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Dr.Naresh Resume.Pdf
PERSONAL INFORMATION NAME First Name: NARESH KUMAR Last Name: AMRADI ADDRESS KRF-Postdoctoral Researcher Environmental Processes and Materials Laboratory, School of Civil and Environmental Engineering, Yonsei University, Seoul, South Korea. TELEPHONE Mobile: +82-10-2246-3222, Office: +82-02-2123-7287 E-MAIL [email protected], [email protected] PERSONAL DETAILS I. Date of Birth: 16/07/1985 II. Marital Status: Married III. Total Research Experience: 9 Years IV. Total Publications: 28 (Including Book Chapters) V. Total Citations: 986, h-Index:14 (Google Scholar) 1. Currently working as KRF-Postdoctoral Researcher at School of RESEARCH EXPERIENCE Civil and Environmental Engineering, Yonsei University, Seoul 03722, Republic of Korea, from April 2019. 2. Completed doctoral studies on “Microbial Transformation of Solid Waste for Renewable Chemicals, Energy and Polymers in the Biorefinery Framework” under the supervision of Dr.S. Venkata Mohan, Senior Principal Scientist, Bio-Engineering and Environmental Sciences Lab, CSIR-Indian Institute of Chemical Technology (CSIR-IICT), Hyderabad, India. 3. Previously worked on domestic wastewater treatment and utilization for agriculture-Wastewater for Agriculture Project at International Crops Research Institute for Semi-Arid Tropics (ICRISAT), Hyderabad from June 2010 to Sept- 2012. Analytical Skills: COD, BOD, Nitrates, Volatile fatty acids (VFA), TECHNICAL SKILLS Phosphates, Sulphates, Chlorides, Proteins, Carbohydrates, Enzyme Assays, Total solids, Totals Dissolved Solids and Volatile solids. Tools: Liquid Chromatography, Gas Chromatography, Cyclic voltammetry (CV), Florescent In situ Hybridization (FISH), Spectroscopic analysis (ƛ max/UV-Vis). Molecular biology techniques: DNA/Protein Isolation, Discontinuous Gradient Gel Electrophoresis (DGGE), 2DE, SDS and Native PAGE. PATENT FILED Low Cost Electrolyte Membranes for Microbial Fuel Cell Application Synthesized by complexing Starch (Wheat, Corn and Rice) with Salt. -
Synergistic Cytotoxicity of Renieramycin M and Doxorubicin in MCF-7 Breast Cancer Cells
marine drugs Article Synergistic Cytotoxicity of Renieramycin M and Doxorubicin in MCF-7 Breast Cancer Cells Jortan O. Tun 1,*, Lilibeth A. Salvador-Reyes 1,2 , Michael C. Velarde 3 , Naoki Saito 4 , Khanit Suwanborirux 5 and Gisela P. Concepcion 1,2,* 1 The Marine Science Institute, University of the Philippines Diliman, Quezon City 1101, Philippines; [email protected] 2 Philippine Genome Center, University of the Philippines Diliman, Quezon City 1101, Philippines 3 Institute of Biology, University of the Philippines Diliman, Quezon City 1101, Philippines; [email protected] 4 Graduate School of Pharmaceutical Sciences, Meiji Pharmaceutical University, Tokyo 204-8588, Japan; [email protected] 5 Department of Pharmacognosy and Pharmaceutical Botany, Faculty of Pharmaceutical Sciences, Center for Bioactive Natural Products from Marine Organisms and Endophytic Fungi (BNPME), Chulalongkorn University, Pathumwan, Bangkok 10330, Thailand; [email protected] * Correspondence: [email protected] (J.O.T.); [email protected] (G.P.C.); Tel.: +632-922-3959 (J.O.T. & G.P.C.) Received: 23 July 2019; Accepted: 7 August 2019; Published: 16 September 2019 Abstract: Renieramycin M (RM) is a KCN-stabilized tetrahydroisoquinoline purified from the blue sponge Xestospongia sp., with nanomolar IC50s against several cancer cell lines. Our goal is to evaluate its combination effects with doxorubicin (DOX) in estrogen receptor positive MCF-7 breast cancer cells. MCF-7 cells were treated simultaneously or sequentially with various combination ratios of RM and DOX for 72 h. Cell viability was determined using the MTT assay. Synergism or antagonism was determined using curve-shift analysis, combination index method and isobologram analysis. -
Accurate Methods for Ancestry and Relatedness Inference
ACCURATE METHODS FOR ANCESTRY AND RELATEDNESS INFERENCE A DISSERTATION SUBMITTED TO THE PROGRAM IN BIOMEDICAL INFORMATICS AND THE COMMITTEE ON GRADUATE STUDIES OF STANFORD UNIVERSITY IN PARTIAL FULFILLMENT OF THE REQUIREMENTS FOR THE DEGREE OF DOCTOR OF PHILOSOPHY Jesse M. Rodriguez December 2013 © 2013 by Jesse M. Rodriguez. All Rights Reserved. Re-distributed by Stanford University under license with the author. This work is licensed under a Creative Commons Attribution- Noncommercial 3.0 United States License. http://creativecommons.org/licenses/by-nc/3.0/us/ This dissertation is online at: http://purl.stanford.edu/cn371vd3410 ii I certify that I have read this dissertation and that, in my opinion, it is fully adequate in scope and quality as a dissertation for the degree of Doctor of Philosophy. Serafim Batzoglou, Primary Adviser I certify that I have read this dissertation and that, in my opinion, it is fully adequate in scope and quality as a dissertation for the degree of Doctor of Philosophy. Russ Altman I certify that I have read this dissertation and that, in my opinion, it is fully adequate in scope and quality as a dissertation for the degree of Doctor of Philosophy. Carlos Bustamante Approved for the Stanford University Committee on Graduate Studies. Patricia J. Gumport, Vice Provost for Graduate Education This signature page was generated electronically upon submission of this dissertation in electronic format. An original signed hard copy of the signature page is on file in University Archives. iii Abstract The predisposition to many diseases is strongly influenced by the genome of an in- dividual. -
Inmate Release Report Snapshot Taken: 9/28/2021 6:00:10 AM
Inmate Release Report Snapshot taken: 9/28/2021 6:00:10 AM Projected Release Date Booking No Last Name First Name 9/29/2021 6090989 ALMEDA JONATHAN 9/29/2021 6249749 CAMACHO VICTOR 9/29/2021 6224278 HARTE GREGORY 9/29/2021 6251673 PILOTIN MANUEL 9/29/2021 6185574 PURYEAR KORY 9/29/2021 6142736 REYES GERARDO 9/30/2021 5880910 ADAMS YOLANDA 9/30/2021 6250719 AREVALO JOSE 9/30/2021 6226836 CALDERON ISAIAH 9/30/2021 6059780 ESTRADA CHRISTOPHER 9/30/2021 6128887 GONZALEZ JUAN 9/30/2021 6086264 OROZCO FRANCISCO 9/30/2021 6243426 TOBIAS BENJAMIN 10/1/2021 6211938 ALAS CHRISTOPHER 10/1/2021 6085586 ALVARADO BRYANT 10/1/2021 6164249 CASTILLO LUIS 10/1/2021 6254189 CASTRO JAYCEE 10/1/2021 6221163 CUBIAS ERICK 10/1/2021 6245513 MYERS ALBERT 10/1/2021 6084670 ORTIZ MATTHEW 10/1/2021 6085145 SANCHEZ ARAFAT 10/1/2021 6241199 SANCHEZ JORGE 10/1/2021 6085431 TORRES MANLIO 10/2/2021 6250453 ALVAREZ JOHNNY 10/2/2021 6241709 ESTRADA JOSE 10/2/2021 6242141 HUFF ADAM 10/2/2021 6254134 MEJIA GERSON 10/2/2021 6242125 ROBLES GUSTAVO 10/2/2021 6250718 RODRIGUEZ RAFAEL 10/2/2021 6225488 SANCHEZ NARCISO 10/2/2021 6248409 SOLIS PAUL 10/2/2021 6218628 VALDEZ EDDIE 10/2/2021 6159119 VERNON JIMMY 10/3/2021 6212939 ADAMS LANCE 10/3/2021 6239546 BELL JACKSON 10/3/2021 6222552 BRIDGES DAVID 10/3/2021 6245307 CERVANTES FRANCISCO 10/3/2021 6252321 FARAMAZOV ARTUR 10/3/2021 6251594 GOLDEN DAMON 10/3/2021 6242465 GOSSETT KAMERA 10/3/2021 6237998 MOLINA ANTONIO 10/3/2021 6028640 MORALES CHRISTOPHER 10/3/2021 6088136 ROBINSON MARK 10/3/2021 6033818 ROJO CHRISTOPHER 10/3/2021 -
CURRICULUM VITAE George M. Weinstock, Ph.D
CURRICULUM VITAE George M. Weinstock, Ph.D. DATE September 26, 2014 BIRTHDATE February 6, 1949 CITIZENSHIP USA ADDRESS The Jackson Laboratory for Genomic Medicine 10 Discovery Drive Farmington, CT 06032 [email protected] phone: 860-837-2420 PRESENT POSITION Associate Director for Microbial Genomics Professor Jackson Laboratory for Genomic Medicine UNDERGRADUATE 1966-1967 Washington University EDUCATION 1967-1970 University of Michigan 1970 B.S. (with distinction) Biophysics, Univ. Mich. GRADUATE 1970-1977 PHS Predoctoral Trainee, Dept. Biology, EDUCATION Mass. Institute of Technology, Cambridge, MA 1977 Ph.D., Advisor: David Botstein Thesis title: Genetic and physical studies of bacteriophage P22 genomes containing translocatable drug resistance elements. POSTDOCTORAL 1977-1980 Postdoctoral Fellow, Department of Biochemistry TRAINING Stanford University Medical School, Stanford, CA. Advisor: Dr. I. Robert Lehman. ACADEMIC POSITIONS/EMPLOYMENT/EXPERIENCE 1980-1981 Staff Scientist, Molec. Gen. Section, NCI-Frederick Cancer Research Facility, Frederick, MD 1981-1983 Staff Scientist, Laboratory of Genetics and Recombinant DNA, NCI-Frederick Cancer Research Facility, Frederick, MD 1981-1984 Adjunct Associate Professor, Department of Biological Sciences, University of Maryland, Baltimore County, Catonsville, MD 1983-1984 Senior Scientist and Head, DNA Metabolism Section, Lab. Genetics and Recombinant DNA, NCI-Frederick Cancer Research Facility, Frederick, MD 1984-1990 Associate Professor with tenure (1985) Department of Biochemistry -
From Population and Personalized Genomics to Personalized/Precision Medicine Manolis Dermitzakis
From Population and Personalized Genomics to Personalized/Precision Medicine Manolis Dermitzakis University of Geneva Dept Genetic Medicine and Development University of Geneva Medical School and Swiss Institute of Bioinformatics [email protected] http://funpopgen.unige.ch/ Our “engine” Revolu'on in Medicine • Advances in technology • Deep learning of human biology Complex traits/disease Space& Time& Popula'on)of)cells) Individual)cells) ) GENE)A) Modified from Dermitzakis Nat Genet 2008 HapMap: cataloguing “common” genec variaon HapMap Consortium Nature 2005 1000 genomes: cataloguing “all” genec variaon Genome-Wide association studies (GWAS) Gene expression as a key molecular phenotype – expression QTL (eQTL) analysis 1Mb$ TSS$ 1Mb$ 1Mb$window$ RNAseq$ gene$ SNPs$ cis$ trans$ eQTL$ Molecular"phenotype" AA" AC" CC" Func'onal varia'on to organismal phenotype GENETIC ASSOCIATION IS A CAUSAL LINK 1Mb$ TSS$ 1Mb$ 1Mb$window$ RNAseq$ gene$ SNPs$ Space& cis$ Interpretation of GWAS using molecular QTLs trans$ eQTL$ Time& eQTLs Molecular"phenotype" 1Mb TSS 1Mb AA" AC" CC" 1Mb window Mechanistic RNAseq insights to gene SNPs Genetic variation Methylation Chromatin Background Rationale Genome-wide association studies (GWAS) have identified hundreds of common DNA variants associated with multiple complex diseases and traits. ~90% of GWAS SNPs lie in noncoding regions (e.g. intergenic, introns). Associated (tag) SNP (*) Chr * Genes * Causal mechanism? Causal gene/s? Many studies show trait-associated SNPs enriched for eQTLs LCL eQTLs LCL -
New Spain and Early Independent Mexico Manuscripts New Spain Finding Aid Prepared by David M
New Spain and Early Independent Mexico manuscripts New Spain Finding aid prepared by David M. Szewczyk. Last updated on January 24, 2011. PACSCL 2010.12.20 New Spain and Early Independent Mexico manuscripts Table of Contents Summary Information...................................................................................................................................3 Biography/History.........................................................................................................................................3 Scope and Contents.......................................................................................................................................6 Administrative Information...........................................................................................................................7 Collection Inventory..................................................................................................................................... 9 - Page 2 - New Spain and Early Independent Mexico manuscripts Summary Information Repository PACSCL Title New Spain and Early Independent Mexico manuscripts Call number New Spain Date [inclusive] 1519-1855 Extent 5.8 linear feet Language Spanish Cite as: [title and date of item], [Call-number], New Spain and Early Independent Mexico manuscripts, 1519-1855, Rosenbach Museum and Library. Biography/History Dr. Rosenbach and the Rosenbach Museum and Library During the first half of this century, Dr. Abraham S. W. Rosenbach reigned supreme as our nations greatest bookseller. -
28 June 2019 25 – 28 Fjuneaculty of 2019 Scienc E Kasetsart University, Bangkok Faculty of Science Kasetsart University, Bangkok
25 - 28 June 2019 25 – 28 FJuneaculty of 2019 Scienc e Kasetsart University, Bangkok Faculty of Science Kasetsart University, Bangkok This page is intentionally left blank. T A B L E O F C ONTENT Workshop schedule 2 Opening and Welcome speeches 6 Sponsors’ Info 11 Organizing Committee 15 Presenters’ Biographies 17 Participants’ Info 27 Invited Talks and Talks 31 Lecture Summary 44 Poster Abstracts 50 1 W O R K S H O P S CHEDULE 08:00-08:40 Registration 08:40-08:45 Celebration of Coronation of King Rama X 08:45-09:05 Welcome and opening ceremony Group Photo 09:05-09:15 Overview of the Empowering agricultural research through (meta)genomics workshop Alexie Papanicolaou 09:15-10:15 Plenary Talk: “Genomes, Genes, Allele and Mechanisms.” Roger Hellens 10:15:10:30 Tea break and networking (Room 352) 10:30-10:50 Invited Talk: High Resolution Profiling of Bacterial Communities using Full-Length 16S rRNA Sequence Data from PacBio SMRT Sequencing System Wirulda Pootakarm 10:50-11:10 Invited Talk: From Omics to System Biology: Impact on Industrial Biotechnology and Human Health Wanwipa Vongsangnak 11:10-11:50 Invited talk: Functional Genomics of Tropical Plants Goh Hoe Han 11:50-13:00 Lunch and Product presentation by Sponsors (Room 352) 12:00-12.20: Advancing metagenomics research with Illumina solutions - Cara Lim 12:20-12:40: QIAGEN’s Innovative Solutions for Advancing Microbiome Research from Challenging Samples to Insight with Confidence - Wasin Sakulkoo 2 12:40-12:50: Sequencing solution on DNBSEQ platform - Honghong Liu 13:00:13:40 Invited -
PRESS RELEASE Geneva | April 29Th, 2019
PRESS RELEASE Geneva | April 29th, 2019 WARNING: embargoed until May 2nd, 2019, 7pm GMT More than genes themselves, how, where and when they are expressed determine our biological traits – our phenotypes. If gene expression is A model to decipher controlled by many regulatory elements, what, ultimately, controls those? And how does genetic variation affect them? The SysGenetiX project, led the complexity by the University of Geneva (UNIGE) in collaboration with the University of Lausanne (UNIL), Switzerland, precisely aimed to investigate these regu- of gene regulation latory elements, as well as the manifold interactions between them and with genes, with the ultimate goal of understanding the mechanisms that Scientist at the Universities render some people more predisposed to manifesting particular diseases of Geneva and Lausanne, than others. By studying chromatin modifications (i.e. how the genome is “packaged”) in the cells of about 300 individuals, scientists from Geneva Switzerland, designed a and Lausanne not only identified the very structure of these regulatory ele- framework to analyse gene ments, they were also able to model how their interactions throughout the regulation, and offer a model whole genome influence gene regulation and risk of disease. A pioneering to better understand the role approach, to read in Science, that will shape tomorrow’s precision medicine. of the non-coding portion of the genome in disease risk. Emmanouil Dermitzakis, professor at the Department of Genetic medicine and Development at UNIGE Faculty of Medicine, Director of the Health 2030 Genome Centre and leader of the SysGenetiX project is a specialist of the genetic variation of gene regulation. -
SARS-Cov-2 Transmission, Vaccination Rate and the Fate of Resistant Strains
medRxiv preprint doi: https://doi.org/10.1101/2021.02.08.21251383; this version posted February 10, 2021. The copyright holder for this preprint (which was not certified by peer review) is the author/funder, who has granted medRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC-ND 4.0 International license . SARS-CoV-2 transmission, vaccination rate and the fate of resistant strains 1 2 3* 1* Simon A. Rella , Yuliya A. Kulikova , Emmanouil T. Dermitzakis , Fyodor A. Kondrashov 1 Institute of Science and Technology Austria, 1 Am Campus, Klosterneuburg, 3400, Austria 2 Banco de España, Calle Alcala 48, 28014 Madrid, Spain 3 Department of Genetic Medicine and Development, University of Geneva Medical School, Geneva, Switzerland *Corresponding authors, ETD [email protected], FAK [email protected] Vaccines are thought to be the best available solution for controlling the ongoing 1,2 3–6 SARS-CoV-2 pandemic . However, the emergence of vaccine-resistant strains may come too rapidly for current vaccine developments to alleviate the health, economic and 7,8 social consequences of the pandemic . To quantify and characterize the risk of such a 9,10 scenario, we created a SIR-derived model with initial stochastic dynamics of the vaccine-resistant strain to study the probability of its emergence and establishment. Using parameters realistically resembling SARS-CoV-2 transmission, we model a wave-like pattern of the pandemic and consider the impact of the rate of vaccination and the strength of non-pharmaceutical intervention measures on the probability of emergence of a resistant strain. -
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PRESS Release Geneva August 31st 2012 under strict embargo until Sept 2nd 2012, 19 pm Swiss Time a new light shed Why are some people more likely than others to suffer from diseases such as type 2 diabetes or heart dysfunction? It is partly due to gene- on genetic tics. Several genetic variants are indeed known to increase the risk of developing these common and multifactorial diseases. Among these regulation’s role differences, the majority concerns modifications in the genome that in the affect the level of expression of certain genes. With colleagues from King’s College, Oxford University and the Wellcome Trust Sanger Ins- predisposition to titute, Emmanouil Dermitzakis, Louis-Jeantet Professor at the Faculty of Medicine of the University of Geneva (UNIGE), and his team disco- common diseases vered several thousands variants affecting the expression levels of genes, 358 of which seem to play a key role in the predisposition to certain diseases. This study is being published in the journal Nature An international team co- Genetics. led by Professor Emmanouil Genetic disease risk differences between one individual and another Dermitzakis of the University are based on complex aetiology. Indeed, they may reflect differences of Geneva has discovered in the genes themselves, or else differences at the heart of the regions several thousands new involved in the regulation of these same genes. genetic variants impacting By gene regulation we mean the decision that the cell makes as to gene expression some of when, where and at what level to activate or suppress the expression of a gene. In theory, two people could thus share a gene that is per- which are responsible for fectly identical and yet show differences in their predisposition to a predisposition to common disease due to genetic differences concerning the regulation (overex- diseases, bringing closer to pression or underexpression) of this same gene. -
Procesos De Mercado
PROCESOS DE MERCADO REVISTA EUROPEA DE ECONOMÍA POLÍTICA Procesos de Mercado Yeso y pigmentos sobre tela y madera Julio Toquero, 2005 «Subyace en este cuadro una reflexión relativa al cambio, a la energía —acción vigorosa— y a la consciencia: realidades fundamentales en los procesos de mercado y en el arte». PROCESOS DE MERCADO REVISTA EUROPEA DE ECONOMÍA POLÍTICA VOLUMEN XVI, NÚMERO 1, PRIMAVERA 2019 REVISTA SEMESTRAL PUBLICADA POR UNIÓN EDITORIAL, CON LA COLABORACIÓN DE LA FACULTAD DE CIENCIAS JURÍDICAS Y SOCIALES DE LA UNIVERSIDAD REY JUAN CARLOS HIMNO Dio, che nell’alma infondere Dios, que has querido poner amor volesti e speme, en nuestra alma el amor y la esperanza, desio nel core accendere debes alumbrar en nuestro corazón tu dei di libertà. el deseo de libertad. Giuramo insiem di vivere Juramos juntos vivir e di morire insieme. y morir juntos. In terra, in ciel En la tierra, en el cielo ... congiungere ci può, ... reunirnos podrá, ci può la tua bontà. podrá tu bondad. Ah! Dio, che nell’alma infondere ¡Ah!, Dios, que has querido poner amor volesti e speme, en nuestra alma el amor y la esperanza, desio nel core accendere debes alumbrar en nuestro corazón tu dei di libertà. el deseo de libertad. Don Carlo (1884), GIUSEPPE VERDI (Dúo de Don Carlo y Don Rodrigo, final de la Escena Primera, Acto Segundo) © 2004, Jesús Huerta de Soto Unión Editorial, S.A. © 2004, Unión Editorial, S.A. Príncipe de Vergara, 109 – 2.ª planta ISSN: 1697-6797 28002 Madrid Depósito legal: M-17.229-2004 Tel: 913 500 228 Fax: 911 812 212 Administración de Procesos de Mercado: Correo: [email protected] c/o Jesús Huerta de Soto www.unioneditorial.es Universidad Rey Juan Carlos procesosdemercado.com Campus de Vicálvaro P.º de los Artilleros, s/n Diseño y Maquetación: RALI, S.A.