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Control of Equilibrium Phases (M,T,S) in the Modified Aluminum Alloy
Materials Transactions, Vol. 44, No. 1 (2003) pp. 181 to 187 #2003 The Japan Institute of Metals EXPRESS REGULAR ARTICLE Control of Equilibrium Phases (M,T,S) in the Modified Aluminum Alloy 7175 for Thick Forging Applications Seong Taek Lim1;*, Il Sang Eun2 and Soo Woo Nam1 1Department of Materials Science and Engineering, Korea Advanced Institute of Science and Technology, 373-1Guseong-dong, Yuseong-gu, Daejeon, 305-701,Korea 2Agency for Defence Development, P.O. Box 35-5, Yuseong-gu, Daejeon, 305-600, Korea Microstructural evolutions, especially for the coarse equilibrium phases, M-, T- and S-phase, are investigated in the modified aluminum alloy 7175 during the primary processing of large ingot for thick forging applications. These phases are evolved depending on the constitutional effect, primarily the change of Zn:Mg ratio, and cooling rate following solutionizing. The formation of the S-phase (Al2CuMg) is effectively inhibited by higher Zn:Mg ratio rather than higher solutionizing temperature. The formation of M-phase (MgZn2) and T-phase (Al2Mg3Zn3)is closely related with both constitution of alloying elements and cooling rate. Slow cooling after homogenization promotes the coarse precipitation of the M- and T-phases, but becomes less effective as the Zn:Mg ratio increases. In any case, the alloy with higher Zn:Mg ratio is basically free of both T and S-phases. The stability of these phases is discussed in terms of ternary and quaternary phase diagrams. In addition, the modified alloy, Al–6Zn–2Mg–1.3%Cu, has greatly reduced quench sensitivity through homogeneous precipitation, which is uniquely applicable in 7175 thick forgings. -
Hitting a Wall: an Ambiguous Case of Wallenberg Syndrome
Open Access Case Report DOI: 10.7759/cureus.16268 Hitting a Wall: An Ambiguous Case of Wallenberg Syndrome James R. Pellegrini 1 , Rezwan Munshi 1 , Bohao Cao 1 , Samuel Olson 2 , Vincent Cappello 1 1. Internal Medicine, Nassau University Medical Center, East Meadow, USA 2. Physical Medicine and Rehabilitation, Stony Brook University, Stony Brook, USA Corresponding author: Bohao Cao, [email protected] Abstract Wallenberg syndrome is the most common stroke of the posterior circulation. Diagnosis of Wallenberg syndrome is often overlooked as initial MRI may show no visible lesion. We present an atypical case of Wallenberg syndrome in which the initial MRI of the brain was normal. Our patient is a 65-year-old male who was brought in by emergency medical services complaining of right- sided facial droop, slurred speech, and left-sided weakness for one day. Physical examination showed decreased left arm and leg strength compared to the right side, decreased left facial temperature sensations, decreased left arm and leg temperature sensations, and difficulty sitting upright with an associated leaning towards the left side. An initial magnetic resonance imaging (MRI) of the brain with and without contrast revealed no abnormality. In light of such a high suspicion for stroke based on the patient’s neurologic deficits, a repeat MRI of the brain was performed three days later and exposed a small focus of bright signal (hyperintensity) on T2-weighted fluid-attenuated inversion recovery and diffusion-weighted imaging (DWI) in the left posterior medulla. Wallenberg syndrome, also known as lateral medullary syndrome or posterior inferior cerebellar artery syndrome, is a constellation of symptoms caused by posterior vascular accidents. -
214 CHAPTER 5 [T, D]-DELETION in ENGLISH in English, a Coronal Stop
CHAPTER 5 [t, d]-DELETION IN ENGLISH In English, a coronal stop that appears as last member of a word-final consonant cluster is subject to variable deletion – i.e. a word such as west can be pronounced as either [wEst] or [wEs]. Over the past thirty five years, this phenomenon has been studied in more detail than probably any other variable phonological phenomenon. Final [t, d]-deletion has been studied in dialects as diverse as the following: African American English (AAE) in New York City (Labov et al., 1968), in Detroit (Wolfram, 1969), and in Washington (Fasold, 1972), Standard American English in New York and Philadelphia (Guy, 1980), Chicano English in Los Angeles (Santa Ana, 1991), Tejano English in San Antonio (Bayley, 1995), Jamaican English in Kingston (Patrick, 1991) and Trinidadian English (Kang, 1 1994), etc.TP PT Two aspects that stand out from all these studies are (i) that this process is strongly grammatically conditioned, and (ii) that the grammatical factors that condition this process are the same from dialect to dialect. Because of these two facts [t, d]-deletion is particularly suited to a grammatical analysis. In this chapter I provide an analysis for this phenomenon within the rank-ordering model of EVAL. The factors that influence the likelihood of application of [t, d]-deletion can be classified into three broad categories: the following context (is the [t, d] followed by a consonant, vowel or pause), the preceding context (the phonological features of the consonant preceding the [t, d]), the grammatical status of the [t, d] (is it part of the root or 1 TP PT This phenomenon has also been studied in Dutch – see Schouten (1982, 1984) and Hinskens (1992, 1996). -
Tail Risk Premia and Return Predictability∗
Tail Risk Premia and Return Predictability∗ Tim Bollerslev,y Viktor Todorov,z and Lai Xu x First Version: May 9, 2014 This Version: February 18, 2015 Abstract The variance risk premium, defined as the difference between the actual and risk- neutral expectations of the forward aggregate market variation, helps predict future market returns. Relying on new essentially model-free estimation procedure, we show that much of this predictability may be attributed to time variation in the part of the variance risk premium associated with the special compensation demanded by investors for bearing jump tail risk, consistent with idea that market fears play an important role in understanding the return predictability. Keywords: Variance risk premium; time-varying jump tails; market sentiment and fears; return predictability. JEL classification: C13, C14, G10, G12. ∗The research was supported by a grant from the NSF to the NBER, and CREATES funded by the Danish National Research Foundation (Bollerslev). We are grateful to an anonymous referee for her/his very useful comments. We would also like to thank Caio Almeida, Reinhard Ellwanger and seminar participants at NYU Stern, the 2013 SETA Meetings in Seoul, South Korea, the 2013 Workshop on Financial Econometrics in Natal, Brazil, and the 2014 SCOR/IDEI conference on Extreme Events and Uncertainty in Insurance and Finance in Paris, France for their helpful comments and suggestions. yDepartment of Economics, Duke University, Durham, NC 27708, and NBER and CREATES; e-mail: [email protected]. zDepartment of Finance, Kellogg School of Management, Northwestern University, Evanston, IL 60208; e-mail: [email protected]. xDepartment of Finance, Whitman School of Management, Syracuse University, Syracuse, NY 13244- 2450; e-mail: [email protected]. -
Acute Stroke Thrombolysis Guideline (Template) TIME IS BRAIN Call “Code Stroke” DOOR to NEEDLE TARGET IS Less Than 60 MINUTES!
Acute Stroke Thrombolysis Guideline (Template) TIME IS BRAIN Call “Code Stroke” DOOR TO NEEDLE TARGET IS less than 60 MINUTES! Guideline: Use of IV Recombinant Tissue Plasminogen Activator (rtPA, tPA, Alteplase, Actilyse®) in Acute Ischaemic Stroke This treatment is indicated in selected acute stroke patients. See New Zealand Clinical Guidelines for Stroke Management 2010 www.nzgg.org.nz Purpose This stroke thrombolysis guideline is intended to guide clinicians when planning stroke thrombolysis with intravenous tissue plasminogen activator (Alteplase). [NB: this template has been developed by the NZ National Thrombolysis Working Group, requires local adaptation, and can be used as a guideline or converted into a pathway by adding tick boxes and patient sticker areas]. Thrombolysis should be considered in patients with acute stroke symptoms arriving within 3.5 hours (treat IV within 4.5 hrs) of symptom onset. Alteplase CANNOT be substituted with any other fibrinolytic agents, including other forms of recombinant tPA. Definitions “Code Stroke” is a key component of effective thrombolysis and denotes a rapid treatment pathway to minimise onset to needle time. It is a communication protocol to activate the appropriate Stroke Thrombolysis Team to meet the patient at the hospital door. This will utilise different resources in different hospitals. It also includes: 1. Avoidance of transit to non-thrombolysis hospitals 2. Pre-hospital notification that a potential stroke thrombolysis patient is en-route with an estimated time of arrival 3. Bloods and IV lines x 2 performed in transit if no delays would result, or otherwise at hospital triage 4. Pre-notification of CT staff of pending arrival 5. -
Tail of a Linear Diffusion with Markov Switching
The Annals of Applied Probability 2005, Vol. 15, No. 1B, 992–1018 DOI: 10.1214/105051604000000828 c Institute of Mathematical Statistics, 2005 TAIL OF A LINEAR DIFFUSION WITH MARKOV SWITCHING By Benoˆıte de Saporta and Jian-Feng Yao Universit´ede Rennes 1 Let Y be an Ornstein–Uhlenbeck diffusion governed by a sta- tionary and ergodic Markov jump process X: dYt = a(Xt)Yt dt + σ(Xt) dWt, Y0 = y0. Ergodicity conditions for Y have been obtained. Here we investigate the tail propriety of the stationary distribution of this model. A characterization of either heavy or light tail case is established. The method is based on a renewal theorem for systems of equations with distributions on R. 1. Introduction. The discrete-time models Y = (Yn,n ∈ N) governed by a switching process X = (Xn,n ∈ N) fit well to the situations where an autonomous process X is responsible for the dynamic (or regime) of Y . These models are parsimonious with regard to the number of parameters, and extend significantly the case of a single regime. Among them, the so- called Markov-switching ARMA models are popular in several application fields, for example, in econometric modeling [see Hamilton (1989, 1990)]. More recently, continuous-time versions of Markov-switching models have been proposed in Basak, Bisi and Ghosh (1996) and Guyon, Iovleff and Yao (2004), where ergodicity conditions are established. In this paper we investi- gate the tail property of the stationary distribution of this continuous-time process. One of the main results (Theorem 2) states that this model can provide heavy tails, which is one of the major features required in nonlinear time-series modeling. -
Consensus-Based Clinical Practice Recommendations for the Examination and Management of Falls in Patients with Parkinson’S Diseaseq
Parkinsonism and Related Disorders 20 (2014) 360e369 Contents lists available at ScienceDirect Parkinsonism and Related Disorders journal homepage: www.elsevier.com/locate/parkreldis Editor’s comment: In this thoughtful and provocative Point-of-View contribution, van der Eijk and colleagues address the shortcomings of the classical model of “professional physician-centered care” and describe an alternative “collaborative patient-centered care” approach that involves, among many other things, shared decision making with patients in the context of a multidisciplinary care setting. They propose that this alternative approach may improve quality of care and produce better outcomes for individuals with disorders such as Parkinson’s disease, while also being cost-effective. The authors discuss their experience with such an approach and describe both the benefits and barriers they have encountered. Whether one agrees or disagrees with the authors’ proposals, this article will provide much food for thought and reflection. Ronald F. Pfeiffer, Editor-in-Chief Department of Neurology, University of Tennessee HSC, 855 Monroe Avenue, Memphis, TN 38163, USA Point of view Consensus-based clinical practice recommendations for the examination and management of falls in patients with Parkinson’s diseaseq Marjolein A. van der Marck a, Margit Ph.C. Klok a, Michael S. Okun b, Nir Giladi c, Marten Munneke a,d, Bastiaan R. Bloem e,*, on behalf of the NPF Falls Task Force1 a Radboud university medical center, Nijmegen Centre for Evidence Based Practice, Department -
SOS – Save Our Shoulders: a Guide for Polio Survivors
1 • Save Our Shoulders: A Guide for Polio Survivors A Guide for Polio Survivors S.O.S. Save Our Shoulders: A Guide for Polio Survivors by Jennifer Kuehl, MPT Roberta Costello, MSN, RN Janet Wechsler, PT Funding for the production of this manual was made possible by: The National Institute for Disability and Rehabilitation Research Grant #H133A000101 and The U.S. Department of the Army Grant #DAMD17-00-1-0533 Investigators: Mary Klein, PhD Mary Ann Keenan, MD Alberto Esquenazi, MD Acknowledgements We gratefully acknowledge the contributions and input provided from all of those who participated in our research. The time and effort of our participants was instrumental in the creation of this manual. Jennifer Kuehl, MPT Moss Rehabilitation Research Institute, Philadelphia Roberta Costello, MSN, RN Moss Rehabilitation Research Institute, Philadelphia Janet Wechsler, PT Moss Rehabilitation Research Institute, Philadelphia Mary Klein, PhD Moss Rehabilitation Research Institute, Philadelphia Mary Ann Keenan, MD University of Pennsylvania, Philadelphia Alberto Esquenazi, MD MossRehab Hospital, Philadelphia Cover and manual design by Ron Kalstein, MEd Albert Einstein Medical Center, Philadelphia Table of Contents 1. Introduction . .5 2. General Information About the Shoulder . .6 3. Facts About Shoulder Problems . .8 4. Treatment Options . .13 5. About Exercise . .16 6. Stretching Exercises . .19 7. Cane Stretches . .22 8. Strengthening Exercises . .25 9. Tips to Avoid Shoulder Problems . .29 10. Conclusion . .31 11. Resources . .31 The information contained within this manual is for reference only and is not a substitute for professional medical advice. Before beginning any exercise program consult your physician. Save Our Shoulders: A Guide for Polio Survivors • 4 Introduction Many polio survivors report new symptoms as they age. -
Fatigue After Stroke
SPECIAL REPORT Fatigue after stroke PJ Tyrrell Fatigue is a common symptom after stroke. It is not invariably related to stroke severity & DG Smithard† and can occur in the absence of depression. It is one of the most troublesome symptoms for †Author for correspondence many patients and yet nothing is known of its causation. There are no specific treatments. William Harvey Hospital, Richard Steven’s Ward, This article assesses the available literature in the context of what is known about fatigue Ashford, Kent, in other disorders. Post-stroke fatigue may be a manifestation of sickness behavior, TN24 0LZ, UK mediated through the central effects of the cytokine interleukin-1, perhaps via effects on Tel.: +44 123 361 6214 Fax: +44 123 361 6662 glutamate neurotransmission. Possible therapeutic strategies are discussed which might be [email protected] a logical basis from which to plan randomized control trials. Following stroke, approximately a third of common and disabling symptom of Parkinson’s patients die, a third recover and a third remain disease [7,8] and of systemic lupus [9]. More than significantly disabled. Even those who recover 90% of patients with poliomyelitis develop a physically may be left with significant emotional delayed syndrome of post-myelitis fatigue [10]. and psychologic dysfunction – including anxi- Fatigue is the most prevalent symptom of ety, readjustment reactions and depression. One patients with cancer who receive radiation, cyto- common but often overlooked symptom is toxic or other therapies [11], and it may persist fatigue. This may occur soon or late after stroke, for years after the cessation of treatment [12]. -
Study Guide Medical Terminology by Thea Liza Batan About the Author
Study Guide Medical Terminology By Thea Liza Batan About the Author Thea Liza Batan earned a Master of Science in Nursing Administration in 2007 from Xavier University in Cincinnati, Ohio. She has worked as a staff nurse, nurse instructor, and level department head. She currently works as a simulation coordinator and a free- lance writer specializing in nursing and healthcare. All terms mentioned in this text that are known to be trademarks or service marks have been appropriately capitalized. Use of a term in this text shouldn’t be regarded as affecting the validity of any trademark or service mark. Copyright © 2017 by Penn Foster, Inc. All rights reserved. No part of the material protected by this copyright may be reproduced or utilized in any form or by any means, electronic or mechanical, including photocopying, recording, or by any information storage and retrieval system, without permission in writing from the copyright owner. Requests for permission to make copies of any part of the work should be mailed to Copyright Permissions, Penn Foster, 925 Oak Street, Scranton, Pennsylvania 18515. Printed in the United States of America CONTENTS INSTRUCTIONS 1 READING ASSIGNMENTS 3 LESSON 1: THE FUNDAMENTALS OF MEDICAL TERMINOLOGY 5 LESSON 2: DIAGNOSIS, INTERVENTION, AND HUMAN BODY TERMS 28 LESSON 3: MUSCULOSKELETAL, CIRCULATORY, AND RESPIRATORY SYSTEM TERMS 44 LESSON 4: DIGESTIVE, URINARY, AND REPRODUCTIVE SYSTEM TERMS 69 LESSON 5: INTEGUMENTARY, NERVOUS, AND ENDOCRINE S YSTEM TERMS 96 SELF-CHECK ANSWERS 134 © PENN FOSTER, INC. 2017 MEDICAL TERMINOLOGY PAGE III Contents INSTRUCTIONS INTRODUCTION Welcome to your course on medical terminology. You’re taking this course because you’re most likely interested in pursuing a health and science career, which entails proficiencyincommunicatingwithhealthcareprofessionalssuchasphysicians,nurses, or dentists. -
The Brain That Changes Itself
The Brain That Changes Itself Stories of Personal Triumph from the Frontiers of Brain Science NORMAN DOIDGE, M.D. For Eugene L. Goldberg, M.D., because you said you might like to read it Contents 1 A Woman Perpetually Falling . Rescued by the Man Who Discovered the Plasticity of Our Senses 2 Building Herself a Better Brain A Woman Labeled "Retarded" Discovers How to Heal Herself 3 Redesigning the Brain A Scientist Changes Brains to Sharpen Perception and Memory, Increase Speed of Thought, and Heal Learning Problems 4 Acquiring Tastes and Loves What Neuroplasticity Teaches Us About Sexual Attraction and Love 5 Midnight Resurrections Stroke Victims Learn to Move and Speak Again 6 Brain Lock Unlocked Using Plasticity to Stop Worries, OPsessions, Compulsions, and Bad Habits 7 Pain The Dark Side of Plasticity 8 Imagination How Thinking Makes It So 9 Turning Our Ghosts into Ancestors Psychoanalysis as a Neuroplastic Therapy 10 Rejuvenation The Discovery of the Neuronal Stem Cell and Lessons for Preserving Our Brains 11 More than the Sum of Her Parts A Woman Shows Us How Radically Plastic the Brain Can Be Appendix 1 The Culturally Modified Brain Appendix 2 Plasticity and the Idea of Progress Note to the Reader All the names of people who have undergone neuroplastic transformations are real, except in the few places indicated, and in the cases of children and their families. The Notes and References section at the end of the book includes comments on both the chapters and the appendices. Preface This book is about the revolutionary discovery that the human brain can change itself, as told through the stories of the scientists, doctors, and patients who have together brought about these astonishing transformations. -
Cognitive Behaviour Therapy (CBT) and Stroke Rehabilitation
Cognitive Behaviour Therapy (CBT) and Stroke Rehabilitation Amy Quilty OT Reg. (Ont.), Occupational Therapist Cognitive Behavioural Therapy (CBT) Certificate Program, University of Toronto Quinte Health Care: [email protected] Learning Objectives • To understand that CBT: • has common ground with neuroscience • principles are consistent with stroke best practices • treats barriers to stroke recovery • is an opportunity to optimize stroke recovery Question? Why do humans dominate Earth? The power of THOUGHT • Adaptive • Functional behaviours • Health and well-being • Maladaptive • Dysfunctional behaviours • Emotional difficulties Emotional difficulties post-stroke • “PSD is a common sequelae of stroke. The occurrence of PSD has been reported as high as 30–60% of patients who have experienced a stroke within the first year after onset” Canadian Stroke Best Practice Recommendations: Mood, Cognition and Fatigue Following Stroke practice guidelines, update 2015 http://onlinelibrary.wiley.com/doi/10.1111/ijs.12557/full • Australian rates: (Kneeborne, 2015) • Depression ~31% • Anxiety ~18% - 25% • Post Traumatic Stress ~10% - 30% • Emotional difficulties post-stroke have a negative impact on rehabilitation outcomes. Emotional difficulties post-stroke: PSD • Post stroke depression (PSD) is associated with: • Increased utilization of hospital services • Reduced participation in rehabilitation • Maladaptive thoughts • Increased physical impairment • Increased mortality Negative thoughts & depression • Negative thought associated with depression has been linked to greater mortality at 12-24 months post-stroke Nursing Best Practice Guideline from RNAO Stroke Assessment Across the Continuum of Care June : http://rnao.ca/sites/rnao- ca/files/Stroke_with_merged_supplement_sticker_2012.pdf Cognitive Behavioral Therapy (CBT) https://www.youtube.com/watch?v=0ViaCs0k2jM Cognitive Behavioral Therapy - CBT A Framework to Support CBT for Emotional Disorder After Stroke* *Figure 2, Framework for CBT after stroke.