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The Cat Show
THE BREEDS WHY DO PEOPLE ACFA recognizes 44 breeds. They are: Abyssinian SHOW CATS? American Curl Longhair American Curl Shorthair • American Shorthair To see how their cats match up to American Wirehair other breeders. Balinese Bengal • To share information. THE Birman Bombay • British Shorthair To educate the public about their Burmese breed, cat care, etc. Chartreux CAT Cornish Rex • To show off their cats. Cymric Devon Rex Egyptian Mau Exotic Shorthair Havana Brown SHOW Highland Fold FOR MORE Himalayan Japanese Bobtail Longhair INFORMATION Japanese Bobtail Shorthair Korat Longhair Exotic ACFA has a great variety of literature Maine Coon Cat you may wish to obtain. These Manx include show rules, bylaws, breed Norwegian Forest Cat standards and a beautiful hardbound Ocicat yearbook called the Parade of Oriental Longhair Royalty. They are available from: Oriental Shorthair Persian ACFA Ragdoll Russian Blue P O Box 1949 Scottish Fold Nixa, MO 65714-1949 Selkirk Rex Longhair Phone: 417-725-1530 Selkirk Rex Shorthair Fax: 417-725-1533 Siamese Siberian Or check our home page: Singapura http://www.acfacat.com Snowshoe Somali Membership in ACFA is open to any Sphynx individual interested in cats. As a Tonkinese Turkish Angora member, you have the right to vote Turkish Van on changes impacting the organization and your breed. AWARDS & RIBBONS WELCOME THE JUDGING Welcome to our cat show! We hope you Each day there will be four or more rings Each cat competes in their class against will enjoy looking at all of the cats we have running concurrently. Each judge acts other cats of the same sex, color and breed. -
Ch07 Final.Qxd 7/2/07 13:55 Page 87
Ch07 final.qxd 7/2/07 13:55 Page 87 87 CHAPTER 7 ARTHROLOGY Feline arthrology has been an overlooked subject in term osteoarthritis is reserved for the specific type of the past with most reviews of joint disease in small DJD that affects diarthrodial synovial articulations. animals focusing on the dog. However, cats are now Diseases of synovial joints can conveniently be known to suffer from many different types of joint divided into degenerative arthritis and inflammatory disease and, although there are many similarities with arthritis on the basis of the predominant pathologic the dog, there are also many features that are unique process (Table 20). Degenerative arthropathies are the to the feline patient. most common types and include traumatic arthritis and osteoarthritis. Inflammatory arthropathies are less CLASSIFICATION OF JOINT DISEASE common than degenerative arthropathies and have The terms arthritis and arthropathy literally mean joint either an infective or immune-mediated etiology. inflammation and joint disease, respectively. These terms Infective arthritis caused by bacterial infection (septic are used interchangeably in this chapter to describe arthritis) is the commonest type of inflammatory arthritis a number of well defined joint diseases characterized in the cat. Septic arthritis is classed as an erosive type of by a combination of inflammatory and degenerative arthritis because there is destruction of articular cartilage changes. The terms degenerative joint disease (DJD) in joints infected by bacteria. Immune-mediated and osteoarthritis are also often used synonymously. In arthropathies can be subdivided into both erosive this chapter, DJD is used as a general descriptive term to and nonerosive forms. -
Tyrosinase Mutations Associated with Siamese and Burmese Patterns in the Domestic Cat (Felis Catus)
doi:10.1111/j.1365-2052.2005.01253.x Tyrosinase mutations associated with Siamese and Burmese patterns in the domestic cat (Felis catus) L. A. Lyons, D. L. Imes, H. C. Rah and R. A. Grahn Department of Population Health and Reproduction, School of Veterinary Medicine, University of California, Davis, Davis, CA, USA Summary The Siamese cat has a highly recognized coat colour phenotype that expresses pigment at the extremities of the body, such as the ears, tail and paws. This temperature-sensitive colouration causes a ÔmaskÕ on the face and the phenotype is commonly referred to as ÔpointedÕ. Burmese is an allelic variant that is less temperature-sensitive, producing more pigment throughout the torso than Siamese. Tyrosinase (TYR) mutations have been sus- pected to cause these phenotypes because mutations in TYR are associated with similar phenotypes in other species. Linkage and synteny mapping in the cat has indirectly sup- ported TYR as the causative gene for these feline phenotypes. TYR mutations associated with Siamese and Burmese phenotypes are described herein. Over 200 cats were analysed, representing 12 breeds as well as randomly bred cats. The SNP associated with the Siamese phenotype is an exon 2 G > A transition changing glycine to arginine (G302R). The SNP associated with the Burmese phenotype is an exon 1 G > T transversion changing glycine to tryptophan (G227W). The G302R mutation segregated concordantly within a pedigree of Himalayan (pointed) Persians. All cats that had ÔpointedÕ or the Burmese coat colour phenotype were homozygous for the corresponding mutations, respectively, suggesting that these phenotypes are a result of the identified mutations or unidentified mutations that are in linkage disequilibrium. -
Ollier Disease: a Case Report
Case Report Ollier Disease: A Case Report Bajracharya L*, Shrestha M**, Paudel S***, Shrestha PS*** *Teaching Assistant, **Assistant Professor, Department of Paediatrics, ***Assistant Professor, Department of Radiology, ****Professor, Department of Paediatrics, TUTH, Kathmandu, Nepal. ABSTRACT Ollier disease is a rare disease featuring multi ple enchondromas mainly aff ecti ng limbs. We describe a boy who presented in our OPD with multi ple painless joint deformiti es mostly in the both upper and lower limbs. Conventi onal X-ray evaluati on revealed typical multi ple radiolucent, homogenous oval and elongated shapes in the deformed limbs. : Ollier Disease , Enchondromatosis INTRODUCTION Enchondromas are common, benign, usually deformity of limbs. There is no history of weight asymptomati c carti lage tumours that develop in loss or traumati c injury. There is no similar family metaphyses and may also involve the diaphysis of history . He is acti ve , appropriate in all domains of long tubular bones.1 Ollier disease is defi ned by the development except delayed motor milestones, short presence of multi ple enchondromas and characterized limb gait and postural lumbar scoliosis. His vitals are by an asymmetric distributi on of carti lage lesions which stable. Weight is 20kg and height is 106cm (both less can be extremely variable. 2 The prevalence of Ollier’s than 3rd percenti le of NCHS). His head circumference disease is esti mated to be 1/100000 .1Children with is 52cm (50th percenti le). Upper segment to lower symptomati c enchondromatosis cases usually present segment rati o is 0.89cm. Multi ple joints are swollen before puberty with deformity, growth disorders and but nontender and non-erythematous. -
Multiple Hereditary Exostoses
Multiple Hereditary Exostoses Dror Paley MD, FRCSC Medical Director, Paley Orthopedic and spine Institute, St. Mary’s Medical Center, West palm Beach, FL David Feldman, MD Director, Spinal deformity center, Paley Orthopedic and spine Institute, St. Mary’s Medical Center, West palm Beach, FL Multiple hereditary exostoses (MHE), also known as multiple osteochondromas (MO), is an autosomal dominant skeletal disorder. Approximately 10–20% of individuals are a result of a spontaneous mutation while the rest are familial. The prevalence of MHE/MO is 1/50 000. There are two known genes found to cause MHE/MO, EXT1 located on chromosome 8q23-q24 and EXT2 located on chromosome 11p11-p12. In 10–15% of the patients, no mutation can be located by current methods of genetic testing.1–7 These mutations are scattered across both genes. EXT1/EXT2 is essential for the biosynthesis of heparan sulfate (HS). HS production in patients’ cells is reduced by 50% or more.8–19 MHE/MO is associated with characteristic progressive skeletal deformities of the extremities and shortening of one or both the sides, leading to limb length discrepancy (LLD) and short stature.20–28 Two bone segments, such as the lower leg or forearm, are at greater risk of problems due to either osteochondromas (OCs) from one or both bones impinging on or deforming the other bone or a primary issue of altered growth causing one bone to grow at a faster or slower rate. OCs can also affect joint motion due to impingement of an OC with the opposite side of the joint or subluxation/dislocation related to deformity, impingement, and incongruity.20,24,26,29,30 OCs can also cause nerve or vessel entrapment and/or compression, including the spinal cord and nerve roots. -
Breeding Policy
ORIENTAL BREEDING POLICY This breeding policy accompanies and supplements the Oriental Registration Policy and should be read in conjunction with that document. The aim of this breeding policy is to give advice and guidance to ensure breeders observe what is considered “best practice” in breeding Orientals with the over-riding objective of improving the Oriental cat to meet all aspects of the Oriental Standard of Points, which describes the ideal for the recognised varieties in the Oriental Group. The origins of the Oriental Until the late 1960’s very few Orientals were seen at shows other than the Havanas (which had their own classes), and the Lilacs and Whites which were exhibited as ‘Any Other Variety (AOV)’. By the end of the decade the Havanas and the Tabby Pointed Siamese (only recognised as a variety of Siamese in 1966), were among the best Siamese types in the country. With the help of prudent outcrossing between Havanas and Tabby Point Siamese, and by backcrossing to both parental varieties, further improvement was made in the Havanas and the emergence of the Oriental Tabby as a beautiful variety in its own right was assured. Hot on the heels of the Oriental Tabbies came the Blues, Blacks, Tortoiseshells (Torties), Silver Tabbies, Smokes and Shaded Silvers. The Oriental is now well established in the UK and over 50 years of breeding has developed and fixed good phenotype in the breed but with a decreasing gene-pool. The Oriental breed has one of the largest numbers of gene variations of any breed of pedigree cat recognised by GCCF. -
21362 Arthritis Australia a to Z List
ARTHRITISINFORMATION SHEET Here is the A to Z of arthritis! A D Goodpasture’s syndrome Achilles tendonitis Degenerative joint disease Gout Achondroplasia Dermatomyositis Granulomatous arteritis Acromegalic arthropathy Diabetic finger sclerosis Adhesive capsulitis Diffuse idiopathic skeletal H Adult onset Still’s disease hyperostosis (DISH) Hemarthrosis Ankylosing spondylitis Discitis Hemochromatosis Anserine bursitis Discoid lupus erythematosus Henoch-Schonlein purpura Avascular necrosis Drug-induced lupus Hepatitis B surface antigen disease Duchenne’s muscular dystrophy Hip dysplasia B Dupuytren’s contracture Hurler syndrome Behcet’s syndrome Hypermobility syndrome Bicipital tendonitis E Hypersensitivity vasculitis Blount’s disease Ehlers-Danlos syndrome Hypertrophic osteoarthropathy Brucellar spondylitis Enteropathic arthritis Bursitis Epicondylitis I Erosive inflammatory osteoarthritis Immune complex disease C Exercise-induced compartment Impingement syndrome Calcaneal bursitis syndrome Calcium pyrophosphate dehydrate J (CPPD) F Jaccoud’s arthropathy Crystal deposition disease Fabry’s disease Juvenile ankylosing spondylitis Caplan’s syndrome Familial Mediterranean fever Juvenile dermatomyositis Carpal tunnel syndrome Farber’s lipogranulomatosis Juvenile rheumatoid arthritis Chondrocalcinosis Felty’s syndrome Chondromalacia patellae Fibromyalgia K Chronic synovitis Fifth’s disease Kawasaki disease Chronic recurrent multifocal Flat feet Kienbock’s disease osteomyelitis Foreign body synovitis Churg-Strauss syndrome Freiberg’s disease -
Orphanet Journal of Rare Diseases Biomed Central
Orphanet Journal of Rare Diseases BioMed Central Review Open Access Ollier disease Caroline Silve*1 and Harald Jüppner2 Address: 1INSERM U. 773, Faculté de Médecine Xavier Bichat, 16 rue Henri Huchard, 75018 Paris, France and 2Endocrine Unit, Department of Medicine, and Pediatric Neprology Unit, MassGeneral Hospital for Children, Massachusetts General Hospital and Harvard Medical School, Boston, MA 02114, USA Email: Caroline Silve* - [email protected]; Harald Jüppner - [email protected] * Corresponding author Published: 22 September 2006 Received: 31 July 2006 Accepted: 22 September 2006 Orphanet Journal of Rare Diseases 2006, 1:37 doi:10.1186/1750-1172-1-37 This article is available from: http://www.OJRD.com/content/1/1/37 © 2006 Silve and Jüppner; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Abstract Enchondromas are common intraosseous, usually benign cartilaginous tumors, that develop in close proximity to growth plate cartilage. When multiple enchondromas are present, the condition is called enchondromatosis also known as Ollier disease (WHO terminology). The estimated prevalence of Ollier disease is 1/100,000. Clinical manifestations often appear in the first decade of life. Ollier disease is characterized by an asymmetric distribution of cartilage lesions and these can be extremely variable (in terms of size, number, location, evolution of enchondromas, age of onset and of diagnosis, requirement for surgery). Clinical problems caused by enchondromas include skeletal deformities, limb-length discrepancy, and the potential risk for malignant change to chondrosarcoma. -
Preventing Fading Kitten Syndrome in Hairless Peterbald Cats
Preventing Fading Kitten Syndrome in Hairless Peterbald Cats Mark Kantrowitz 1 Abstract Newborn hairless Peterbald kittens have a very high mortality rate, rarely surviving to one month of age. Since the early days of the breed, breeders have not attempted to breed born-hairless sires and dams together because all of the kittens will be hairless, with few surviving to adulthood. Even in litters from heterozygous parents, it is unusual for more than one hairless kitten to survive. Several potential causes of fading kitten syndrome in Peterbald cats are identified. A new 7-step protocol has been developed to address all of the potential causes of fading kitten syndrome. This protocol has been used successfully to breed a hairless sire, CelestialBlue Eureka, with a hairless dam, CelestialBlue Mimsy. All four hairless kittens in this litter have survived to four months of age, and there is every expectation that they will reach adulthood and live normal lifespans. This is the first time a litter of born-hairless Peterbald kittens has achieved a 0% mortality rate since the origin of the Peterbald breed. Litter of four hairless Peterbald kittens at one month of age 1 CelestialBlue Peterbald Cattery, Peterbald.com Introduction The Peterbald is a rare breed of hairless housecat that originated in St. Petersburg, Russia, in 1994. It is the result of a cross of the hairless Donskoy (Don Sphynx) with Oriental Shorthair cats. Siamese cats were subsequently allowed as outcrosses. The result is an intelligent and affectionate hairless cat with a long, elegant, tubular body, large, low-set ears and a triangular head with a blunt tip. -
Craniofacial Chondrosarcomas: Imaging Findings in 15 Untreated Cases
165 Craniofacial Chondrosarcomas: Imaging Findings in 15 Untreated Cases Ya-Yen Lee1 Radiographic findings of 15 untreated chondrosarcomas of the cranial and facial Pamela Van Tassel bones were reviewed. These tumors have a propensity to occur in the wall of a maxillary sinus, at the junction of sphenoid and ethmoid sinuses and vomer, and at the undersur face of the sphenoid bone. Because of its slow-growing nature, chondrosarcomas tend to be large, multi lobulated, and sharply demarcated when detected. Frequent bone changes are a combination of erosion and destruction, with sharp transitional zones and absent periosteal reaction. Tumor matrix calcifications, not necessarily chondroid, are almost always present. Both CT and MR may be necessary for thorough evaluation of tumor extent. Chondrosarcoma, a malignant but usually slow-growing cartilaginous tumor, constitutes approximately 11 % of malignant bone tumors [1] but rarely occurs in the craniofacial region . Because of its propensity to occur in the deep facial structures or base of the skull, the true extent and origin of the tumor may be overlooked if not properly evaluated radiographically. We review a relatively large series of craniofacial chondrosarcomas and discuss the differential diagnosis and choice of imaging technique. Materials and Methods This retrospective radiologic review was based on the pretreatment radiographic studies of 15 patients with craniofacial chondrosarcomas seen at our institution over a period of 40 years , excluding three intracranial dural chondrosarcomas, which are to be reported sepa rately. An attempt was also made to correlate the radiographic findings with the hi stologic grades of the tumors. The ages of the patients ranged from 10 to 73 years , with a mean of 40 years. -
CFA EXECUTIVE BOARD MEETING FEBRUARY 4/5, 2017 Index To
CFA EXECUTIVE BOARD MEETING FEBRUARY 4/5, 2017 Index to Minutes Secretary’s note: This index is provided only as a courtesy to the readers and is not an official part of the CFA minutes. The numbers shown for each item in the index are keyed to similar numbers shown in the body of the minutes. (1) MEETING CALLED TO ORDER. .................................................................................... 3 (2) ADDITIONS/CORRECTIONS; RATIFICATION OF ON-LINE MOTIONS. ................ 5 (3) APPEAL HEARING. ....................................................................................................... 12 (4) PROTEST COMMITTEE. ............................................................................................... 13 (5) INVESTMENT PRESENTATION. ................................................................................. 14 (6) CENTRAL OFFICE OPERATIONS. .............................................................................. 15 (7) MARKETING................................................................................................................... 18 (8) BOARD CITE. .................................................................................................................. 20 (9) JUDGING PROGRAM. ................................................................................................... 29 (10) REGIONAL ASSIGNMENT ISSUE. .............................................................................. 34 (11) PERSONNEL ISSUES. ................................................................................................... -
TICA Oriental Longhair & Oriental Shorthair Breed Introduction Www
TICA Oriental Longhair & Oriental Shorthair Breed Introduction www.tica.org General Description: The Oriental is a member of the Siamese breed group and comes in two coat lengths: the Oriental Shorthair and the Oriental Longhair. Like all of the group members (Siamese, Balinese, Oriental Shorthairs and Oriental Longhairs), Orientals are long, slender, stylized cats. They are lively, talkative and intelligent and are very attached to their people. All of the members of the breed group have the same physical standard except for coat length and color. What makes the Oriental Shorthair distinct from the rest of the Siamese group is their wide array of colors combined with a short sleek coat while the Oriental Longhair has a semi-long coat draping the elegant body. History : Orientals are a man-made breed that originated in the 1950s in England. After World War II the number of breeders and breeding cats was reduced. Some of the remaining breeders became quite creative as they rebuilt their breeding programs. Many modern breeds developed from the crosses done at that time. One such breed is the Oriental Shorthair/Longhair. Russian Blues, British Shorthairs, Abyssinians, and regular domestic cats were crossed to Siamese. The resulting cats were not pointed and were crossed back to Siamese. In surprisingly few generations, there were cats that were indistinguishable from Siamese in all ways except color. As the Siamese pointed color is genetically recessive, pointed kittens were also produced. The best Siamese-colored cats from these crosses went back into the Siamese breed, enlarging and strengthening the Siamese gene pool.