Main Panel B

Total Page:16

File Type:pdf, Size:1020Kb

Main Panel B Panel B Main Panel B Chair Professor Robert Stout Queen’s University Belfast Members Professor Ray Fitzpatrick University of Oxford Professor Philip Hannaford University of Aberdeen Professor Fritz Henn Brookhaven National Laboratory, USA Professor Jeffrey Koplan Woodruff Health Sciences Center, Atlanta, USA Dr Ruairidh Milne University of Southampton Professor Peter McGuffin King’s College London Professor Alan Silman University of Manchester Professor Chris van Weel Radboud University, Nijmegen, Netherlands Observers Professor Lord Lesley Turnberg Association of Medical Research Charities Dr Chris Watkins Medical Research Council Secretariat Mrs Lois Neal University of Newcastle upon Tyne Ms Milly Bodfish University of Cambridge 12 RAE 03/2007 Panel B Sub-panel 6 – Epidemiology and Public Health Chair Professor Alan Silman University of Manchester Members Dr David Armstrong King’s College London Professor Valerie Beral University of Oxford Professor Kar Keung Cheng University of Birmingham Professor David Coggon University of Southampton Professor George Davey Smith University of Bristol Professor Alun Evans Queen’s University Belfast Professor Sian Griffiths The Chinese University of Hong Kong Professor Ian Harvey University of East Anglia Professor Anne Johnson University College London Professor David Leon London School of Hygiene & Tropical Medicine Professor Stephen Palmer Cardiff University Professor Peter Smith London School of Hygiene & Tropical Medicine Professor Simon Thompson Medical Research Council Professor Graham Watt University of Glasgow RAE 03/2007 13 Panel B Sub-panel 7 – Health Services Research Chair Professor Ray Fitzpatrick University of Oxford Members Professor Bipin Bhakta University of Leeds Professor John Bligh Peninsula Medical School, Universities of Exeter and Plymouth Professor Ann Bowling University College London Professor Paul Dieppe University of Bristol Professor Jenny Donovan University of Bristol Professor Adrian Grant University of Aberdeen Professor John Harris University of Manchester Professor David Jones University of Leicester Professor Peter Littlejohns National Institute for Health and Clinical Excellence (NICE) Professor Anne Mills London School of Hygiene & Tropical Medicine Professor Jonathan Nicholl University of Sheffield Professor Mandy Ryan University of Aberdeen Professor Trevor Sheldon University of York Professor Andrew Stevens University of Birmingham Professor Tom Walley University of Liverpool Professor J Williams Swansea University 14 RAE 03/2007 Panel B Sub-panel 8 – Primary Care and Other Community Based Clinical Subjects Chair Professor Philip Hannaford University of Aberdeen Members Professor Anthony Avery University of Nottingham Professor Nicky Britten Peninsula Medical School, Universities of Exeter and Plymouth Professor Christopher Dowrick University of Liverpool Professor Christopher Dunn University of the West of England, Bristol Professor Elaine Hay Keele University Professor Paul Little University of Southampton Professor David Mant University of Oxford Professor Tim Peters University of Bristol Professor Martin Roland University of Manchester Professor Sally Wyke University of Stirling RAE 03/2007 15 Panel B Sub-panel 9 – Psychiatry, Neuroscience and Clinical Psychology Chair Professor Peter McGuffin King’s College London Members Professor Anthony Bell St George’s Hospital Medical School Professor Brendan Bradley University of Southampton Professor Traolach Brugha University of Leicester Professor Graham Collingridge University of Bristol Professor Alastair Compston University of Cambridge Professor Sally-Ann Cooper University of Glasgow Professor Ian Ferrier University of Newcastle upon Tyne Professor Richard Frackowiak University College London Professor Paul Grasby Imperial College London Professor Chris Hollis University of Nottingham Professor Richard Morris University of Edinburgh Professor Michael Owen Cardiff University Professor Trevor Robbins University of Cambridge Professor Graham Turpin University of Sheffield Professor Til Wykes King’s College London 16 RAE 03/2007.
Recommended publications
  • Current Strategies for Investigating the Genetic and Environmental Risk
    BRITISH JOURNAL OF PSYCHIATRY (2005), 186, 179^181 EDITORIAL Current strategies for investigating the genetic and positional candidate gene G72G72 in bothinboth disorders (Elkin et aletal, 2004). Thus, the environmental risk factors for affective disorders* molecular and the quantitative genetic findings appear to be convergent in suggest- ing three sets of genes: one conferring liab- ANNE FARMER, THALIA C. ELEY and PETER McGUFFIN ility to both schizophrenia and bipolar disorder, and two that are specific for each of the two main Kraepelinian syndromes. Interestingly, in the quantitative analyses the environmental risk factors appeared to be specific to each psychotic disorder. A rather different type of analysis seeks It is probable that the genetic components schemes of subtyping are of any use (Ken- to tease out dimensions within the broad of affective disorders (bipolar affective dell, 1976; Farmer & McGuffin, 1989). category of recurrent depression. Using disorder, major depressive disorder and One typology that has stood the test of time phenotypic data from participants in cur- anxiety states) result from multiple genes and seems clinically useful is the unipolar/ rent large-scale genetic studies of depres- that confer a susceptibility or liability to bipolar subdivision. Until recently, a sion (see below), this has produced some develop the disorder when other (environ- common view (Gershon et aletal, 1982) was interesting early results. Factor analysis of mental) risk factors are also present. In that bipolar affective disorder and major psychopathology from worst and second- general, bipolar affective disorder has been depressive disorder lie on the same severity worst episodes of depression in sibling pairs found to have the highest heritability (i.e.
    [Show full text]
  • A Breath of Fresh Air
    European Journal of Human Genetics (2006) 14, 1145–1146 & 2006 Nature Publishing Group All rights reserved 1018-4813/06 $30.00 www.nature.com/ejhg familiar with genetic terminology. Chap- BOOK REVIEW ter references are indicated with an * or a diamond } depending on relevance and an excellent way to find a good reference A breath of fresh air quickly. ...................................... I enjoyed the miscellaneous section Respiratory Genetics most – covering rare diseases and syn- dromes. It is up to date and includes EK Silverman, SD Shapiro, DA Lomas, ST Weiss. Hodder Arnold, 2005. discussion of causes of hereditary and ........... sporadic pneumothorax including certain Patrick J Morrison mutations in Birt-Hogg-Dube´ syndrome – ......................................................................... something that was not really recognised until recently. European Journal of Human Genetics (2006) 14, 1145 . There are only six colour plates – doi:10.1038/sj.ejhg.5201672 disappointing in a book of this size but overall, this is a great book for respiratory and interested general physicians. General his is an excellent new textbook to carry out a good Pub Med search, and a clinical geneticists will find it useful to on respiratory genetics – an area succinct guide to SNPs and microarrays. have a copy in the library for reference, that has suddenly emerged over Other chapters cover pharmacokinetics and may find their laboratory colleagues T the last decade. and functional genomics. having a look at the more clinical sec- It is well written and divided into four Parts 2–4 cover three clinical respira- tions. This book should become the clear sections: tory disease sections – Part 2 covers standard reference text in this area’ Part 1 deals with key concepts in obstructive lung disease – asthma, chronic respiratory genetics – this section is tar- pulmonary disease and cystic fibrosis are Patrick J Morrison is a Professor in the geted at nongeneticists and will not be of covered comprehensively.
    [Show full text]
  • Eliot Slater's Contribution
    Causes of common mental health problems in adults Peter McGuffin MRC SGDP Centre Institute of Psychiatry, King‟s College London Sir Francis Galton (1822-1911) „…Nature prevails enormously over nurture‟(1883) The history of twins as a criterion of the relative powers of nature and nurture (1876) „..a born devil,on who‟s nature, nurture cannot stick.‟ Prospero of Caliban in Shakespeare‟s „The Tempest‟ Act iv scene i (Michael Horton as Caliban) Psychiatrists‟ opening gambits 1 Have you suffered vexation, grief or reverse of fortune? Phillipe Pinel (quoted by Sir Michael Rutter) Psychiatrists‟ opening gambits 2 Are you a twin? Eliot Slater (quoted by Sir Denis Hill) Excerpt from a Bethlem Royal Hospital front sheet 1823 Cardiff Study of Depression in Siblings (Farmer et al 2000) % reported % CATEGO current past cases D-siblings 7.4 17.6 18.5 C-siblings 0 4.8 1.9 Behaviours that run in families . Huntington‟s disease . Alzheimer‟s disease . Depression . Schizophrenia . Personality . Intelligence . Religious involvement . Attending medical school Why might a disorder run in families? . Shared genes . Shared environment . A combination of the two behaviour Natural experiments teasing apart genes and environment Twin studies : is there more similarity monozygotic ( one egg) than dizygotic ( two egg) pairs? Adoption studies: do individuals resemble their biological relatives more than adopting relatives? The Cholmondeley Ladies c.1600-10 MZ TWINS . MZ (monozygotic) twins have 100% of their genes in common (they‟re „natural clones‟) . Shared environment
    [Show full text]
  • A Point of Rarity in Genetic Risk for Bipolar Disorder and Schizophrenia
    ORIGINAL ARTICLE Rare Copy Number Variants A Point of Rarity in Genetic Risk for Bipolar Disorder and Schizophrenia Detelina Grozeva, MSc; George Kirov, PhD, MRCPsych; Dobril Ivanov, MSc; Ian R. Jones, PhD, MRCPsych; Lisa Jones, PhD; Elaine K. Green, PhD; David M. St Clair, MD, PhD; Allan H. Young, PhD, FRCPsych; Nicol Ferrier, PhD, FRCPsych; Anne E. Farmer, PhD, FRCPsych; Peter McGuffin, PhD, FRCPsych; Peter A. Holmans, PhD*; Michael J. Owen, PhD, FRCPsych*; Michael C. O’Donovan, PhD, FRCPsych*; Nick Craddock, PhD, FRCPsych*; for the Wellcome Trust Case Control Consortium Context: Recent studies suggest that copy number varia- Main Outcome Measures: Overall load of CNVs and tion in the human genome is extensive and may play an presence of rare CNVs. important role in susceptibility to disease, including neu- ropsychiatric disorders such as schizophrenia and autism. Results: The burden of CNVs in bipolar disorder was The possible involvement of copy number variants (CNVs) not increased compared with controls and was signifi- in bipolar disorder has received little attention to date. cantly less than in schizophrenia cases. The CNVs pre- viously implicated in the etiology of schizophrenia were Objectives: To determine whether large (Ͼ100 000 base not more common in cases with bipolar disorder. pairs) and rare (found in Ͻ1% of the population) CNVs are associated with susceptibility to bipolar disorder and Conclusions: Schizophrenia and bipolar disorder dif- to compare with findings in schizophrenia. fer with respect to CNV burden in general and associa- tion with specific CNVs in particular. Our data are con- Design: A genome-wide survey of large, rare CNVs in a sistent with the possibility that possession of large, rare case-control sample using a high-density microarray.
    [Show full text]
  • A Comparison of Ten Polygenic Score Methods for Psychiatric Disorders Applied Across Multiple Cohorts
    medRxiv preprint doi: https://doi.org/10.1101/2020.09.10.20192310; this version posted May 5, 2021. The copyright holder for this preprint (which was not certified by peer review) is the author/funder, who has granted medRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC-ND 4.0 International license . A comparison of ten polygenic score methods for psychiatric disorders applied across multiple cohorts Authors: Guiyan Ni,1 Jian Zeng,1 Joana A Revez,1 Ying Wang,1 Zhili Zheng,1 Tian Ge,2 Restuadi Restuadi,1 Jacqueline Kiewa,1 Dale R Nyholt,3 Jonathan R I Coleman,4 Jordan W Smoller,2,5,6 Schizophrenia Working Group of the Psychiatric Genomics Consortium,7 Major Depressive Disorder Working Group of the Psychiatric Genomics Consortium,8 Jian Yang,1,9 Peter M Visscher,1 Naomi R Wray1,10 1. Institute for Molecular Bioscience, University of Queensland, Brisbane, Queensland, 4072, Australia 2. Psychiatric and Neurodevelopmental Genetics Unit (PNGU), Massachusetts General Hospital, Boston, MA, 02114, US 3. Faculty of Health, School of Biomedical Sciences, Centre for Genomics and Personalised Health, Queensland University of Technology, Brisbane, Queensland, 4000, Australia 4. Social, Genetic, and Developmental Psychiatry Centre, Institute of Psychiatry, Psychology, and Neuroscience, King's College London, London, SE58AF United Kingdom 5. Department of Psychiatry, Massachusetts General Hospital, Boston, MA, 02114, US 6. Stanley Center for Psychiatric Research, Broad Institute, Cambridge, MA, 02142, US 7. A list of members and affiliations appears in the Supplementary Data. 8. A list of members and affiliations appears in the Supplementary Data.
    [Show full text]
  • Training and Engaging in Research When You Are a Clinician
    Training and Engaging in Research when you are a clinician Anita Thapar Professor of Child & Adolescent Psychiatry, School of Medicine Overview • Background introduction • Why do research? • What should SpRs do? • …and consultants Background introduction Back to 1980-85 At graduation • Clinical Child and Adolescent Psychiatry • … Research? What is that? Teaching hospital training? First exposure to research 1988/9 Peter McGuffin brings Psychiatric Genetics to Cardiff What captured me? • Senior figures who were enthusiastic about research • It seemed like fun • Interesting to think • Directly supportive in practical ways Clinical research training fellowship PhD • Visits to IOP Michael Rutter-Child Psychiatry at Institute of Psychiatry, London Manchester The late Richard Harrington Professor of Child & Adolescent Psychiatry Return to Cardiff 1999 Why do research if you are an NHS clinician? Professor of Child & Adolescent Psychiatry Why do research if you are an NHS clinician? Why do research? For you • Learn to appreciate research methods, be critical of evidence and develop an enquiring approach to clinical work • Enhances interest in clinical work • Using your scientific background Why do research? For you • Do something different to direct clinical work • “Wonderful to have a change” from my routine clinical work • “Keeps my brain active” • Provides perspective Why do research? For you • It can be fun even if you think you are not a born researcher • Team working • Keeps one interested and mentally engaged as keeps changing Why do research?
    [Show full text]
  • Sensation-Seeking, Life Events and Depression the Cardiff Depression
    BRITISH JOURNAL OF PSYCHIATRY )2001), 178, 549^552 Sensation-seeking, life events and depression no current or past history of depression and had a sibling C-sib) who was willing The Cardiff Depression Study to participate in the study. Wherever possible, the sibling nearest in age to the D-proband or the C-proband was studied. ANNE FARMER, KATE REDMAN, TANYA HARRIS, ARSHAD MAHMOOD, However, if this sibling was unavailable STEPHANIE SADLER and PETER MCCGUFFIN or unwilling, the sibling next in age was asked to participate. Most interviews were conducted face to face but for 18.5% of the D-sibs and 33.3% of the C-sibs telephone interviews were undertaken. Background The relationship Adverse events are associated with the onset between adversityandgeneticriskadversityandgenetic risk factors of depressive episodes Brown & Harris, Interviews and self-rating 1978; Paykel, 1978) and there is evidence questionnaires in depression could be mediated by familial that both depression and adversity are All subjects were interviewed using the `hazard prone'traits, as reflectedin high familial and genetically influenced Plomin Schedules for Clinical Assessment in levels of sensation-seeking. & Bergman, 1991; Thapar & McGuffin, Neuropsychiatry SCAN; Wing et aletal,, 1996). This familial relationship could be 1990) and the Life Events and Difficulties Aims To eexamine x a min e wwhether h e t h e r highigh h mediated by aspects of personality that also Schedule LEDS; Brown & Harris, 1978). sensation-seeking scores are associated run in families. For example, certain person- The severity and threat of events scored with more adverse life events resultingresultingin in ality characteristics could lead to risk-taking on the LEDS were rated contextually by depression.depression.
    [Show full text]
  • A Point of Rarity in Genetic Risk for Bipolar Disorder and Schizophrenia
    ORIGINAL ARTICLE Rare Copy Number Variants A Point of Rarity in Genetic Risk for Bipolar Disorder and Schizophrenia Detelina Grozeva, MSc; George Kirov, PhD, MRCPsych; Dobril Ivanov, MSc; Ian R. Jones, PhD, MRCPsych; Lisa Jones, PhD; Elaine K. Green, PhD; David M. St Clair, MD, PhD; Allan H. Young, PhD, FRCPsych; Nicol Ferrier, PhD, FRCPsych; Anne E. Farmer, PhD, FRCPsych; Peter McGuffin, PhD, FRCPsych; Peter A. Holmans, PhD*; Michael J. Owen, PhD, FRCPsych*; Michael C. O’Donovan, PhD, FRCPsych*; Nick Craddock, PhD, FRCPsych*; for the Wellcome Trust Case Control Consortium Context: Recent studies suggest that copy number varia- Main Outcome Measures: Overall load of CNVs and tion in the human genome is extensive and may play an presence of rare CNVs. important role in susceptibility to disease, including neu- ropsychiatric disorders such as schizophrenia and autism. Results: The burden of CNVs in bipolar disorder was The possible involvement of copy number variants (CNVs) not increased compared with controls and was signifi- in bipolar disorder has received little attention to date. cantly less than in schizophrenia cases. The CNVs pre- viously implicated in the etiology of schizophrenia were Objectives: To determine whether large (Ͼ100 000 base not more common in cases with bipolar disorder. pairs) and rare (found in Ͻ1% of the population) CNVs are associated with susceptibility to bipolar disorder and Conclusions: Schizophrenia and bipolar disorder dif- to compare with findings in schizophrenia. fer with respect to CNV burden in general and associa- tion with specific CNVs in particular. Our data are con- Design: A genome-wide survey of large, rare CNVs in a sistent with the possibility that possession of large, rare case-control sample using a high-density microarray.
    [Show full text]
  • Rebecca Georgina Smith
    REBECCA GEORGINA SMITH CONTACT INFORMATION Work Home C2.26 MRC SGDP Centre, 1 Prior’s Way, Institute of Psychiatry, Psychology and Cowden, Neuroscience Kent, King’s College London, TN8 7HX London, SE5 8AF Phone Number: 07595 232 708 Phone Number: 020 7848 5271 Email: [email protected] BIOGRAPHY I earned a BSc (Hons) in Genetics from the University of Leeds which was completed in 2005. In 2007, I began working at the Social, Genetic and Developmental Psychiatry Centre (SGDP), King’s College London as a research assistant working under Prof Ian Craig, Dr Katherine Aitchison and Prof Peter McGuffin on the GENDEP project in which I performed genotyping of genetic polymorphisms such as SNPs and VNTRs in a large unipolar depression cohort. In 2009, I started my PhD at the SGDP in psychiatric genetics with Prof Jonathan Mill and Prof Ian Craig working on my thesis investigating behavioural and molecular changes associated with advanced paternal age in a mouse model, and also contributing to work on allele-specific methylation and the role of epigenetics in psychiatric disease. I completed my first post-doctoral research position at the SGDP with Dr Katie Lunnon and Prof Jonathan Mill investigating epigenetic changes associated with Alzheimer’s disease in a post-mortem brain sample, as well as continuing my research in paternal age and psychiatric disease with Dr Cathy Fernandes and Dr Abraham Reichenberg. My second post-doctoral position was working on a genome-wide epigenetic project investigating DNA methylation changes and differences at birth and in childhood, for children with different trajectories of conduct problems.
    [Show full text]
  • WORKING TOGETHER Co Kilkenny
    DATES FOR YOUR DIARY 2017 17TH FEBRUARY: NCHD Conference, Dublin “The Science Of Psychiatry” International Conference of the College of Psychiatrists of Ireland 6TH & 7TH APRIL: & the Royal College of Psychiatrists College of Psychiatrists in Northern Ireland of Ireland Spring Conference WORKING TOGETHER Co Kilkenny “Psychiatry In The Digital Age” SLIEVE RUSSELL HOTEL, CAVAN 10th & 11th NOVEMBER 2016 Name: College of Psychiatrists of Ireland 5 Herbert Street, Dublin 2 Tel: 01 661 8450 | Fax: 01 685 4291 ........................................................................................ www.irishpsychiatry.ie Approved for 6 External CPD credits per day POSTER PRESENTATION OF WELCOME November 2016 Dear colleagues It is our great pleasure to welcome you to the Slieve Russell Hotel for the joint conference of the College of Psychiatrists of Ireland and the Royal College of Psychiatrists in Northern Ireland. This conference is an opportunity for the psychiatrists on this island (and indeed those visiting) to get together, make new friends, meet colleagues and learn from each other. Though we are dealing with two different health systems, we can share our experiences and try to pool our resources for the benefit of our patients. There is no doubt that this is a trying and difficult time for psychiatry and for those with mental health difficulties. It is by working together that we can at least reduce some of the stresses that our patients face in attempting to obtain adequate mental health care. With all this in mind, the conference looks at some resource poor areas, such as services for eating disorders and early intervention for psychosis. There is a consensus among many psychiatrists that these challenges have a better chance of developing if they are dealt with on an “all island” basis.
    [Show full text]
  • Rare Copy Number Variants a Point of Rarity in Genetic Risk for Bipolar Disorder and Schizophrenia
    ORIGINAL ARTICLE Rare Copy Number Variants A Point of Rarity in Genetic Risk for Bipolar Disorder and Schizophrenia Detelina Grozeva, MSc; George Kirov, PhD, MRCPsych; Dobril Ivanov, MSc; Ian R. Jones, PhD, MRCPsych; Lisa Jones, PhD; Elaine K. Green, PhD; David M. St Clair, MD, PhD; Allan H. Young, PhD, FRCPsych; Nicol Ferrier, PhD, FRCPsych; Anne E. Farmer, PhD, FRCPsych; Peter McGuffin, PhD, FRCPsych; Peter A. Holmans, PhD*; Michael J. Owen, PhD, FRCPsych*; Michael C. O’Donovan, PhD, FRCPsych*; Nick Craddock, PhD, FRCPsych*; for the Wellcome Trust Case Control Consortium Context: Recent studies suggest that copy number varia- Main Outcome Measures: Overall load of CNVs and tion in the human genome is extensive and may play an presence of rare CNVs. important role in susceptibility to disease, including neu- ropsychiatric disorders such as schizophrenia and autism. Results: The burden of CNVs in bipolar disorder was The possible involvement of copy number variants (CNVs) not increased compared with controls and was signifi- in bipolar disorder has received little attention to date. cantly less than in schizophrenia cases. The CNVs pre- viously implicated in the etiology of schizophrenia were Objectives: To determine whether large (Ͼ100 000 base not more common in cases with bipolar disorder. pairs) and rare (found in Ͻ1% of the population) CNVs are associated with susceptibility to bipolar disorder and Conclusions: Schizophrenia and bipolar disorder dif- to compare with findings in schizophrenia. fer with respect to CNV burden in general and associa- tion with specific CNVs in particular. Our data are con- Design: A genome-wide survey of large, rare CNVs in a sistent with the possibility that possession of large, rare case-control sample using a high-density microarray.
    [Show full text]
  • Genome-Wide Association Study of Suicide Attempt in Psychiatric Disorders Identifies Association with Major Depression Polygenic Risk Scores
    bioRxiv preprint doi: https://doi.org/10.1101/416008; this version posted September 14, 2018. The copyright holder for this preprint (which was not certified by peer review) is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under aCC-BY-NC-ND 4.0 International license. Genome-wide association study of suicide attempt in psychiatric disorders identifies association with major depression polygenic risk scores Authors Niamh Mullins 1, 2, Tim B. Bigdeli 3, 4, Anders D Børglum 5, 6, 7, Jonathan R I Coleman 1, Ditte Demontis 5, 6, 7, Ayman H. Fanous 3, 4, Divya Mehta 8, 9, Robert A. Power 1, Stephan Ripke 10, 11, 12, Eli A Stahl 12, 13, 14, Anna Starnawska 5, 6, 7, Adebayo Anjorin 15, Aiden Corvin 16, Alan R Sanders 17, 18, Andreas J Forstner 19, 20, 21, 22, 23, Andreas Reif 24, Anna C Koller 22, 23, Beata Świątkowska 25, Bernhard T Baune 26, Bertram Müller- Myhsok 27, 28, 29, Bettina Konte 30, Brenda WJH Penninx 31, Carlos Pato 32, 33, Clement Zai 34, Dan Rujescu 30, 35, Digby Quested 36, Douglas F Levinson 37, Elisabeth B Binder 28, 38, Enda M Byrne 39, Esben Agerbo 7, 40, 41, Fabian Streit 42, Fermin Mayoral 43, Frank Bellivier 44, 45, 46, 47, Franziska Degenhardt 22, 23, Gerome Breen 1, 48, Gunnar Morken 49, 50, Gustavo Turecki 51, Guy A Rouleau 52, 53, Hans J Grabe 54, Henry Völzke 55, Ina Giegling 30, 35, Ingrid Agartz 56, 57, 58, Ingrid Melle 59, 60, Jacob Lawrence 61, James B Potash 62, James TR Walters 63, Jana Strohmaier 42, Jianxin Shi 64, Joanna Hauser 65, Joanna M Biernacka 66, John B Vincent 34, John Kelsoe 67, John S Strauss 34, 68, Jolanta Lissowska 69, Jonathan Pimm 70, Jordan W Smoller 71, 72, 73, José Guzman Parra 43, Klaus Berger 74, Laura J Scott 75, M.
    [Show full text]