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Alzheimer-Medicatie Op Drift Met Nitromemantine

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Alzheimer-Medicatie Op Drift Met Nitromemantine Blog op seniorennet www.haesbrouck.be Blog op adhdfraude www.megablunder.net Jaargang 7 nr. 684 22 juni 2013 http://www.youtube.com/watch?v=cpmIEwPIJEo Nieuwsbrief Alzheimer-medicatie op drift met nitromemantine De aangeduide inhoud op psychiatrie.be is intussen verdwenen en de URL verwijst voortaan (merkwaardig genoeg met 17/06/2013 als laatste herziening) naar: http://www.schizofrenie24x7.be/?q=google_appliance Immers op 15/06/2013 projecteerde ik tijdens een lezing in Gent de vorige afbeelding, waardoor ik de geciteerde tekst hier afdruk. Nu zou nieuwe medicatie voor Alzheimer voortaan aan de synapsen gaan prutsen. Terwijl de grote lichten, die zich specialisten noemen, nog niet eens beseffen, dat door het kapotverbranden van neuronen door 'veilige' mirakelmedicatie, de synapsen om een prikkeloverdracht te kunnen realiseren als een oceaan zo groot en dus veel te groot zijn geworden om nog ooit bruikbare prikkeloverdrachten mogelijk te maken. Als de synaps groter is dan de spleet die het handboek uit 1976 en de elektronenmicroscoop-foto aangeeft dan kan een elektrische prikkel, nodig om een gedrag onder controle te houden, helemaal niet meer. Leuke medicatie die droefenis, ADHD, koude tenen, menopauze, opvliegers, blozen, PTSD , liefdesverdriet en nog veel meer tot genot van elkeen kan aanpakken, maakt neuronen kapot, waardoor de vitale synapswerking die een rol te spelen heeft, zelfs bij normaal gedrag, om zeep is geholpen. Waardoor uiteindelijk na lang of chronisch gebruik ... jawel... Alzheimer kon ontstaan. En nu zou nieuwe Alzheimer-medicatie eens aan de synapsen gaan prutsen. Synapsen, die er niet meer zijn. Hoera, leve de chemie. Ik vermoed dat de meest verstandigen onder de geleerde peuten, synapsen op de plaatjes gaan bijtekenen, in mooie couleuren en met flitsende schichten die fantoomprikkeloverdrachten zouden moeten voorstellen. Die nieuwe Alzheimer-geleerden werden vast en zeker nog opgeleid in de tijd dat de ziekten zomaar verzonnen konden worden en daarbij dan vanzelfsprekend ook de uitleg daarover. Maar Alzheimer bestaat al enige tijd, en nog niemand heeft tot op vandaag al bekend gemaakt op welke manier je die commercieel heel rendabele dementie tot stand kan brengen. Vandaar de grote leemte in kennis daarover, iets wat voeding geeft aan de meest wilde fantasieën op het vlak van een behandeling. Hoe werkt nitromemantine (volgens de nieuwe leer)? Deze wetenschappelijke tekst veronderstelt dat de kapotte neuronen, waarvan de verbinding met de andere neuronen is wegverbrand, uiteindelijk met nitromemantine toch wel weer functioneel tot stand zou moeten kunnen komen. Zelfs wanneer de synaps, waarover de elektrische prikkelgeleiding moet gaan, veel te groot is geworden om ooit nog een prikkel te kunnen veroorzaken. En zelfs dan volstaat een nieuwe aangroei (neuroplasticiteit) daarbij niet, omdat een nieuwe groei op de juiste plaatsen helemaal niet meer kan. Waardoor het zenuwstelsel permanent beschadigd en daardoor gehandicapt is geworden. Lievemoederen helpt in dat geval jammer genoeg niet meer. Maar hoe werkt nitromemantine dan wel volgens mijn bescheiden theorie? Zoals de officiële tekst aangeeft is die stof een dubbele molecule, die creatief gebruik maakt van de instabiele zuurstofbruggen, geënt op een relatief inerte andere component - memantine -(waarvan de literatuur de werking omschrijft als 'This patented work contributed to the FDA approval of memantine in 2003 for the treatment of moderate to severe Alzheimer’s disease. However, memantine’s effectiveness has been limited. ' Het vroegere gebruik van memantine bij Alzheimer schijnt vooral te 'genezen' wanneer de extended release van de stof (memantine ER) gecombineerd werd met donepezil (Aricept). Commercieel als Acrescent (Lundbeck), Arimenda of Balaxur. Intussen is bij Aricept bekend dat de stof een chemisch patroon gebruikt om de overblijvende gezonde neuronen te vernielen, waardoor de ontstane gevarenreflex (doping) de schijn wekt dat patiënt alerter is geworden en dat daarmee het ziekteproces even wordt tegengehouden. Een valse illusie dus. Maar commercieel wel handig. Vandaar dat memantine alleen, niet zoveel blijkt uit te halen en men aangewezen blijft op het therapeutisch verwoesten van neuronen. Omdat het voortaan steeds moeilijker werd om de echte farmacologie van succesmedicatie te verbergen en om een op til zijnde Alzheimer-epidemie in te dijken, worden intussen heel wat onderzoeken stopgezet. Ik noem ze even: Dexanabinol (sinabidol) bij de Hebreeuwse universiteit in Jerusalem. Neramexane bij Merz & Co. EVT102 bij Hoffman-LaRoche. Ipenoxazone bij Nippon Chemiphar. Traxoprodil bij Pfizer. Alaproclate bij Astra. Wat meteen een vlucht naar drankjes op gang deed komen. In deze bijdrage gaat het over de ultieme lucide vondst om deze min of meer inerte stof, memantine, instabiel te koppelen aan een reducerend reagens, dat medisch al blijk gaf van een therapeutische activiteit als vaatverwijder. Waarbij door verestering met de metabolieten uit de voeding, nitroglycerine – als een prodrug - nieuwe en wel (weeral) actieve psychotica zal vormen, die net zo goed als de reeds bekende psychotica schijnbaar helend ook de nog overblijvende goede neuronen vernietigend kunnen aanpakken. Alvast grappig hoezeer wetenschappelijk en geleerd men deze farce aan de mensheid kan verkopen. Toch benieuwd wie zich aan deze truc zal laten vangen en welke comorbiditeiten er vervolgens nog aan dementie zullen toegevoegd worden. Vraag is alleen: wie zal de bevolking ervan op de hoogte brengen dat zelfs dementie als een voorwendsel zal gebruikt worden om levensonwaardige levens uit deze maatschappij te extermineren. In functie van een beter en performanter menselijk ras. Wie zal zich daar als eerste ooit om bekommeren? Apotheker Fernand Haesbrouck, 22 juni 2013 .
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