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Deep Tissue Pressure Injury Or an Imposter?

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Deep Tissue Pressure Injury Or an Imposter? ® NPNATIONAL PRESSURE INJURYIAP ADVISORY PANEL Improving Patient Outcomes Through Education, Research and Public Policy DEEP TISSUE Intact or non-intact skin with localized area of persistent non-blanchable deep PRESSURE INJURY red, maroon, purple discoloration or epidermal separation revealing a dark OR AN IMPOSTER? wound bed or blood-filled blister. Pain and temperature change often precede skin color changes. Discoloration may appear differently in darkly pigmented skin. This injury results from intense and/or prolonged pressure and shear forces at the bone-muscle interface. The wound may evolve rapidly to reveal the actual extent of tissue injury or may resolve without tissue loss. If necrotic tissue, subcutaneous tissue, granulation tissue, fascia, muscle or other underlying structures are visible, this indicates a full thickness pressure injury (Unstageable, Stage 3 or Stage 4). Initial DTPI Initially intact purple or Sacral DTPI after cardiac surgery in Low sacral-coccygeal DTPI in a patient Forehead DTPI after surgery in sitting in High-Fowler’s position prone position 24 hours ago maroon skin or blood blister supine position 48 hours ago DTPI of right buttock with early separation DTPI of right para-sacrum with early DTPI of para-sacrum with blistering, of the dermis, 72 hours after surgery done separation of the dermis, 72 hours after 72 hours after cardiac surgery in with patient rotated to the right mechanical ventilation for hypoxia supine position Evolving DTPI as epidermis sloughs Blistered appearance DTPI of para-sacrum with blistering, DTPI of buttocks with blistering, 72 hours Blood blister - Tissue may be 72 hours after cardiac surgery in after mechanical ventilation for hypoxia hard to the touch or boggy supine position ® NPNATIONAL PRESSURE INJURYIAP ADVISORY PANEL Improving Patient Outcomes Through Education, Research and Public Policy DEEP TISSUE Many conditions can lead to purple or ecchymotic skin and rapidly developing PRESSURE INJURY eschar. Some of the most common differential diagnoses are shown below. OR AN IMPOSTER? Ischemia Trauma COVID-19 Embolic Disease Warfarin Induced Skin Necrosis Hematoma COVID-19 accelerates clotting in Marked disease of internal Erythematous flushing then History of trauma to area, small vessels Skin color change iliacs or postoperative aorto- progressing within 24 hours to often anticoagulated - Area is is not always on pressure iliac bypass with emboli full thickness hemorrhagic bullae palpable and often tender bearing tissues several days after high loading doses of Warfarin. Vasopressor Induced Ischemia From Hypotension Blunt Trauma Chronic Friction Injury Peripheral Ischemia Sudden purpura near end of life, History of traumatic injury Immobile or chairbound patient Levophed in use - Ischemia of no pressure events had occurred. Irregular shape who uses a slide board ears, nose, fingers also common Patient died 4 days later Painful to touch. Morel Lavallée Skin thick and irregular lesions Lesions are possible DIC/Sepsis with Calciphylaxis (AKA Calcific Bruise Skin Tear Microvascular Emboli Uremic Arteriopathy) History of trauma in the area Patient fell attempting to Reticular presentation Seen in patients in dialysis Color changes to yellow and green in ambulate. Usually, profuse Spontaneous onset, rapidly dependent renal failure due to hyper- a few days bleeding. necrotic parathyroidism, hypercalcemia and hyper-phosphatemia.
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