Specimen Collection Requirements - SVAN for More Information Visit Or Call: (317) 803-1010 Or (877) 803-1010 (Option #4)

Total Page:16

File Type:pdf, Size:1020Kb

Specimen Collection Requirements - SVAN for More Information Visit Or Call: (317) 803-1010 Or (877) 803-1010 (Option #4) Specimen Collection Requirements - SVAN For more information visit www.maclonline.com or call: (317) 803-1010 or (877) 803-1010 (Option #4) OR Light Royal Blue '(1) Red '(2) Gold SST '(3) Green '(5) Blue '(8) Light Blue (Sodium Citrate 3.2%) Red (No Additive) Gold (SST gel separator) Green (Lithium Heparin) Royal Blue (No Additive) T/F Activated Protein C Resistance Acetominophen Acetone C-peptide Mono Screen Carboxyhemoglobin (CO) (On Ice) Copper T/F Adams 13 Alcohol (Do not open prior to testing) Aldolase DHEAS Osmolality Leukocyte Alkaline Phos Stain (LAP) T/F Alpha 2 Antiplasmin Aldosterone Alpha 1 Antitrypsin DIrect LDL Parvovirus, Human Methemoglobin (On Ice) Royal Blue (EDTA) T/F Antithrombin III Amikacin ANA EBV Screen Phospholipids Arsenic T/F Chromogenic Factor VIII Aminodarone Anticardiolipin Antibody (IgA) Folate Prealbumin * Do not open prior to testing Cadmium T/F Dilute PT Androstenedione Anticardiolipin Antibody (IgG + IgM) HCG, Serum Qualitative Prolactin Heavy Metals, Blood T/F D-Dimer Apolipoprotein B Anti-DNase-B Titer Hepatitis A, IgM Prostatic Acid Phosphatase Mercury T/F DRVVT (Dilute Russel Viper Venom) Barbiturate Anti-Double-Stranded DNA Hepatitis A, Total Protein Electrophoresis Lavender '(6) Zinc T/F Factor II C1Q Binding Assay Anti-Histone AB Hepatitis B Core IgM Antibody PSA Lavender (EDTA) T/F Factor V C5 (Complement) Antiphosphatidylserine Ab (IgA) Hepatitis B Surface Antibody PTH, Intact and Ca++ (plus EDTA) ACTH (place on ice) T/F Factor V Inhibitor Caffeine (Neonatal) Antiphosphatidylserine Ab (IgM + IgG) Hepatitis B Surface Antigen RA Factor/Screen CBC Tan T/F Factor VII Carbamazepine (Tegretol) Anti-Smooth Muscle Antibody Hepatitis BE Antibody RPR CBC with Differential '(10) T/F Factor VIII R Complement, C3 ASO Titer Hepatitis BE Antigen Rubella IgG Cell Count (Body Fluids) Tan (EDTA K2) T/F Factor VIII Inhibitor R Complement, C4 Beta 2 Glycoprotein 1 Ab (IgA) Hepatitis C Antibody Rubeola IgG CMV, PCR, Quant and Qual Lead T/F Factor IX * Complement, Total (CH50) Beta 2 Glycoprotein 1 Ab (IgG + IgM) Hepatitis Panel, Acute Scleroderma Antibody Crystal Analysis (Body Fluids) T/F Factor IX Inhibitor * Cryoglobulin Beta 2 Microglobulin Herpes 1 + 2 IgM Antibody Sjogren's Screen Cyclosporine T/F Factor X Digitoxin Betahydroxybutyrate (BHOB) Histoplasma AB Comp Fix Testosterone Factor V Leiden Mutation T/F Factor XI Digoxin CA 125 HIV Antibody (separate tube) Thyroglobulin Fetal Hemoglobin T/F Factor XII Dihydrotestosterone CA 27.29 Homocysteine G6PD Thyroglobulin Antibody an T/F Factor XIII Doxepin Cancer Antigen 15-3 Immunofixation, Serum Thyroid Receptor Antibody T/F Glucagon (2 tubes) Gray '(11) T/F Fibrin Monomer Complex Ethylene Glycol (place on ice) Carbohydrate Antigen 19-9 Immunoglobulin A Total Bile Acid Hemoglobinopathy Evaluation Gray (NA Fluoride/K Oxalate) T/F Fibrinogen Flecainide Cardio CRP Immunoglobulin G TSH Hemogram Glucose Tolerance Tests (all) T/F Heparin Dependent Antibodies Free Fatty Acid Cardiolipin Antibody Immunoglobulin M Transferrin Hgb A1C T/F Heparin Level Gentamicin CEA Immunoglobulin Panel Varicella Zoster Antibodies Hgb/Hct T/F Hexagonal Phospholipid Neutralization GHB Rohypnal Cholesterol Insulin Vitamin B12 Histamine ** Lupus Anticoagulant (4 tubes) Halidol (Haloperidol) Cortisol *Ionized Calcium Vitamin D, 25 Hydroxy HIV 1 DNA, Qual, PCR T/F Mixing Study PT Hydroxyprogesterone Cortisol (Thyrotropin) Stimulation LH Vitamin E HIV Viral Load T/F Mixing Study PTT Imipramine (Tofranil) Lyme Disease Antibody T/F HSV 1 and 2 PCR, Qual Plasminogen Activator Inhibitor 1 Ag Lamotrigine (Lamictal) DO NOT FREEZE SST TUBE. PLEASE TRANSFER TO POUR OFF TUBE. * Do not open prior to testing Malaria Stain - make slides T/F Plasminogen Activity Librium (Chlordiazepoxide) T/F = Transfer serum to plastic vial - freeze within 2 hours of draw MTHFR Lidocaine Platelet Aggregation See Below Platelet Count Lithium Mint Platelet Function Test See Below Prothrombin 20210 Gene Mutation T/F Protein C Activity Lysozyme (Muramidase) Green Green OR '(4) R PTH, Intact T/F Protein C Ag (Total) Mellaril (Thioridazine) Mint Green (PST gel separator) OR Dark Green, Gold, Tiger Top are also acceptable Rapamycin (Sirolimus) T/F Protein S Activity Meprobamate Acetominophen Electolytes Phosphorus RBC Folate (2 tubes) T/ FProtein S Ag (Free) Methotrexate (protect from light) Albumin Estradiol Potassium RBC Cholinesterase (2 tubes) T/F Protein S Ag (Total) Norpace (Disopramide) *Alcohol Ferritin Procalcitonin T/F Renin T/F PT Oxcarbazephine (Trileptal) Alkaline Phosphatase FSH Progesterone Reticulocyte Count T/F PTT Pentobarbital ALT (SGPT) Gentamycin Renal Panel Reticulocyte Hgb Content T/F Reptilase Time Phenobarbital Ammonia (on ice) GGT Salicylate Sed Rate Ristocetin Dose Response (2 tubes) Phenytoin (Dilantin) Amylase Glucose Sodium Sickle Cell Screen T/F TCT (Thrombin Clotting Time) Primidone (Mysoline) AST (SGOT) Glucose Tolerance Tests (all) T3 Uptake Tacrolimus (Prograf) T/F Von Willebrand Antigen Propafenone Basic Metabolic Profile (BMET) HCG, Quantitative T3, Free Total Eosinophil Count T/F VonWillebrand Factor (VWF) Activity Protriptyline (Vivacity) Bilirubin, Direct HDL T3, Total T/F Vasoactive Peptide T/F VWF Multimers Prozac (Fluoxetine) Bilirubin, Total Hepatic Panel T4 (Thyroxine) T/F Vitamin B1 (protect from light) Ritalin (Methylphenidate) BUN Iron T4, Free T/F Vitamin B6 (protect from light) Salicylate Calcium Iron Study Panel TIBC T/F Vitamin K (protect from light) T/F = Transfer plasma to plastic vial - Theophylline Chloride Lactic Acid (on ice) Total Bilirubin Antibody Screen freeze within 2 hours of draw Thorazine (Chlorpromazine) CKMB LDH Total Protein ABO Rh **2 full light blue tops and 2 full SST Tobramycin CO2 Lipase Triglyceride Direct Antiglobulin Test (DAT) Trazodone Comp Metabolic Profile (CMET) Lipid Panel Troponin-I Indirect Antiglobulin Test (IAT) Must call for STAT pickup and Valium CPK Magnesium TSH Type and Screen indicate draw time on tubes Valproic Acid C-Reactive Protein (CRP) NTpro-BNP Uric Acid Type and Crossmatch Vancomycin Creatinine Phenytoin (Dilantin) Valproic Acid Rhogam Must use discard tube when using Zonegran (Zonisamide) T/F Hepatitis B Virus DNA Qual PCR butterfly for light blue tubes. * Do not open prior to testing T/F Hepatitis C RNA PCR, Quant * Deliver to lab immediately. T/F HIV Genotyping T/F HIV1 RNA PCR, Quant Numbers in "( )" beside tube R = Refrigerated Transport in Mylar Bag type picture indicate order of draw T/F = Transfer plasma to plastic vial - freeze within 2 hours of draw 10.2020 SVAN.
Recommended publications
  • Section 8: Hematology CHAPTER 47: ANEMIA
    Section 8: Hematology CHAPTER 47: ANEMIA Q.1. A 56-year-old man presents with symptoms of severe dyspnea on exertion and fatigue. His laboratory values are as follows: Hemoglobin 6.0 g/dL (normal: 12–15 g/dL) Hematocrit 18% (normal: 36%–46%) RBC count 2 million/L (normal: 4–5.2 million/L) Reticulocyte count 3% (normal: 0.5%–1.5%) Which of the following caused this man’s anemia? A. Decreased red cell production B. Increased red cell destruction C. Acute blood loss (hemorrhage) D. There is insufficient information to make a determination Answer: A. This man presents with anemia and an elevated reticulocyte count which seems to suggest a hemolytic process. His reticulocyte count, however, has not been corrected for the degree of anemia he displays. This can be done by calculating his corrected reticulocyte count ([3% × (18%/45%)] = 1.2%), which is less than 2 and thus suggestive of a hypoproliferative process (decreased red cell production). Q.2. A 25-year-old man with pancytopenia undergoes bone marrow aspiration and biopsy, which reveals profound hypocellularity and virtual absence of hematopoietic cells. Cytogenetic analysis of the bone marrow does not reveal any abnormalities. Despite red blood cell and platelet transfusions, his pancytopenia worsens. Histocompatibility testing of his only sister fails to reveal a match. What would be the most appropriate course of therapy? A. Antithymocyte globulin, cyclosporine, and prednisone B. Prednisone alone C. Supportive therapy with chronic blood and platelet transfusions only D. Methotrexate and prednisone E. Bone marrow transplant Answer: A. Although supportive care with transfusions is necessary for treating this patient with aplastic anemia, most cases are not self-limited.
    [Show full text]
  • Method Development and Validation of Vitamin D2 and Vitamin D3 Using Mass Spectrometry
    Method Development and Validation of Vitamin D2 and Vitamin D3 Using Mass Spectrometry Devon Victoria Riley A thesis submitted in partial fulfillment of the requirements for the degree of Master of Science University of Washington 2016 Committee: Andrew Hoofnagle Geoffrey Baird Dina Greene Program Authorized to Offer Degree: Laboratory Medicine ©Copyright 2016 Devon V. Riley ii University of Washington Abstract Method Development and Validation of Vitamin D2 and Vitamin D3 Using Mass Spectrometry Devon V. Riley Chair of the Supervisory Committee: Associate Professor Andrew Hoofnagle, MD, PhD Vitamin D has long been known to maintain bone health by regulating calcium and phosphorous homeostasis. In recent years, scientists have discovered additional physiological roles for vitamin D. The complex interaction between the active vitamin D hormone and its metabolic precursors continues to be a rich area of research. Fundamental to this research is the availability of accurate and precise assays. Few published assays for vitamins D2 and D3 have contained sufficient details on method validation or performance characteristics. The liquid chromatography-tandem mass spectrometry (LC-MS/MS) assay developed for this thesis has undergone a rigorous validation and proven to yield a sensitive and specific method that exceeds the capabilities of all previously published methods. Developing and validating a novel assay is often complicated by the lack of established acceptability standards. This thesis explores this challenge, specifically for establishing meaningful interpretations and qualification standards of the lower limit of the measuring interval. Altogether, future research focused on vitamins D2, D3 and the Vitamin D pathway can benefit from this robust LC-MS/MS assay and the associated quality parameters outlined in this thesis.
    [Show full text]
  • Evaluation of Prolonged Surface Activated Coagulation Time
    Biomedical Science Faculty of Health and Society Malm¨oUniversity SE-205 06 Malm¨o Sweden Master programme in Biomedical Surface Science http://edu.mah.se/en/Program/VABSE Evaluation of prolonged surface activated coagulation time Master degree thesis, 30 ECTS Author: Amalie Jesting Supervisors: Sebastian Bj¨orklund,PhD, Lecturer, Malm¨oUniversity Jens Peter Gøtze, MD, DMSc Professor, Chief-Physician, Rigshospitalet Søren Frank Jørgensen, MSc, Senior Lecturer, Metropolitan University College August 2018 MALMO¨ UNIVERSITY Abstract Faculty of Health and Society Master programme in Biomedical Surface Science Evaluation of prolonged surface activated coagulation time by Amalie Jesting Background: Blood coagulation is an essential defense mechanism to prevent bleeding. Disorders in the coagulation system can be severe and blood tests measuring the blood's ability to coagulate are important. Activated partial thromboplastin time (APTT) is a blood test that measures blood coagulation time. An abnormal prolonged APTT can both be associated with a bleeding tendency or a risk of thrombosis. Additional blood tests are needed to discover the cause of a prolonged APTT. One potential test is the APTT mixing study, which can separate samples with and without inhibitors. The aim of this project is to investigate how the cause of a prolonged APTT is evaluated today and to examine if it is possible to indicate the cause of a prolonged APTT using the APTT mixing study performed on routine samples. The goal is to be able to indicate the cause of a prolonged APTT immediately when is it first discovered. This will save time and help guide the physicians in their work with the patient.
    [Show full text]
  • A Life-Threatening Case of Pregnancy-Related Atypical
    Puri et al. BMC Nephrology (2020) 21:488 https://doi.org/10.1186/s12882-020-02100-4 CASE REPORT Open Access A life-threatening case of pregnancy- related atypical Haemolytic uremic syndrome and successful treatment with Eculizumab Prianka Puri1,2* , Anida Hanxhiu1, Daniel V. O’Hara1,3, Danny Hsu4 and Mirna Vucak-Dzumhur1,5 Abstract Background: Pregnancy-related Atypical Haemolytic Uremic Syndrome (P-aHUS) is a rare condition affecting genetically predisposed women during pregnancy. It is often difficult to diagnose and has a significant impact on maternal and foetal outcomes. It is characterised by microangiopathic haemolytic anaemia and kidney injury from thrombotic microangiopathy. Case presentation: A 27-year-old female of Lebanese descent presented at 36 weeks’ gestation with foetal death in-utero (FDIU) with placental abruption on a background of previously normal antenatal visits. She was coagulopathic and anaemic with anuric acute kidney injury, requiring emergency Caesarean section, intubation and dialysis. Her coagulopathy rapidly resolved, however, her anaemia and renal dysfunction persisted. A diagnosis of P-aHUS was made, and she was empirically treated with Eculizumab. Her ADAMTS13 level was normal, effectively excluding thrombotic thrombocytopenic purpura. Within 2 weeks of treatment her haematological parameters improved, and her renal function began to recover and within 2 months she became dialysis independent. Conclusion: This case highlights the challenges of a timely diagnosis of P-aHUS from other pregnancy-related diseases. Although our patient is dialysis-independent, her risk of relapse remains high with subsequent pregnancies. Currently we are awaiting her genetic sequencing to complete her assessment for underlying mutations and are determining the safest approach to a future planned pregnancy.
    [Show full text]
  • Mixing Studies
    Welcome Jim DeMase, Senior National Technical Sales Manager Sunday March 19, 2017/CAMLT/Kaiser Regional Reference Lab Welcome Your Presenter Today Jim DeMase, Senior National Technical Sales Manager, Precision BioLogic Account Manger for Western and Central USA and British Columbia, Alberta, Saskatchewan and Manitoba CANADA. H A L L A Hemostasis Is Unique-The Cascade, the Diseases and the Tests-All Mixed Up Part 1- What is Hemostasis Part 2- Intro to Hemophilia Part 3- Mixing Studies Objectives 1. Describe the stages of hemostasis as well as common bleeding and thrombotic disorders. 2. Explain the types of hemophilia, symptoms, diagnosis and common treatments. 3. Relate the methods and clinical application for a mixing study and the steps to perform one. What is Hemostasis? Pathology Study of diseases Especially the structural and functional changes (to the body) caused by the diseases Many disciplines Biochemistry Blood Transfusion Services Histology Microbiology Hematology Hematology Science of blood and its diseases Sub-disciplines: Immunology Microscopy Molecular Biology Hemostasis (Coagulation) Hemostasis The process of stopping bleeding Greek roots heme, blood + stasis, halt = halt of the blood Blood Red viscous liquid in arteries, veins and capillaries Pumped by the heart Irrigates every tissue Transport of gases, nutritive materials and elements for immunity Blood Leading the way in experimental and clinical research in hematology Blood composition Cellular Red cells: hemoglobin White cells: neutrophils, monocytes, lymphocytes Platelets: small cells, essential role in prevention of blood loss Liquid Plasma: yellow liquid, composed mainly of lightly salted water containing nutritional materials, waste products and numerous different proteins Proteins in the Plasma Albumin Globulins Coagulation proteins Procoagulant (e.g.
    [Show full text]
  • Outpatient Laboratory Requisition
    Yale-New Haven Hospital Department of Laboratory Medicine Outpatient Laboratory Requisition 20 York Street, New Haven, Connecticut 06510-3202 · 203-688-3670 · 1-800-305-3278 DATE: _____/____/_____ Unit #___________________________ Visit #_______________________________ Patient Name _______________________________________________________________________________ (Last, First, Middle Initial) Date of Birth______/_____/______ ❑ Female ❑ Male Patient Telephone: (______) ________-_____________ Address ___________________________________________________________________________________ (Street, City, State, Zip) ❑ Mr. Insured’s Name ❑ M r s . _______________________________________________________________________ Ms. Insured’s I.D. # __❑____________________________________________________________________________ Payor Number ______________________________________________________________________________ Primary Insurance: Name & State _______________________________________________________________ Relationship to patient: ❑ Self ❑ Spouse ❑ Child ❑ Other __________________________________________ Group # _________________________________ Insured’s Employer ________________________________ Diagnosis / ICD 9 Code _________________________ TIME DRAWN: ________ ❑ YNHH Blood Draw ❑ Fasting Specimen Type: ❑ Blood ❑ Urine ❑ Other_______________________ ❑ CSF ❑ Fluid____________ ❑ Bone Marrow ❑ Call / ❑ Fax Results ❑ Timed Urine Date Collected _______ Time Collected ________ To:________________________________ CC: X TEST NAME SCC CODE X TEST NAME SCC CODE
    [Show full text]
  • Pt Mixing Studies
    Lab Dept: Coagulation Test Name: PT MIXING STUDIES General Information Lab Order Codes: PTM Synonyms: Protime Mixing Study; Prothrombin Time Mixing Study; Simple PT Mixing Study CPT Codes: 85611 - Prothrombin time: substitution, plasma fractions, each Test Includes: PT 1:2 dilution reported in seconds. Logistics Test Indications: Useful for detecting the presence/absence of an inhibitor or the absence of specific coagulation factors. Lab Testing Sections: Coagulation Phone Numbers: MIN Lab: 612-813-6280 STP Lab: 651-220-6550 Test Availability: Daily, 24 hours Turnaround Time: 4 hours Special Instructions: ● Patient’s with hematocrit levels >55% must have a special tube made to adjust for the hematocrit; contact the laboratory for special tube. ● Specimen must arrive within 30 minutes of collection. ● Indicate when specimen is drawn from a line or a heparin lock. Specimen Specimen Type: Whole blood Container: Light Blue top tube (Buffered Na Citrate 3.2%) Draw Volume: 2.7 mL blood Processed Volume: 1 mL plasma Collection: ● A clean venipuncture is essential, avoid foaming. ● Entire sample must be collected with single collection, pooling of sample is unacceptable. ● Capillary collection is unacceptable. ● Patient’s with a hematocrit level >55% must have a special tube made to adjust for the hematocrit; contact lab for a special tube. ● Mix thoroughly by gentle inversion. Deliver immediately to the laboratory at room temperature via courier or pneumatic tube. Off campus collections: ● Must be tested within 4 hours. ● Do not refrigerate. ● If not received in our lab within 4 hours of collection, sample must be centrifuged and *platelet-poor plasma removed from cells and transferred to an aliquot tube being careful not to disturb the cell layer.
    [Show full text]
  • Mixing Study
    Improving Acute Care Using Coagulation Mixing Studies George A Fritsma MS, MLS The Fritsma Factor, Your interactive Hemostasis Resource℠ Sponsored by Precision BioLogic Dartmouth, Nova Scotia [email protected] www.fritsmafactor.com 1 Surgeons and physicians order PT and PTT assays to predict bleeding risk, often generating isolated, unexplained prolonged PTs or PTTs. In follow-up, the laboratory practitioner mixes patient plasma with normal plasma and repeats the assay. Mixing studies may be provided at community hospitals and acute care facilities. The information gained from mixing studies is an essential first step in the diagnosis of many hemostatic abnormalities. During this presentation, we discuss the importance of mixing studies, describe how they are performed, and show how their results contribute to the diagnosis. 2 Bottom Line at the Start (BLAST) The participant… • Lists the clinical applications for a PTT mixing study. • Lists the steps to perform a PTT mixing study. • Explains why the mixing study is an acute care assay. • Correlates mixing study results with lupus anticoagulant and specific inhibitor testing. 3 Mixing Study: An Acute Care Assay Differentiates a coagulopathy from a specific inhibitor or from a lupus anticoagulant + = Kershaw GK, Orellana D. Mixing tests: diagnostic aides in the investigation of prolonged prothrombin times and activated partial thromboplastin times. Semin Thrombos Hemost 2013;39:283–90. 4 Case: 32-yo Female Pre-op Screen • Six weeks post-partum • Easy bruising, frequent nosebleeds,
    [Show full text]
  • Blood Coagulation Overview and Inherited Hemorrhagic Disorders 2002 Abshire
    Blood Coagulation Overview and Inherited Hemorrhagic Disorders 2002 Abshire 1) Thrombin is one of the key proteins in the coagulation cascade and has both procoagulant and anticoagulant properties. Which one of the following is an important anticoagulation function: a. Activation of factors V and VIII b. Factor XIII activation c. Stimulation of the thrombin activated fibrinolytic inhibitor (TAFI) d. Complex with thrombomodulin (TM). 2) The endothelial cell lining blood vessels possesses several important anticoagulation functions . Which one of the following is one of its' important hemostasis (procoagulant) functions? a. Production of nitric oxide (NO) b. Activation of thrombomodulin (TM) c. Release of plasminogen activator inhibitor (PAI-I) d. Release of TPA e. Interaction of heparin sulfate with anti-thrombin III (AT III) 3) A term well infant is born without complications after a normal pregnancy and initial nursery stay. Upon drawing the metabolic screen, the baby is noted to have prolonged oozing from the heelstick sample. CBC, plt ct, PT, a PTT and fibrinogen were normal. Family history is negative. He returns for the 2 week check and the mother notices that the umbilical cord has been oozing for 4 days. A screening test for factor XIII deficiency is ordered and the baby's clot does not dissolve in 5 M urea. The euglobulin lysis time (ELT) was less than one hour. The most likely diagnosis is: a. Factor XIII heterozygous deficiency b. Homozygous antiplasmin deficiency c. Mild hemophilia B (Factor IX deficiency) d. von Willebrand Disease e. Tissue plasminogen activator (TPA) excess. 4) The newborn's coagulation system is both similar and quite different from a child's.
    [Show full text]
  • Elevated APTT? How Best to Follow Up
    Online ExClusivE Habib Ur Rehman, MBBS, FACP, FRCPC Elevated APTT? Department of Medicine, Regina General Hospital, How best to follow up Saskatchewan, Canada [email protected] This step-by-step guide will help you refine The author reported no potential conflict of interest your approach to an abnormal activated partial relevant to this article. thromboplastin time. CASE u During an office visit, 23-year-old CASE u mr. K’s physical examination is un- john K tells you that he recently experienced remarkable and his renal and liver panel— excessive bleeding after a dental extraction. including serum albumin—are within the he says that he has no personal or family his- normal range. as noted earlier, he had no tory of a bleeding disorder and is not taking history of bleeding before the tooth extrac- any medication that might have contributed tion; his family history is negative for excessive to the problem. bleeding, as well. his lab work shows a normal complete blood count and international normalized ratio (inr). his activated partial thrombo- Consider an artifactual cause plastin time (apTT), however, is 67 seconds It’s important to rule out artifactual causes (normal=24-37 seconds). of an abnormal APTT before undertaking a how would you manage his care? more detailed investigation. Although not applicable in this case, unfractionated hepa- orrect interpretation of abnormal rin in a central venous or arterial catheter can APTT readings requires an under- prolong APTT. C standing of the clinical context in z A high hematocrit value will give which the test is ordered and the test’s limita- falsely prolonged APTT.
    [Show full text]
  • Charge Master
    Epic Charge Code Description Epic Price HC PBB FNA BIOPSY W/O IMAGE GUIDE EA ADDL LESION $587.00 HC FNA BIOPSY W/US GUIDANCE 1ST LESION $1,540.00 HC FNA BIOPSY W/US GUIDANCE EA ADDL LESION $610.25 HC FNA BIOPSY W/FLUORO GUIDANCE 1ST LESION $1,540.00 HC FNA BIOPSY W/FLUORO GUIDANCE EA ADDL LESION $610.25 HC FNA BIOPSY W/CT GUIDANCE 1ST LESION $1,540.00 HC FNA BIOPSY W/CT GUIDANCE EA ADDL LESION $610.25 HC FNA BIOPSY W/MR GUIDANCE 1ST LESION $1,540.00 HC FNA BIOPSY W/MR GUIDANCE EA ADDL LESION $610.25 HC FNA BIOPSY W/O IMAGE GUIDE 1ST LESION $788.00 HC IMAGE-GUIDED CATHETER FLUID COLLECTION DRAINAGE $1,205.50 HC PERQ SFT TISS LOC DEVICE PLMT 1ST LES W/GDNCE $3,652.00 HC PLACEMENT SOFT TISSUE LOCALIZATION DEVICE PERQ EA ADDL W/IMAGE $1,995.00 HC PBB ACNE SURGERY $597.00 HC DRAIN SKIN ABSCESS SIMPLE $441.00 HC DRAIN SKIN ABSCESS COMPLIC $842.75 HC DRAIN PILONIDAL CYST SIMPL $1,552.75 HC REMOVE FOREIGN BODY SIMPLE $842.75 HC REMOVE FOREIGN BODY COMPLIC $1,945.00 HC DRAINAGE OF HEMATOMA/FLUID $1,945.00 HC PUNCTURE DRAINAGE OF LESION $842.75 HC COMPLEX DRAINAGE, WOUND $3,148.75 HC DEBRIDEMENT, INFECTED SKIN, UP TO 10% BSA $1,127.00 HC DEBRIDE ASSOC OPEN FX/DISLO SKIN/MUS/BONE $2,717.50 HC DEBRIDEMENT, SKIN, SUB-Q TISSUE,=<20 SQ CM $938.75 HC DEBRIDEMENT, SKIN, SUB-Q TISSUE,MUSCLE,=<20 SQ CM $1,453.65 HC DEBRIDEMENT BONE 1ST </=20 SQ CM $2,650.00 HC DEBRIDEMENT, SKIN, SUB-Q TISSUE,EACH ADD 20 SQ CM $469.50 HC DEBRIDEMENT, SKIN, SUB-Q TISSUE,MUSCLE,EACH ADD 20 SQ CM $726.80 HC DEBRIDEMENT, SKIN, SUB-Q TISSUE,MUSCLE,BONE,EACH ADD 20 SQ CM $1,450.00
    [Show full text]
  • Reflex Test Protocols 2016
    MGH Pathology Service Laboratory Reflex Test Protocols 2016 Reflex Test Protocols Reflex tests fall into 2 categories: standard industry practice (example: sensitivities, interpretations, and confirmations) and institutional practice based on staff clinical practice Tests that are reflexed should be reasonable and medically necessary. The medical community should approve reflex protocols. The physician must have the option of selecting a reflex protocol or a single test. Blood Transfusion Services Test ordered on Requisition Initial Test Performed Criteria for Reflex Test Ordered by Reflex Antibody Screen Antibody Screen Positive screen Antibody identification Cold agglutinin screen Cold agglutinin screen Positive (>1+) Cold agglutinin titer Direct antiglobulin test Direct antiglobulin test IgG positive and transfused w/in 3 Eluate mo Fetal Screen Fetal screen Positive Kleihauer-Betke Core Lab - Chemistry Test ordered on Requisition Initial Test Performed Criteria for Reflex Test Ordered by Reflex Thyroid Screen TSH TSH>5.00 Free T4 TSH<0.40 Free T4. T3 Monitoring L-T4 Therapy TSH TSH<0.05 Free T4 Thyroglobulin Thyroglobulin Specimen received and test Thyroglobulin Antibodies requested Direct LDL Lipid Panel with calculated LDL Triglycerides >399 Direct (measured LDL) Free testosterone Total testosterone, sex hormone Albumin <3.5 Free testosterone measured by binding globulin (SHBG), albumin, equilibrium dialysis free testosterone (calculated), % free testosterone (calculated). Notes: The albumin is needed for the calculation but will
    [Show full text]