What Is the Best Approach to the Evaluation of Resting Tachycardia For

Total Page:16

File Type:pdf, Size:1020Kb

What Is the Best Approach to the Evaluation of Resting Tachycardia For From the CLINIcAL InQUiRiES Family Physicians Inquiries Network Jennifer E. DeVoe, What is the best approach MD, DPhil Department of Family Medicine, Oregon Health and Science to the evaluation of resting University, Portland Dolores Zegar Judkins, tachycardia for an adult? MLS Oregon Health and Science University Library, Portland EVIDENCE - BASED ANSWER The best evidence about the diagnostic evaluation suggests a sinus tachycardia evaluation of resting tachycardias in adults with narrow QRS complexes and no is currently outlined by practice guide- identifiable secondary cause, a 24-hour lines.1 Initial evaluation includes clinical Holter monitor is usually recommended history, physical examination, and 12-lead (strength of recommendation: C, based electrocardiogram (ECG). If the initial ® onDowden expert opinion). Health Media CLINICAL COMMENTARYCopyrightFor personal use only Wide-complex tachycardias and irregular more relaxed pace. For nonurgent cases it heartbeats should be urgently managed is important to keep in mind the differential This Clinical Inquiry organizes a rational diagnosis and rationally evaluate the likely approach to tachycardia, which is frequent- causes. In my patient population, I tend to ly an incidental and asymptomatic finding see sinus tachycardias in young healthy on patient intake. The recommendation of patients in whom no secondary cause aside evaluating a 12-lead ECG for sinus vs non- from anxiety is identified. Oftentimes I sinus tachycardia, then further investigating follow up after initiating treatment for underlying causes, helps frame the workup anxiety or its underlying cause and find in an approachable manner. Particularly that the tachycardia has resolved. In these helpful is the pointer that the wide- cases, I have been less aggressive about complex tachycardias and irregular ordering a 24-hour Holter monitor. heartbeats should be urgently managed, Laurel Woods, MD whereas the rest can be assessed at a Group Health Family Medicine Residency Program, Seattle, Wash æ Evidence Summary was an international practice guideline de- Heart rate varies by age; however, tachy- veloped by the American College of Car- cardia in adults is usually defined as a rate diology, the American Heart Association exceeding 100 beats/minute.1 Tachycardia Task Force on Practice Guidelines, and the at rest requires a diagnostic evaluation. European Society of Cardiology Commit- However, our search found no systematic tee for Practice Guidelines.1 reviews, randomized trials, or prospective This joint guideline recommends that cohort studies relevant to this question. the diagnostic evaluation of a hemody- The highest level of evidence we located namically stable patient should begin with www.jfponline.com VOL 56, NO 1 / JAnUARY 2007 59 For mass reproduction, content licensing and permissions contact Dowden Health Media. ES i R i QU n TABLE I supraventricular (from the sinus node, the Potential secondary causes of atria, and the atrioventricular junction) in AL resting sinus tachycardia5–7 c origin, and wide QRS complex tachycar- Hyperthyroidism dias are usually ventricular (from all sites below the AV junction).2,3 If an irregular CLINI Fever heartbeat or wide-complex tachycardia Sepsis is detected, appropriate management (in- Anxiety cluding possible urgent referrals) should begin immediately.1 A stable patient with Pheochromocytoma a regular rhythm and a narrow QRS com- Anemia plex can be further investigated at a more Hypotension and shock relaxed pace. Refer to the TABLe for a listing of com- Pulmonary embolism mon secondary causes for sinus tachycar- Acute coronary ischemia and myocardial infarction dia, which should direct lab investigations. Heart failure If no secondary cause is easily identifiable, a 24-hour monitor is recommended as the Chronic pulmonary disease next step. Hypoxia Exposure to medications, stimulants, or illicit drugs Recommendations from Others Malignancy Textbook chapters and other review ar- ticles regarding this topic describe a simi- Pregnancy lar initial evaluation and provide further details about interpreting the 12-lead a clinical history, physical examination ECG.2–7 The most relevant and recent with relevant labs, and 12-lead ECG.1 review article suggests that further inves- Many patients with tachycardia are tigation of narrow QRS complex tachy- FAST TRACK asymptomatic; however, common symp- cardias with a regular rate currently If a 12-lead ECG toms and complaints include palpitations, involves 4 diagnostic categories: normal fatigue, lightheadedness, chest discomfort, sinus tachycardia (ie, secondary cause suggests sinus dyspnea, presyncope, or syncope.1 If the can be identified), inappropriate sinus tachycardia patient has experienced symptoms, it is of tachycardia (IST), postural orthostatic with narrow QRS crucial importance to obtain a clinical his- tachycardia syndrome (POTS), and sinus complexes and no tory describing the pattern in terms of the node reentry tachycardia (SNRT).4 number of episodes, duration, frequency, If a secondary cause is identified, it secondary cause, mode of onset, and possible triggers.1 should be treated appropriately. If no a 24-hour The main goals of the physical ex- underlying cause is discovered, a 24-hour Holter monitor amination, labs, and the 12-lead ECG are Holter monitor is recommended. to determine if the patient has a sinus or Persistent sinus tachycardia (some- is recommended nonsinus tachycardia and to look for oth- times with nocturnal normalization of er findings that may suggest either a cause heart rate) is diagnosed as IST.4 If the for the tachycardia or any complications monitor shows paroxysmal episodes of resulting from the tachycardia. sinus tachycardia, determine if they are First, determine if the patient’s heart- triggered by orthostasis and relieved by beat is regular or irregular. Atrial flutter recumbency (confirm with head upright and atrial fibrillation are common causes tilt test) to make the diagnosis of POTS. If of an irregular heartbeat that can easily be it is not POTS, the recordings from the 24- diagnosed with a 12-lead ECG. Second, hour Holter monitor help make the diag- determine the width of the QRS interval: nosis of SNRT, which consists of sudden, narrow QRS complex tachycardias are paroxysmal, and usually nonsustained 60 VOL 56, NO 1 / JAnUARY 2007 THE JOURNAL OF FAMILY PRActIcE Evaluation of resting tachycardia in an adult p FIGURE Diagnostic algorithm for evaluating tachycardias4 HEART RATE >100 beats/minute SINUS TACHYCARDIA Non-SINUS TACHYCARDIA P waves precede QRS complexes; No clear P-QRS patterns is there an underlying cause? UNDERLYING CAUSE IDENTIFIED NO UNDERLYING CAUSE MAKE DIAGNOSIS • Diagnose as normal IDENTIFIED Initiate treatment sinus tachycardia Consider 24-hour Holter monitor • Initiate treatment Persistent or paroxysmal? PERSISTENT TACHYCARDIA PAROXYSMAL TACHYCARDIA • Diagnose with inappropriate sinus Is there a postural trigger? tachycardia (IST) (Head upright tilt test) • Initiate treatment (bradycardic agents or surgical ablation) NO POSTURAL TRIGGER POSTURAL TRIGGER • Diagnose sinus node reentry • Diagnose postural orthostatic tachycardia (SNRT) tachycardia syndrome (POTS) • Initiate termination treatments • Initiate treatment for acute episodes (pharmacologic and • Consider prevention for frequent nonpharmacologic options) episodes tachycardia.4 The FIGUre shows an algo- eds. Textbook of Primary Care Medicine. 3rd ed. rithm of one common diagnostic strategy St. Louis, Mo: Mosby; 2001:528–537. 2–7 4. Stoenescu ML, Kowey PR. Tachycardias. In: Rakel for evaluation of tachycardia. RE, ed. Conn’s Current Therapy. 57th ed. Philadel- phia, Pa: Saunders; 2005:354–355. REFERENCES 5. Olgin JE, Zipes DP. Specific arrhythmias: diagnosis and treatment. In: Braunwald E, ed. Braunwald’s 1. American College of Cardiology/American Heart Heart Disease: A Textbook of Cardiovascular Medi- Association Task Force on Practice Guidelines and cine. Philadelphia, Pa: Saunders; 2005:803–806. the European Society of Cardiology Committee for Practice Guidelines (ACC/AHA/ESC). ACC/AHA/ 6. Castellanos A, Moleiro F, Chakko S, et al. Heart rate ESC guidelines for the management of patients variability in inappropriate sinus tachycardia. Am J with surpraventricular arrythmias-executive sum- Cardiol 1998; 82:531–534. mary. J Am Coll Cardiol 2003; 42:1493–1531. 7. Krahn AD, Yee R, Klein GJ, Morillo C. Inappropri- 2. Yusuf S, Camm JA. Deciphering the sinus tachy- ate sinus tachycardia: evaluation and treatment. J cardias. Clin Cardiol 2005; 28:267–276. Cardiovasc Electrophysiol 1995; 6:1124–1128. 3. Martin DT. Arrhythmias. In: Noble J, Greene HL, www.jfponline.com VOL 56, NO 1 / JAnUARY 2007 61.
Recommended publications
  • Common Arrhythmias Disclosures
    Common Arrhythmias Disclosures • I work for Virginia Garcia Memorial Health Center. • And I am a medical editor for Jones & Bartlett Publishing. Jon Tardiff, BS, PA-C OHSU Clinical Assistant Professor What a 12-Lead ECG can help you do • Diagnose ACS / AMI • Interpret arrhythmias • Identify life-threatening syndromes (WPW, LGL, Long QT synd., Wellens synd., etc) • Infer electrolyte imbalances • Infer hypertrophy of any chamber • Infer COPD, pericarditis, drug effects, and more! Arabic, Somali, Mai Mai, Pashtu, Urdu, ASL, and more! For example… WPW with Atrial Fib 55 66 Wolff-Parkinson-WhiteWPW Graphic synd. Same pt, converted to SR Drs. Wolff, Parkinson, & White 77 Another example: Dr. William Stokes—1800s 71 y.o. man with syncope This patient is conscious and alert! Third Degree Block 9 Treatment: permanent pacemaker 10 Lots of ways to read ECGs… Limitations of a 12-Lead ECG • QRSs wide or narrow? • Is it sinus rhythm or not? • Truly useful only ~40% of the time • Regular or irregular? • If not, is it atrial fibrillation? • Each ECG is only a 10 sec. snapshot • Fast or slow? • BBB? • P waves? • MI? • Serial ECGs are necessary, especially for ACS • Other labs help corroborate ECG findings (cardiac markers, Cx X-ray) • Confounders must be ruled out (LBBB, dissecting aneurysm, pericarditis, WPW, Symptoms: digoxin, LVH, RVH) • Syncope is bradycardia, heart blocks, or VT • Rapid heart beat is AF, SVT, or VT Conduction System Lead II P wave axis …upright in L II II R T P R U Q S …upright in L II R wave axis SA Node AV Node His Bundle BBs Purkinje Fibers 14 13 Q S Normal Sinus Rhythm Triplicate Method: 6-second strip: 6 seconds 300, 150, 100, Count PQRST cycles in a 6 75, 60, 50 second strip & multiply x 10 Quick, easy, sufficient Easy, & more accurate 300 150 100 75 60 6 seconds What is the heart rate? Horizontal axis is time (mS); vertical axis is electrical energy (mV) 16 1.
    [Show full text]
  • Constrictive Pericarditis Causing Ventricular Tachycardia.Pdf
    EP CASE REPORT ....................................................................................................................................................... A visually striking calcific band causing monomorphic ventricular tachycardia as a first presentation of constrictive pericarditis Kian Sabzevari 1*, Eva Sammut2, and Palash Barman1 1Bristol Heart Institute, UH Bristol NHS Trust UK, UK; and 2Bristol Heart Institute, UH Bristol NHS Trust UK & University of Bristol, UK * Corresponding author. Tel: 447794900287; fax: 441173425926. E-mail address: [email protected] Introduction Constrictive pericarditis (CP) is a rare condition caused by thickening and stiffening of the pericar- dium manifesting in dia- stolic dysfunction and enhanced interventricu- lar dependence. In the developed world, most cases are idiopathic or are associated with pre- vious cardiac surgery or irradiation. Tuberculosis remains a leading cause in developing areas.1 Most commonly, CP presents with symptoms of heart failure and chest discomfort. Atrial arrhythmias have been described as a rare pre- sentation, but arrhyth- mias of ventricular origin have not been reported. Figure 1 (A) The 12 lead electrocardiogram during sustained ventricular tachycardia is shown; (B and C) Case report Different projections of three-dimensional reconstructions of cardiac computed tomography demonstrating a A 49-year-old man with a striking band of calcification around the annulus; (D) Carto 3DVR mapping—the left hand panel (i) demonstrates a background of diabetes, sinus beat with late potentials at the point of ablation in the coronary sinus, the right hand panel (iii) shows the hypertension, and hyper- pacemap with a 89% match to the clinical tachycardia [matching the morphology seen on 12 lead ECG (A)], and cholesterolaemia and a the middle panel (ii) displays the three-dimensional voltage map.
    [Show full text]
  • J Wave Syndromes
    Review Article http://dx.doi.org/10.4070/kcj.2016.46.5.601 Print ISSN 1738-5520 • On-line ISSN 1738-5555 Korean Circulation Journal J Wave Syndromes: History and Current Controversies Tong Liu, MD1, Jifeng Zheng, MD2, and Gan-Xin Yan, MD3,4 1Tianjin Key Laboratory of Ionic-Molecular Function of Cardiovascular disease, Department of Cardiology, Tianjin Institute of Cardiology, The Second Hospital of Tianjin Medical University, Tianjin, 2Department of cardiology, The Second Hospital of Jiaxing, Jiaxing, China, 3Lankenau Institute for Medical Research and Lankenau Medical Center, Wynnewood, Pennsylvania, USA, 4The First Affiliated Hospital, Medical School of Xi'an Jiaotong University, Xi'an, China The concept of J wave syndromes was first proposed in 2004 by Yan et al for a spectrum of electrocardiographic (ECG) manifestations of prominent J waves that are associated with a potential to predispose affected individuals to ventricular fibrillation (VF). Although the concept of J wave syndromes is widely used and accepted, there has been tremendous debate over the definition of J wave, its ionic and cellular basis and arrhythmogenic mechanism. In this review article, we attempted to discuss the history from which the concept of J wave syndromes (JWS) is evolved and current controversies in JWS. (Korean Circ J 2016;46(5):601-609) KEY WORDS: Brugada syndrome; Sudden cardiac death; Ventricular fibrillation. Introduction History of J wave and J wave syndromes The concept of J wave syndromes was first proposed in 2004 The J wave is a positive deflection seen at the end of the QRS by Yan et al.1) for a spectrum of electrocardiographic (ECG) complex; it may stand as a distinct “delta” wave following the QRS, manifestations of prominent J waves that are associated with a or be partially buried inside the QRS as QRS notching or slurring.
    [Show full text]
  • The Syndrome of Alternating Bradycardia and Tachycardia by D
    Br Heart J: first published as 10.1136/hrt.16.2.208 on 1 April 1954. Downloaded from THE SYNDROME OF ALTERNATING BRADYCARDIA AND TACHYCARDIA BY D. S. SHORT From the National Heart Hospita. Received September 15, 1953 Among the large number of patients suffering from syncopal attacks who attended the National Heart Hospital during a four-year period, there were four in whom examination revealed sinus bradycardia alternating with prolonged phases of auricular tachycardia. These patients presented a difficult problem in treatment. Each required at least one admission to hospital and in one case the symptoms were so intractable as to necessitate six admissions in five years. Two patients had mitral valve disease, one of them with left bundle branch block. One had aortic valve sclerosis while the fourth had no evidence of heart disease. THE HEART RATE The sinus rate usually lay between 30 and 50 a minute, a rate as slow as 22 a minute being observed in one patient (Table I). Sinus arrhythmia was noted in all four patients, wandering of TABLE I http://heart.bmj.com/ RATE IN SINus RHYTHM AND IN AURICULAR TACHYCARDIA Rate in Case Age Sex Associated Rate in auricular tachycardia heart disease sinus rhythm Auricular Venliicular 1 65 M Aortic valve sclerosis 28-48 220-250 60-120 2 47 F Mitral valve disease 35-75 180-130 90-180 on September 26, 2021 by guest. Protected copyright. 3 38 F Mitral valve disease 22-43 260 50-65 4 41 F None 35-45 270 110 the pacemaker in three, and periods of sinus standstill in two (Fig.
    [Show full text]
  • Unstable Angina with Tachycardia: Clinical and Therapeutic Implications
    Unstable angina with tachycardia: Clinical and therapeutic implications We prospectively evaluated 19 patients with prolonged chest pain not evolving to myocardiai infarction and accompanied with reversible ST-T changes and tachycardia (heart rate >lOO beats/min) in order to correlate heart rate reduction with ischemic electrocardiographic (ECG) changes. Fourteen patients (74%) received previous long-term combined treatment with nifedipine and nitrates. Continuous ECG monitoring was carried out until heart rate reduction and at least one of the following occurred: (1) relief of pain or (2) resolution of ischemic ECG changes. The study protocol consisted of carotid massage in three patients (IS%), intravenous propranolol in seven patients (37%), slow intravenous amiodarone infusion in two patients (lo%), and intravenous verapamil in four patients (21%) with atrial fibrillation. In three patients (16%) we observed a spontaneous heart rate reduction on admission. Patients responded with heart rate reduction from a mean of 126 + 10.4 beats/min to 64 k 7.5 beats/min (p < 0.005) and an ST segment shift of 4.3 k 2.13 mm to 0.89 k 0.74 mm (p < 0.005) within a mean interval of 13.2 + 12.7 minutes. Fifteen (79%) had complete response and the other four (21%) had partial relief of pain. A significant direct correlation was observed for heart rate reduction and ST segment deviation (depression or elevation) (f = 0.7527 and 0.8739, respectively). These patients represent a unique subgroup of unstable angina, in which the mechanism responsible for ischemia is excessive increase in heart rate. Conventional vasodilator therapy may be deleterious, and heart rate reduction Is mandatory.
    [Show full text]
  • Respiration Driven Excessive Sinus Tachycardia Treated with Clonidine Matthew Emile Li Kam Wa,1 Patricia Taraborrelli,1 Sajad Hayat,2 Phang Boon Lim1
    Novel treatment (new drug/intervention; established drug/procedure in new situation) BMJ Case Reports: first published as 10.1136/bcr-2016-216818 on 28 April 2017. Downloaded from CASE REPORT Respiration driven excessive sinus tachycardia treated with clonidine Matthew Emile Li Kam Wa,1 Patricia Taraborrelli,1 Sajad Hayat,2 Phang Boon Lim1 1Department of Cardiology, SUMMARY no evidence of dual AV node physiology, accessory Imperial College Healthcare A 26-year-old man presented to our syncope service pathway or inducible supraventricular tachycardia. NHS Trust, London, UK 2Department of Cardiology, with debilitating daily palpitations, shortness of breath, A subsequent permanent pacemaker led to no University Hospitals Coventry presyncope and syncope following a severe viral further episodes of frank syncope. However his and Warwickshire NHS Trust, respiratory illness 4 years previously. Mobitz type II block ongoing debilitating exertional and respiratory- Coventry, UK had previously been identified, leading to a permanent driven palpitations with presyncope remained. pacemaker and no further episodes of frank syncope. Conservative measures including increased fluid Correspondence to Dr Phang Boon Lim, Transthoracic echocardiography, electophysiological study intake and compression stockings had no effect. [email protected] and repeated urine metanepherines were normal. His Trials of medication including fludrocortisone, fle- palpitations and presyncope were reproducible on deep cainide, β blockers and ivabradine were either not Accepted 18 December 2016 inspiration, coughing, isometric hand exercise and tolerated or had no significant effect on his passive leg raises. We demonstrated rapid increases in symptoms. heart rate with no change in morphology on his 12 lead During a simple active stand over 3 min, his ECG.
    [Show full text]
  • Tachycardia (Fast Heart Rate)
    Tachycardia (fast heart rate) Working together to improve the diagnosis, treatment and quality of life for all those aff ected by arrhythmias www.heartrhythmalliance.org Registered Charity No. 1107496 Glossary Atrium Top chambers of the heart that receive Contents blood from the body and from the lungs. The right atrium is where the heart’s natural pacemaker (sino The normal electrical atrial node) can be found system of the heart Arrhythmia An abnormal heart rhythm What are arrhythmias? Bradycardia A slow heart rate, normally less than 60 beats per minute How do I know what arrhythmia I have? Cardiac Arrest the abrupt loss of heart function, breathing and consciousness Types of arrhythmia Cardioversion a procedure used to return an abnormal What treatments are heartbeat to a normal rhythm available to me? Defi brillation a treatment for life-threatening cardiac arrhythmias. A defibrillator delivers a dose of electric current to the heart Important information This booklet is intended for use by people who wish to understand more about Tachycardia. The information within this booklet comes from research and previous patients’ experiences. The booklet off ers an explanation of Tachycardia and how it is treated. This booklet should be used in addition to the information given to you by doctors, nurses and physiologists. If you have any questions about any of the information given in this booklet, please ask your nurse, doctor or cardiac physiologist. 2 Heart attack A medical emergency in which the blood supply to the heart is blocked, causing serious damage or even death of heart muscle Tachycardia Fast heart rate, more than 100 beats per minute Ventricles The two lower chambers of the heart.
    [Show full text]
  • Basic Rhythm Recognition
    Electrocardiographic Interpretation Basic Rhythm Recognition William Brady, MD Department of Emergency Medicine Cardiac Rhythms Anatomy of a Rhythm Strip A Review of the Electrical System Intrinsic Pacemakers Cells These cells have property known as “Automaticity”— means they can spontaneously depolarize. Sinus Node Primary pacemaker Fires at a rate of 60-100 bpm AV Junction Fires at a rate of 40-60 bpm Ventricular (Purkinje Fibers) Less than 40 bpm What’s Normal P Wave Atrial Depolarization PR Interval (Normal 0.12-0.20) Beginning of the P to onset of QRS QRS Ventricular Depolarization QRS Interval (Normal <0.10) Period (or length of time) it takes for the ventricles to depolarize The Key to Success… …A systematic approach! Rate Rhythm P Waves PR Interval P and QRS Correlation QRS Rate Pacemaker A rather ill patient……… Very apparent inferolateral STEMI……with less apparent complete heart block RATE . Fast vs Slow . QRS Width Narrow QRS Wide QRS Narrow QRS Wide QRS Tachycardia Tachycardia Bradycardia Bradycardia Regular Irregular Regular Irregular Sinus Brady Idioventricular A-Fib / Flutter Bradycardia w/ BBB Sinus Tach A-Fib VT PVT Junctional 2 AVB / II PSVT A-Flutter SVT aberrant A-Fib 1 AVB 3 AVB A-Flutter MAT 2 AVB / I or II PAT PAT 3 AVB ST PAC / PVC Stability Hypotension / hypoperfusion Altered mental status Chest pain – Coronary ischemic Dyspnea – Pulmonary edema Sinus Rhythm Sinus Rhythm P Wave PR Interval QRS Rate Rhythm Pacemaker Comment . Before . Constant, . Rate 60-100 . Regular . SA Node Upright in each QRS regular . Interval =/< leads I, II, . Look . Interval .12- .10 & III alike .20 Conduction Image reference: Cardionetics/ http://www.cardionetics.com/docs/healthcr/ecg/arrhy/0100_bd.htm Sinus Pause A delay of activation within the atria for a period between 1.7 and 3 seconds A palpitation is likely to be felt by the patient as the sinus beat following the pause may be a heavy beat.
    [Show full text]
  • Common Types of Supraventricular Tachycardia: Diagnosis and Management RANDALL A
    Common Types of Supraventricular Tachycardia: Diagnosis and Management RANDALL A. COLUCCI, DO, MPH, Ohio University College of Osteopathic Medicine, Athens, Ohio MITCHELL J. SILVER, DO, McConnell Heart Hospital, Columbus, Ohio JAY SHUBROOK, DO, Ohio University College of Osteopathic Medicine, Athens, Ohio The most common types of supraventricular tachycardia are caused by a reentry phenomenon producing acceler- ated heart rates. Symptoms may include palpitations (including possible pulsations in the neck), chest pain, fatigue, lightheadedness or dizziness, and dyspnea. It is unusual for supraventricular tachycardia to be caused by structurally abnormal hearts. Diagnosis is often delayed because of the misdiagnosis of anxiety or panic disorder. Patient history is important in uncovering the diagnosis, whereas the physical examination may or may not be helpful. A Holter moni- tor or an event recorder is usually needed to capture the arrhythmia and confirm a diagnosis. Treatment consists of short-term or as-needed pharmacotherapy using calcium channel or beta blockers when vagal maneuvers fail to halt or slow the rhythm. In those who require long-term pharmacotherapy, atrioventricular nodal blocking agents or class Ic or III antiarrhythmics can be used; however, these agents should generally be managed by a cardiologist. Catheter ablation is an option in patients with persistent or recurrent supraventricular tachycardia who are unable to tolerate long-term pharmacologic treatment. If Wolff-Parkinson-White syndrome is present, expedient referral
    [Show full text]
  • Basic Cardiac Rhythms – Identification and Response Module 1 ANATOMY, PHYSIOLOGY, & ELECTRICAL CONDUCTION Objectives
    Basic Cardiac Rhythms – Identification and Response Module 1 ANATOMY, PHYSIOLOGY, & ELECTRICAL CONDUCTION Objectives ▪ Describe the normal cardiac anatomy and physiology and normal electrical conduction through the heart. ▪ Identify and relate waveforms to the cardiac cycle. Cardiac Anatomy ▪ 2 upper chambers ▪ Right and left atria ▪ 2 lower chambers ▪ Right and left ventricle ▪ 2 Atrioventricular valves (Mitral & Tricuspid) ▪ Open with ventricular diastole ▪ Close with ventricular systole ▪ 2 Semilunar Valves (Aortic & Pulmonic) ▪ Open with ventricular systole ▪ Open with ventricular diastole The Cardiovascular System ▪ Pulmonary Circulation ▪ Unoxygenated – right side of the heart ▪ Systemic Circulation ▪ Oxygenated – left side of the heart Anatomy Coronary Arteries How The Heart Works Anatomy Coronary Arteries ▪ 2 major vessels of the coronary circulation ▪ Left main coronary artery ▪ Left anterior descending and circumflex branches ▪ Right main coronary artery ▪ The left and right coronary arteries originate at the base of the aorta from openings called the coronary ostia behind the aortic valve leaflets. Physiology Blood Flow Unoxygenated blood flows from inferior and superior vena cava Right Atrium Tricuspid Valve Right Ventricle Pulmonic Valve Lungs Through Pulmonary system Physiology Blood Flow Oxygenated blood flows from the pulmonary veins Left Atrium Mitral Valve Left Ventricle Aortic Valve Systemic Circulation ▪ Blood Flow Through The Heart ▪ Cardiology Rap Physiology ▪ Cardiac cycle ▪ Represents the actual time sequence between
    [Show full text]
  • Acute Non-Specific Pericarditis R
    Postgrad Med J: first published as 10.1136/pgmj.43.502.534 on 1 August 1967. Downloaded from Postgrad. med. J. (August 1967) 43, 534-538. CURRENT SURVEY Acute non-specific pericarditis R. G. GOLD * M.B., B.S., M.RA.C.P., M.R.C.P. Senior Registrar, Cardiac Department, Brompton Hospital, London, S.W.3 Incidence neck, to either flank and frequently through to the Acute non-specific pericarditis (acute benign back. Occasionally pain is experienced on swallow- pericarditis; acute idiopathic pericarditis) has been ing (McGuire et al., 1954) and this was the pre- recognized for over 100 years (Christian, 1951). In senting symptom in one of our own patients. Mild 1942 Barnes & Burchell described fourteen cases attacks of premonitory chest pain may occur up to of the condition and since then several series of 4 weeks before the main onset of symptoms cases have been published (Krook, 1954; Scherl, (Martin, 1966). Malaise is very common, and is 1956; Swan, 1960; Martin, 1966; Logue & often severe and accompanied by listlessness and Wendkos, 1948). depression. The latter symptom is especially com- Until recently Swan's (1960) series of fourteen mon in patients suffering multiple relapses or patients was the largest collection of cases in this prolonged attacks, but is only partly related to the country. In 1966 Martin was able to collect most length of the illness and fluctuates markedly from of his nineteen cases within 1 year in a 550-bed day to day with the patient's general condition. hospital. The disease is thus by no means rare and Tachycardia occurs in almost every patient at warrants greater attention than has previously some stage of the illness.
    [Show full text]
  • Recurrent Takotsubo Cardiomyopathy: a Rare Diagnosis with a Common Emergency Department Presentation Emily M Miner, Harini Gurram, Tennie Renkens and Julie L
    Case Report iMedPub Journals ARCHIVES OF MEDICINE 2017 http://www.imedpub.com/ Vol.9 No.4:8 ISSN 1989-5216 DOI: 10.21767/1989-5216.1000229 Recurrent Takotsubo Cardiomyopathy: A Rare Diagnosis with a Common Emergency Department Presentation Emily M Miner, Harini Gurram, Tennie Renkens and Julie L. Welch* Department of Emergency Medicine, Indiana University School of Medicine, Indianapolis, Indiana, USA *Corresponding author: Julie L Welch, Department of Emergency Medicine, Indiana University School of Medicine, Indianapolis, Indiana, USA, Tel: 317-962-8880; E-mail: [email protected] Received date: August 08, 2017; Accepted date: August 14, 2017; Published date: August 16, 2017 Citation: Miner EM, Gurram H, Renkens T, Welch JL. Recurrent Takotsubo Cardiomyopathy: A Rare Diagnosis with a Common Emergency Department Presentation. Arch Med. 2017, 9:4 Copyright: © 2017 Miner EM, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. The incidence of recurrent Takotsubo cardiomyopathy after initial diagnosis is about 5% at 6 years with recurrence being Abstract more common when the initial episode has more severe left ventricular dysfunction [4]. While the long-term prognosis of Takotsubo cardiomyopathy is a non-ischemic Takotsubo is favorable in >95% of cases, acute symptoms of an cardiomyopathy that is often triggered by a physical or episode can be life-threatening and lead to cardiogenic shock emotional stressor and commonly affects post-menopausal [5]. Proper diagnosis is crucial in these life-threatening women. A 57 year old female with a significant past medical history for atrial fibrillation, anxiety, and Takotsubo situations.
    [Show full text]