Colorado 4Th Tier Medication List

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Colorado 4th Tier Medication List Brand Name Generic Name Drug Category Actemra Tocilizumab Immunomodulator Acthar HP Corticotropin Endocrine agent Actimmune Interferon gamma 1b Immunomodulator Alferon Interferon alfa N3 Immunomodulator Amevive Alefacept Immunomodulator Ampyra Dalfampridine Potassium channel blocker (multiple sclerosis) Aranesp Darbepoetin Hematopoietic Arixtra Fondiparinux Anticoagulant Avonex, Rebif Interferon beta-1a Immunomodulator (multiple sclerosis) Betaseron, Extavia Interferon beta-1b Immunomodulator (multiple sclerosis) Cimzia Certolizumab Immunomodulator Copaxone Glatiramer acetate Immunomodulator (multiple sclerosis) Egrifta Tesamorelin Endocrine agent Enbrel Etanercept Immunomodulator Epogen, Procrit Erythropoetin Hematopoietic Extavia Interferon beta-1b Immunomodulator (multiple sclerosis) Forteo Teriparatide Parathyroid hormone Fragmin Dalteparin Anticoagulant (low molecular weight heparin) Fuzeon Enfuvirtide Antiretroviral Gilenya Fingolimod Immunomodulator (multiple sclerosis) Genotropin, Humatrope, Norditropin, Somatropin Growth hormone Nutropin, Nutropin AQ, Nutropin Depot, Omnitrope, Saizen, Serostim, Serostim LQ, Tev-Tropin, Zorbtive Humira Adalimumab Immunomodulator Incivek Telaprevir Antiviral (Hepatitis C) Infergen Interferon alfacon-1 Immunomodulator Innohep Tinzaparin Anticoagulant (low molecular weight heparin) Intron-A Interferon alfa 2b Immunomodulator Kineret Anakinra Immunomodulator Leukine Sargramostim (GM-CSF) Hematopoietic Lovenox, Enoxaparin Enoxaparin Anticoagulant (low molecular weight heparin) Colorado 4th Tier Medication List Brand Name Generic Name Drug Category Makena Hydroxyprogesterone Caproate Endocrine agent (pre-term birth) Miacalcin injection Calcitonin Polypeptide hormone (bone loss prevention) Neulasta Pegfilgrastim Hematopoietic Neumega Oprelvekin Hematopoietic Neupogen Filgrastim (G-CSF) Hematopoietic Orencia Abatacept Immunomodulator Pegasys Peginterferon alfa 2a Immunomodulator Peg-Intron Peginterferon alfa 2b Immunomodulator Prolia Denosumab Osteoporosis agent Remicade Infliximab Immunomodulator Roferon-A Interferon alfa 2a Immunomodulator Simponi Golimumab Immunomodulator Somavert Pegvisomant Growth hormone antagonist Stelara Ustekinumab Immunomodulator Sylatron Pebinterferon alfa 2b Anticancer agent (melanoma) Thyrogen Thyrotropin alfa Diagnostic aid (thyroid function) Victrelis Boceprevir Antiviral (Hepatitis C) Xgeva Denosumab Immunomodulator Xolair Omalizumab Monoclonal antibody (anti-asthmatic) Updated as of July 2011. This list is subject to change. For the most recent list, please go to Anthem.com or contact Customer Service at the number on your ID card. Inclusion of a drug on this list is not a guarantee of coverage and certain drugs may require Prior Authorization. Please refer to the member’s certificate of coverage. .

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STIM1 Controls Calcineurin/Akt/Mtor/NFATC2‑Mediated Osteoclastogenesis Induced by RANKL/M‑CSF
736 EXPERIMENTAL AND THERAPEUTIC MEDICINE 20: 736-747, 2020 STIM1 controls calcineurin/Akt/mTOR/NFATC2‑mediated osteoclastogenesis induced by RANKL/M‑CSF YANJIAO HUANG1, QIANG LI2, ZUNYONG FENG3 and LANRONG ZHENG1 Departments of 1Pathological Anatomy, 2Anatomy and 3Forensic Medicine, Wannan Medical College, Wuhu, Anhui 241002, P.R. China Received October 15, 2018; Accepted June 20, 2019 DOI: 10.3892/etm.2020.8774 Abstract. Store-operated Ca2+ entry (SOCE) is the stable (p.E136X, p.R429C) or constitutively activated (p.R304W) calcium channel influx in most cells. It consists of the SOCE, failed to respond to RANKL/M-CSF-mediated induc- cytoplasmic ion channel ORAI and endoplasmic reticulum tion of normal osteoclastogenesis. In addition, activation of the receptor stromal interaction molecule 1 (STIM1). Abolition calcineurin/Akt/mTOR/NFATC2 signalling cascade induced of SOCE function due to ORAI1 and STIM1 gene defects by RANKL/M-CSF was abnormal in the BMDMs with may cause non-perspiration, ectoderm dysplasia and STIM1 mutants compared with that in BMDMs from healthy skeletal malformations with severe combined immunodefi- subjects. In addition, overexpression of wild-type STIM1 ciency (CID). Calcineurin/mammalian target of rapamycin restored SOCE in p.R429C- and p.E136X-mutant BMDMs, but (mTOR)/nuclear factor of activated T cells 2 (NFATC2) is not in p.R304W-mutant BMDMs. Of note, calcineurin, cyclo- an important signalling cascade for osteoclast development. sporin A, mTOR inhibitor rapamycin and NFATC2‑specific Calcineurin is activated by Ca2+ via SOCE during osteoclas- small interfering RNA restored the function of SOCE in togenesis, which is induced by receptor activator of NF-κB p.R304W-mutant BMDMs.
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Denosumab and Anti-Angiogenetic Drug-Related Osteonecrosis of the Jaw: an Uncommon but Potentially Severe Disease
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Managing Osteoporosis and Joint Damage in Patients with Rheumatoid Arthritis: an Overview
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Paracrine and Endocrine Actions of Boneâ€”The Functions of Secretory
Bone Research www.nature.com/boneres REVIEW ARTICLE OPEN Paracrine and endocrine actions of bone—the functions of secretory proteins from osteoblasts, osteocytes, and osteoclasts Yujiao Han1, Xiuling You1, Wenhui Xing1, Zhong Zhang1 and Weiguo Zou 1 The skeleton is a dynamic organ that is constantly remodeled. Proteins secreted from bone cells, namely osteoblasts, osteocytes, and osteoclasts exert regulation on osteoblastogenesis, osteclastogenesis, and angiogenesis in a paracrine manner. Osteoblasts secrete a range of different molecules including RANKL/OPG, M-CSF, SEMA3A, WNT5A, and WNT16 that regulate osteoclastogenesis. Osteoblasts also produce VEGFA that stimulates osteoblastogenesis and angiogenesis. Osteocytes produce sclerostin (SOST) that inhibits osteoblast differentiation and promotes osteoclast differentiation. Osteoclasts secrete factors including BMP6, CTHRC1, EFNB2, S1P, WNT10B, SEMA4D, and CT-1 that act on osteoblasts and osteocytes, and thereby inﬂuenceaA osteogenesis. Osteoclast precursors produce the angiogenic factor PDGF-BB to promote the formation of Type H vessels, which then stimulate osteoblastogenesis. Besides, the evidences over the past decades show that at least three hormones or “osteokines” from bone cells have endocrine functions. FGF23 is produced by osteoblasts and osteocytes and can regulate phosphate metabolism. Osteocalcin (OCN) secreted by osteoblasts regulates systemic glucose and energy metabolism, reproduction, and cognition. Lipocalin-2 (LCN2) is secreted by osteoblasts and can inﬂuence energy metabolism by suppressing appetite in the brain. We review the recent progresses in the paracrine and endocrine functions of the secretory proteins of osteoblasts, osteocytes, and osteoclasts, revealing connections of the skeleton with other tissues and providing added insights into the pathogenesis of degenerative diseases affecting multiple organs and the drug discovery process.
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Denosumab, Cortical Bone and Bone Erosions in Rheumatoid Arthritis
ARD Online First, published on June 23, 2016 as 10.1136/annrheumdis-2016-210022 Correspondence Ann Rheum Dis: first published as 10.1136/annrheumdis-2016-210022 on 23 June 2016. Downloaded from Denosumab, cortical bone and bone erosions University of Verona, Policlinico Borgo Roma, Piazzale Scuro, 10, Verona 37134, in rheumatoid arthritis Italy; [email protected] Competing interests None declared. Ta ke u c h i et al1 demonstrate an additional inhibitory effect on the Provenance and peer review Not commissioned; internally peer reviewed. progression of bone erosions in rheumatoid arthritis (RA) with the To cite Rossini M, Adami G, Viapiana O, et al. Ann Rheum Dis Published Online addition to methotrexate of an anti-receptor activator of nuclear First: [please include Day Month Year] doi:10.1136/annrheumdis-2016-210022 factor kappa-B ligand (RANKL) antibody, denosumab, conﬁrming Accepted 6 June 2016 2 results already reported 8 years ago by Cohen and colleagues. Ann Rheum Dis 2016;0:1. doi:10.1136/annrheumdis-2016-210022 Insufﬁcient inhibition of structural joint damage by disease- modifying antirheumatic drugs (DMARDs) was reported despite – clinical improvement,3 5 and recently two meta-analysis67showed REFERENCES 1 Takeuchi T, Tanaka Y, Ishiguro N, et al. Effect of denosumab on Japanese patients that methotrexate combined with biologics is statistically superior with rheumatoid arthritis: a dose-response study of AMG 162 (Denosumab) in to methotrexate in inhibiting radiographic progression, but the patients with RheumatoId arthritis on methotrexate to Validate inhibitory effect on estimated mean change with all treatments was less than the bone Erosion (DRIVE)-a 12-month, multicentre, randomised, double-blind, minimal clinically important difference.
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THE CONCISE GUIDE to PHARMACOLOGY 2017/18 Alexander, Stephen P
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Osteonecrosis of the Jaws Produced by Sunitinib
Med Oral Patol Oral Cir Bucal. 2019 May 1;24 (3):e326-38. Osteonecrosis of the jaws produced by sunitinib Journal section: Oral Medicine and Pathology doi:10.4317/medoral.22858 Publication Types: Review http://dx.doi.org/doi:10.4317/medoral.22858 Osteonecrosis of the jaws produced by sunitinib: a systematic review Carmen Vallina 1, Lucía Ramírez 2, Jesús Torres 3, Elisabeth Casañas 4, Gonzalo Hernández 5, Rosa-María López-Pintor 6 1 DDS, Oral Medicine Postgraduate. Department of Dental Clinical Specialties. School of Dentistry. Complutense University, Madrid, Spain 2 DDS, PhD Student, Oral Medicine Postgraduate. Department of Dental Clinical Specialties. School of Dentistry. Complutense University, Madrid, Spain 3 DDS, PhD, Professor. Department of Dental Clinical Specialties. School of Dentistry. Complutense University, Madrid, Spain 4 DDS, PhD, Researcher, Oral Medicine Postgraduate. Department of Dental Clinical Specialties. School of Dentistry. Com- plutense University, Madrid, Spain 5 MD, DDS, PhD, Professor, Director Specialty in Oral Medicine. Department of Dental Clinical Specialties. School of Dentistry. Complutense University, Madrid, Spain 6 DDS, PhD, Associate Professor, Co-director Speciality in Oral Medicine. Department of Dental Clinical Specialties. School of Dentistry. Complutense University, Madrid, Spain Correspondence: Departamento de Especialidades Clínicas Odontológicas Facultad de Odontología Universidad Complutense de Madrid Plaza Ramón y Cajal S/N 28040-Madrid Spain Vallina C, Ramírez L, Torres J, Casañas E, Hernández G, López-Pintor [email protected] RM. Osteonecrosis of the jaws produced by sunitinib: a systematic re- view. Med Oral Patol Oral Cir Bucal. 2019 May 1;24 (3):e326-38. http://www.medicinaoral.com/medoralfree01/v24i3/medoralv24i3p326.pdf Received: 22/08/2018 Article Number: 22858 http://www.medicinaoral.com/ Accepted: 10/12/2018 © Medicina Oral S.
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Denosumab (Prolia™, XGEVA™)
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White Spot Lesions After Orthodontic Treatment Assessed by Clinical
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Facility Description Price CC SWING BED RUGS CODE CC PRIVATE 1ST WEST 861 CC PRIVATE OBSTETRICS 861 CC BAKRI BALLOON FOR PP HEMR 661 CC BAKRI BALLOON FOR PP HEMR 661 CC RPR CMPX LID/N/L 2.6‐7.5 1914 CC RPR CMPX LID/N/ E/A 5CM/< 1053 CC RPR CMPX LID/N/L 2.6‐7.5 1914 CC RPR CMPX LID/N/ E/A 5CM/< 1053 CC PRIVATE ECU 494 CC IV THRPY FIRST HR DX 691 CC IV THRPY EA/ADD HR DX 190 CC IV THRPY ADD/SEQ TO 1 HR 205 CC IV THRPY CONCURRENT 122 CC IV THRPY FIRST HR HYDRTN 690 CC IV THRPY EA/ADD HR HYDRTN 134 CC INJ IV EA ADD DIFF MED 269 CC RECOVERY PER HOUR 280 CC MINOR PROCEDURE 1627 CC ARTERIAL LINE PLACEMENT 463 CC INJ EPIDURAL BLOOD PATCH 2850 CC INJ IV PUSH INITIAL 692 CC ADDITIONAL CPT CODE CC MED ICU ROOM CHARGE 1872 CC NURSERY 1200 CC CIRC TRAY 91 CC INIT HOSP CARE LV 1 P/DAY 205 CC INIT HOSP CARE LV 2 P/DAY 279 CC INIT HOSP CARE LV 3 P/DAY 412 CC SUBS HOSP CARE LV 1 P/DAY 81 CC SUBS HOSP CARE LV 2 P/DAY 147 CC SUBS HOSP CARE LV 3 P/DAY 212 CC HOSP DAY MGMT 30 MNT OR < 147 CC HOSP DAY MGMT > 30 MNTS 214 CC OBS CARE D/C DAY MGMT 155 CC INITL OBS CARE LV 1 P/DAY 197 CC INITL OBS CARE LV 2 P/DAY 239 CC INITL OBS CARE LV 3 P/DAY 362 CC SUBS OBS CARE LV 1 P/DAY 88 CC SUBS OBS CARE LV 2 P/DAY 145 CC SUBS OBS CARE LV 3 P/DAY 236 CC SNF D/C DAY MGMT 30 OR < 240 CC SNF D/C DAY MGMT >30 318 CC PERC REPLAC J TUBE W/O FL 1948 CC INITIAL NF CARE LEVEL 1 204 CC INITIAL NF CARE LEVEL 2 290 CC INITIAL NF CARE LEVEL 3 368 CC SUBSQENT NF CARE LEVEL 197 CC SUBSQENT NF CARE LEVEL 2 151 CC SUBSQENT NF CARE LEVEL 3 198 CC SUBSQENT NF CARE LEVEL 4 295 CC OBS OR I/P
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