriculum vitae in grote lijnen 9 10 1.1.Het curriculum vitae in grote lijnen Den Haag, Utrecht 1949 Geboren in Den Haag (10 April) 1968 Diploma Gymnasium Haganum, Den Haag 1972 Kandidaatsexamen Scheikunde (Biochemie; Universiteit Utrecht) 1974 Kandidaatsexamen Psychologie (Universiteit Utrecht) 1974 Kandidaatsexamen Filosofie (‘Verkort kandidaatsexamen’; Universiteit Utrecht) 1975 Doctoraalexamen Scheikunde, Cum Laude (Biochemie en Neurochemie; Universiteit Utrecht) 1976 Getrouwd, twee kinderen (Mei 1982, February1985) 1977 Doctoraalexamen Psychologie (Cognitieve Psychologie en Neuropsychologie; Universiteit Utrecht) 1980 Wetenschappelijke promotie op het proefschrift ‘Neuropeptides, Brain Membrane Phosphorylation and Grooming Behaviour’ (Prof. W.H. Gispen and Prof. D. de Wied, promotores; Universiteit Utrecht) 1983 Registratie als Klinisch Psycholoog (NIP, Nederlands Instituut van Psychologen) Maastricht 1985-2009 Hoogleraar Neuropsychologie en Psychobiologie (Faculteit Geneeskunde, Universiteit Maastricht) 1986-1995 Themaleider ‘Veroudering’, Universiteit Maastricht 1995-2009 Hoogleraar Neuropsychologie en Biologische Psychologie (Faculteit Psychologie, Universiteit Maastricht) 1995-2001 Directeur Instituut Hersenen & Gedrag, Universiteit Maastricht 1999 Registratie als Gezondheidszorgpsycholoog BIG (Wet Beroepen Individuele Gezondheidszorg). 2002-2006 Voorzitter landelijke commissie Hersenen en Leren, NWO en OCW 2005 Registratie als Klinisch Psycholoog BIG 2008 Registratie als Klinisch Neuropsycholoog BIG 2006-heden Directeur Centrum Brein & Leren (Maastricht, Amsterdam) Amsterdam 2009-heden Hoogleraar Neuropsychologie en Educational Neuropsychology (Vrije Universiteit Amsterdam) 2009-2013 Wetenschappelijk directeur instituut LEARN!, onderzoeksinstituut op gebied van educatie (Vrije Universiteit Amsterdam) 2009-2016 Landelijk coördinator van koepelprogramma LEREN dat berust onder het Nationaal Initiatief Hersenen en Cognitie (NIHC) van NWO 2011 Toekenning Societal Impact Award (Vrije Universiteit Amsterdam) 2013 Benoeming tot Universiteitshoogleraar Neuropsychologie (Vrije Universiteit Amsterdam) 2018 Emeritaat 2018 Uitbouw van NeuroPsy, adviespraktijk t.a.v. Leren, brein & onderwijs 11 1.2. Aanstellingen Utrecht 1975 Wetenschappelijk medewerker; Rudolf Magnus Instituutvoor Farmacologie, Utrecht. 1980 Wetenschappelijk ambtenaar I; Psychiatrische Universiteitskliniek, Utrecht. 1983 Wetenschappelijk hoofdambtenaar; Psychiatrische Universiteitskliniek, Utrecht. Maastricht 1985 Hoogleraar Neuropsychologie & Psychobiologie (Universiteit Maastricht, Faculteit Geneeskunde, Maastricht). 1985 Hoofd van de Academische Afdeling Neuropsychologie; Psychomedisch Centrum Vijverdal, Maastricht 1986 Directeur, Onderzoeksthema Veroudering Maastricht (tot en met December 1994) 1987 Hoofd van de Divisie Neuropsychologie; Afdeling Psychiatrie & Neuropsychologie; Academisch Ziekenhuis Maastricht. 1991-1992 Hoofd ad interim van de Afdeling Psychiatrie; Academisch Ziekenhuis Maastricht 1995-2001 Directeur, Onderzoeksinstituut Hersenen & Gedrag Maastricht 1997-2008 Directeur, Divisie Cognitieve Stoornissen, instituut Hersenen & Gedrag Maastricht 2000-2008 Directeur van het Alzheimer Centrum Limburg 2006-2018 Directeur of the Centrum Brein & Leren Maastricht Amsterdam 2009-2013 Hoofd, afdeling Onderwijsneurowetenschappen, Faculteit Psychologie en Pedagogiek, Vrije Universiteit Amsterdam 2009-2018 Hoogleraar Hersenen, gedrag en educatie (Educational Neuropsychology), Vrije Universiteit Amsterdam (Faculteit Psychologie en Pedagogiek) 2009-2013 Directeur, LEARN!, onderzoeksinstituut op het gebied van Educatie. 2009 Directeur van Centrum Brein en Leren Amsterdam 2013-2018 Hoofd, afdeling Universiteitshoogleraar ‘Brein, leren en educatie’. 2018 Emeritaat (1 januari 2018) 12 Hoofdstuk 2 Bestuur en management 13 14 2.1. Bestuur en science management Science management in verband van NWO en verwante organisaties 1986-1998 Lid van de commissie Psychogeriatrische Ziektebeelden van de Gezondheidsraad. 1986-2015 Evaluator van projectaanvragen, programma-aanvragen en VENI/VIDI/VICI beurzen 1985-1987 Lid van het ‘Alzheimer-Committee’, NWO Medische wetenschappen 1987-1991 Lid van de commissie Neurowetenschappen van NWO/Medische Wetenschappen
Load more

Recommended publications
  • What Is the Relationship Between Child Nutrition and School Outcomes?
    WIDER BENEFITS OF LEARNING RESEARCH REPORT NO.18 What is the relationship between child nutrition and school outcomes? In recent times, the diet of children has risen to the top of the political agenda, not only for the WIDER BENEFITS OF LEARNING RESEARCH REPORT NO.18 potential health repercussions later in life, but also for its immediate effects on the physical and mental health of children and their consequent school experience and attainment. In this report we review the literature on the relationship between aspects of nutrition and physical health, mental health, behavioural and social outcomes in children. Our research attempts to address the following questions: • How does nutrition impact upon healthy outcomes in children? • How can the health outcomes that manifest as a result of nutrition impact upon school life experiences and outcomes? We find that the early development of preferences for foods of poor nutritional value can have long-term health implications. Ultimately, the aim must be to prevent nutritional deficiencies from What is the relationship between arising but the relationships between nutrition, health, education and social and cultural factors are complex and multi-directional. There is evidence that appropriately designed interventions can help to address early deficiencies and engage both children and parents in healthy eating. child nutrition and school outcomes? Given that the diet of children depends not only on the availability of foods but, crucially, on their preferences, any effective interventions must address the multiple determinants of children’s preferences for particular foods. In particular, the role of parents is important and there may be a need to adopt a collaborative approach between schools and parents to address children’s nutritional choices.
    [Show full text]
  • Activity of Selected Group of Monoterpenes in Alzheimer's
    International Journal of Molecular Sciences Review Activity of Selected Group of Monoterpenes in Alzheimer’s Disease Symptoms in Experimental Model Studies—A Non-Systematic Review Karolina Wojtunik-Kulesza 1,*, Monika Rudkowska 2, Kamila Kasprzak-Drozd 1,* , Anna Oniszczuk 1 and Kinga Borowicz-Reutt 2 1 Department of Inorganic Chemistry, Medical University of Lublin, Chod´zki4a, 20-093 Lublin, Poland; [email protected] 2 Independent Experimental Neuropathophysiology Unit, Medical University of Lublin, Jaczewskiego 8b, 20-090 Lublin, Poland; [email protected] (M.R.); [email protected] (K.B.-R.) * Correspondence: [email protected] (K.W.-K.); [email protected] (K.K.-D.) Abstract: Alzheimer’s disease (AD) is the leading cause of dementia and cognitive function im- pairment. The multi-faced character of AD requires new drug solutions based on substances that incorporate a wide range of activities. Antioxidants, AChE/BChE inhibitors, BACE1, or anti-amyloid platelet aggregation substances are most desirable because they improve cognition with minimal side effects. Plant secondary metabolites, used in traditional medicine and pharmacy, are promising. Among these are the monoterpenes—low-molecular compounds with anti-inflammatory, antioxidant, Citation: Wojtunik-Kulesza, K.; enzyme inhibitory, analgesic, sedative, as well as other biological properties. The presented review Rudkowska, M.; Kasprzak-Drozd, K.; focuses on the pathophysiology of AD and a selected group of anti-neurodegenerative monoterpenes Oniszczuk, A.; Borowicz-Reutt, K. and monoterpenoids for which possible mechanisms of action have been explained. The main body Activity of Selected Group of of the article focuses on monoterpenes that have shown improved memory and learning, anxiolytic Monoterpenes in Alzheimer’s and sleep-regulating effects as determined by in vitro and in silico tests—followed by validation in Disease Symptoms in Experimental in vivo models.
    [Show full text]
  • The Essentials of a Global Index for Cognitive Function
    Mini-Review • DOI: 10.1515/tnsci-2017-0014 • Translational Neuroscience • 8 • 2017 • 87-96 Translational Neuroscience Joseph Mathew Antony1*, Ian Weaver2, THE ESSENTIALS OF A GLOBAL Matthew Rueffer3, Najla Guthrie1, INDEX FOR COGNITIVE FUNCTION Malkanthi Evans1 Abstract 1KGK Science Inc. 1440, One London Place, Cognition is comprised of the faculties: perception, creativity, intuition, and ratiocination. Optimal levels of London, ON, N6A 5R8, Canada cognition are needed for independent functioning and balanced living. With an aging population that continues 2Department of Psychology and Neuroscience, to grow, dietary supplements that tilt the balance towards maintenance of cognition are being marketed for Dalhousie University, Halifax, NS B3H 4R2, Canada vulnerable populations facing these challenges. Randomized clinical trials provide the causal inference necessary 3Robarts Clinical Trials, 100 Dundas St, to define the efficacy of emerging nutraceuticals. Cognition testing, in particular, requires a battery of tests that London, ON N6A 5B6 encompass all brain regions involved in cognition so as to provide endpoints necessary for product validation. The lack of well controlled studies for comparison analyses, limited sample sizes, ambiguous dosages, and poor cognitive measures result in data that cannot be compared across studies to determine the efficacy of supplements claiming to enhance cognition. Clinical trials for the nutraceutical industry should consider the multifaceted nature of supplements, where clinical endpoints must be comprehensive while remaining feasible. Combining endpoints of cognition with physiological biomarkers of immunity and metabolism to arrive at a global index for cognitive health may be necessary for claim substantiation in order to fully justify and scientifically validate improvements in cognitive health. The issues and needs of a global index will be discussed here.
    [Show full text]
  • Nutrition and Cognition in Older Persons
    Nutrition and Aging: I.H. Rosenberg; A. Sastre (eds), Nestle´ Nutrition Workshop Series Clinical & Performance Program, Vol. 6, pp. 121–134, Nestec Ltd.; Vevey/S. Karger AG, Basel, 2002. Nutrition and Cognition in Older Persons John E. Morley GRECC, St. Louis VA Medical Center, and Division of Geriatric Medicine, Saint Louis University, St. Louis, Mo., USA The concept that ‘we are what we eat’ is by no means a new one and appears to be particularly true when one examines the effect of food intake on cognitive function. Even in the case of mild protein energy malnutrition, poor nutritional state is associated with a decline in cognitive function [1]. Food intake, or the lack thereof, can modulate thought processes in multiple ways (Table 1). This chapter will focus on a number of mechanisms that appear to play a role in modulating cognition in older persons. Modulation of Memory by Gastrointestinal Peptide Release Cholecystokinin (CCK) has been shown to enhance memory retention [2]. It produces this effect by stimulating ascending fibers in the vagus nerve (Fig. 1). These then send messages to the nucleus tractus solitarius and from there to the amygdala and the hippocampus [3]. When mice are fed immediately after learning a task, they demonstrate improved memory [2]. This enhanced retention can be abolished by utilizing a CCK antagonist. Circulating CCK levels are elevated in older persons and increase to a greater degree following fat administration [4]. CCK levels are elevated to a greater degree in malnourished compared to healthy older individuals [5]. Food intake improves cognition in humans [6].
    [Show full text]
  • Dementia: Advances and Treatment
    Sarubbo F Miralles A Moranta D Esteban S DEMENTIA: ADVANCES AND TREATMENT Dementia: Advances and Treatment Chapter 1 Dietary polyphenols as promising molecules to prevent dementia Sarubbo F*; Miralles A; Moranta D; Esteban S *Laboratorio de Neurofisiología, Departamento de Biología, Instituto Universitario de Investi- gación en Ciencias de la Salud, Universidad de las Islas Baleares (UIB), Mallorca, Spain. Correspondence to: Sarubbo F, Laboratorio de Neurofisiología, Departamento de Biología, Instituto Universitario de Investigación en Ciencias de la Salud, Universidad de las Islas Baleares (UIB), Mallorca, Spain. Email:[email protected]; Tel: +34 971 173145; Fax: +34 971 173184 Abstract Due to the increased number of elderly people worldwide, nowadays one of the major medical and socio-economic challenges is to search strategies to combat the consequences of aging process, reducing the incidence of neurodegenerative dis- eases such as dementia. Dementia is a clinical syndrome of chronic and progressive symptoms characterized by multiple cognitive deficits associated with aging, which includes impairment in memory and in other cognitive functions to the extent that it interferes with daily function. In the last years oxidative stress and inflammation have been pointed out as the leading causes of brain aging and neurodegeneration. Therefore,an approach for preventing some brain age-related diseases, such as de- mentia, may be the consumption or administration of polyphenols,which are natural compounds present in edible plants. Due to their antioxidant and anti-inflammatory properties, polyphenolshave been suggested such as a beneficial strategy against the development of brain aging and neurodegeneration. This chapter summarizes the latest discoveries regarding how polyphenols exert positive effects combating the biochemical mechanisms that originate aging and dementia,such as oxidative stress, inflammation and the aggregation of abnormal folding proteins, among oth- ers.
    [Show full text]
  • World Alzheimer Report 2014 Dementia and Risk Reduction an ANALYSIS of PROTECTIVE and MODIFIABLE FACTORS
    World Alzheimer Report 2014 Dementia and Risk Reduction AN ANALYSIS OF PROTECTIVE AND MODIFIABLE FACTORS SUPPORTED BY Authors Chapter 1 Prof Martin Prince Dr Maëlenn Guerchet The Global Observatory for Ageing and Dementia Care, King’s College Prof Emiliano Albanese London Chapter 2 Prof Emiliano Albanese 1 University of Geneva, Switzerland Richard Pender Dr Maëlenn Guerchet *2 Dr Maëlenn Guerchet 1,2 The Global Observatory for Ageing and Dementia Care, King’s College Prof Martin Prince London 1 Education, Occupation; 2 Dr Matthew Prina Leg length and head circumference, early-life events The Global Observatory for Ageing and Dementia Care, King’s College London Chapter 3 Dr Matthew Prina 1,2 Richard Pender 2 Contributors Dr Cleusa Ferri 3 Richard Pender Dr Diego R. Mazzotti 3 The Global Observatory for Ageing and Dementia Care, King’s College 4 London Prof Emiliano Albanese 1 Depression; 2 Anxiety disorders; 3 Sleep disorders; Dr Cleusa Ferri 4 Universidade Federal de Sao Paulo, Department of Psychobiology, Brazil Psychological distress: personality and life events Institute of Education and Health Sciences at the Hospital Alemao Chapter 4 Oswaldo Cruz, Brazil Dr Cleusa Ferri 1 Dr Diego R. Mazzotti Dr Ronaldo D. Piovezan 1 Universidade Federal de Sao Paulo, Department of Psychobiology, Brazil Ivan Padilla 1 Dr Ronaldo D. Piovezan Prof Martin Prince 1,3 Universidade Federal de Sao Paulo, Department of Psychobiology, Brazil Prof Emiliano Albanese 2,3 Dr Matthew Prina 3 Ivan Padilla 1 Universidade Federal de Sao Paulo, Department of Psychobiology, Brazil Smoking and alcohol; 2 Physical activity and cognitive stimulation; 3 Diet Prof José A.
    [Show full text]
  • A Mediation Analysis to Identify Links Between Gut Bacteria and Memory in Context of Human Milk Oligosaccharides
    microorganisms Article A Mediation Analysis to Identify Links between Gut Bacteria and Memory in Context of Human Milk Oligosaccharides Stephen A. Fleming 1,* , Jonas Hauser 2, Jian Yan 3, Sharon M. Donovan 4,5 , Mei Wang 4 and Ryan N. Dilger 1,5,6,7 1 Traverse Science, Inc., Champaign, IL 61820, USA; [email protected] 2 Société des Produits Nestlé SA, 1000 Lausanne, Switzerland; [email protected] 3 Nestlé Product Technology Center Nutrition, CH-1800 Vevey, Switzerland; [email protected] 4 Department of Food Science and Human Nutrition, University of Illinois, Urbana, IL 61801, USA; [email protected] (S.M.D.); [email protected] (M.W.) 5 Division of Nutritional Sciences, University of Illinois, Urbana, IL 61801, USA 6 Piglet Nutrition and Cognition Laboratory, Department of Animal Sciences, University of Illinois, Urbana, IL 61801, USA 7 Neuroscience Program, University of Illinois, Urbana, IL 61801, USA * Correspondence: [email protected] Abstract: Elucidating relationships between the gut and brain is of intense research focus. Multiple studies have demonstrated that modulation of the intestinal environment via prebiotics or probiotics can induce cognitively beneficial effects, such as improved memory or reduced anxiety. However, the mechanisms by which either act remain largely unknown. We previously demonstrated that different types of oligosaccharides affected short- and long-term memory in distinct ways. Given that the oligosaccharide content of human milk is highly variable, and that formula-fed infants typically do not consume similar amounts or types of oligosaccharides, their potential effects on brain Citation: Fleming, S.A.; Hauser, J.; development warrant investigation. Herein, a mediation analysis was performed on existing datasets, Yan, J.; Donovan, S.M.; Wang, M.; including relative abundance of bacterial genera, gene expression, brain volume, and cognition in Dilger, R.N.
    [Show full text]
  • Nutrition, Learning and Behavior in Children: Accessibility Info
    Nutrition, Learning and Behavior in Children: Accessibility Info Food and Nutrition Information Center National Agricultural Library/USDA 10301 Baltimore Avenue, Room 105 Beltsville, MD 20705-2351 Nutrition, Learning and Behavior in Children: A Resource List for Professionals March 2004 This Food and Nutrition Information Center (FNIC) Resource List is a quick guide designed to help professionals find information related to nutrition, learning and behavior in children. Opinions expressed in the publications do not necessarily reflect the views of the U.S. Department of Agriculture. Materials which are part of the National Agricultural Library (NAL) collection have a call number. Lending and copy service information is provided at the end of this document. If you are not eligible for direct borrowing privileges, check with your local library on how to borrow materials through interlibrary loan with the NAL. Materials cannot be purchased from the NAL. Please contact the publisher /producer if you wish to purchase any materials on this list. This Resource List is available from the Food and Nutrition Information Center's (FNIC) web site at http://www.nal.usda.gov/fnic/pubs_and_db.html. Each resource has been placed in one of the following categories, in alphabetical order by title: I. General Nutrition, Hunger, Learning and Behavior in the United States A. Articles Dated 2000 - Present B. Articles Dated Prior to 2000 II. Micronutrient Status, Learning and Behavior in the United States A. Articles Dated 2000 - Present B. Articles Dated Prior to 2000 file:////CSCSRVLNH5DSS1/Shared_Data/Share/Content - Co....05cb/Nutrition, Learning and Behavior in Children.htm (1 of 35)10/11/2005 3:21:27 PM Nutrition, Learning and Behavior in Children: III.
    [Show full text]
  • Nutrition, the Brain and Epigenomics
    Nutrition, the Brain and Epigenomics: A Lifelong Approach to Optimal Cognition Margaret Joy Dauncey, PhD, ScD, FRSB University of Cambridge Wolfson College Cambridge, United Kingdom [email protected] Abstract Glossary of Abbreviations This review focuses on interactions Nutrition affects the brain throughout BDNF: Brain-Derived Neurotrophic between nutrition, the brain, and life, with profound implications for Factor cognition, and the underlying mech- optimal cognition. Advances in DHA: Docosahexaenoic Acid anisms involved. Three interrelated epigenomics are helping to elucidate IGF: Insulin-Like Growth Factor topics are discussed: the underlying mechanisms involved. miRNA: MicroRNA 1. The role of nutrition in brain Nutrition is one of many epigenetic ncRNA: Nonprotein-Coding RNA health and cognition regulators that modify gene expression SIRT1: Sirtuin Silent Information 2. Underlying mechanisms in without changes in DNA sequence. Regulator 1 relation to gene expression Although epigenetic modifications and epigenetics can be stable and heritable, they can be reversible, 3. Interactions between nutrition emphasizing critical roles for nutrition in both prevention and epigenomics and their relevance to nutritional and treatment of cognitive impairment. Insights into nutritional optimization of cognition throughout life regulation of gene expression, involving a One Health strategy across species, should provide novel lifelong 1. Nutrition, the Brain and Cognition approaches to optimal cognition and mental well-being One Health Approach:
    [Show full text]
  • The Impact of Diet-Based Glycaemic Response and Glucose Regulation on Cognition: Evidence Across the Lifespan
    Proceedings of the Nutrition Society (2017), 76, 466–477 doi:10.1017/S0029665117000829 © The Authors 2017 First published online 27 June 2017 The Joint Winter Meeting between the Nutrition Society and the Royal Society of Medicine held at The Royal Society of Medicine, London on 6–7 December 2016 Conference on ‘Diet, nutrition and mental health and wellbeing’ Symposium 2: Nutrition, cognition and emotion The impact of diet-based glycaemic response and glucose regulation on cognition: evidence across the lifespan Sandra I. Sünram-Lea1* and Lauren Owen2 1Department of Psychology, Lancaster University, Lancaster, UK 2Department of Psychology, University of Central Lancashire, Preston, Lancashire, UK The brain has a high metabolic rate and its metabolism is almost entirely restricted to oxi- dative utilisation of glucose. These factors emphasise the extreme dependence of neural tis- sue on a stable and adequate supply of glucose. Whereas initially it was thought that only glucose deprivation (i.e. under hypoglycaemic conditions) can affect brain function, it has become apparent that low-level fluctuations in central availability can affect neural and con- sequently, cognitive performance. In the present paper the impact of diet-based glycaemic response and glucose regulation on cognitive processes across the lifespan will be reviewed. The data suggest that although an acute rise in blood glucose levels has some short-term improvements of cognitive function, a more stable blood glucose profile, which avoids greater peaks and troughs in circulating glucose is associated with better cognitive function and a lower risk of cognitive impairments in the longer term. Therefore, a habitual diet that secures optimal glucose delivery to the brain in the fed and fasting states should be most advantageous for the maintenance of cognitive function.
    [Show full text]
  • Nutrition, the Brain and Cognitive Decline: Insights from Epigenetics MJ Dauncey
    European Journal of Clinical Nutrition (2014) 68, 1179–1185 © 2014 Macmillan Publishers Limited All rights reserved 0954-3007/14 www.nature.com/ejcn REVIEW Nutrition, the brain and cognitive decline: insights from epigenetics MJ Dauncey Nutrition affects the brain throughout life, with profound implications for cognitive decline and dementia. These effects are mediated by changes in expression of multiple genes, and responses to nutrition are in turn affected by individual genetic variability. An important layer of regulation is provided by the epigenome: nutrition is one of the many epigenetic regulators that modify gene expression without changes in DNA sequence. Epigenetic mechanisms are central to brain development, structure and function, and include DNA methylation, histone modifications and non-protein-coding RNAs. They enable cell-specific and age-related gene expression. Although epigenetic events can be highly stable, they can also be reversible, highlighting a critical role for nutrition in prevention and treatment of disease. Moreover, they suggest key mechanisms by which nutrition is involved in the pathogenesis of age-related cognitive decline: many nutrients, foods and diets have both immediate and long-term effects on the epigenome, including energy status, that is, energy intake, physical activity, energy metabolism and related changes in body composition, and micronutrients involved in DNA methylation, for example, folate, vitamins B6 and B12, choline, methionine. Optimal brain function results from highly complex interactions between numerous genetic and environmental factors, including food intake, physical activity, age and stress. Future studies linking nutrition with advances in neuroscience, genomics and epigenomics should provide novel approaches to the prevention of cognitive decline, and treatment of dementia and Alzheimer's disease.
    [Show full text]
  • Nutrition and the Function of the Central Nervous System
    nutrients Nutrition and the Function of the Central Nervous System Edited by Billy R. Hammond Printed Edition of the Special Issue Published in Nutrients www.mdpi.com/journal/nutrients Nutrition and the Function of the Central Nervous System Nutrition and the Function of the Central Nervous System Special Issue Editor Billy R. Hammond MDPI • Basel • Beijing • Wuhan • Barcelona • Belgrade Special Issue Editor Billy R. Hammond University of Georgia USA Editorial Office MDPI St. Alban-Anlage 66 Basel, Switzerland This is a reprint of articles from the Special Issue published online in the open access journal Nutrients (ISSN 2072-6643) from 2017 to 2018 (available at: http://www.mdpi.com/journal/nutrients/special issues/nutri cns) For citation purposes, cite each article independently as indicated on the article page online and as indicated below: LastName, A.A.; LastName, B.B.; LastName, C.C. Article Title. Journal Name Year, Article Number, Page Range. ISBN 978-3-03897-051-4 (Pbk) ISBN 978-3-03897-052-1 (PDF) Articles in this volume are Open Access and distributed under the Creative Commons Attribution (CC BY) license, which allows users to download, copy and build upon published articles even for commercial purposes, as long as the author and publisher are properly credited, which ensures maximum dissemination and a wider impact of our publications. The book taken as a whole is c 2018 MDPI, Basel, Switzerland, distributed under the terms and conditions of the Creative Commons license CC BY-NC-ND (http://creativecommons.org/licenses/by-nc-nd/4.0/). Contents About the Special Issue Editor .....................................
    [Show full text]