Clinical Practice Guidelines for the BC Eating Disorders Continuum of Services September 1, 2012
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Obesity with Comorbid Eating Disorders: Associated Health Risks and Treatment Approaches
nutrients Commentary Obesity with Comorbid Eating Disorders: Associated Health Risks and Treatment Approaches Felipe Q. da Luz 1,2,3,* ID , Phillipa Hay 4 ID , Stephen Touyz 2 and Amanda Sainsbury 1,2 ID 1 The Boden Institute of Obesity, Nutrition, Exercise & Eating Disorders, Faculty of Medicine and Health, Charles Perkins Centre, The University of Sydney, Camperdown, NSW 2006, Australia; [email protected] 2 Faculty of Science, School of Psychology, the University of Sydney, Camperdown, NSW 2006, Australia; [email protected] 3 CAPES Foundation, Ministry of Education of Brazil, Brasília, DF 70040-020, Brazil 4 Translational Health Research Institute (THRI), School of Medicine, Western Sydney University, Locked Bag 1797, Penrith, NSW 2751, Australia; [email protected] * Correspondence: [email protected]; Tel.: +61-02-8627-1961 Received: 11 May 2018; Accepted: 25 June 2018; Published: 27 June 2018 Abstract: Obesity and eating disorders are each associated with severe physical and mental health consequences, and individuals with obesity as well as comorbid eating disorders are at higher risk of these than individuals with either condition alone. Moreover, obesity can contribute to eating disorder behaviors and vice-versa. Here, we comment on the health complications and treatment options for individuals with obesity and comorbid eating disorder behaviors. It appears that in order to improve the healthcare provided to these individuals, there is a need for greater exchange of experiences and specialized knowledge between healthcare professionals working in the obesity field with those working in the field of eating disorders, and vice-versa. Additionally, nutritional and/or behavioral interventions simultaneously addressing weight management and reduction of eating disorder behaviors in individuals with obesity and comorbid eating disorders may be required. -
ABSTRACT Autism Spectrum Disorder (ASD)
ADLN - Perpustakaan Universitas Airlangga ABSTRACT Autism Spectrum Disorder (ASD) was a disturbance of pervasife development in children characterized by the disturbance and delayed in cognitive, language, behaviour, communication, and social interaction. In the past 10 to 20 years, increased the number of autism currently reached an average of 8 persons among 1000 resident or 1:125. The cause of autism is is yet not found until now. The purpose of this research is analize risk factor of the trigger autism in children. This research used case control design. In the case category (ASD) accounted for 35 children according to record data in special school and the control category as many as 105 children taken from public school. Independent variables were genetic, premature, postmature, antenatal bleeding, maternal age of pregnancy, caesar childbirth, low birth weight (LBW), interval of pregnancy, organic brain syndrome, asphyxia, vaccination, smoking, consumtion of medicine and medicinal herbs, and spontaneous abortion. Whereas dependent variable was ASD incident. The data analysis by calculated odds ratio with 95% CI. The result of the research obtained were genetic, postmature, antenatal bleeding, caesar childbirth, low birth weight, interval of pregnancy, organic brain syndrome, asphyxia, vaccination, smoking, consumtion of medicine and medicinal herbs, spontaneous abortion was not the risk factor of the trigger ASD incident. The result obtained on the variables that significally premature (OR= 7.12 , 95% CI: 2.56<OR<26.07) which means that the children born prematurely had a 7.12 times higher for the onset of autism than children born with normal gestational age and another variable maternal age over 35 years (OR=8.58 , 95% CI: 1.30 <OR< 92.57) which means that the mother was pregnant at the age over 35 years had a 5.58 times higher risk to had children had autism than the mother who become pregnant less than 35 years. -
Consultation-Liaison Psychiatry Fellowship
Consultation-Liaison Psychiatry Fellowship Name of institution: McGill University Health Center Type of Fellowship: Consultation-Liaison Psychiatry Number of fellowship positions requested: 1-2 Duration of Fellowship: 1 year Hospitals involved in training: McGill University Health Center, including Montreal General, Glen - Royal Victoria, Montreal Chest Institute, and Montreal Neurological Hospital Sites Time spent by the fellow in each institution: MGH- 25% Glen - 65% MNH- 10% Mission of Fellowship: This Clinical Fellowship year is intended to teach advanced skills in C/L psychiatry. Its purpose is to train academic leaders in C/L psychiatry, stimulate interest in academic work in the field and provide the administrative skills required for the development of C/L services at other institutions. Fellowship Program Director: Dr. Annette Granich Names of Teaching Faculty: The teaching faculty comprises M-J. Brouillette, S. Ducharme, G. Galbaud du Fort, A. Granich, L. Hoffman, T. Nguyen, L. Pinard, Z. Prelevic, L. Tourian, A. Wilner. This is an expert group of Consultation-Liaison Psychiatrists with an average of 15 years of clinical experience in the field. Roles of Teaching Faculty: Supervising psychiatrists will review clinical cases with the fellow and assess his/her skills specific to interviewing and interacting in the medical setting, including diagnostic and therapeutic skills, clinical judgment, communication effectiveness with other professionals, and advocacy for patients so as to ensure optimal care delivery. Role of Project Supervisors: Supervisors will guide and oversee the fellow in the preparation and completion of a project to be submitted for publication, in their area of specialized interest and expertise. They will ensure comprehensive exposure to and provide supervision for clinical experience in their area of subspecialty. -
New Eating Disorder Service
ORIGINAL PAPERS New eating disorder service Harry R, Millar Aims and method The paper describes the first three the Grampian area. We welcome direct enqui years experience of a new out-patient eating disorder ries from patients, relatives and friends, but service for adults. Clinical data on referrals using strongly encourage referral through the GP and standardised assessments are presented. if the GP is not involved a psychiatrist sees the Results The referral rate rapidly rose to over 200 referral. patients per annum. Forty-nine per cent of referrals We wish to enable generalist colleagues to were bulimic disorders and 26% had either anorexia continue seeing patients and offer a consul nervosa or atypical anorexia nervosa. Seventy per cent tative service to them but some managers were directly referred by general practitioners. seemed to believe that we should see all eating Clinical implications The creation of a specialist disorder patients. We had to explain that this eating disorder service quickly leads to a high referral is not logistically possible and not clinically rate suggesting a previously unmet need. A service necessary. based on assessment and treatment by nurse therapists is acceptable and feasible with appropriate training and support from psychiatry, clinical psychology and Assessment dietetics. Expectations of a specialist service include We have tried to provide a "comprehensive teaching, consultation, research and audit as well as multidimensional assessment" as recommended the provision of direct clinical care. by the American Psychiatric Association (1993). A Royal College of Psychiatrists (1992) report This includes two to three sessions with the recommends that across the country "a con primary therapist, a nutritional assessment by sultant-led local service for eating disorders the dietician, a physical assessment by a junior should be established, together with appropriate doctor and, if indicated, a psychiatric assess non-medial support." In 1994, an adult out ment by the consultant. -
Eating Disorders Toolkit
Coach & Trainer TOOLKIT NEDA TOOLKIT for Coaches and Trainers Table of contents Introduction . 1 PREVENTION . 2 Types of eating disorders . 3 Eating disorder signs and symptoms specific to an athletic setting . 4 Factors that protect or put athletes at risk for eating disorders . 5 Physiological impact of eating disorders on athletic performance . 6 The Female Athlete Triad . 7 Encouraging healthy and appropriate exercise and training for athletes . 9 Eating disorders prevention and the middle or high school athlete . 10 E A R LY INTERVENTION . 11 The potential role of the coach . 12 Tips on how to positively intervene . 13 Tips on how to provide a healthy sport environment conducive to recovery . 14 Confidentiality issues . 15 Talking with… coach interviews . 16 Coaches guide to sports nutrition . 19 Talking with… nutritionist interviews . 21 T R E AT M E N T . 23 Talking with… psychologist interviews . 24 Eating disorders and the team dynamic . 26 Athletes’ own stories: Patrick Bergstrom . 27 Diane Israel . 28 Whitney Post . 29 Kimiko Soldati . 30 The Walker family . 31 M O R E INFORMATION . 32 Frequently asked questions . 33 Common myths about eating disorders . 35 Glossary . 37 RESOURCES . 41 Books, Resources, Curricula, Position Papers . 42 Acknowledgements . 43 © 2010 National Eating Disorders Association. Permission is granted to copy and reprint materials for educational purposes only. National Eating Disorders Association must be cited and web address listed. NEDA TOOLKIT for Coaches and Trainers Introduction The benefits of sport are well recognized: organized Though most athletes with eating disorders are female, male athletes are also at risk—especially those competing in sports athletics builds self-esteem, promotes physical condi- that tend to emphasize diet, appearance, size and weight. -
Curriculum Vitae Peter Robert Martin Address
CURRICULUM VITAE PETER ROBERT MARTIN ADDRESS: Department of Psychiatry and Behavioral Sciences Vanderbilt Psychiatric Hospital Suite 3035, 1601 23rd Avenue South Nashville, Tennessee 37232-8650 U.S.A. Phone: 615-343-4527 Mobile: 615-364-7175 E-Mail: [email protected] [email protected] https://orcid.org/0000-0003-2292-4741 WEBSITES: https://wag.app.vanderbilt.edu/PublicPage/Faculty/Details/27348 http://www.vanderbilt.edu/ics/ DATE AND PLACE OF BIRTH: September 6, 1949, Budapest, Hungary FAMILY: Married Barbara Ruth Bradford, December 23, 1985 Alexander Bradford Martin, born October 21, 1989 EDUCATION: 1967 - 1971 Honours B.Sc. (Molecular Genetics) McGill University, Montreal, Quebec, Canada 1971 - 1975 M.D., C.M. McGill University, Montreal, Quebec, Canada 1975 - 1976 Resident in Internal Medicine, Sunnybrook Medical Centre, University of Toronto, Toronto, Ontario, Canada 1976 - 1978 Research Fellow in Clinical Pharmacology, Clinical Pharmacology Program, Addiction Research Foundation Clinical Institute - Toronto Western Hospital, University of Toronto 1976 - 1979 M.Sc. (Pharmacology) University of Toronto Dissertation: Intravenous phenobarbital treatment of barbiturate and other hypnosedative withdrawal: A pharmacokinetic approach. 1978 - 1979 Resident in Psychiatry, Affective Disorders Unit, Clarke Institute of Psychiatry, University of Toronto 1979 Resident in Psychiatry, Hospital for Sick Children, University of Toronto 1980 Resident in Psychiatry, Toronto General Hospital, University of Toronto LICENSURE AND CERTIFICATION: The College of Physicians and Surgeons of Ontario (License No. 28907), 1976. The Board of Medical Examiners of the State of Maryland (License No. D26685), 1981. The Board of Medical Examiners of the State of Tennessee (License No. MD17128), 1986. Fellow of the Royal College of Physicians (Canada), Psychiatry, 1981. -
PAVOL JOZEF ŠAFARIK UNIVERSITY in KOŠICE Dissociative Amnesia: a Clinical and Theoretical Reconsideration DEGREE THESIS
PAVOL JOZEF ŠAFARIK UNIVERSITY IN KOŠICE FACULTY OF MEDICINE Dissociative amnesia: a clinical and theoretical reconsideration Paulo Alexandre Rocha Simão DEGREE THESIS Košice 2017 PAVOL JOZEF ŠAFARIK UNIVERSITY IN KOŠICE FACULTY OF MEDICINE FIRST DEPARTMENT OF PSYCHIATRY Dissociative amnesia: a clinical and theoretical reconsideration Paulo Alexandre Rocha Simão DEGREE THESIS Thesis supervisor: Mgr. MUDr. Jozef Dragašek, PhD., MHA Košice 2017 Analytical sheet Author Paulo Alexandre Rocha Simão Thesis title Dissociative amnesia: a clinical and theoretical reconsideration Language of the thesis English Type of thesis Degree thesis Number of pages 89 Academic degree M.D. University Pavol Jozef Šafárik University in Košice Faculty Faculty of Medicine Department/Institute Department of Psychiatry Study branch General Medicine Study programme General Medicine City Košice Thesis supervisor Mgr. MUDr. Jozef Dragašek, PhD., MHA Date of submission 06/2017 Date of defence 09/2017 Key words Dissociative amnesia, dissociative fugue, dissociative identity disorder Thesis title in the Disociatívna amnézia: klinické a teoretické prehodnotenie Slovak language Key words in the Disociatívna amnézia, disociatívna fuga, disociatívna porucha identity Slovak language Abstract in the English language Dissociative amnesia is a one of the most intriguing, misdiagnosed conditions in the psychiatric world. Dissociative amnesia is related to other dissociative disorders, such as dissociative identity disorder and dissociative fugue. Its clinical features are known -
Social Cognition in Eating Disorders: Encoding and Representational Processes in Binging and Purging Patients
RESEARCH ARTICLE Social Cognition in Eating Disorders: Encoding and Representational Processes in Binging and Purging Patients Lily Rothschild-Yakar1,2*, Zohar Eviatar2, Adi Shamia2 & Eitan Gur3 1Safra Children’s Hospital, Sheba Medical Center, Tel Hashomer, Israel 2Department of Psychology, University of Haifa, Israel 3Sheba Medical Center, Tel Hashomer, Israel Abstract Objective: The present study investigates social cognition impairments in 29 women with bingeing/purging spectrum eating disorders (ED) compared to 27 healthy controls. Method: Measures were used to examine encoding and representational processes in relation to affect perception and affect attribution, as well as the ability to recognize mental causality in social relationships. Results: ED patients failed to correctly encode causality in interpersonal relations, exhibited deficits in their ability to ascribe behaviour to mental states, and showed a greater tendency to attribute negative affects in interpersonal relationships. Stepwise regression analyses suggested that ED symptoms could account for deficits in the recognition of causality in interpersonal relations. Conclusions: In addition to addressing ED symptoms, social cognition deficits should be addressed in the psychological treatment of EDs. Copyright # 2010 John Wiley & Sons, Ltd and Eating Disorders Association. Keywords eating disorders; social cognition; mentalization *Correspondence Lily Rothschild-Yakar, PhD, Department of Psychology, University of Haifa, Haifa 31905, Israel. Tel: 972-3-649-9563; Fax: 974-4-824-0966. Email: [email protected] Published online 29 July 2010 in Wiley Online Library (wileyonlinelibrary.com) DOI: 10.1002/erv.1013 cognition in AN (restricting type), suggesting similarities Introduction with autistic spectrum disorders. They suggested that Dysfunctional social cognition may play an important deficits in social cognition may be central to the role in eating disorders (ED). -
Liaison Psychiatry Services - Guidance
! ! ! ! ! ! ! ! ! ! ! ! Liaison Psychiatry Services - Guidance 1st edition, February 2014 ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! ! Title: Liaison Psychiatry Services - Guidance ! Edition: 1st edition ! Date: February 2014 ! URL: http://mentalhealthpartnerships.com/resource/liaison-psychiatry-services- guidance/ ! Commissioners: Strategic Clinical Network for Mental Health, Dementia and Neurological Conditions South West ! Editors: Dr Peter Aitken, Dr Sarah Robens, Tobit Emmens Devon Partnership NHS Trust, Dryden Road, Wonford House, Exeter, EX2 5AF. www.devonpartnership.nhs.uk ! ! Contents ! Preface 2 With thanks and appreciation 3 What people told us they wanted from the Guidance 4 Introduction 6 Connecting liaison psychiatry services with other pathways of care 6 Models of hospital based liaison psychiatry services for which there is evidence of cost and quality outcome benefits 8 Effective liaison psychiatry services 9 Where liaison psychiatry services can impact 11 Key references 12 !1 Preface ! This guidance provides advice on commissioning liaison psychiatry services, specifying minimum and optimum service specifications, costs and quality outcome measures. It was commissioned by the Strategic Clinical Network for Mental Health, Dementia and Neurological Conditions South West. ! The guidance is provided as four related documents, each with increasing levels of detail: ! • Liaison Psychiatry Services - Guidance - sets out the key considerations to be made when commissioning liaison psychiatry services. • An Evidence Base for Liaison Psychiatry - Guidance - sets out the evidence gathered from lay people, professionals, commissioners and the literature about what is needed from liaison psychiatry services. • Developing Models for Liaison Psychiatry Services - Guidance - provides the technical information needed for commissioning liaison psychiatry services. • Model Service Specifications for Liaison Psychiatry Services - sets out exemplar service specifications for four models of liaison psychiatry. -
Dr. Jennifer Kruse, MD Board-Certified in Psychiatry
California Medical Evaluators 11620 Wilshire Blvd. Suite 340 Los Angeles, CA 90025 Phone: 888.853.7944 Fax: 213.478.0550 [email protected] Dr. Jennifer Kruse , MD Board-Certified in Psychiatry Board-certified psychiatrist with more than 9 years of clinical expertise. EDUCATION • UCLA Clinical Pharmacology Training Program (2016-2017) T32 Research Fellowship Award • CTSI Training Program in Translational Science (2015-2017) Track 2 Certificate Program • Mayo Clinic, Rochester, MN (2013-2014) Psychosomatic Medicine Fellowship • Semel Institute for Neuroscience & Human Behavior, UCLA (2009-2013) Psychiatric Residency • Mayo Medical School, Mayo Clinic (2005-2009) Doctor of Medicine • University of Iowa, Iowa City, IA (2001-2005) Bachelor of Arts, Spanish, with Honors and Highest Distinction PROFESSIONAL EXPERIENCE • Psychiatrist, Transitional Age Youth, Full Service Partnership – San Fernando Valley Community Mental Health Center, Inc., Van Nuys, CA (2011-2013) ACADEMIC APPOINTMENTS • Health Sciences Assistant Clinical Professor - Dept. of Psychiatry, UCLA (2015-present) • Supervising Faculty - Mood Disorders Clinic, Dept. of Psychiatry, UCLA (2015-present) • Associate Director of Consultation/Liaison Psychiatry – RRUMC, UCLA (2014-present) • Wilder Scholar – UCLA (2014-present) • Clinical Instructor – Department of Psychiatry, UCLA (2014-2015) • Staff Associate Physician – UCLA (2013-2014) LICENSURE AND CERTIFICATION • California State Medical License (2010) • Board-Certified, American Board of Psychiatry and Neurology (2013) • Psychosomatic -
Amnesia: Its Detection by Psychophysiological Measures
Amnesia: Its Detection by Psychophysiological Measures BRIAN E. LYNCH, M.A.* and JOHN McD. W. BRADFORD, M.B., Ch.B. (VCT), D.P.M. (VCT), F.F. PSYCH. (SA), M.R.C. P S Y C H. ( V . K . ) ** Introduction The term amnesia encompasses many varied examples and causes of memory loss. In an edited text on amnesia, Whitty and Zangwill explore the subject in terms of cerebral disease, trauma, electroconvulsive therapy and psychogenesis. 1 It becomes obvious from the various causative factors involved in amnesia that such a multi-factorial problem requires an eclectic approach in answering. At present, one area being investigated in some detail is alcohoVdrug-induced memory loss. The focus of the present study is the exploration of memory dysfunction resulting from alcohoVdrug abuse. In any discussion of alcohoVdrug-induced amnesia certain features of the dysfunction become important for consideration. Before the amnesia can be examined it must be determined to be either a legitimate loss or a feigned amnesia state. The basis of feigned versus genuine amnesia rests with an individual's biological and psychological resources. Expediency suggests that genuine amnesia tends toward an organic base and feigned toward a psychogenic base. That is to say, the probability of a genuine amnesia is greatly increased when found in concert with organicity. Likewise, feigned memory loss is more likely to be found in individuals suffering from a psychogenic disorder. In addition to the issue of genuine versus feigned amnesia, the type of memory loss is important in any clinical assessment of the disorder. The present study utilizes a clinical breakdown of amnesia type in accordance with characteristics outlined by Bradford and Smith.2 The amnesia states are discussed in relation to the following category types: (A) hazy amnesia: no absolute amnesia either in circumscribed periods or in one complete period; (B) partial (patchy) amnesia: segments of memory loss with no complete period of amnesia; (C) complete amnesia: total ·Mr. -
Madness As Disability
HPY0010.1177/0957154X14545846History of PsychiatryGilman 545846research-article2014 Article History of Psychiatry 2014, Vol. 25(4) 441 –449 Madness as disability © The Author(s) 2014 Reprints and permissions: sagepub.co.uk/journalsPermissions.nav DOI: 10.1177/0957154X14545846 hpy.sagepub.com Sander L Gilman Emory University, Atlanta, USA Abstract How does society imagine mental illness? Does this shift radically over time and with different social attitudes as well as scientific discoveries about the origins and meanings of mental illness? What happens when we begin to think about mental illness as madness, as a malleable concept constantly shifting its meaning? We thus look at the meanings associated with ‘general paralysis of the insane’ in the nineteenth century and autism today in regard to disability. In this case study we examine the claims by scholars such as the anthropologist Emily Martin and the psychiatrist Kay Jamison as to the relationship between mental illness, disability and creativity. Today, the health sciences have become concerned with mental illness as a form of disability. How does this change the meaning of madness for practitioners and patients? Keywords Autism, bipolar disease, disability, madness With interest in mental illness from the standpoint of the growing field of disability studies comes a problem for the medical humanities (Callard et al., 2012; Longmore and Umansky, 2001). Is it possible to discuss what may well be the most stigmatizing form of illness or disability in the West without understanding the complexity of the tradition in which it stands? The diagnostic history of mental illness is entangled with the history of madness as a social convention, a history that colours even the contemporary debates about the meanings and forms of mental illness in our age of brain imagery and neuro-anatomy.