In Abdominal Disease

Total Page:16

File Type:pdf, Size:1020Kb

In Abdominal Disease JULY 2, zgzo.J "PANCREATIC REACTION " IN ABDOMINAL DISEASE. 1B and, if we cannot finally conquer, at least prolong for chemical syntheses effected by its cells in the formation years a life which unaided would have rapidly been of urea; it protects the body from the action of various sacrifioed. toxins in virtue of its detoxicating powers; and by virtue of its glycogenic function it is a centre of stored - REFERENCES. energy IGlomerular Lesions of Diffuse Nephritis, Herringham and Thurs- producing material. Its influence through the bile on field, Trans. Path. Soc. of Lond., vol. lv, 1904, p. 283, with photo- digestive processes is relatively insignificant. mnicrographs. 2Tigestedt and Bergmann, Skandinavisches Arch. f. Physiol., 1898. Band viii, 223; corroborated by Batty Shaw. Goulatonian The pancreas, on the other hand, is a great secretory Jiectures, 1906, Lancet, 1906. vol. i. 8 L. S. Foster, Joural of Medical gland; it performs no such functions as the liver performs; Besearch, vol. xxi, No. 2. p. 297, 1909. it is comparable to the salivary glands, not to the liver. Its disturbances affect the value of its secretion, either internal or external. There is nothing else to be disturbed. THE " PANCREATIC REACTION" IN The liver, in virtue of the scope of its functions, is exposed to injurious influences vastly more numerous than ABDOMINAL DISEASE. the pancreas. In neither is injury by extension from the BY WILLIAM RUSSELL, M.D., F.R.C.P.EDIN., duodenum along the ducts common. Injury to the liver PHYSICIAN AND LECTURER ON CLINICAL ZJEDICINE, ROYAL INFIRMARY; by way of the blood or lymph is common from acute AND LECTURER ON PRACTICE OF MEDICINE, SCHOOL OF parenchymatous inflammation ending in abscess to slow ME,DICINE, EDINBURGH. interstitial hyperplasia ending in cirrhosis. In the pancreas acute necrotic and haemorrhagic in- IN no department of internal medicine is accurate differen- flammation is known as a highly mortal malady, but it tial diagnosis so called for in the interest of the patient as has not been shown that these conditions arise from duct in diseases and disorder of the abdominal viscera. infection; while the cirrhoses of this organ have no known This general proposition is, I think, fully accepted by clinical significance apart from the production of diabetes my surgical colleagues, for the immature period of the in some instances. Apart, indeed, from the severe forms exploratory laparotomy has been succeeded by a desire for of inflammation, it may be fairly postulated that other accurate diagnosis, so that the abdomen may not be opened pathological changes in the gland can hardly be expected unless the prospect can confidently be entertained that the to do more than influence its secretory function. symptoms which justify surgical interposition will be Of the internal secretion and its modifications we are removed. That this highly desirable accuracy in differen- still in comparative ignorance, althongh much suggestive tial diagnosis has not yet been universally attained may be work has been done on this highly subtle question; while assumed from some recent writings. It surely, however, as regards the external secretion, diminution or increase does not ask for unattainable skill on the part of the in quantity of one or other or of all its ferments can be physician that an accurate differential diagnosis should be thought of only as affecting the processes of digestion in made between primary and secondary gastric symptoms, the intestine. between disturbances of gastric function and duodenal There is no failure in excretory function to consider, as ,ulcer, and between gastric catarrh and appendicitis. such failure has to be reckoned with in hepatic disorder. In view of the importance to be attached to accurate Any absorption from the gland must be the absorption differential diagnosis in the abdomen, all new aids claim- of some of its secretion, or of an altered enzyme; and the ing to further this object demand to be carefully tested enzymes are protein bodies, not carbohydrate bodies, and and exatmined. It was with this object in view that,'some presumably cannot, therefore, be the source of the -eighteen months ago, I, for the second time, took up the substance in question. -examination of the urine by Cammidge's method to ascer- The clinical pathology of the two glands is thus tain whether it would facilitate differential diagnosis in fundamentally different, and care has to be taken that -abdominal cases. clinical contentions regarding the pancreas are not based The claim of the author of this test was that, with upon an imaginary pathology of the organ. certain reservations, the reaction was indicative of the In the investigation undertaken I confined myself to existence of pancreatitis. This was not merely a new the examination of the urine in patients with abdominal test for a hitherto unrecognized product in the urine, but symptoms, or definite abdominal lesion, as my aim was ,the presence of this product was claimed to indicate the to ascertain whether any assistance was to be obtained presence of a certain condition in the pancreas, which from the presence or absence of the reaction in making condition, in its turn, was placed in the position of a a differential diagnosis. Cases were selected in which common and serious disease, and recognized as such for it was thought that the reaction might be helpful in one the first time. We were, in fact, brought face to face with or other direction. what was practically almost a new disease. We had I have tabulated 78 cases which are fairly representa- -before this recognized necrotic, haemorrhagic, and purulent tive of the types of abdominal cases met with. They pancreatitis; the metastasis of mumps to the pancreas; are as follows: of cases of diabetes associated with atrophic cirrhosis of the gland; and the contentions relating to the islands of TABLE A.-Mlarked Positive Reaction (4 Cases). Langerhans were well known. But here was a condition suggested to be common and important, which had hitherto Name and Diagnosis. Reaction. Remarks. escaped notice, the test of its existence being the presence Sex. of a urinary reaction. The Cammidge reaction claimed to be a pancreatic Veitch (m.)... Necrotic Positive, marked Abdomen opened; pancreatitis died. Decoction of reaction. With such reservations as are usually applic- the pancreas gave -able in clinical work, a positive reaction was to be markedreaction. No -regarded other organ simi- as strongly suggestive of the existence of larly tested. pancreatitis. Hay (m.) ... Necrotic malig- Positive, Gastric symptoms not If this is not to be the claim based upon 'the nant ulcer, size marked; crystals marked; patient de- reaction, of florin, pos- appeared in ten veloped profound -the performance of the test can at best merely be the terior gastric minutes intoxication sugges- demonstration of a chemical vagary in the individual; wall; extensive tive 'of severe pan- while, if the infarction, and creatic disease. Pan- claim be abandoned, and at the same time growths in liver creas showed neither the value of the reaction be asserted, we get into a region macroscopic nor ,somewhat outside either the pale of scientific medicine or microscopic change. Guthrie (m.)IHyperchlor. (1) Positive, Relieved by medical of logical thinking. The reaction cannot both be and hydria marked; treatment; returned ,not be a sign of pancreatitis. appeared imme- - 6 months later with In approaching this diately. symptoms of gastric subject it is necessary to bear in (2) Positive, ulcer, for which a mind that, although the pancreas and the liver are so medium gastro - enterostomy was done. -closely associated anatomically that their ducts may have Hoggan (f.)..... Pain in Positive, Died soon after ad- a common opening in the duodenal wall, their functions abdomen, with marked, and mission. No post- are totally dissimilar. !profound intoxi- rapidly mortem exramina- The liver is a great excretory gland, through its biliary Ication (cause appearing tion. funection; it is indirectly the same in virtue of the unknown) B 1 6 mau ;Oww I " PANCREATIC REACTION " IN ABDOMINAL DISEASE. [JULY 2, logo. TABLE B.-Hepatic Cirrhosis; Gall-Stone Colic; Catarrhal TABLE D.-Ga,tro-intestinal Cases with Positive Reaction Jaundice (12 Cases). (18 Cases). I~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~ Name and Sex. Diagnosis. Reaction. Remarks. Name and Sex Diagnosis. Reaction. Remarks. Welsh (f.) ... Cirrhotic liver Positive Died. Todd (f.) Gastric symptoms Twice positive, Cured. and ascites and constipation small Greig (m.) ... Cirrhotic liver Positive Ascitic fluid posi- Fox (f) ... Indigestion and Positive Cured. and ascites tive. constipation McKay (f.) ... Cirrhotic liver Positive Died; ascitic fluid Alexander (f.) Gastric symptoms Positive Cured. and ascites positive. and constipation Result: 3 positive and ascitic fluid positive in 2, Reid (f.) ... Gastric symptoms Positive Cured. Keddie (f.) ... Gall-stone colic I (1) Positive Operated on second Privatecase(f.) Great nausea and Positive 1(2) Negative time and stones skin irritation removed. Brown (f.) ... Obstinate vomit- Positive Cured. Cairns (f.) ... Gall stone colic Positive Operated on, stones ing. neurotic and tender gall removed. Mack (f.) ... Obstinate vomit- Twice positive Cured. bladder ing, neurotic Slater (f.) .. History of gall- Negative Improved. Knowles (f.) Gastric symptoms Positive Improvecl. stone colic McGruer (f.)... History of biliary Positive Cured. Turnbull (f.)... Gastric symptoms, Positive Improved. colic and consti- previous appendi- pation citis Henderson (f.) Gall-stone colic Positive Left hospital, Lees (f.) Gastric symptoms, i Positive Cured. sent in as ulcer Henderson (f.) Readmitted Negative Stone removed. Christie (f.) Gastric symptoms Positive Former gastrec- tomy, died. Private (f.) ... Hepatic colic Positive Latent case. McDonald (f.) Duodenal ulcer Twice positive, Gastro-enteros- small tomy, cured. Hanley (f.) ... Gall-stone colic Positive Stones found in Allen (f.) ... Gastric synmptoms Positive Old gastro- faeces. enterostomy. 6 Gillies (m) ... Constipation, ten- Positive Sent in as pneu- Result: positive, 3 negative.
Recommended publications
  • Study of Cholelithiasis After Gastrectomy
    Kurume Medical Journal, 47,105-108, 2000 Original Article Study of Cholelithiasis after Gastrectomy HISAFUMI KINOSHITA, HIROYASU IMAYAMA, KOTARO HASHINO AND SHIGEAKI AOYAGI Department of Surgery, Kurume University School of Medicine , Kurume 830-0011, Japan Summary: We studied cholelithiasis that occurred after gastrectomy in 52 patients (35 males and 17 females) encountered at our department between January , 1978 and December, 1998. Gastrectomy had been performed for gastric or duodenal ulcer in 35, gastric cancer in 14, gastroptosis in 2, and gastric trauma in 1 of these patients. Reconstruction after gastrectomy was performed by the Billroth II method (B-ll method) in 31 patients, Billroth I method (B-I method) in 17, Roux-en-Y method (Roux-Y method) in 3, and esophagogastrostomy in 1. The period between gas- trectomy and discovery of gallstones was 1-5 years in 9, 5-10 years in 10, and 10 years or longer in 33, or more than 60% of the patients. Gallstones were present in the gallbladder alone in 33 , bile duct alone in 9, gallbladder and bile duct in 10; the percentage of bile duct stones was high . The type of stones was bilirubin-calcium stones in 21, black stones in 12, pure cholesterol stones in 1, combined stones in 4, mixed stones in 12, and others in 2; pigment stones accounted for 63 .5%. Gallstones were symptomatic in 78.8% of the patients, and abdominal pain was the most frequent symptom. Bile was positive on bacterial culture in 68.4%, and Gram-negative bacilli were the most frequently isolated. Lymph node dissection, vagotomy, cholestasis , and biliary tract infection are considered to be related to cholelithiasis after gastrectomy.
    [Show full text]
  • Chapter 5 Herbs That Expel Dampness
    Chapter 5 Herbs that expel dampness introduction Herbs that are fragrant,dry the dampness harmonize spleen. Functions: abreact Qi stagnation 疏畅气机、 tonify spleen and stomach, resolve dampness宣化湿浊. Dampness sticky,spleen like dryness, dislike dampness,dampness in Spleen & G in Middle Jiao—affect the functions. Symptoms…? Western medicine diseases: acute gastritis, diarrhea, dysentery, ulcer, gastroptosis etc. Main effects: 1. adjust stomach & intestines movement Herbs has essential oil. Can regulate the stomach & intestine functions. Examples: Pei lan 佩兰、Beidoukou白豆蔻 enhance the stomach & intestines tension Saren 砂仁 intestines movement Relax intestine & stomach spasm, examples: Houpe厚朴、Cangzu苍术、Saren砂仁. 2. promote the digestive function Houpe厚朴、Huoxiang广藿香、Beikou白豆 蔻、Caokou草豆蔻、Caoguo草果 have essential oil,stimulate taste Nerve-enhance digestive glands secretion. 3. anti-ulcer Cangzu苍术、Houpo厚朴、Saren砂仁 ① Protect stomach membrane Cangzu-aminohexose 氨基己糖 关苍术提取物能增加氨基己糖在胃液和粘膜中的含量 砂仁能促进胃粘膜细胞释放前列腺素 保护胃粘膜免遭许多外源性因素的损伤。 ② Inhibit gastric acid Houpo 厚朴酚 can anti-tetragastrin四肽胃泌素 & carbamylcholine氨甲酰胆碱---gastric acid Cangzu 毛苍术 selinenol β-桉叶醇 can anti- H2 receptor, anti acid. 4. Anti-microbes magnolol厚朴酚、cangzu苍术、Pogostemon 广藿香酮 can inhibit or kill G+/G- bacteria, virus, broad spectrum antibiotics. Cangzu can anti fungi, mold yeast. Houpo 厚朴(Magnolia officnalis) Components: essential oil & some alkaloids. Functions & indications Houpo厚朴 dry dampness, remove food stognation, harmonize Qi, stop wheezing. Treatment : dampness in
    [Show full text]
  • Of Gastroptosis, Which, Though Only an Incidental Part Enteroptosis, Is
    Dr. J. R. Pennington, Chicago : The general practitioner mobility of the stomach, which is affected by the posi¬ is entitled to the same information as the specialist; conse¬ tion of the individual, by the weight of the stomach quently, I intended that this paper should contain statistical contents and by the tension or relaxation of the abdom¬ information not obtained the and other readily by average inal muscles. We have endeavored to determine the physician. Dr. Hirschman is to be commended for his normal of the stomach in a of valuable suggestions, and were they put into practice "by position study 1,000 insurance companies and others they would do much for the cases, and have come to the conclusion that in the control of this dreaded disease. supine position the textbook teachings are correct ; but in the upright position there is a wide normal variation depending on the anatomic make-up of various types of individuals. there seems to be as much A CLINICAL AND ROENTGENOGRAPHIC Indeed, difference in the size, contour and position of the STUDY OF GASTROPTOSIS stomachs of men as there is of their mouths. come so as OBSERVATIONS IN ONE THOUSAND GASTRO- We have also to the conclusion that far is it matters INTESTINAL PATIENTS gastric digestion concerned, very little whether a stomach is in the high position or in the brim SEALE HARRIS, M.D. of the pelvis, so long as its muscular tonus is good and AND the intra-abdominal pressure is normal. We have J. P. CHAPMAN, M.D. repeatedly found patients whose stomachs in the BIRMINGHAM, ALA.
    [Show full text]
  • Non-Ulcer Dyspepsia. Chronic Gastritis.”
    МІНІСТЕРСТВО ОХОРОНИ ЗДОРОВ’Я УКРАЇНИ ХАРКІВСЬКИЙ НАЦІОНАЛЬНИЙ МЕДИЧНИЙ УНІВЕРСИТЕТ “Затверджено” на методичній нараді кафедри внутрішньої медицини № 3 Завідувач кафедри професор______________________ (Л.В.Журавльова) “27” серпня 2010 р. МЕТОДИЧНІ РЕКОМЕНДАЦІЇ ДЛЯ СТУДЕНТІВ з англомовною формою навчання Навчальна дисципліна Основи внутрішньої медицини Модуль № 2 Змістовний модуль № 2 Основи діагностики, лікування та профілактики основних хвороб органів травлення Тема заняття Шлункова диспепсія та хронічні гастрити Курс 4 Факультет Медичний Харків 2014 KHARKOV NATIONAL MEDICAL UNIVERSITY DEPARTMENT OF INTERNAL MEDICINE N3 METHODOLOGICAL RECOMMENDATIONS FOR STUDENTS “Non-ulcer dyspepsia. Chronic gastritis.” Kharkiv 2014 Content module №2 «Bases of diagnostics, treatment and preventive maintenance of the basic illnesses organs of digestive truct» Practical class № 4. «Gastric dyspepsia and chronic gastritis» Urgency of gastric (non-ulcer, functional) dyspepsia. Non-ulcer dyspepsia or functional dyspepsia (FD) is most common in people to 25 years old and younger, but it can be common in older persons. Women suffer from FD in 1,5-2 times more than men. Prevalence is from 1,5 % to 58,8 % from number of all gastrointestinal disorders. Special symptoms (aerophagia, neurogenic symptoms, vomiting) meet rather seldom. They more characteristically for women with hysterical type of mentality. The educational purposes: to teach students to distinguish the basic symptoms and syndromes of FD ; to acquaint students with the methods of physical examination of FD; to acquaint students with the methods of research which are applied to the diagnostics of FD; with indications and contra-indications they have; with the techniques of their performance; with the diagnostic value of each of them; to teach students to interpret the results of the lead researches independently; to teach students to distinguish and diagnose the complications of FD; to teach students to appoint the treatment for FD.
    [Show full text]
  • Treatment of Mitochondrial Neurogastrointestinal Encephalomyopathy with Dialysis
    OBSERVATION Treatment of Mitochondrial Neurogastrointestinal Encephalomyopathy With Dialysis Haluˆk Yavuz, MD; Ahmet O¨ zel, MD; Mette Christensen, MSc; Ernst Christensen, PhD; Marianne Schwartz, PhD; Mithat Elmaci, MD; John Vissing, MD, PhD Objective: To study the effect of continuous ambula- Results: Dialysis stopped vomiting and reduced ab- tory peritoneal dialysis on nucleoside levels and clinical dominal pain, and the patient gained 5 kg in weight and course in a patient with mitochondrial neurogastroin- started to menstruate again. Symptoms returned if dialy- testinal encephalomyopathy (MNGIE). sis was paused. Dialysis did not affect plasma nucleoside levels. Patient: We studied a patient with genetically verified MNGIE, who prior to treatment had lost weight progres- Conclusions: This study shows an unambiguous clini- sively, developed amenorrhea, vomited multiple times cal benefit of peritoneal dialysis on gastrointestinal daily, and had abdominal pain. symptoms in MNGIE. Dialysis did not affect nucleoside levels, indicating elevated thymidine and deoxyuridine Intervention: The patient was treated with peritoneal levels are not solely responsible for the pathogenesis of dialysis for 3 years, and the effect on symptoms and MNGIE. plasma concentrations of thymidine and deoxyuridine were monitored. Arch Neurol. 2007;64:435-438 ITOCHONDRIAL NEURO- treatment for the condition is still lack- gastrointestinal en- ing. In this study, we report the use of con- cephalomyopathy tinuous ambulatory peritoneal dialysis (MNGIE) is a rare au- (CAPD) as a treatment for MNGIE. tosomal recessive mul- tisystem disorder characterized by exter- M REPORT OF A CASE nal ophthalmoplegia, gastrointestinal dysmotility and pain, cachexia, periph- eral neuropathy, and leukoencephalopa- A girl with consanguineous parents pre- thy. The disease is caused by mutations in sented with episodic vomiting and epigas- the gene encoding thymidine phosphory- tric pain at age 15 years.
    [Show full text]
  • Years Ago. Mortality Following Perforation. of an Abdominal Viscus
    MAY 6, ig9ii.] DEATHS FOLLOWING GABTRO4EJUNOSTOMY. [zTHoB045 This patiept has never been troubled with the old Robson and Moynihan, perforations of the stomach were symptoms since the operation, and is a healthy, happy successfally treated. In carrying out this treatment many young woman. From this history we see that over four surgeons used flushing Qf the abaominal cavity, with sub- years of suffering was endured before it was possible to sequent closure of the abdominal incision without drainage. determine the true seat of disease. This method gave excellent results and is still the beat The second case is one with more purely gastric method in many cases of gastric perforation. symptoms. It was a natural mistake to apply this treatment to CASE II. peritonitis following perforation of the appendix, but few Mrs. D., aged 36, came to me on October 13th, 1910, giving a went so far as to advocate closure of the abdomen without history, of epigastric pain and discomfort, and constant vomiting after meals for the past two years. Sbe had not been able to drainage. The results were extremely bad. eat any solid food, and subsisted upon milk and broths. She I think that the credit of giving the death-blow to this has had eight children, the youngest of whom is 3 years old. heroic and mistaken treatment is chiefly due to Murphy She had not menstruated for the past two years. She has in America, whilst the scientific work of Dudgeon and suffered from obstinate constipation. nailed the lid on its coffin. There was considerable epigastric tenderness, also some Sargent in England firmly tenderness on deep pressure over the right iliac fossa.
    [Show full text]
  • Managment of Boerhaave's Syndrome
    ISSN 2377-8369 Open Journal | July 2017 | Volume 2 | Issue 1 | Editor-in-Chief Jinghong Chen, PhD Associate Editors Ajay Goel, PhD Tatiana Goretsky (Zagranichnaya), PhD Jacintha O. Sullivan, PhD Shreyas Saligram, MD, MRCP www.openventio.org Gastro ISSN 2377-8369 Open Journal Table of Contents Editorial 1. Management of Boerhaave’s Syndrome e1-e3 – Sami Mansour and Alexandros Charalabopoulos* Case Report 2. Salvage Cryotherapy for Treatment of Persistent Barrett’s Esophagus 1-3 – Shreyas Saligram* Mini Review 3.Overriding Elements in Colon Cancer Progression: Some Less Known Facts 4-8 – Rohit Gundamaraju*, Ravichandra Vemuri and Rajaraman Eri Research 4. Analysis of the Intracellular Zinc in HCV Replicon 9-13 – Kazuhisa Yuasa, Daichi Takizawa, Hitoshi Takagi*, Satoru Kakizaki, Ken Sato, Masanobu Yamada, Youko Katsuya, Hiroshi Koyama, Takuro Sakai and Kazuo Arakawa Research 5. The Clinical or Radiographic Diagnosis of Gastroptosis: Still Relevant? 14-19 – Anke J. M. van Welie, Willemijn M. Klein and Jos M. Th. Draaisma* Gastro Open J GASTRO ISSN 2377-8369 http://dx.doi.org/10.17140/GOJ-2-e003 Open Journal Editorial Management of Boerhaave’s Syndrome *Corresponding author Alexandros Charalabopoulos, MD, MSc, PhD, MRCS, FRCS Sami Mansour, MBBS, MRCS; Alexandros Charalabopoulos, MD, MSc, PhD, MRCS, Department of Upper Gastrointestinal FRCS* Surgery Regional Oesophagogastric Unit Broomfield Hospital Department of Upper Gastrointestinal Surgery, Regional Oesophagogastric Unit, Broomfield Mid Essex Hospital Services NHS Trust Chelmsford, Essex, England, UK Hospital, Mid Essex Hospital Services NHS Trust, Chelmsford, Essex, England, UK E-mail: [email protected] Volume 2 : Issue 1 Boerhaave’s syndrome or spontaneous oesophageal perforation is characterised by Article Ref.
    [Show full text]
  • Article / Autopsy Case Report Artigo / Relato De Caso De Autópsia
    Autopsy and Case Reports 2013; 3(2): 21-29 Article / Autopsy Case Report Artigo / Relato de Caso de Autópsia Intrathoracic gastric volvulus: an autopsy case report Cristiane Rúbia Ferreiraa, Linda Ferreira Maximianob, Victor Manuel Lobo dos Santosb, João Augusto dos Santos Martinesc Ferreira CR, Maximiano LF, Santos VML, Martines JAS. Intrathoracic gastric volvulus: an autopsy case report. Autopsy Case Rep [Internet]. 2013;3(2):21-29. http://dx.doi.org/10.4322/acr.2013.014 ABSTRACT First described by Berti in 1866, gastric volvulus (GV) is an uncommon and potentially lethal entity. GV occurs when the stomach twists by more than 180º resulting in obstruction of the alimentary tract, visceral ischemia, necrosis, and perforation. It is classified according to the rotation axis in organoaxial, mesenteroaxial or a combination of both. The clinical presentation can be acute, and is usually severe or chronic, which sometimes may be asymptomatic. It predominantly occurs in the fifth decade of life, but children, mainly those under the age of 1 year, may be affected. No ethnicity or gender was observed to show predominance. This entity is related to gastric, diaphragmatic disorders as well as laxity of gastric ligaments. Acute GV may complicate with incarceration and strangulation of the stomach when gastric necrosis ensues. These cases show a mortality rate of 60%. The authors report the fatal case of a surgically treated GV in a 43-year-old female patient who looked for medical care only after 1 month of initial symptoms. Diagnosis was confirmed with a thoracic and abdominal axial computed tomography. Besides the entire stomach being herniated and twisted into the thoracic cavity, the pancreas was pulled up through the hiatal orifice, provoking acute pancreatitis.
    [Show full text]
  • Abdominal Echography Cpt Code
    MEDICAID CODING GUIDELINE ABDOMINAL ECHOGRAPHY CPT CODE: 76700 Echography, Abdominal, B-scan and/or real time with image documentation; complete 76705 limited (e.g. single organ, quadrant, follow-up) COVERED DIAGNOSIS: 150.2-150.5 Malignant neoplasm of abdominal esophagus 150.8-150.9 Malignant neoplasm of esophagus, unspecified 151.0-151.9 Malignant neoplasm of stomach 152.0-152.9 Malignant neoplasm of small intestine, including duodenum 153.0-153.9 Malignant neoplasm of colon 155.0-155.2 Malignant neoplasm of liver and intra-hepatic bile ducts 156.0-156.9 Malignant neoplasm of gallbladder and extra-hepatic bile ducts 157..0-157.9 Malignant neoplasm of pancreas 158.0-158.9 Malignant neoplasm of retroperitoneum and peritoneum 159.0-159.9 Malignant neoplasm of other and ill-defined site within digestive organs and peritoneum 171.5 Malignant neoplasm of abdomen 179 Malignant neoplasm of uterus, unspecified 180.0-180.9 Malignant neoplasm of cervix uteri 182.0-182.8 Malignant neoplasm of body of uterus 183.0-183.9 Malignant neoplasm of ovary, adnexa 189.0-189.9 Neoplasm of kidney and other and unspecified urinary organs 194.9 Malignant neoplasm of endocrine gland 195.2 Malignant neoplasm of abdomen 196.2 Secondary and unspecified neoplasm of intra-abdominal lymph nodes 197.4-197.5 Secondary malignant neoplasm of small and large intestine 197.7 Secondary malignant neoplasm of liver 197.8 Secondary malignant neoplasm of other digestive organs and spleen 198.0 Secondary malignant neoplasm of kidney 198.1 Secondary malignant neoplasm of other
    [Show full text]
  • Superior Mesenteric Artery Syndrome Coexists with Nutcracker Syndrome in a Female
    Shi et al. BMC Gastroenterology (2019) 19:15 https://doi.org/10.1186/s12876-019-0932-1 CASE REPORT Open Access Superior mesenteric artery syndrome coexists with Nutcracker syndrome in a female: a case report Ying Shi1,2* , Guangya Shi3, Zhenyu Li4, Yanfang Chen1,2, Shaohui Tang1,2 and Wei Huang1,2* Abstract Background: Superior mesenteric artery (SMA) syndrome, also known as Wilkie’s syndrome or Benign duodenal stasis, is a rare benign disease. It could threaten the life if the manifestation is severe and the treatment is inappropriate. In the patients with SMA syndrome, the third portion (transverse part) of the duodenum is compressed externally between the SMA and abdominal aorta (AA) leading to duodenal stasis and gastrointestinal obstruction. SMA syndrome may rarely combine with Nutcracker syndrome when left renal vein (LRV) was compressed between SMA and AA. Case presentation: A 32-year-old female patient presented with complaints of gradually severe bloating, epigastric pain, left flank ache, nausea and occasional vomiting of 1 month’s duration. The epigastric and left flank ache was aggravated when the patient was supine and relieved in a prone or left lateral decubitus. The abdominal bloating was associated with early satiety. The vomiting always started 40 min after meal. The patient gave a history of urine stone with drotaverine hydrochloride tablets treatment for two weeks before the gastrointestinal symptoms arising. The patient had no significant surgical history, but had a rapid weight loss of approximately 10 kg with a body mass index (BMI) from 21 kg/m2 to less than 18 kg/m2 over the last two months.
    [Show full text]
  • Some Points in the Differential Diagnosis of Chronic Indigestion
    Postgrad Med J: first published as 10.1136/pgmj.11.118.276 on 1 August 1935. Downloaded from 276 POST-GRADUATE MEDICAL JOURNAL August, 1935 SOME POINTS IN THE DIFFERENTIAL DIAGNOSIS OF CHRONIC INDIGESTION. By HERBERT J. PATERSON, C.B.E., M.C., M.D., F.R.C.S. Given the disease it is easy to deduce the symptoms which may accompany that particular disease. In the investigation of an individual case the problem must be approached from a different angle, for then it is the symptoms which are known whereas the disease which is the cause of these symptoms is the unknown quantity. Consequently, at the bedside the clinician has first to obtain an accurate and detailed history and then attempt inductively to ascertain the cause or causes which produce these symptoms. Inasmuch as there may be many possible causes for similar symptoms he has to consider the symptoms in detail together with the information gained by examination of the patient and then make a diagnosis by a process of exclusion. When in this way a provisional diagnosis has been made the signs and symptoms should be compared with those usually associated with the disease suspected, to see whether these confirm the provisional diagnosis. By far the most important symptoms of chronic indigestion are pain and dis- comfort. It is necessary, therefore, to differentiate between these symptoms, for real pain which is recurrent or constant is almost always due to some organicProtected by copyright. lesion. The crucial diagnostic point is the time relation of the discomfort or pain to the ingestion of food.
    [Show full text]
  • Treating-Acid-Reflux.Pdf
    www.acupunctureceus.com OPTIONS FOR WELLNESS, INC. 7059 SW 53 LN MIAMI, FL 33155 305-665-0615 [email protected] CEU PROVIDER Florida Board of Acupuncture 50-2489-1 NCCAOM ACHB-038 CALIFORNIA CEP 722 Acid Reflux More than 60 million Americans experience heartburn at least once a month and some studies have suggested that more than 15 million Americans experience heartburn symptoms each day. (American College of Gastroenterology ). Acid reflux disease is described as heartburn 2 or more days a week despite treatment and diet change. Acid reflux disease is caused by reflux of stomach acid into the esophagus. In most patients this is due to a transient relaxation of the "gate" or sphincter (LES) that keeps the lower end of the esophagus closed when a person is not swallowing food or liquids. This transient relaxation happens a few times each day in people without acid reflux disease. The esophagus is not able to cope with acid as well as the stomach and is easily injured. It's the acid refluxing into the esophagus that produces the symptoms and potentially damages the esophagus. GERD can masquerade as other diseases Increasingly, we are becoming aware that the irritation and damage to the esophagus from continual presence of acid can prompt an entire array of symptoms other than simple heartburn. Experts recognize that often the role of acid reflux has been overlooked as a potential factor in the diagnosis and treatment of patients with chronic cough, hoarseness and asthma-like symptoms. In some instances, patients have never reported heartburn, and in others the potential causal link between reflux and the onset of these so-called “extra-esophageal manifestations” has not been fully recognized.
    [Show full text]