Coding for Alveoloplasty with Extractions I
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Comparison of Etoricoxib and Indomethacin for the Treatment of Experimental Periodontitis in Rats
Brazilian Journal of Medical and Biological Research (2007) 40: 117-125 Etoricoxib in experimental periodontitis 117 ISSN 0100-879X Comparison of etoricoxib and indomethacin for the treatment of experimental periodontitis in rats M.C.F. Azoubel1, 1Departamento de Fisiologia e Farmacologia, A.M.A. Menezes1, 2Departamento de Morfologia, Faculdade de Medicina, D. Bezerra1, R.B. Oriá2, Universidade Federal do Ceará, Fortaleza, CE, Brasil R.A. Ribeiro1 and G.A.C. Brito2 Abstract Correspondence We investigated the effect of etoricoxib, a selective cyclooxygenase- Key words G.A.C. Brito 2 inhibitor, and indomethacin, a non-selective cyclooxygenase inhib- • Alveolar bone loss Departamento de Fisiologia itor, on experimental periodontitis, and compared their gastrointesti- • Inflammation e Farmacologia nal side effects. A ligature was placed around the second upper left • Periodontitis Faculdade de Medicina, UFC molars of female Wistar rats (160 to 200 g). Animals (6 per group) • Cyclooxygenase inhibitors Rua Coronel Nunes de Melo, 1127 • were treated daily with oral doses of 3 or 9 mg/kg etoricoxib, 5 mg/kg Etoricoxib 60430-270 Fortaleza, CE • Indomethacin Brasil indomethacin, or 0.2 mL saline, starting 5 days after the induction of Fax: +55-85-3366-8333 periodontitis, when bone resorption was detected, until the sacrifice E-mail: [email protected] on the 11th day. The weight and survival rate were monitored. Alveolar bone loss (ABL) was measured as the sum of distances Research supported by CNPq and between the cusp tips and the alveolar bone. The gastric mucosa was Fundação Cearense de Pesquisa e examined macroscopically and the periodontium and gastric and Cultura (FUNCAP). -
Differential Diagnosis of Pulmonic Stenosis by Means of Intracardiac Phonocardiography
Differential Diagnosis of Pulmonic Stenosis by Means of Intracardiac Phonocardiography Tadashi KAMBE, M.D., Tadayuki KATO, M.D., Norio HIBI, M.D., Yoichi FUKUI, M.D., Takemi ARAKAWA, M.D., Kinya NISHIMURA,M.D., Hiroshi TATEMATSU,M.D., Arata MIWA, M.D., Hisao TADA, M.D., and Nobuo SAKAMOTO,M.D. SUMMARY The purpose of the present paper is to describe the origin of the systolic murmur in pulmonic stenosis and to discuss the diagnostic pos- sibilities of intracardiac phonocardiography. Right heart catheterization was carried out with the aid of a double- lumen A.E.L. phonocatheter on 48 pulmonic stenosis patients with or without associated heart lesions. The diagnosis was confirmed by heart catheterization and angiocardiography in all cases and in 38 of them, by surgical intervention. Simultaneous phonocardiograms were recorded with intracardiac pressure tracings. In valvular pulmonic stenosis, the maximum ejection systolic murmur was localized in the pulmonary artery above the pulmonic valve and well transmitted to both right and left pulmonary arteries, the superior vena cava, and right and left atria. The maximal intensity of the ejection systolic murmur in infundibular stenosis was found in the outflow tract of right ventricle. The contractility of the infundibulum greatly contributes to the formation of the ejection systolic murmur in the outflow tract of right ventricle. In tetralogy of Fallot, the major systolic murmur is caused by the pulmonic stenosis, whereas the high ventricular septal defect is not responsible for it. In pulmonary branch stenosis, the sys- tolic murmur was recorded distally to the site of stenosis. Intracardiac phonocardiography has proved useful for the dif- ferential diagnosis of various types of pulmonic stenosis. -
Lecture 2 – Bone
Oral Histology Summary Notes Enoch Ng Lecture 2 – Bone - Protection of brain, lungs, other internal organs - Structural support for heart, lungs, and marrow - Attachment sites for muscles - Mineral reservoir for calcium (99% of body’s) and phosphorous (85% of body’s) - Trap for dangerous minerals (ex:// lead) - Transduction of sound - Endocrine organ (osteocalcin regulates insulin signaling, glucose metabolism, and fat mass) Structure - Compact/Cortical o Diaphysis of long bone, “envelope” of cuboid bones (vertebrae) o 10% porosity, 70-80% calcified (4x mass of trabecular bone) o Protective, subject to bending/torsion/compressive forces o Has Haversian system structure - Trabecular/Cancellous o Metaphysis and epiphysis of long bone, cuboid bone o 3D branching lattice formed along areas of mechanical stress o 50-90% porosity, 15-25% calcified (1/4 mass of compact bone) o High surface area high cellular activity (has marrow) o Metabolic turnover 8x greater than cortical bone o Subject to compressive forces o Trabeculae lined with endosteum (contains osteoprogenitors, osteoblasts, osteoclasts) - Woven Bone o Immature/primitive, rapidly growing . Normally – embryos, newborns, fracture calluses, metaphyseal region of bone . Abnormally – tumors, osteogenesis imperfecta, Pagetic bone o Disorganized, no uniform orientation of collagen fibers, coarse fibers, cells randomly arranged, varying mineral content, isotropic mechanical behavior (behavior the same no matter direction of applied force) - Lamellar Bone o Mature bone, remodeling of woven -
The Cementum: Its Role in Periodontal Health and Disease*
THE JOURNAL OF PERIODONTOLOGY JULY, NINETEEN HUNDRED SIXTY ONE The Cementum: Its Role In Periodontal Health and Disease* by DONALD A. KERR, D.D.S., M.S.,** Ann Arbor, Michigan HE cementum is a specialized calcified tissue of mesenchymal origin which provides for the attachment of the periodontal fibers to the surface of the Troot. It consists of 45 to 50 per cent inorganic material and 50 to 55 per cent organic material with the inorganic material in a hydroxyl apatite structure. The primary cementum is formed initially by appositional growth from the dental sac and later from the periodontal membrane under the influence of cementoblasts. It is formed in laminated layers with the incorporation of Sharpey's fibers into a fibrillar matrix which undergoes calcification. Cementum deposition is a Continuous process throughout life with new cementum being deposited over the old cemental surface. Cementum is formed by the organiza• tion of collagen fibrils which are cemented together by a matrix produced by the polymerization of mucopolysaccharides. This material is designated as cementoid and becomes mature cementum upon calcification. The significance of the continuous deposition of cementum has received various interpretations. 1. Continuous deposition of cementum is necessary for the reattachment of periodontal fibers which have been destroyed or which require reorientation due to change in position of teeth. It is logical that there should be a continuous deposition of cementum because it is doubtful that the initial fibers are retained throughout the life of the tooth, and therefore new fibers must be continually formed and attached by new cementum. -
Clinical Significance of Dental Anatomy, Histology, Physiology, and Occlusion
1 Clinical Significance of Dental Anatomy, Histology, Physiology, and Occlusion LEE W. BOUSHELL, JOHN R. STURDEVANT thorough understanding of the histology, physiology, and Incisors are essential for proper esthetics of the smile, facial soft occlusal interactions of the dentition and supporting tissues tissue contours (e.g., lip support), and speech (phonetics). is essential for the restorative dentist. Knowledge of the structuresA of teeth (enamel, dentin, cementum, and pulp) and Canines their relationships to each other and to the supporting structures Canines possess the longest roots of all teeth and are located at is necessary, especially when treating dental caries. The protective the corners of the dental arches. They function in the seizing, function of the tooth form is revealed by its impact on masticatory piercing, tearing, and cutting of food. From a proximal view, the muscle activity, the supporting tissues (osseous and mucosal), and crown also has a triangular shape, with a thick incisal ridge. The the pulp. Proper tooth form contributes to healthy supporting anatomic form of the crown and the length of the root make tissues. The contour and contact relationships of teeth with adjacent canine teeth strong, stable abutments for fixed or removable and opposing teeth are major determinants of muscle function in prostheses. Canines not only serve as important guides in occlusion, mastication, esthetics, speech, and protection. The relationships because of their anchorage and position in the dental arches, but of form to function are especially noteworthy when considering also play a crucial role (along with the incisors) in the esthetics of the shape of the dental arch, proximal contacts, occlusal contacts, the smile and lip support. -
Cardiac Amyloidosis and Surgery. What Do We Know About Rare
Cardiac amyloidosis and surgery. What do we know about rare diseases? Carlos Mestres1 and Mathias van Hemelrijck2 1University Hospital Zurich 2UniversitatsSpital Zurich May 3, 2021 Commentary to JOCS-2020-RA-1888 JOCS-2020-RA-1888 Cardiac amyloidosis in non-transplant cardiac surgery Cardiac amyloidosis and surgery. What do we know about rare diseases? Running Title: Cardiac amyloidosis and cardiac surgery Carlos { A. Mestres MD PhD FETCS1, 2, Mathias Van Hemelrijck MD1 1 - Clinic of Cardiac Surgery, University Hospital Zurich,¨ Zurich¨ (Switzerland) 2 - Department of Cardiothoracic Surgery, The University of the Free State, Bloemfontein, (South Africa) Word count (All): 1173 Word count (Text): 774 Key words : Cardiac amyloidosis, cardiac surgery, rare disease Correspondence: Carlos A. Mestres, MD, PhD, FETCS Clinic for Cardiac Surgery University Hospital Zurich,¨ R¨amistrasse 100 CH 8091 Zurich¨ (Switzerland) Email: [email protected] Rare diseases are serious, chronic and potentialy lethal. The European Union (EU) definition of a rare disease is one that affects fewer than 5 in 10,000 people (1). In the EU, these rare diseases are estimated to affect up to 8% of the roughly 500 million population (2). In the United States, a rare disease is defined as a condition affecting fewer than 200,000 people in the US (3). This a definition created by Congress in the Orphan Drug Act of 1983 (4). Therefore, the estimates for the US are that 25-30 million people are affected by a rare disease. There are more than 6000 rare diseases and 80% are genetic disorders diagnosed during childhood. Despite all community efforts, there are still a lack of an universal definition of rare diseases. -
Heart Valve Disease: Mitral and Tricuspid Valves
Heart Valve Disease: Mitral and Tricuspid Valves Heart anatomy The heart has two sides, separated by an inner wall called the septum. The right side of the heart pumps blood to the lungs to pick up oxygen. The left side of the heart receives the oxygen- rich blood from the lungs and pumps it to the body. The heart has four chambers and four valves that regulate blood flow. The upper chambers are called the left and right atria, and the lower chambers are called the left and right ventricles. The mitral valve is located on the left side of the heart, between the left atrium and the left ventricle. This valve has two leaflets that allow blood to flow from the lungs to the heart. The tricuspid valve is located on the right side of the heart, between the right atrium and the right ventricle. This valve has three leaflets and its function is to Cardiac Surgery-MATRIx Program -1- prevent blood from leaking back into the right atrium. What is heart valve disease? In heart valve disease, one or more of the valves in your heart does not open or close properly. Heart valve problems may include: • Regurgitation (also called insufficiency)- In this condition, the valve leaflets don't close properly, causing blood to leak backward in your heart. • Stenosis- In valve stenosis, your valve leaflets become thick or stiff, and do not open wide enough. This reduces blood flow through the valve. Blausen.com staff-Own work, CC BY 3.0 Mitral valve disease The most common problems affecting the mitral valve are the inability for the valve to completely open (stenosis) or close (regurgitation). -
Splitting of the Alveolar Process of the Lower Jaw
SPLITTING OF THE ALVEOLAR PROCESS OF THE LOWER JAW. Peekskill, N. Y., Nov. 1, 1858. Messrs. Editors:—I send you the following communica tion, which I received from G. J. Fisher, M. D., of Sing Sing, New York, eminent as a surgeon in this part of the country, which you are at liberty to publish, if you think it of suffi cient interest to the dental profession, to be intitled to a place in your valuable journal. Respectfully, yours, E. D. Fuller, D. D. S. Sing Sing, N. Y., October 28, 1858. My Dear Friend :—I send you an account of a case which is so near the boundary line which separates dental from general surgery, that you may be interested with it, and even consider it worthy of insertion in some one of the journals devoted to your speciality. You are at liberty to use it as your own. Miss V., of Tarrytown, New York, a tall and comely lass, while engaged in juvenile sports with several children in the dining room, was tripped by her dress, causing her to fall, with great violence, striking the front teeth of the lower jaw against the margin of a mahogony table. On arising, she found the teeth driven into the mouth, and the jaw broken*. She applied to a physician, who directed her to a dentist for treatment. She came to Sing Sing the following day (Oct. 20th), and applied to Dentist Greenleaf, who, after examin ing the case, came to the conclusion that it was a little more surgical than dental, and therefore called me in to treat the injury. -
Hereditary Gingival Hyperplasia – Case Report Dziedziczny Przerost Dziąseł – Opis Przypadków
CLINICAL CASE Dent. Med. Probl. 2011, 48, 3, 443–449 © Copyright by Wroclaw Medical University ISSN 1644-387X and Polish Dental Society Karolina Thum-Tyzo¹, Beata Petkowicz², Joanna Wysokińska-Miszczuk¹ Hereditary Gingival Hyperplasia – Case Report Dziedziczny przerost dziąseł – opis przypadków ¹ Chair and Department of Periodontology of the Medical University of Lublin, Poland ² Oral Medicine Independent Unit of the Medical University of Lublin, Poland Abstract Hereditary gingival hyperplasia is a rare case in clinical practice. The study describes two cases of this pathology occurring in several members of one family. This disease is inherited in an autosomal dominant or recessive way. Patients affected by this disease require a thorough diagnosis. In addition to basic medical history and dental examination, outside and inside oral cavity, additional study may be needed. The first reported case concerns a young mother with enlargement of gum tissue, which also appeared in her children, mother and grandmother. In children, hyperplasia has caused the delay in the eruption of permanent teeth, diastema secondary, dental abnor- malities, changes in facial appearance and problems with oral hygiene. Similar changes were observed in the second case described a father and son for whom gingival hyperplasia was a serious problem, with respect to functional and aesthetic disturbances, deteriorating quality of life. The aim of this study is to present the problems associated with enlargement of the gums and the difficulty in the treatment this disease which is not always successful (Dent. Med. Probl. 2011, 48, 3, 443–449). Key words: gingival hyperplasia, inherited disease, gingivectomy. Streszczenie Dziedziczny rozrost dziąseł jest przypadkiem rzadko spotykanym w praktyce klinicznej. -
Cardiac Surgery a Guide for Patients and Their Families Welcome to the Johns Hopkins Hospital
HEART AND VASCULAR INSTITUTE Cardiac Surgery A guide for patients and their families Welcome to The Johns Hopkins Hospital We are providing this book to you and your family to guide you through your surgical experience at the Johns Hopkins Heart and Vascular Institute. The physicians, nurses and other health care team members strive to provide you with the safest and best medical care possible. Please do not hesitate to ask your surgeon, nurse or other health care team member any questions before, during and after your operation. The booklet consists of two major sections. The first section informs you about the surgery and preparing for the hospital stay. The second section prepares you for the recovery period after surgery in the hospital and at home. TABLE OF CONTENTS Welcome to Johns Hopkins Cardiac Surgery 1 The Function of the Heart 2 Who’s Taking Care of You 3 Heart Surgery 4 Preparation for Surgery 5 Preoperative Testing and Surgical Consultation 7 The Morning of Surgery 9 After Surgery 10 Going Home After Cardiac Surgery 19 How We Help with Appointments and 24 Other Arrangements for Out-of-Town Patients Appendix 25 Looking back, it was the choice of my life. It’s not easy to put your heart in someone else’s hands. But for me, the choice was clear: I trusted it to Hopkins. My Heart. My Choice Patient Lou Grasmick, Founder & CEO, Louis J. Grasmick Lumber Company, Inc. Welcome to Johns Hopkins Cardiac Surgery The Johns Hopkins Hospital has a distinguished history of advancements in the treat- ment of cardiovascular diseases in adults and children, beginning with the Blalock-Taussig shunt in 1944. -
Cover Title Is Vesta Std Regular 48/52 with 45Pt After. 2020 Facility And
2020 Facility and Physician Cover title is Vesta Billing Guide Std Regular 48/52 Surgicalwith Heart Valve45pt Therapy after. Cover subtitle is Vesta Std Regular 18/22. Surgical Valve Repair and Replacement Procedures Physician Billing Codes Clinicians use Current Procedural Terminology (CPT)1 codes to bill for procedures and services. Each CPT code is assigned unique Relative Value Units (RVUs), which are used to determine payment by the Centers for Medicare & Medicaid Services (CMS) and other payers. Some commonly billed CPT codes used to describe procedures related to Edwards Lifesciences’ Heart Valve technologies are listed below.2 This list may not be comprehensive or complete. These procedures may be subject to the CMS multiple procedure reduction rule. When applicable, a payment reduction of 50% is applied to all payment amounts except the procedure with the greatest RVUs, which is paid at 100% unless exempt by CPT instructions or payer policy. Medicare National Average CPT Code Description Physician Payment3 Facility Setting Aortic 33390 Valvuloplasty, aortic valve, open, with cardiopulmonary bypass; simple $2,018 (ie, valvotomy, debridement, debulking, and/or simple commissural resuspension) 33391 Valvuloplasty, aortic valve, open, with cardiopulmonary bypass; complex $2,398 (eg, leaflet extension, leaflet resection, leaflet reconstruction, or annuloplasty) 33405 Replacement, aortic valve, open, with cardiopulmonary bypass; with prosthetic valve other $2,373 than homograft or stentless valve 33406 Replacement, aortic valve, open, -
Alveolar Process May Be Defined As That Part of the Maxilla and the Mandible That Forms and Supports the Sockets of the Teeth
The alveolar process may be defined as that part of the maxilla and the mandible that forms and supports the sockets of the teeth. It developed with the eruption of teeth and disappears or lost after tooth extraction ALVEOLAR PROCESS BASAL BONE Alveolar (bone) process: is that part of the maxilla and the mandible that forms and supports the sockets of the teeth. Basal Bone. it is the bone of the facial skeleton which support the alveolar bone. There is no anatomical boundary between basal bones and alveolar bone. Both alveolar process and basal bone are covered by the same periosteum. In some areas alveolar processes may fuse or masked with jaw bones as in (1) Anterior part of maxilla (palatal). (2) Oblique line of the mandible. * Alveolar process is resorbed after extraction of teeth. Functions of alveolar bone – Houses and protects developing permanent teeth, while supporting primary teeth. – Organizes eruption of primary and permanent teeth. – Anchors the roots of teeth to the alveoli, which is achieved by the insertion of Sharpey’s fibers into the alveolar bone proper (attachment). – Helps to move the teeth for better occlusion (support). – Helps to absorb and distribute occlusal forces generated during tooth contact (shock absorber). – Supplies vessels to periodontal ligament. •DEVELOPMENT OF ALVEOLAR BONE •Near the end of the second month of fetal life, the maxilla as well as the mandible form a groove that is open towards the surface of the oral cavity. •Tooth germs develop within the bony structures at late bell stage. •Bony septa and bony bridge begin to form and separatethe individual tooth germs from one another, keeping individual tooth germs in clearly outlined bony compartments.