Diarrhea After Hours Telephone Triage Protocols | Adult | 2015
Total Page:16
File Type:pdf, Size:1020Kb
Load more
Recommended publications
-
Peptic Ulcer Disease
Peptic Ulcer Disease orking with you as a partner in health care, your gastroenterologist Wat GI Associates will determine the best diagnostic and treatment measures for your unique needs. Albert F. Chiemprabha, M.D. Pierce D. Dotherow, M.D. Reed B. Hogan, M.D. James H. Johnston, III, M.D. Ronald P. Kotfila, M.D. Billy W. Long, M.D. Paul B. Milner, M.D. Michelle A. Petro, M.D. Vonda Reeves-Darby, M.D. Matt Runnels, M.D. James Q. Sones, II, M.D. April Ulmer, M.D., Pediatric GI James A. Underwood, Jr., M.D. Chad Wigington, D.O. Mark E. Wilson, M.D. Cindy Haden Wright, M.D. Keith Brown, M.D., Pathologist Samuel Hensley, M.D., Pathologist Jackson Madison Vicksburg 1421 N. State Street, Ste 203 104 Highland Way 1815 Mission 66 Jackson, MS 39202 Madison, MS 39110 Vicksburg, MS 39180 Telephone 601/355-1234 • Fax 601/352-4882 • 800/880-1231 www.msgastrodocs.com ©2010 GI Associates & Endoscopy Center. All rights reserved. A discovery that Table of contents brought relief to millions of ulcer What Is Peptic Ulcer Disease............... 2 patients...... Three Major Types Of Peptic Ulcer Disease .. 6 The bacterium now implicated as a cause of some ulcers How Are Ulcers Treated................... 9 was not noticed in the stomach until 1981. Before that, it was thought that bacteria couldn’t survive in the stomach because Questions & Answers About Peptic Ulcers .. 11 of the presence of acid. Australian pathologists, Drs. Warren and Marshall found differently when they noticed bacteria Ulcers Can Be Stubborn................... 13 while microscopically inspecting biopsies from stomach tissue. -
Chronic Diarrhea
Chronic Diarrhea Barbara McElhanon, MD Subra Kugathasan, MD Emory University School of Medicine 2013 Resident Education Series Reviewed by Edward Hoffenberg, MD of the Professional Education Committee Case • A 15 year old boy with PMH of obesity, anxiety disorder & ADHD presents with 3 months of non-bloody loose stool 5-15 times/day and diffuse abdominal pain that is episodically severe Case - History • Wellbutrin was stopped prior to the onset of her symptoms and her Psychiatrist was weaning Cymbalta • After stopping Cymbalta, she went to Costa Rica for a month long medical mission trip • Started having symptoms of abdominal pain and diarrhea upon return from her trip. • Ingestion of local Georgia creek water, but after her symptoms had started • Subjective fever x 4 days Case - Lab work by PCP • At onset of illness: – + occult blood in stool – + stool calprotectin (a measure of inflammation in the colon) – Negative stool WBC – Negative stool culture – Negative C. difficile – Negative ova & parasite study – Negative giardia antigen – Normal CBC with diff, Complete metabolic panel, CRP, ESR Case - History • Non-bloody diarrhea and abdominal pain continues • No relation to food • No fevers • No weight loss • Normal appetite • No night time occurrences • No other findings on ROS • No sick contacts Case – Work-up prior to visit Labs Imaging and Procedures • MRI enterography (MRI of the • Fecal occult blood, stool abdomen/pelvis with special cuts calprotectin, stool WBC, stool to evaluate the small bowel) culture, stool O&P, stool giardia -
Gastrointestinal Illness (GI)
Gastrointestinal Illness (GI) Gastrointestinal illness (GI) is one of the most common causes of outbreaks in LTCFs. There are many causal agents for GI illnesses including: viruses like Hepatitis A and norovirus, bacteria like E. coli and Salmonella and parasites such as Giardia and Cryptosporidium. • Mode of Transmission: Person-to-person through the fecal-oral route, but can also be 2 transferred through contaminated food and objects • Symptoms: ◦ Bacteria – loss of appetite, nausea and vomiting, diarrhea, abdominal pain/cramps, blood in stool, fever ◦ Virus – watery diarrhea, nausea and vomiting, headache, muscle aches ◦ Ova and Parasites – diarrhea, mucous/blood in stool, nausea or vomiting, severe abdominal pain • Duration: Less than two weeks Gastrointestinal Illness: Any combination of diarrhea (≥ 3 loose stools in 24 hours), vomiting, abdominal pain, with or without fever Gastrointestinal Illness Outbreak: The occurrence of more cases of GI illness in a 24-hour period than would normally be expected based on a facility’s individual surveillance data Precautions • Practice proper hand hygiene. • Clean and disinfect contaminated surfaces. • Follow the CDC’s Standard Precautions guidelines. • Understand that any patient with foodborne illness may represent the sentinel case of a more widespread outbreak. • Communicate with patients about ways to prevent food-related diseases. • Wash fruits and vegetables and cook all food, including seafood thoroughly. • When you are sick, do not prepare food or care for others who are sick. • Wash laundry thoroughly. Reporting Process Upon suspicion of a GI outbreak, facilities are required to notify DOH-Collier at (239)252-8226. Once notified, DOH-Collier will provide initial guidance, educational materials and two forms (listed below). -
Frequently Used Diagnosis Codes
Frequently Used Diagnosis Codes LIVER DIAGNOSIS CODES: Kidney/Pancreas Diagnosis Codes Abdominal pain, generalized R10.84 Abdominal pain, unspec site R10.84 Abdominal pain, RUQ R10.11 Diabetes type I (juvenile) controlled E10.9 Alpha-1-antitrypsin deficiency E88.01 Diabetes type II controlled E11.9 Ascites, other R18.8 Diabetic Nephrosis E11.21 Autoimmune disease, NOS M35.9 Kidney Transplant Complication T86.10 Biliary atresia Q44.2 Kidney Transplant, Failure T86.12 Blood in stool K92.1 Kidney Transplant, Infection T86.13 Cholangitis and/or PSC K83.0 Kidney Transplant, Rejection T86.11 Cirrhosis, Biliary (PBC-PSC) K74.5 Pancreas Transplant Complication T86.899 Cirrhosis, Liver w/o Alcohol K74.60 Renal Failure, ESRD N18.6 Cirrhosis, Liver w/Alcohol K70.30 Transplant-Kidney Status Post Z94.0 Colitis, Ulcerative Unspec K51.90 Transplant -Pancreas Status Post Z94.83 Crohn's Disease, large intestine K50.10 Cystic disease, liver, congential Q44.6 Heart/Lung Diagnosis Codes Encephalopathy, Hepatic K72.90 Airway Stenosis J98.8 Esophageal Varices w/ bleeding I85.00 Cardiomyopathy, NOS, Idiopathic I42.8 Esophageal Varices W/O bleeding I85.01 Complication Heart Transplant - T86.21 Hepatitis A, Viral w/p hepatic coma B15.9 Complication Lung Transplant -T86.810 Hepatitis B, Chronic B18.1 COPD J44.9 Hepatitis B, Acute B16.9 Failure, Congestive Heart I50.9 Hepatitis C, Acute w/o hepatic coma B17.10 Fibrosis, Cystic E84.9 Hepatitis C, Chronic B18.2 Fibrosis, Pulmonary I84.10 Hepatosplenomegaly R16.2 Fibrosis, Idiopathic J84.112 Jaundice, not newborn -
Active Peptic Ulcer Disease in Patients with Hepatitis C Virus-Related Cirrhosis: the Role of Helicobacter Pylori Infection and Portal Hypertensive Gastropathy
dore.qxd 7/19/2004 11:24 AM Page 521 View metadata, citation and similar papers at core.ac.uk ORIGINAL ARTICLE brought to you by CORE provided by Crossref Active peptic ulcer disease in patients with hepatitis C virus-related cirrhosis: The role of Helicobacter pylori infection and portal hypertensive gastropathy Maria Pina Dore MD PhD, Daniela Mura MD, Stefania Deledda MD, Emmanouil Maragkoudakis MD, Antonella Pironti MD, Giuseppe Realdi MD MP Dore, D Mura, S Deledda, E Maragkoudakis, Ulcère gastroduodénal évolutif chez les A Pironti, G Realdi. Active peptic ulcer disease in patients patients atteints de cirrhose liée au HCV : Le with hepatitis C virus-related cirrhosis: The role of Helicobacter pylori infection and portal hypertensive rôle de l’infection à Helicobacter pylori et de la gastropathy. Can J Gastroenterol 2004;18(8):521-524. gastropathie liée à l’hypertension portale BACKGROUND & AIM: The relationship between Helicobacter HISTORIQUE ET BUT : Le lien entre l’infection à Helicobacter pylori pylori infection and peptic ulcer disease in cirrhosis remains contro- et l’ulcère gastroduodénal dans la cirrhose reste controversé. Le but de la versial. The purpose of the present study was to investigate the role of présente étude est de vérifier le rôle de l’infection à H. pylori et de la gas- H pylori infection and portal hypertension gastropathy in the preva- tropathie liée à l’hypertension portale dans la prévalence de l’ulcère gas- lence of active peptic ulcer among dyspeptic patients with compen- troduodénal évolutif chez les patients dyspeptiques souffrant d’une sated hepatitis C virus (HCV)-related cirrhosis. -
Challenges in the Management of Acute Peptic Ulcer Bleeding
Review Challenges in the management of acute peptic ulcer bleeding James Y W Lau, Alan Barkun, Dai-ming Fan, Ernst J Kuipers, Yun-sheng Yang, Francis K L Chan Acute upper gastrointestinal bleeding is a common medical emergency worldwide, a major cause of which are bleeding Lancet 2013; 381: 2033–43 peptic ulcers. Endoscopic treatment and acid suppression with proton-pump inhibitors are cornerstones in the Institute of Digestive Diseases, management of the disease, and both treatments have been shown to reduce mortality. The role of emergency surgery The Chinese University of Hong continues to diminish. In specialised centres, radiological intervention is increasingly used in patients with severe and Kong, Hong Kong, China (Prof J Y W Lau MD, recurrent bleeding who do not respond to endoscopic treatment. Despite these advances, mortality from the disorder Prof F K L Chan MD); Division of has remained at around 10%. The disease often occurs in elderly patients with frequent comorbidities who use Gastroenterology, McGill antiplatelet agents, non-steroidal anti-infl ammatory drugs, and anticoagulants. The management of such patients, University and the McGill especially those at high cardiothrombotic risk who are on anticoagulants, is a challenge for clinicians. We summarise University Health Centre, Quebec, Canada the published scientifi c literature about the management of patients with bleeding peptic ulcers, identify directions for (Prof A Barkun MD); Institute of future clinical research, and suggest how mortality can be reduced. Digestive Diseases, Xijing Hospital, Fourth Military Introduction by how participants were sampled, their inclusion Medical University, Xian, China (Prof D Fan MD); Department of Acute upper gastrointestinal bleeding is characterised by criteria, and defi nitions of case ascertainment. -
An Overview: Current Clinical Guidelines for the Evaluation, Diagnosis, Treatment, and Management of Dyspepsia$
Osteopathic Family Physician (2013) 5, 79–85 An overview: Current clinical guidelines for the evaluation, diagnosis, treatment, and management of dyspepsia$ Peter Zajac, DO, FACOFP, Abigail Holbrook, OMS IV, Maria E. Super, OMS IV, Manuel Vogt, OMS IV From University of Pikeville-Kentucky College of Osteopathic Medicine (UP-KYCOM), Pikeville, KY. KEYWORDS: Dyspeptic symptoms are very common in the general population. Expert consensus has proposed to Dyspepsia; define dyspepsia as pain or discomfort centered in the upper abdomen. The more common causes of Functional dyspepsia dyspepsia include peptic ulcer disease, gastritis, and gastroesophageal reflux disease.4 At some point in (FD); life most individuals will experience some sort of transient epigastric pain. This paper will provide an Gastritis; overview of the current guidelines for the evaluation, diagnosis, treatment, and management of Gastroesophageal dyspepsia in a clinical setting. reflux disease (GERD); r 2013 Elsevier Ltd All rights reserved. Nonulcer dyspepsia (NUD); Osteopathic manipulative medicine (OMM); Peptic ulcer disease (PUD); Somatic dysfunction Dyspeptic symptoms are very common in the general common causes of dyspepsia include peptic ulcer disease population, affecting an estimated 20% of persons in the (PUD), gastritis, and gastroesophageal reflux disease United States.1 While a good number of these individuals (GERD).4 However, it is not unusual for a complete may never seek medical care, a significant proportion will investigation to fail to reveal significant organic findings, eventually proceed to see their family physician. Several and the patient is then considered to have “functional reports exist on the prevalence and impact of dyspepsia in the dyspepsia.”5,6 The term “functional” is usually applied to general population.2,3 However, the results of these studies disorders or syndromes where the body’s normal activities in are strongly influenced by criteria used to define dyspepsia. -
GASTROINTESTINAL COMPLAINT Nausea, Vomiting, Or Diarrhea (For Abdominal Pain – Refer to SO-501) I
Deschutes County Sheriff’s Office – Adult Jail SO-559 L. Shane Nelson, Sheriff Standing Order Facility Provider: February 21, 2020 STANDING ORDER GASTROINTESTINAL COMPLAINT Nausea, Vomiting, or Diarrhea (for Abdominal Pain – refer to SO-501) I. ASSESSMENT a. History i. Onset and duration ii. Frequency of vomiting, nausea, or diarrhea iii. Blood in stool or black stools? Blood in emesis or coffee-ground appearance? If yes, refer to SO-510 iv. Medications taken – do they help? v. Do they have abdominal pain? If yes, refer to SO-501 Abdominal Pain. vi. Do they have other symptoms – dysuria, urinary frequency, urinary urgency, urinary incontinence, vaginal/penile discharge, hematuria, fever, chills, flank pain, abdominal/pelvic pain in females or testicular pain in males, vaginal or penile lesions/sores? (if yes to any of the above – refer to Dysuria SO-522) vii. LMP in female inmates – if unknown, obtain HCG viii. History of substance abuse? Are they withdrawing? Refer to appropriate SO based on substance history and withdrawal concerns. ix. History of IBS or other known medical causes of chronic diarrhea, nausea, or vomiting? Have prescriptions been used for this in the past? x. History of abdominal surgeries? xi. Recent exposure to others with same symptoms? b. Exam i. Obtain Vital signs, including temperature ii. If complaints of dizziness or lightheadedness with standing, obtain orthostatic VS. iii. Is there jaundice present? iv. Are there signs of dehydration – tachycardia, tachypnea, lethargy, changes in mental status, dry mucous membranes, pale skin color, decreased skin turgor? v. Are you concerned for an Acute Gastroenteritis? Supersedes: October 17, 2018 Review Date: February 2022 Total Pages: 3 1 SO-559 February 21, 2020 Symptoms Exam Viruses cause 75-90% of acute gastroenteritis here in the US. -
Gastrointestinal Dis
7/24/2010 Esophageal Disorders • Dysphagia – Difficulty Swallowing and passing food from mouth via the esophagus Gastrointestinal Diseases • Diagnostic aids: Endoscopy, Barium x‐ray, Cineradiology, Scintigraphy, ambulatory esophageal pH monitoring, esophageal Fernando Vega, MD manometery HIHIM 409 Esophageal Disorders Peptic Ulcer Disease • Reflux esophagitis –(GERD) • Hypersecretion of HCL, impaired mucosal • Hiatal Hernia resistance factors • Barret’s esophagus – • Role of H.Pylori – • Esophageal Varices • Bldileeding Ulcer – Diseases of the Small Intestine Wireless capsule endoscopy • Duodenal ulcer • Malabsorption • Regional Enteritis (Crohn’s Disease) • Single use • Gastroenteritis • PlldPropelled by peritliistalsis • Excreted naturally • Inguinal Hernia • 2 frames/second • Last 7‐8 hrs (>50,000 images) 1 7/24/2010 Diarrhea Diarrhea • Single greatest cause of morbidity and • The GI tract absorbs 9 Liter of water a day = 2 mortality in the world liters of dietary fluids+ 7 liters of digestive fluids. Only 100 – 200 ml water comes out in form of stool. Diarrhea Diarrhea • Acute diarrheal illnesses are common and self‐limited. Most often caused by • Caused by impaired absorption or adenoviruses, rotaviruses, astroviruses and cdaliciviruses not detectable by ordinary laboratory tests. No antiviral therapy is available. hypersecretion or both • • Consider: • ↓ absorption • Medication • Antibiotics • Mucosal damage • Travelers’ diarrgia • • Tropical sprue Osmotic Diarrhea • Parasitic infections • • Food poisoning Motility abnormalities -
Leading Article Vaccines Against Gut Pathogens
Gut 1999;45:633–635 633 Gut: first published as 10.1136/gut.45.5.633 on 1 November 1999. Downloaded from Leading article Vaccines against gut pathogens Many infectious agents enter the body using the oral route development.15 Salmonella strains harbouring mutations and are able to establish infections in or through the gut. in genes of the shikimate pathway (aro genes) have For protection against most pathogens we rely on impaired ability to grow in mammalian tissues (they are immunity to prevent or limit infection. The expression of starved in vivo for the aromatic ring).6 Salmonella strains protective immunity in the gut is normally dependent both harbouring mutations in one or two aro genes (i.e., aroA, on local (mucosal) and systemic mechanisms. In order to aroC ) are eVective vaccines in several animal models after obtain full protection against some pathogens, particularly single dose oral administration and induce strong Th1 type non-invasive micro-organisms such as Vibrio cholerae, and mucosal responses.7 An aroC/aroD mutant of S typhi mucosal immunity may be particularly important. There is was well tolerated clinically in human volunteers; mild a need to take these factors into account when designing transient bacteraemia in a minority of the subjects was the vaccines targeting gut pathogens. Conventional parenteral only drawback.8 Th1 responses, cytotoxic T lymphocyte vaccines (injected vaccines) can induce a degree of responses, and IgG, IgA secreting gut derived lymphocytes systemic immunity but are generally poor stimulators of appeared in the majority of vaccinees.89 In an attempt to mucosal responses. -
Peptic Ulcer Disease
\ Lecture Two Peptic ulcer disease 432 Pathology Team Done By: Zaina Alsawah Reviewed By: Mohammed Adel GIT Block Color Index: female notes are in purple. Male notes are in Blue. Red is important. Orange is explanation. 432PathologyTeam LECTURE TWO: Peptic Ulcer Peptic Ulcer Disease Mind Map: Peptic Ulcer Disease Acute Chronic Pathophysiology Morphology Prognosis Locations Pathophysiology Imbalance Acute severe Extreme Gastric Duodenal gastritis stress hyperacidity between agrresive and defensive Musocal Due to factors increased Defenses Morphology acidity + H. Pylori infection Mucus Surface bicarbonate epithelium barrier P a g e | 1 432PathologyTeam LECTURE TWO: Peptic Ulcer Peptic Ulcer Definitions: Ulcer is breach in the mucosa of the alimentary tract extending through muscularis mucosa into submucosa or deeper. erosi on ulcer Chronic ulcers heal by Fibrosis. Erosion is a breach in the epithelium of the mucosa only. They heal by regeneration of mucosal epithelium unless erosion was very deep then it will heal by fibrosis. Types of Ulcer: 1- Acute Peptic Ulcers ( Stress ulcers ): Acutely developing gastric mucosal defects that may appear after severe stress. Pathophysiology: All new terms mentioned in the diagram are explained next page Pathophysiology of acute peptic ulcer Complication of a As a reult of Due to acute severe stress extreme gastritis response hyperacidity Mucosal e.g. Zollinger- inflammation as a Curling's ulcer Stress ulcer Cushing ulcer Ellison response to an syndrome irritant e.g. NSAID or alcohol P a g e | 2 432PathologyTeam -
Upper Gastrointestinal Bleeding, Adult – Emergency Department (UGIB
Provincial Clinical Knowledge Topic Upper Gastrointestinal Bleed, Adult Emergency Department Provincial Clinical Knowledge Topic Upper Gastrointestinal Bleed, Adult Emergency Department Topic Title: Upper Gastrointestinal Bleed, Adult Creation September 2015 Emergency Department Date: Clinical Working Emergency SCN – Upper Revised Group: Gastrointestinal Bleed Date: Status: Approved Version: 1.0 1 Completion Date: September 2015 Version 1.0 Provincial Clinical Knowledge Topic Upper Gastrointestinal Bleed, Adult Emergency Department Table of Contents Important Information Before You Begin ......................................................................................... 5 Rationale ........................................................................................................................................ 6 Goals of Management ..................................................................................................................... 7 Nursing Assessment and Documentation ....................................................................................... 7 Physician Assessment and Documentation..................................................................................... 9 Initial Decision Making .................................................................................................................. 10 Order Set Components ................................................................................................................. 11 Order Set Components - General Care ....................................................................................