Salivary Gland Pathology 25.Pdf

Total Page:16

File Type:pdf, Size:1020Kb

Salivary Gland Pathology 25.Pdf k Index 461 Mechanoreceptors, 15 patient history, 284 Melanoma pleomorphic adenoma, 286–290 desmoplastic subtype, 63 polymorphous low-grade adenocarcinoma, 174–175, 306, histopathology, 381 309, 312 lower lip, 380–381 primary lymphomas, 368 metastases, 62–63, 189 radiation therapy, 328 nodular, 379 sites of, 285 Merkel cell tumors, metastases, 62, 63, 375 staging, 193–196 Mesenchymal-epithelial transition (MET), 214 Mixed tumor. See Pleomorphic adenoma(s) Mesenchymal neoplasms, 188 Modified Blair incision, 238, 239 Mesenchymal salivary gland tumors Monomorphic adenoma, 167–169, 290 lymphatic malformations, 398, 400 Monomorphic clear cell tumor, 182 neural tumors, 398, 401, 402 Motion artifacts, 21–22 vascular tumors, 397–398, 397–399 Mouth Messenger ribonucleic acid (mRNA), 208, 209, 209 dry, 141 Metal deposits, brain, 24 ranula, 98, 99, 100 Metallic implants, 21, 22 MRI. See Magnetic resonance imaging (MRI) Metalloproteinases, 142 MRS. See Magnetic resonance spectroscopy (MRS) Metastases, 189 Mucocele, 97, 99, 114, 412 diagnostic imaging, 62–64, 63 Mucoepidermoid carcinoma (MEC), 170–172, 214, 389, distant. See Distant metastases 390, 396, 404 regional. See Regional metastases ADC values, 27 skip, 270 biomarkers, 190, 263 Metastasizing mixed tumor, 182, 289 buccal mucosa, 294 Metastasizing pleomorphic adenoma, 287 children, 297 Methicillin resistant S. aureus (MRSA), acute bacterial clear cell variant, 383 parotitis, 75, 78, 96 diagnostic imaging, 56, 57 k Microliths, 439 fixed to mandible, 277 k Middle ear, aberrant glands, 438 grading, 171, 255–256, 293, 294 Mikulicz disease, 144–146, 145, 146, 449 histopathology, 272, 291–292, 294, 304 Minor salivary glands, 13 lip, 170, 294, 304 anatomy, for diagnostic imaging, 44–45 minor salivary gland, 56 incisional biopsy, 146 minor salivary gland tumors, 290–297 mucous escape reaction, 98 molecular events, 352 necrotizing sialometaplasia, 448, 448 neck dissection, 297 saliva production, 1 palate, 291–292, 293 sarcoidosis, biopsy, 153, 158 parotid gland, 56, 57, 171, 297 sialolithiasis, 118, 134, 137, 137 with perineural invasion, 304 Sjogren syndrome, 145, 146, 147 radiation therapy, 304, 335, 338 Minor salivary gland tumors, 403–405, 404, 406 sublingual gland, 276 acinic cell adenocarcinoma, 319 submandibular gland, 263 adenoid cystic carcinoma, 56, 57, 284, 301 survival rates, 252, 255, 255 anatomic barriers for surgery, 285–286, 286 treatment, 294 benign vs. malignant, 282, 285 Mucopapillary cysts, 106 canalicular adenoma, 290, 294, 295 Mucosa-associated lymphoid tissue (MALT) lymphoma. carcinoma ex-pleomorphic adenoma, 287 See MALT lymphoma cervical metastases, 304, 323 Mucosal melanoma, parotid metastases, 380–381 chemotherapy, 328 Mucous escape phenomenon, diagnostic imaging, 50, 51 clear cell carcinoma, 176 Mucous escape reaction differential diagnosis, 284 clinical features, 98–105 epithelial-myoepithelial carcinoma, 182, 320 cyst of Blandin and Nuhn’s gland, 105, 108 etiology, 282–284, 285 definition, 97 imaging, 284 treatment, 102–104 incidence, 281, 283 Mucous retention cysts, 97, 105–106, 108, 11 4 incisional biopsy, 146, 284 Mucus-producing adenopapillary carcinoma, 177 mucoepidermoid carcinoma, 56, 290–297 Multi-leaf collimator (MLC), 339, 341 neck dissection, 304, 304, 323 Multiple-row detector CT (MDCT), 18 k k 462 Index Multiple sclerosis, MRI DWI, 26 submandibular gland tumors, 404 Multiplicative effect measures, 164 Nerve expander, 416 Mumps, 48, 74 Nerve grafts, 416 complications, 94 Nerves, donor, 416 viral, 90 Neural network analysis, 28 Mural nodules, 50 Neural tumors, 363, 365, 366, 398, 401, 402 Musicians, barotrauma, 432 Neurilemmomas, 365 Mycobacterial disease Neurofibroma, 59, 365 adenitis, 121, 122 Neurofibromatosis, 365 nontuberculous, 90, 93 Neurogenic tumors, diagnostic imaging, 59, 60, 61, 61 tuberculous, 49, 53, 90 Neutron radiation, 345 Mycobacterium avium-intracellulare, 90 Nicotine, effect on BAT uptake, 34 Mycobacterium Kansasii, 90 Nodal irradiation, 337 Mycobacterium scrofulaceum, 90 Non-Hodgkin lymphoma, 372, 372, 373 Mycobacterium tuberculosis, 90 parotid gland, 187, 373 Mylohyoid boutonniere, 44 sublingual gland, 372 Mylohyoid line, 11 Non-neoplastic salivary gland lesions Mylohyoid muscle, discontinuities, 101–102 bacterial sialadenitis, 392, 393, 394, 394–395 Myocarditis, mumps, 94 inflammatory and infectious diseases, 390, 390 Myoepithelial carcinoma, 185–186 mucous escape reaction, 390–391, 391, 392 Myoepithelial cells, 14–15, 145 Nutritional disturbances, and sialadenosis, 156 Myoepithelial islands, benign lymphoepithelial lesion, 187 Myoepithelioma, 169 Obturator Myxoid liposarcoma, 366 definitive, 315 –317, 324–327 immediate, 312, 315 –317, 315–318, 324–327 Nagasaki, 233, 282 Omega face lift incision, 244 Nasal cartilage, sarcoidosis, 153, 153 parotidectomy, 242–244 Nasal cavity tumors, 196–197, 281 k shotgun wound, 411 k Nasopharynx Oncocytes, 56 metastases from, 381 Oncocytoma, 56, 168, 179, 448 tumors of, 281 Oncocytosis, 448 National Cancer Institute (NCI), 165 Oral cancer, 281 Neck salivary biomarkers, 441 aberrant glands, 438 tumor staging, 193–196 CT scan, 18, 19 Oral motor therapy, 445 soft tissue window, 20 Oral mucosa, 44 Neck carcinomas, 203 Orofacial regulation therapy, 445 Neck dissection, 270, 272 adenoid cystic carcinomas, 344 Oropharynx, metastases from, 381 adjuvant radiation, 337 Orthodontic brackets, 72 124 elective nodal irradiation, 337 Osteoma, mandible, 121, metastatic squamous cell carcinoma, 377 Otic ganglion, 8 minor salivary gland tumors, 304, 304, 323 mucoepidermoid carcinoma, 297 Palatal glands, 13 occult nodal disease, 335 Palatal stent, 287, 298 and radiation/chemo-radiation, 218 Palate. See also Hard palate; Soft palate radical, nodular melanoma, 379 mucoceles, 99 supraomohyoid, 275, 276 necrotizing sialometaplasia, 448, 448 type 1 modified radical, 270, 275, 275, 277–278 tumors, 281 Necrotizing sialometaplasia, minor salivary glands, 448, 448 adenoid cystic carcinoma, 301 Neoplasia, 203 bony involvement, 285 Neoplastic salivary gland disease carcinoma ex-pleomorphic adenoma, 287 epithelial tumors, 396 cupping out, 285, 287, 290 mesenchymal tumors, 396–402, 397–402 epithelial-myoepithelial carcinoma, 320 minor salivary gland tumors, 403–405, 404, 406 incisional biopsy, 285 parotid tumors, 403, 404 lesion evaluation, 284 pediatric vs. adult populations, 395, 395 mucoepidermoid carcinoma, 291–292, 293 k k Index 463 pleomorphic adenoma, 286–287, 288–289 adjacent structures, 39, 40 polymorphous low-grade adenocarcinoma, 174–175, density, 21, 40 309 normal scan, 37 Panda sign, 53 deep (medial) surface, 4 Panoramic radiographs, sialolithiasis, 71 embryology, 2 Papillary adenocarcinoma, 177 external carotid artery, 6 Papillary cystadenoma lymphomatosum. See Warthin tumor facial nerve, 3, 4–5, 40 Papillotomy, 126 fascia, 4 Parainfluenza virus, 94 fistulas, 409, 410, 412, 432 Paramyxovirus gadolinium contrast, 40 acute sialadenitis, 48 HIV infection and, 107 mumps, 94 hypoplasia, 437–438 Paranasal sinuses, tumor staging, 196 imaging, 39–42 Parapharyngeal space (PPS), 39 incisional biopsy, 146, 149, 149–150, 150 giant ranula, 51 infectious process, 72 growth of soft palate tumors into, 286, 288 lymph nodes, 6 pleomorphic adenoma, 241, 246 metastases, 62–63 Parasympathetic innervation squamous cell carcinoma, 375, 377, 378 salivary glands, 8, 9 lymphoepithelial cysts, 50 submandibular gland, 12 metastases to, 189 Parasympathetic secretomotor nerve supply, parotid, 8 milking, 73, 75 Parkinson’s disease, reduced saliva production, 443 MRI Parotid cysts, 234 STIR image, 26, 40 HIV-associated, 106–108, 109 –112, 110, 112, 11 3 –11 4 T1 image, 40 Parotid duct, 3, 3, 4, 7–8, 20, 40 mucous retention cysts, 105–106, 108, 11 4 lacrimal probe, 130 mumps, 92 ligation, for drooling, 447 mycobacterial disease, 90 k obstruction, 424 nerve supply to, 8 k repair, 424, 425 pediatric, 40 sialoliths, 131 penetrating injuries, algorithm, 431 strictures, 130 PET, 37 surface markings, 8 radioiodine damage, 431 Parotidectomy, 110, 130 retromandibular vein, 6, 7 for chronic bacterial parotitis, 84–85 saliva composition, 11 8 face lift approach, 236, 238, 239–241 saliva production, 2 first branchial cleft anomalies, 441, 442 sarcoidosis, 53, 153, 154 for Frey syndrome, 418, 419 sialadenitis, 69, 72, 96 hollowing, 420 sialadenosis, 156, 159 mycobacterial disease, 93 biopsy, 158, 159 nodular melanoma, 379 sialoceles, 412, 412, 414, 415, 432 pleomorphic adenoma, 237 gunshot wound, 414, 415 primary desmoid melanoma, 371 sialolithiasis, 118–119, 128, 130–132, 130–132 Parotid emphysema, 432 sialolithotomy, 131, 133–134 Parotid gland, 3, 335, 340, 342 sialoliths, CT scan, 51, 52, 133 acini, 13–15 Sjogren syndrome, 143, 144, 147 acute bacterial parotitis. See Acute bacterial parotitis standardized uptake value (SUV), 36 (ABP) superficial surface, 4 anatomy, 2, 2–4, 3, 4, 39–42 traumatic fistulas, 410 associated structures, 3, 4 ultrasound, 30, 40 auriculotemporal nerve, 5–6 vascular lesions, 47 blunt trauma, algorithm, 432 Parotid gland tumors bony features related to, 2, 2 acinic cell carcinoma, 173–174 borders of, 3–4 angiosarcoma, 366, 368 branchial cleft cysts, 440, 443 deep lobe, 240, 247, 250 capsule, 2–4 diagnosis chronic inflammatory changes, 49 differential, 234 CT fine needle aspiration biopsy, 234–236, 238 k k 464 Index Parotid gland tumors (continued) lymphoma, 62 history, 233–234 pleomorphic adenoma, 244, 249, 250, 251, 252 imaging, 234 Warthin, 235, 252 physical examination, 234 Parotid tumors, 403, 404 distant metastases, 382, 382, 383
Recommended publications
  • Glossary for Narrative Writing
    Periodontal Assessment and Treatment Planning Gingival description Color: o pink o erythematous o cyanotic o racial pigmentation o metallic pigmentation o uniformity Contour: o recession o clefts o enlarged papillae o cratered papillae o blunted papillae o highly rolled o bulbous o knife-edged o scalloped o stippled Consistency: o firm o edematous o hyperplastic o fibrotic Band of gingiva: o amount o quality o location o treatability Bleeding tendency: o sulcus base, lining o gingival margins Suppuration Sinus tract formation Pocket depths Pseudopockets Frena Pain Other pathology Dental Description Defective restorations: o overhangs o open contacts o poor contours Fractured cusps 1 ww.links2success.biz [email protected] 914-303-6464 Caries Deposits: o Type . plaque . calculus . stain . matera alba o Location . supragingival . subgingival o Severity . mild . moderate . severe Wear facets Percussion sensitivity Tooth vitality Attrition, erosion, abrasion Occlusal plane level Occlusion findings Furcations Mobility Fremitus Radiographic findings Film dates Crown:root ratio Amount of bone loss o horizontal; vertical o localized; generalized Root length and shape Overhangs Bulbous crowns Fenestrations Dehiscences Tooth resorption Retained root tips Impacted teeth Root proximities Tilted teeth Radiolucencies/opacities Etiologic factors Local: o plaque o calculus o overhangs 2 ww.links2success.biz [email protected] 914-303-6464 o orthodontic apparatus o open margins o open contacts o improper
    [Show full text]
  • Diseases of Salivary Glands: Review
    ISSN: 1812–1217 Diseases of Salivary Glands: Review Alhan D Al-Moula Department of Dental Basic Science BDS, MSc (Assist Lect) College of Dentistry, University of Mosul اخلﻻضة امخجوًف امفموي تُئة رطبة، حتخوي ػىل طبلة ركِلة من امسائل ثدغى انوؼاب ثغطي امسطوح ادلاخوَة و متﻷ امفراغات تني ااطَة امفموًة و اﻷس نان. انوؼاب سائل مؼلد، ًنذج من امغدد انوؼاتَة، اذلي ًوؼة دورا" ىاما" يف اﶈافظة ػىل سﻻمة امفم. املرىض اذلٍن ؼًاهون من هلص يف اﻷفراز انوؼايب حكون دلهيم مشبلك يف اﻷلك، امخحدث، و امبوع و ًطبحون غرضة مﻷههتاابت يف اﻷغش َة ااطَة و امنخر املندرش يف اﻷس نان. ًوخد ثﻻثة أزواج من امغدد انوؼاتَة ام ئرُسة – امغدة امنكفِة، امغدة حتت امفكِة، و حتت انوساهَة، موضؼيا ٍكون خارج امخجوًف امفموي، يف حمفظة و ميخد هظاهما املنَوي مَفرغ افرازاهتا. وًوخد أًضا" امؼدًد من امغدد انوؼاتَة امطغرية ، انوساهَة، اتحنكِة، ادلىوزيًة، انوساهَة احلنكِة وما كبل امرخوًة، ٍكون موضؼيا مﻷسفل و مضن امغشاء ااطي، غري حماطة مبحفظة مع هجاز كنَوي كطري. افرازات امغدد انوؼاتَة ام ئرُسة مُست مدشاهبة. امغدة امفكِة ثفرز مؼاب مطيل غين ابﻷمِﻻز، وامغدة حتت امفكِة ثنذج مؼاب غين اباط، أما امغدة حتت انوساهَة ثنذج مؼااب" مزخا". ثبؼا" ميذه اﻷخذﻻفات، انوؼاب املوحود يق امفم ٌشار امَو مكزجي. ح كرَة املزجي انوؼايب مُس ثس َطا" واملادة اﻷضافِة اموػة من لك املفرزات انوؼاتَة، اكمؼدًد من امربوثُنات ثنذلل ثرسػة وثوخطق هبدروكس َل اﻷتُذاًت مﻷس نان و سطوح ااطَة امفموًة. ثبدأ أمراض امغدد انوؼاتَة ػادة تخغريات اندرة يف املفرزات و ام كرتَة، وىذه امخغريات ثؤثر اثهواي" من خﻻل جشلك انووحية اجلرثومِة و املوح، اميت تدورىا ثؤدي اىل خنور مذفش َة وأمراض وس َج دامعة. ىذه اﻷمراض ميكن أن ثطبح شدًدة تؼد املؼاجلة امشؼاغَة ﻷن امؼدًد من احلاﻻت اجليازًة )مثل امسكري، امخوَف اهكُيس( ثؤثر يف اجلراين انوؼايب، و ٌش خيك املرض من حفاف يف امفم.
    [Show full text]
  • Practice Parameter for the Diagnosis and Management of Primary Immunodeficiency
    Practice parameter Practice parameter for the diagnosis and management of primary immunodeficiency Francisco A. Bonilla, MD, PhD, David A. Khan, MD, Zuhair K. Ballas, MD, Javier Chinen, MD, PhD, Michael M. Frank, MD, Joyce T. Hsu, MD, Michael Keller, MD, Lisa J. Kobrynski, MD, Hirsh D. Komarow, MD, Bruce Mazer, MD, Robert P. Nelson, Jr, MD, Jordan S. Orange, MD, PhD, John M. Routes, MD, William T. Shearer, MD, PhD, Ricardo U. Sorensen, MD, James W. Verbsky, MD, PhD, David I. Bernstein, MD, Joann Blessing-Moore, MD, David Lang, MD, Richard A. Nicklas, MD, John Oppenheimer, MD, Jay M. Portnoy, MD, Christopher R. Randolph, MD, Diane Schuller, MD, Sheldon L. Spector, MD, Stephen Tilles, MD, Dana Wallace, MD Chief Editor: Francisco A. Bonilla, MD, PhD Co-Editor: David A. Khan, MD Members of the Joint Task Force on Practice Parameters: David I. Bernstein, MD, Joann Blessing-Moore, MD, David Khan, MD, David Lang, MD, Richard A. Nicklas, MD, John Oppenheimer, MD, Jay M. Portnoy, MD, Christopher R. Randolph, MD, Diane Schuller, MD, Sheldon L. Spector, MD, Stephen Tilles, MD, Dana Wallace, MD Primary Immunodeficiency Workgroup: Chairman: Francisco A. Bonilla, MD, PhD Members: Zuhair K. Ballas, MD, Javier Chinen, MD, PhD, Michael M. Frank, MD, Joyce T. Hsu, MD, Michael Keller, MD, Lisa J. Kobrynski, MD, Hirsh D. Komarow, MD, Bruce Mazer, MD, Robert P. Nelson, Jr, MD, Jordan S. Orange, MD, PhD, John M. Routes, MD, William T. Shearer, MD, PhD, Ricardo U. Sorensen, MD, James W. Verbsky, MD, PhD GlaxoSmithKline, Merck, and Aerocrine; has received payment for lectures from Genentech/ These parameters were developed by the Joint Task Force on Practice Parameters, representing Novartis, GlaxoSmithKline, and Merck; and has received research support from Genentech/ the American Academy of Allergy, Asthma & Immunology; the American College of Novartis and Merck.
    [Show full text]
  • Classification of Salivary Gland Disorders
    Salivary Gland Diseases and Disorders Dr. Mahmoud E. Khalifa Prof of OMFS Lecture ILOs At the end of this chapter you should be able to: 1. Distinguish the clinical features of infections of the salivary glands from those in other structures 2. Differentiate on clinical grounds between infection, obstruction, benign and malignant neoplasms of the salivary glands 3. Plan and evaluate the results of the investigation of disorders of the salivary glands 4. List the important/relevant information to be elicited from patients with salivary gland disorders 5. Select cases which require referral for a specialist opinion 6. Describe the causes of a dry mouth and be able to distinguish between organic and functional causes. Anatomy Major glands Minor glands 3 pairs Situated mostly 800 to 1000 in the oral cavity Parotid Submandibular The majority atAlso found in the the junction of pharynx, larynx, the hard and soft trachea, and palates sinuses sublingual Functions These glands function to produce saliva, which serves as Lubricant for speech & swallowing Assists taste Immunologic (antibacterial) Digestive Cleansing properties Based on the type of secretion, the salivary glands may be grouped as: (i) Serous, (ii) Mucous and (iii) Mixed. Parotid gland secretion is serous in nature. The sublingual gland secretes mixed, but predominantly mucous. The submandibular gland secretion is also mixed, but is predominantly serous. The minor glands secrete mucous saliva. Parotid Gland The parotid gland is the largest salivary gland, the secretion of which is serous in nature. It is pyramidal in shape; The base located superficial and apex medially The base is triangular in shape its apex is towards the angle of the mandible, the base at the external acoustic meatus The parotid duct (Stenson‘s duct) Emerges at the anterior part of the gland.
    [Show full text]
  • Chapter 11. Non-Neoplastic Diseases of Salivary Glands
    Surgical pathology of the mouth and jaws R. A. Cawson, J. D. Langdon, J. W. Eveson 11. Non-neoplastic diseases of salivary glands Investigation Investigations will be discussed in relation to specific disorders and those more appropriate to neoplasms are discussed in the following chapter. However, it must be emphasized that chronic inflammatory swellings of the major salivary glands, in particular, sometimes cannot be distinguished from neoplasms clinically. Nevertheless, biopsy of the parotid glands is contraindicated because of the frequency of pleomorphic adenomas which can be seeded into the surrounding tissues to produce multiple recurrences. There are also the risks of damaing branches of the facial nerve or of producing a parotid fistula. For imaging techniques, see Chapters 1 and 12. Developmental disorders Aplasia/agenesis. Complete absence of one or more salivary glands is very rare, but occasionally the parotid glands are absent. Absence of all major salivary glands is even more rare. Duct atresia. This is also rare, but usually affects the submandibular duct in the floor of the mouth. Absence of the duct results in retention cysts of the submandibular and sublingual glands. Salivary gland hypoplasia. This can be a feature of the Melkersson-Rosenthal syndrome. The hypoplasia possibly may be secondary to atrophy of parasympathetic nerves thought to be implicated in this syndrome. Congenital salivary fistulae. These are sometimes seen in association with branchial clefts. Aberrant salivary tissue. This is common in the cervical lymph nodes (where it should not be mistaken for a metastasis) and may be found in the middle ear cleft. Stafne's bone cavity is another example.
    [Show full text]
  • Treatments for Ankyloglossia and Ankyloglossia with Concomitant Lip-Tie Comparative Effectiveness Review Number 149
    Comparative Effectiveness Review Number 149 Treatments for Ankyloglossia and Ankyloglossia With Concomitant Lip-Tie Comparative Effectiveness Review Number 149 Treatments for Ankyloglossia and Ankyloglossia With Concomitant Lip-Tie Prepared for: Agency for Healthcare Research and Quality U.S. Department of Health and Human Services 540 Gaither Road Rockville, MD 20850 www.ahrq.gov Contract No. 290-2012-00009-I Prepared by: Vanderbilt Evidence-based Practice Center Nashville, TN Investigators: David O. Francis, M.D., M.S. Sivakumar Chinnadurai, M.D., M.P.H. Anna Morad, M.D. Richard A. Epstein, Ph.D., M.P.H. Sahar Kohanim, M.D. Shanthi Krishnaswami, M.B.B.S., M.P.H. Nila A. Sathe, M.A., M.L.I.S. Melissa L. McPheeters, Ph.D., M.P.H. AHRQ Publication No. 15-EHC011-EF May 2015 This report is based on research conducted by the Vanderbilt Evidence-based Practice Center (EPC) under contract to the Agency for Healthcare Research and Quality (AHRQ), Rockville, MD (Contract No. 290-2012-00009-I). The findings and conclusions in this document are those of the authors, who are responsible for its contents; the findings and conclusions do not necessarily represent the views of AHRQ. Therefore, no statement in this report should be construed as an official position of AHRQ or of the U.S. Department of Health and Human Services. The information in this report is intended to help health care decisionmakers—patients and clinicians, health system leaders, and policymakers, among others—make well-informed decisions and thereby improve the quality of health care services. This report is not intended to be a substitute for the application of clinical judgment.
    [Show full text]
  • Plastic Surgery Essentials for Students Handbook to All Third Year Medical Students Concerned with the Effect of the Outcome on the Entire Patient
    AMERICAN SOCIETY OF PLASTIC SURGEONS YOUNG PLASTIC SURGEONS STEERING COMMITTEE Lynn Jeffers, MD, Chair C. Bob Basu, MD, Vice Chair Eighth Edition 2012 Essentials for Students Workgroup Lynn Jeffers, MD Adam Ravin, MD Sami Khan, MD Chad Tattini, MD Patrick Garvey, MD Hatem Abou-Sayed, MD Raman Mahabir, MD Alexander Spiess, MD Howard Wang, MD Robert Whitfield, MD Andrew Chen, MD Anureet Bajaj, MD Chris Zochowski, MD UNDERGRADUATE EDUCATION COMMITTEE OF THE PLASTIC SURGERY EDUCATIONAL FOUNDATION First Edition 1979 Ruedi P. Gingrass, MD, Chairman Martin C. Robson, MD Lewis W.Thompson, MD John E.Woods, MD Elvin G. Zook, MD Copyright © 2012 by the American Society of Plastic Surgeons 444 East Algonquin Road Arlington Heights, IL 60005 All rights reserved. Printed in the United States of America ISBN 978-0-9859672-0-8 i INTRODUCTION PREFACE This book has been written primarily for medical students, with constant attention to the thought, A CAREER IN PLASTIC SURGERY “Is this something a student should know when he or she finishes medical school?” It is not designed to be a comprehensive text, but rather an outline that can be read in the limited time Originally derived from the Greek “plastikos” meaning to mold and reshape, plastic surgery is a available in a burgeoning curriculum. It is designed to be read from beginning to end. Plastic specialty which adapts surgical principles and thought processes to the unique needs of each surgery had its beginning thousands of years ago, when clever surgeons in India reconstructed individual patient by remolding, reshaping and manipulating bone, cartilage and all soft tissues.
    [Show full text]
  • Clinical and Molecular Investigation of Rare
    CLINICAL AND MOLECULAR INVESTIGATION OF RARE CONGENITAL DEFECTS OF THE PALATE RIMANTE SESELGYTE A thesis submitted for the degree of Doctor of Philosophy to University College London August 2019 page Title 1 DECLARATION I, Rimante Seselgyte, confirm that the work presented in this thesis is my own. Where information has been derived from other sources, I confirm that this has been indicated in the thesis. Signed……………………….. Declaration 3 ABSTRACT Cleft palate (CP) affects around 1/1500 live births and, along with cleft lip, is one of the most common forms of birth defect. The studies presented here focus on unusual defects of the palate, especially to understand better the rarely reported but surprisingly common condition called submucous cleft palate (SMCP). The frequency and consequences of SMCP from a surgical perspective were first investigated based on the caseload of the North Thames Cleft Service at Great Ormond Street Hospital and St Andrew's Centre, Broomfield Hospital, Mid Essex Hospitals Trust. It was previously reported that up to 80% of individuals with unrepaired SMCP experience speech difficulties as a consequence of velopharyngeal insufficiency (VPI). Attempted repair of the palatal defect can sometimes give poor results, so controversies still exist about the correct choice of surgical technique to use. Over 23 years, 222 patients at The North Thames Cleft Service underwent operations to manage SMCP. Nearly half of them (42.8%) were diagnosed with 22q11.2 deletion syndrome (22q11.2 DS). The first operation was palate repair, with an exception of one case, followed by a second surgical intervention required in approximately half of the patients.
    [Show full text]
  • Dental Manifestations in Bariatric Patients – Review of Literature
    www.scielo.br/jaos Dental manifestations in bariatric patients – review of literature Carolina Silveira BARBOSA1, Gabriel Salles BARBÉRIO1, Vinicius Rizzo MARQUES1, Vitor de Oliveira BALDO1, Marília Afonso Rabelo BUZALAF2, Ana Carolina MAGALHÃES3 1- Undergraduate student, Bauru School of Dentistry, University of São Paulo. 2- DDS, MSc, PhD, Full Professor, Department of Biological Sciences, Bauru School of Dentistry, University of São Paulo. 3- DDS, MSc, PhD, Assistant Professor, Department of Biological Sciences, Bauru School of Dentistry, University of São Paulo. Corresponding address: Ana Carolina Magalhães - Faculdade de Odontologia de Bauru - Universidade de São Paulo - Departamento de Ciências Biológicas - Al. Octávio Pinheiro Brisolla, 9-75 Bauru-SP 17012-901 (Brazil) - Phone: + 55 14 32358246 Fax: + 55 14 32343164 - e-mail: [email protected] Received: August 19, 2009 - Accepted: February 19, 2010 ABSTRACT he rate of bariatric surgery has significantly risen in the past decade as an increasing Tprevalence of extreme obesity can be observed. Although bariatric surgery is an effective therapeutic modality for extreme obesity, it is associated with risk factors affecting also oral health. Based on an overview of the current literature, this paper presents a summary of dental manifestations in bariatric patients. Bariatric surgeries are associated with an increased risk for gastro-esophageal reflux which in turn might account for the higher amount of carious and erosive lesions observed in bariatric patients. As a result, also dentin hypersensitivity might be observed more frequently. The current data indicate that recommended postsurgical meal patterns and gastric reflux might increase the risk for dental lesions, particularly in the presence of other risk factors, such as consumption of sweet-tasting foods and acidic beverages.
    [Show full text]
  • Parotid Duct Ligation - 4 Duct Ligation - Bilateral Excision of the Submandibular Glands +/- Ligation of the Parotid Duct Tympanic Neurectomy
    SialorrheaSialorrhea SalivarySalivary disordersdisorders SialoendoscopySialoendoscopy Sam J Daniel, MD Director Pediatric Otolaryngology Associate Professor Montreal Children’s Hospital McGill University Objectives • Review the rehabilitative, medical, and surgical treatment options for sialorrhea • Discuss novel therapies such as Botox injections in the salivary glands • Discuss the technique and pitfalls of sialoendoscopy • Review selected salivary gland disorders Sialorrhea • Sialorrhea (drooling) is the loss of saliva from the oral cavity • It occurs in healthy children frequently until age 2 Introduction • Neurological disorders in which drooling is problematic: – Parkinson’s disease (close to 80%) – Atypical Parkinsonian syndromes – Amyotrophic lateral sclerosis – Cerebral palsy – Pseudobulbar palsy – Stroke ANATOMYANATOMY && PHYSIOLOGYPHYSIOLOGY ofof thethe salivarysalivary glandsglands Autonomic innervation •Parasympathetic –Abundant, watery saliva –Amylase down •Sympathetic –Scant, viscous saliva –Amylase up Parotid gland Parasympathetic innervation: • Originates in inferior salivary nucleus in medulla. • CN9->sup & inf glossopharyngeal ganglions->tympanic plexus (Jacobson’s nerve) lesser superficial petrosal nerve- >foramen ovale > otic ganglion- synapse with post- ganglionic fibers >auriculotemporal n. • M3 muscarinic receptors of parotid gland Sympathetic innervation: • Postganglionic innervation is provided by the superior cervical ganglion and distributes with the arterial system. Submandibular glands Parasympathetic innervation:
    [Show full text]
  • Toma, 539 Acinic Cell Carcinoma Mucinous Adenocarcinoma Vs., 334
    Cambridge University Press 978-0-521-87999-6 - Head and Neck Margaret Brandwein-Gensler Index More information INDEX acanthomatous/desmoplastic ameloblas- ameloblastomas benign neoplasia toma, 539 desmoplastic ameloblastoma, 539 juvenile nasopharyngeal angiofibroma, acinic cell carcinoma metastasizing ameloblastoma, 537 99–104 mucinous adenocarcinoma vs., 334–335 mural ameloblastoma, 533 salivary gland anlage tumor, 104–106 oncocytoma vs., 295–299 odonto-ameloblastoma, 551–553 benign peripheral nerve sheath tumors papillary cystic variant, vs. cystade- peripheral ameloblastoma, 534 (BPNST), 40–44 noma, 316–317 unicystic ameloblastoma, 532–533 benign sinonasal tract neoplasia, 28–48 salivary glands, 353–359 aneurysmal bone cyst (ABC), 584 benign peripheral nerve sheath tumor, adenocarcinoma not otherwise specified central GCRG vs., 590–591 40–44 (ANOS), 389–390 angiocentric T-cell lymphoma, 81 meningioma, 37–40 adenoid cystic carcinoma (ACC) angiomatoid/angioectatic polyps vs. JNAF, nasal glial heterotopia (NGH), 44–48 adenomatoid odontogenic tumor vs., 100–104 oncocytic Schneiderian papilloma 545 angiosarcoma vs. Kaposi’s sarcoma, 206 (OSP), 5, 33–36 basal cell adenocarcinoma vs., 372 antrochoanal polyp, 5–8 Schneiderian inverted papilloma, 28–32 basal cell adenoma vs., 293 apical periodontal cyst, 510–512 bisphosphonate osteonecrosis (BPP), canalicular adenoma vs., 294 arytenoid chondrosarcomas, 241 565–566 neuroendocrine carcinoma vs., 240–241 atrophic oral lichen planus, 126 blastomas of salivary glands, 319–325 atypical adenoma vs. parathyroid
    [Show full text]
  • Sialoblastoma of the Cheek: a Case Report and Review of the Literature
    MOLECULAR AND CLINICAL ONCOLOGY 4: 925-928, 2016 Sialoblastoma of the cheek: A case report and review of the literature PEERAYUT SITTHICHAIYAKUL1, JULINTORN SOMRAN1, NONGLUK OILMUNGMOOL2, SARAN WORASAKWUTTIPONG3 and NOPPADOL LARBCHAROENSUB4 Departments of 1Pathology, 2Radiology and 3Surgery, Faculty of Medicine, Naresuan University, Phitsanulok 65000; 4Department of Pathology, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok 10400, Thailand Received November 23, 2015; Accepted March 21, 2016 DOI: 10.3892/mco.2016.840 Abstract. Sialoblastoma is a rare salivary gland tumor that basal cell adenoma, basaloid adenocarcinoma, congenital hybrid recapitulates the primitive salivary gland anlage. The authors basal cell adenoma-adenoid cystic carcinoma, and embryoma. herein report a case of sialoblastoma of a minor salivary gland, Sialoblastoma most commonly affects the major salivary glands clinically presenting with progressive enlargement of a mass and is histologically characterized by variably arranged, tight in the cheek of a 1-year-old female infant. Histopathologically, clusters or clumps of basaloid cells and partially formed ductal the mass consisted of tight clusters of basaloid cells and and pseudo‑ductal spaces separated by thin fibrous bands. The partially formed ductal and pseudo-ductal spaces separated overall prognosis of this type of tumor remains controversial. by thin fibrous bands. Immunohistchemical studies demon- Sialoblastoma has a tendency to progress to local invasion, local strated the presence of cytokeratin AE1̸AE3, p63, CD99, recurrence and occasional metastasis. In 1996, according to the α-fetoprotein (AFP) and Hep Par-1 expression in a considerable third series of the Armed Forces Institute of Pathology (AFIP) number of tumor cells. The clinical and pathological charac- classification of salivary gland tumors, sialoblastoma was clas- teristics are presented and relevant literature is reviewed.
    [Show full text]