Superimposed Primary Chancre in a Patient with Adamantiades-Behçet's Disease

Total Page:16

File Type:pdf, Size:1020Kb

Superimposed Primary Chancre in a Patient with Adamantiades-Behçet's Disease 124 Sex Transm Inf 1999;75:124–125 Superimposed primary chancre in a patient with Sex Transm Infect: first published as 10.1136/sti.75.2.124 on 1 April 1999. Downloaded from Case report: Adamantiades-Behçet’s disease cobblestone N Meljanac, E Dippel, Ch C Zouboulis Adamantiades-Behçet’s disease was diagnosed in a 42 year old Turkish patient with recurrent oral aphthae, genital ulcerations, papules, and sterile pustules, histologically presenting as cutaneous vasculitis, and intermittent arthritis with joint eVusion particularly of the knees. Six months after initial improvement under treatment with colchicine 2 mg/day, a solitary genital ulcer with enlarged inguinal lymph nodes appeared and persisted for 7 weeks despite the continuation of colchicine treatment and the introduction of clindamycin 2 mg/day intravenously. The unusual persistence of the ulcer and the failure of clindamycin therapy led to further diVerential diagnos- tic considerations and the identification of primary syphilis. The genital lesion healed 4 weeks after initiation of treatment with tetracycline 2 mg/day by mouth for 15 days. (Sex Transm Inf 1999;75:124–125) Keywords: Behçet’s disease; syphilis Case report B51, B18, Bw4, Bw6, and Cw7. Further labo- A 42 year old Turkish man presented on May ratory values were as follows: cardiolipin 1997 at the department of dermatology at the autoantibodies of the IgG type 11.9 anticardio- Free University of Berlin with several 2–10 mm lipin antibodies IgG units/ml (GPL-U/ml) Department of large buccal aphthae, a 2 mm large ulcer at the (normal values <12 GPL-U/ml), of the IgM Dermatology, oreficium urethrae, a 1 cm wide ulcus on the type 3.2 anticardiolipin antibodies IgM University Medical units/ml (MPL-U/ml) (normal <6 MPL-U/ Center Benjamin penile shaft (fig 1a), and papulopustular skin Franklin, The Free lesions at his shoulders and shanks resembling ml), antinuclear antibody titre 1:80, anti- University of Berlin, folliculitis. There was a history of recurrent oral herpes simplex virus titre 1:40 in the comple- Berlin, Germany aphthae and genital ulcerations since October ment fixation test with positive IgG and N Meljanac 1996 and recurrent erythematous papules and negative IgM antibodies in enzyme immu- E Dippel noassay. Herpes simplex virus polymerase Ch C Zouboulis sterile pustules occurring particularly on the extremities since December 1996. Recurrent chain reaction (PCR) and cytomegalovirus PCR, ELISA, HIV anti- http://sti.bmj.com/ Correspondence to: conjunctivitis and intermittent mono- or poly- Chlamydia trachomatis Priv-Doz Dr Christos C bodies, TPHA, and VDRL tests were negative. arthritis with joint eVusion, especially of the Zouboulis, Department of Hepatitis serology showed IgG antibodies to Dermatology, University knees, completed the clinical picture. Family hepatitis A virus, but no indication of an active Medical Center Benjamin history was negative. Franklin, The Free process. Unspecific inflammatory factors were University of Berlin, The combination of clinical findings led to elevated—namely, erythrocyte sedimentation Hindenburgdamm 30, the initial diagnosis of Adamantiades-Behçet’s D-12200 Berlin, Germany. rate 74 mm Hg in the first hour, C reactive disease. Ophthalmological examination was protein 45 mg/l (normal <10 mg/l), and á2 on September 30, 2021 by guest. Protected copyright. Accepted for publication normal. A pathergy test was negative. HLA globulin in serum electrophoresis varying from 18 January 1999 type I analysis revealed the alleles A2, A24, 10.1 to 11.1% (normal 5–10%). Skin biopsies from the genital ulcer and folliculitis-like lesions on the left shoulder and left shank were suggestive of perivascular inflammatory reaction and nodular vasculitis, findings compatible with Adamantiades- Behçet’s disease. Systemic treatment with colchicine 1 mg twice daily by mouth was initi- ated which led to clearance of the mucocutane- ous lesions within a period of 3 weeks. In September 1997 the patient was again referred to our department witha7weekhistory of a solitary genital ulcer. On examination, a 2 cm large ulcer of the penile shaft was detected, covered with a haemorrhagic crust, sharply demarcated, and surrounded by oedematous tissue. Enlarged, painless inguinal lymph nodes were also present (fig 1b). Oral mucosa and eyes were not aVected. The patient reported an active sexual life with several female partners. A bacterial superinfection of a genital lesion of Figure 1 (a)A1cmlarge ulcer on the penile shaft of the patient at his first admission. (b)A2cmlarge ulcer of the penile shaft with asymptomatic enlarged lymph nodes (arrow) Adamantiades-Behçet’s disease was suspected on the second referral. and systemic treatment with clindamycin Case report: cobblestone 125 3 × 600 mg intravenously was initiated. Colchi- nosed 6 months before the confirmation of pri- cine treatment was continued. With this thera- mary syphilis, while syphilis serology was nega- peutic regimen over 7 days the genital ulcer tive at that time. Sex Transm Infect: first published as 10.1136/sti.75.2.124 on 1 April 1999. Downloaded from showed no tendency to heal. Another his- This case emphasises the need for re- topathological examination revealed an unspe- evaluating a chronic disorder with uncommon cific granulomatous reaction. With respect to features of apparently typical lesions during its the persistence of the lesion tests for syphilis course in order to avoid misdiagnosis. It also were repeated with following findings: TPHA demonstrates the importance of taking a sexual test 1:640, VDRL test 1:128, and FTA-ABS test history of every genital lesion and the need to positive. Because of a history of a penicillin perform TPHA and VDRL tests with every hypersensitivity reaction the patient was treated atypical genital ulcer, irrespectively of earlier with tetracycline 4 × 500 mg/day for 2 weeks. A established diagnosis.4 Since Adamantiades- complete healing of the ulcer was noted 4 weeks Behçet’s disease is a chronic disorder with after initiation of treatment. The suspect female common monosymptomatic onset and partner was identified, found positive for syphi- recurrence,1 there must be awareness of the lis, and treated successfully. possibility of additional diseases with similar symptomatology; dermatovenereological con- Discussion sideration of every genital lesion is especially Our patient presented with clinical and his- important. topathological features suggesting 1 Adamantiades-Behçet’s disease. Since muco- We thank Dr Peter K Kohl, Department of Dermatology and cutaneous lesions do not usually persist over 4 Venereology, Academic Hospital Neukölln, for critical review of the manuscript. weeks, the 7 week old genital ulcer led to further diagnostic steps which detected the 1 Zouboulis ChC. Adamantiades-Behçet’s disease: clinical additional primary syphilis. and experimental findings in 53 patients from the Berlin 2 3 area. Habilitation thesis. Berlin: Medical Faculty of the Gnad and Sarica et al reported two patients Free University of Berlin, 1995. with uveitis and uveitis combined with muco- 2 Gnad HD. Zuweisungsdiagnose “Morbus Behçet”. Klin Monatsb Augenheilkd 1975;175:548–50. cutaneous lesions, respectively, as a result of 3 Sarica R, Azizlerli G, Közer LB, et al. Uveitis als Manifesta- secondary syphilis, who were misdiagnosed as tion der Lues II–fehldiagnostiziert als Morbus Behçet. Derm Monatsschrift 1993;179:59–63. having Adamantiades-Behçet’s disease. In our 4 Joyce JP, Waldman PC, Hood AF. Persistent genital ulcera- case Adamantiades-Behçet’s disease was diag- tion. Chancroid. Arch Dermatol 1989;125:552–6. http://sti.bmj.com/ on September 30, 2021 by guest. Protected copyright..
Recommended publications
  • Reporting of Diseases and Conditions Regulation, Amendment, M.R. 289/2014
    THE PUBLIC HEALTH ACT LOI SUR LA SANTÉ PUBLIQUE (C.C.S.M. c. P210) (c. P210 de la C.P.L.M.) Reporting of Diseases and Conditions Règlement modifiant le Règlement sur la Regulation, amendment déclaration de maladies et d'affections Regulation 289/2014 Règlement 289/2014 Registered December 23, 2014 Date d'enregistrement : le 23 décembre 2014 Manitoba Regulation 37/2009 amended Modification du R.M. 37/2009 1 The Reporting of Diseases and 1 Le présent règlement modifie le Conditions Regulation , Manitoba Règlement sur la déclaration de maladies et Regulation 37/2009, is amended by this d'affections , R.M. 37/2009. regulation. 2 Schedules A and B are replaced with 2 Les annexes A et B sont remplacées Schedules A and B to this regulation. par les annexes A et B du présent règlement. Coming into force Entrée en vigueur 3 This regulation comes into force on 3 Le présent règlement entre en vigueur January 1, 2015, or on the day it is registered le 1 er janvier 2015 ou à la date de son under The Statutes and Regulations Act , enregistrement en vertu de Loi sur les textes whichever is later. législatifs et réglementaires , si cette date est postérieure. December 19, 2014 Minister of Health/La ministre de la Santé, 19 décembre 2014 Sharon Blady 1 SCHEDULE A (Section 1) 1 The following diseases are diseases requiring contact notification in accordance with the disease-specific protocol. Common name Scientific or technical name of disease or its infectious agent Chancroid Haemophilus ducreyi Chlamydia Chlamydia trachomatis (including Lymphogranuloma venereum (LGV) serovars) Gonorrhea Neisseria gonorrhoeae HIV Human immunodeficiency virus Syphilis Treponema pallidum subspecies pallidum Tuberculosis Mycobacterium tuberculosis Mycobacterium africanum Mycobacterium canetti Mycobacterium caprae Mycobacterium microti Mycobacterium pinnipedii Mycobacterium bovis (excluding M.
    [Show full text]
  • Syphilis Staging and Treatment Syphilis Is a Sexually Transmitted Disease (STD) Caused by the Treponema Pallidum Bacterium
    Increasing Early Syphilis Cases in Illinois – Syphilis Staging and Treatment Syphilis is a sexually transmitted disease (STD) caused by the Treponema pallidum bacterium. Syphilis can be separated into four different stages: primary, secondary, early latent, and late latent. Ocular and neurologic involvement may occur during any stage of syphilis. During the incubation period (time from exposure to clinical onset) there are no signs or symptoms of syphilis, and the individual is not infectious. Incubation can last from 10 to 90 days with an average incubation period of 21 days. During this period, the serologic testing for syphilis will be non-reactive but known contacts to early syphilis (that have been exposed within the past 90 days) should be preventatively treated. Syphilis Stages Primary 710 (CDC DX Code) Patient is most infectious Chancre (sore) must be present. It is usually marked by the appearance of a single sore, but multiple sores are common. Chancre appears at the spot where syphilis entered the body and is usually firm, round, small, and painless. The chancre lasts three to six weeks and will heal without treatment. Without medical attention the infection progresses to the secondary stage. Secondary 720 Patient is infectious This stage typically begins with a skin rash and mucous membrane lesions. The rash may manifest as rough, red, or reddish brown spots on the palms of the hands, soles of the feet, and/or torso and extremities. The rash does usually does not cause itching. Rashes associated with secondary syphilis can appear as the chancre is healing or several weeks after the chancre has healed.
    [Show full text]
  • Disseminated Mycobacterium Tuberculosis with Ulceronecrotic Cutaneous Disease Presenting As Cellulitis Kelly L
    Lehigh Valley Health Network LVHN Scholarly Works Department of Medicine Disseminated Mycobacterium Tuberculosis with Ulceronecrotic Cutaneous Disease Presenting as Cellulitis Kelly L. Reed DO Lehigh Valley Health Network, [email protected] Nektarios I. Lountzis MD Lehigh Valley Health Network, [email protected] Follow this and additional works at: http://scholarlyworks.lvhn.org/medicine Part of the Dermatology Commons, and the Medical Sciences Commons Published In/Presented At Reed, K., Lountzis, N. (2015, April 24). Disseminated Mycobacterium Tuberculosis with Ulceronecrotic Cutaneous Disease Presenting as Cellulitis. Poster presented at: Atlantic Dermatological Conference, Philadelphia, PA. This Poster is brought to you for free and open access by LVHN Scholarly Works. It has been accepted for inclusion in LVHN Scholarly Works by an authorized administrator. For more information, please contact [email protected]. Disseminated Mycobacterium Tuberculosis with Ulceronecrotic Cutaneous Disease Presenting as Cellulitis Kelly L. Reed, DO and Nektarios Lountzis, MD Lehigh Valley Health Network, Allentown, Pennsylvania Case Presentation: Discussion: Patient: 83 year-old Hispanic female Cutaneous tuberculosis (CTB) was first described in the literature in 1826 by Laennec and has since been History of Present Illness: The patient presented to the hospital for chest pain and shortness of breath and was treated for an NSTEMI. She was noted reported to manifest in a variety of clinical presentations. The most common cause is infection with the to have redness and swelling involving the right lower extremity she admitted to having for 5 months, which had not responded to multiple courses of antibiotics. She acid-fast bacillus Mycobacterium tuberculosis via either primary exogenous inoculation (direct implantation resided in Puerto Rico but recently moved to the area to be closer to her children.
    [Show full text]
  • Pdf/Bookshelf NBK368467.Pdf
    BMJ 2019;365:l4159 doi: 10.1136/bmj.l4159 (Published 28 June 2019) Page 1 of 11 Practice BMJ: first published as 10.1136/bmj.l4159 on 28 June 2019. Downloaded from PRACTICE CLINICAL UPDATES Syphilis OPEN ACCESS Patrick O'Byrne associate professor, nurse practitioner 1 2, Paul MacPherson infectious disease specialist 3 1School of Nursing, University of Ottawa, Ottawa, Ontario K1H 8M5, Canada; 2Sexual Health Clinic, Ottawa Public Health, Ottawa, Ontario K1N 5P9; 3Division of Infectious Diseases, Ottawa Hospital General Campus, Ottawa, Ontario What you need to know Box 1: Symptoms of syphilis by stage of infection (see fig 1) • Incidence rates of syphilis have increased substantially around the Primary world, mostly affecting men who have sex with men and people infected • Symptoms appear 10-90 days (mean 21 days) after exposure with HIV http://www.bmj.com/ • Main symptom is a <2 cm chancre: • Have a high index of suspicion for syphilis in any sexually active patient – Progresses from a macule to papule to ulcer over 7 days with genital lesions or rashes – Painless, solitary, indurated, clean base (98% specific, 31% sensitive) • Primary syphilis classically presents as a single, painless, indurated genital ulcer (chancre), but this presentation is only 31% sensitive; – On glans, corona, labia, fourchette, or perineum lesions can be painful, multiple, and extra-genital – A third are extragenital in men who have sex with men and in women • Diagnosis is usually based on serology, using a combination of treponemal and non-treponemal tests. Syphilis remains sensitive to • Localised painless adenopathy benzathine penicillin G Secondary on 24 September 2021 by guest.
    [Show full text]
  • Nonbacterial Pus-Forming Diseases of the Skin Robert Jackson,* M.D., F.R.C.P[C], Ottawa, Ont
    Nonbacterial pus-forming diseases of the skin Robert Jackson,* m.d., f.r.c.p[c], Ottawa, Ont. Summary: The formation of pus as a Things are not always what they seem Fungus result of an inflammatory response Phaedrus to a bacterial infection is well known. North American blastomycosis, so- Not so well appreciated, however, The purpose of this article is to clarify called deep mycosis, can present with a is the fact that many other nonbacterial the clinical significance of the forma¬ verrucous proliferating and papilloma- agents such as certain fungi, viruses tion of pus in various skin diseases. tous plaque in which can be seen, par- and parasites may provoke pus Usually the presence of pus in or on formation in the skin. Also heat, the skin indicates a bacterial infection. Table I.Causes of nonbacterial topical applications, systemically However, by no means is this always pus-forming skin diseases administered drugs and some injected true. From a diagnostic and therapeutic Fungus materials can do likewise. Numerous point of view it is important that physi¬ skin diseases of unknown etiology cians be aware of the nonbacterial such as pustular acne vulgaris, causes of pus-forming skin diseases. North American blastomycosis pustular psoriasis and pustular A few definitions are required. Pus dermatitis herpetiformis can have is a yellowish [green]-white, opaque, lymphangitic sporotrichosis bacteriologically sterile pustules. The somewhat viscid matter (S.O.E.D.). Pus- cervicofacial actinomycosis importance of considering nonbacterial forming diseases are those in which Intermediate causes of pus-forming conditions of pus can be seen macroscopicaily.
    [Show full text]
  • 2012 Case Definitions Infectious Disease
    Arizona Department of Health Services Case Definitions for Reportable Communicable Morbidities 2012 TABLE OF CONTENTS Definition of Terms Used in Case Classification .......................................................................................................... 6 Definition of Bi-national Case ............................................................................................................................................. 7 ------------------------------------------------------------------------------------------------------- ............................................... 7 AMEBIASIS ............................................................................................................................................................................. 8 ANTHRAX (β) ......................................................................................................................................................................... 9 ASEPTIC MENINGITIS (viral) ......................................................................................................................................... 11 BASIDIOBOLOMYCOSIS ................................................................................................................................................. 12 BOTULISM, FOODBORNE (β) ....................................................................................................................................... 13 BOTULISM, INFANT (β) ...................................................................................................................................................
    [Show full text]
  • Reportable Disease Surveillance in Virginia, 2013
    Reportable Disease Surveillance in Virginia, 2013 Marissa J. Levine, MD, MPH State Health Commissioner Report Production Team: Division of Surveillance and Investigation, Division of Disease Prevention, Division of Environmental Epidemiology, and Division of Immunization Virginia Department of Health Post Office Box 2448 Richmond, Virginia 23218 www.vdh.virginia.gov ACKNOWLEDGEMENT In addition to the employees of the work units listed below, the Office of Epidemiology would like to acknowledge the contributions of all those engaged in disease surveillance and control activities across the state throughout the year. We appreciate the commitment to public health of all epidemiology staff in local and district health departments and the Regional and Central Offices, as well as the conscientious work of nurses, environmental health specialists, infection preventionists, physicians, laboratory staff, and administrators. These persons report or manage disease surveillance data on an ongoing basis and diligently strive to control morbidity in Virginia. This report would not be possible without the efforts of all those who collect and follow up on morbidity reports. Divisions in the Virginia Department of Health Office of Epidemiology Disease Prevention Telephone: 804-864-7964 Environmental Epidemiology Telephone: 804-864-8182 Immunization Telephone: 804-864-8055 Surveillance and Investigation Telephone: 804-864-8141 TABLE OF CONTENTS INTRODUCTION Introduction ......................................................................................................................................1
    [Show full text]
  • 3- Chlamydia, Syphilis & Gonorrhea (STD)
    3- Chlamydia, syphilis & gonorrhea (STD) Microbiology 435’s Teamwork Reproductive Block Learning Objectives: ● Know the causative agents of syphilis, gonorrhea and Chlamydia infections. ● Realize that these three infections are acquired through sexual intercourse. ● Know the pathogenesis of syphilis, gonorrhea and Chlamydia infection. ● Describe the clinical feature of the primary, secondary tertiary syphilis and complications. ● Recall the different diagnostic methods for the different stages of syphilis. ● Describe the clinical features of gonorrhea that affect only men, only women and those ones which affect both sexes. ● Describe the different laboratory tests for the diagnosis of gonorrhea ● Describe the morphology and the distinct life cycle of the Chlamydia. ● Realize what are the different genera, species and serotypes of the family Chlamydophila. ● Recognize that Chlamydia cause different diseases that affect the eye (causing trachoma) and the respiratory system (mainly cause a typical pneumonia). ● Know the different urogenital clinical syndromes caused by Chlamydia trachomatis that affect men, women and both sex. ● Realize that these urogenital syndromes are difficult to differentiate clinically from the similar ones caused by N.gonorrheae. ● Know the treatment of syphilis, gonorrhea and Chlamydia infections. ● Realize that there are no effective vaccines against all these three diseases. Important Resources: 435 females & males slides and ​ ​ ​ Males notes notes, wikipedia, ​ ​ Females notes Lippincott’s Illustrated Reviews: Microbiology- ​ ​ Extra Third Edition ​ ​ Editing file: Here ​ ​ Credit: Team members ​ Introduction (take-home message) ● Syphilis, Chlamydia and Gonorrhea are main STDs, caused by delicate organisms that cannot survive ​ ​ ​ outside the body. Infection may not be localized. ​ ​ ● Clinical presentation may be similar (urethral or genital discharge, ulcers). ● One or more organisms (Bacteria, virus, parasite) may be transmitted by sexual contact.
    [Show full text]
  • Young Adults Young Adults (20 - 35 Years)
    YOUNG ADULTS YOUNG ADULTS (20 - 35 YEARS) A large number of skin diseases reach their peak of expression during the young adult years, and these individuals also represent a large and growing proportion of the South African population, in common with other developing countries. Young adults are especially distressed by skin conditions that are uncomfortable, disfiguring or contagious. This group has also been severely affected by HIV/AIDS, with a consequent increase in the frequency and severity of the HIV- associated dermatoses. Some of these conditions have been discussed in other sections of this review. ACNE KELOIDALIS Inflammatory papules and pustules located mainly on the nape of the neck and occiput characterise this acne-like condition. It primarily affects black males. The lesions heal with scarring, creating huge keloids if this tendency exists. The cause is unknown, and unlikely to result from repeated shaving or short hair cutting. It DENGA A MAKHADO may be caused by unusually thick collagen, which traps and disrupts terminal MB ChB, FCDerm (SA) hair follicles. Treatment is not very successful, and large keloids are best excised Consultant Dermatologist by a surgeon. Mild cases can be helped by intralesional injection of steroids like Division of Dermatology Celestone Soluspan into individual papules and scars. Long-term oral tetracy- clines, as for acne, can also be beneficial, as can topical antibiotics like erythro- University of the Witwatersrand and mycin and clindamycin. Chris Hani Baragwanath Hospital Johannesburg ACNE VULGARIS Denga Anna Makhado is a consultant der- There has been a recent tendency for acne to persist beyond the teens, or even matologist at Chris Hani Baragwanath occur for the first time in adulthood, called persistent adult acne.
    [Show full text]
  • Folliculitis
    Folliculitis Common Cutaneous • Inflammation of hair follicle(s) Bacterial Infections • Symptoms: Often pruritic (itchy) Pseudomonas folliculitis Eosinophilic Folliculitis (HIV) Folliculitis: Causes • Bacteria: – Gram positives (Staph): most common – Gram negatives: Pseudomonas – “hot tub” folliculitis • Fungal: Pityrosporum aka Malassezia • HIV: eosinophilic folliculitis (not bacterial) • Renal Failure: perforating folliculitis (not bacterial) Treatment of Folliculitis 21 year old female with controlled Crohn’s disease and history of • Bacterial hidradenitis suppuritiva presents stating – culture pustule she has recurrent flares of her HS – topical clindamycin or oral cephalexin / doxycycline – shower and change shirt after exercise – keep skin dry; loose clothing • Fungal: topical antifungals (e.g., ketoconazole) • Eosinophilic folliculitis – Phototherapy – Treat the HIV MRSA MRSA Eradication • Swab nares mupirocin ointment bid x 5 days • GI noted Crohn’s was controlled but increased – Swab axillae, perineum, pharynx infliximab intensity, but that was not controlling • Chlorhexidine 4% bodywash qd x 1 week recurrent “flares” • Chlorhexidine mouthwash qd x 1 week; soak toothbrush (or disposable) •I & D MRSA on three occasions • Bleach bath: 1/3 cup to tub, soak x 10 min tiw x 1 week, then prn (perhaps weekly) • THIS WAS INFLIXIMAB-RELATED • Oral antibiotics x 14 days: Bactrim, Doxycycline, depends FURUNCULOSIS FROM MRSA COLONIZATION on sensitivities – D/C infliximab • Swab partners – Anti-MRSA regimen • Hand sanitizer frequently – Patient is better • Bleach wipes to surfaces (doorknobs, faucet handles) • Towels use once then wash; paper towels when possible Pointing abscess (furuncle) --pointing requires I & D-- Acute Paronychia Furuncle Treatment Impetigo • Incise & Drain (I & D) Culture pus • Warm soaks • Antibiotics – e.g., cephalexin orally AND mupirocin topically • If recurrent, suspect nasal carriage of Staph aureus swab culture and mupirocin to nares b.i.d.
    [Show full text]
  • NYS DOH STD Reporting Form
    To order more copies of this form call the Provider Access Line: 1-866-NYC-DOH1 NYC Department of Health & Mental Hygiene Universal Reporting Form PHA No. Form PD-16 (9/09) Mail completed form to: NYC Dept. of Health & Mental Hygiene; 125 Worth Street, Room 315, CN-6; New York, NY 10013 • Or report online: www.nyc.gov/nycmed P Patient Last Name First Name Middle Name DATE OF REPORT A T Patient AKA: Last Name AKA: First Name M.I . I ____ / ____ / ____ E Date of Birth Age Country of Birth Soc.Sec.No. N T ____ / ____ / ________ If patient is a child, Guardian Last Name Guardian First Name M.I. Ⅺ Homeless I Borough: Manhattan N Patient Home Address Apt. No. Zip Code Bronx F Unknown Brooklyn O Home Telephone Number Medical Record Number ( _______ ) ________ – _____________ Queens R Unknown M Staten Island Other Telephone Number Medicaid Number Ⅺ NYC, borough unknown A Unknown ( _______ ) ________ – _____________ Unknown T Ⅺ I Sex Race (Check all that apply) Ethnicity Hispanic Please report non-NYC Not NYC (Specify City/State) Male Transexual Asian White American Indian/Alaska Native Unknown (Check one) Non-Hispanic residents to the appropriate ________________, _____ O N Female Unknown Black Other race Native Hawaiian/Pacific Islander Unknown health jurisdiction Ⅺ Unknown Admitted to hospital? Admission Date Is patient alive? If no, date of death Unknown Is patient pregnant? If yes, due date Yes No ____ / ____ / ________ Unknown Yes No Yes No Unknown Discharge Date Unknown ____ / ____ / ________ Unknown Unknown
    [Show full text]
  • Community Dermatology Journal
    www.ifd.org Community Dermatology ISSUE No. 16 Journal Comm Dermatol J 2013; 9: 1-12 HIFA and Community Dermatology Journal Neil Pakenham-Walsh Director HIFA Michele Murdoch Assistant Editor, Community Dermatology Journal Christopher Lovell Assistant Editor, Community Dermatology Journal Healthcare Information for all (HIFA) links over 6,000 healthcare professionals, libraries and publishers in 167 countries. We are pleased to announce that Community Dermatology Journal has been welcomed as a supporting organization to this global initiative, which was first established in 2006 at the 10th Congress of the Association for Health Information and Libraries in Africa, in Mombasa, Kenya. HIFA’s strategy is based on Distributing Community Dermatology Journal at Dabat Health a shared vision of a world where no-one dies for lack of healthcare Centre, Ethiopia Photo: Chris Lovell knowledge. It aims to achieve this via three main routes; 1) Through dynamic global email forums for discussion, in The aims and objectives of HIFA are in accord with those of collaboration with the World Health Organization and other Community Dermatology Journal, which is distributed free interested parties. HIFA’s five global forums (HIFA2015, of charge to healthcare workers worldwide, especially in rural CHILD2015, HIFA-Portuguese, HIFA-EVIPNet-French, and communities and resource-poor settings. We encourage all readers HIFA-Zambia) collaborate with WHO, International Child to join: www.hifa2015.org Health Group, International Society for Social Paediatrics and Child Health, and also with the Zambia UK Health Workforce Alliance. Contents 2) Through “HIFA Voices”, a knowledge base that harnesses the collective intelligence of HIFA members about information 1 Lead Article needs and how to meet them.
    [Show full text]