Low Back Pain: an Interventionalist's Perspective
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Download the Herniated Disc Brochure
AN INTRODUCTION TO HERNIATED DISCS This booklet provides general information on herniated discs. It is not meant to replace any personal conversations that you might wish to have with your physician or other member of your healthcare team. Not all the information here will apply to your individual treatment or its outcome. About the Spine CERVICAL The human spine is comprised 24 bones or vertebrae in the cervical (neck) spine, the thoracic (chest) spine, and the lumbar (lower back) THORACIC spine, plus the sacral bones. Vertebrae are connected by several joints, which allow you to bend, twist, and carry loads. The main joint LUMBAR between two vertebrae is called an intervertebral disc. The disc is comprised of two parts, a tough and fibrous outer layer (annulus fibrosis) SACRUM and a soft, gelatinous center (nucleus pulposus). These two parts work in conjunction to allow the spine to move, and also provide shock absorption. INTERVERTEBRAL ANNULUS DISC FIBROSIS SPINAL NERVES NUCLEUS PULPOSUS Each vertebrae has an opening (vertebral foramen) through which a tubular bundle of spinal nerves and VERTEBRAL spinal nerve roots travel. FORAMEN From the cervical spine to the mid-lumbar spine this bundle of nerves is called the spinal cord. The bundle is then referred to as the cauda equina through the bottom of the spine. At each level of the spine, spinal nerves exit the spinal cord and cauda equina to both the left and right sides. This enables movement and feeling throughout the body. What is a Herniated Disc? When the gelatinous center of the intervertebral disc pushes out through a tear in the fibrous wall, the disc herniates. -
Guidline for the Evidence-Informed Primary Care Management of Low Back Pain
Guideline for the Evidence-Informed Primary Care Management of Low Back Pain 2nd Edition These recommendations are systematically developed statements to assist practitioner and patient decisions about appropriate health care for specific clinical circumstances. They should be used as an adjunct to sound clinical decision making. Guideline Disease/Condition(s) Targeted Specifications Acute and sub-acute low back pain Chronic low back pain Acute and sub-acute sciatica/radiculopathy Chronic sciatica/radiculopathy Category Prevention Diagnosis Evaluation Management Treatment Intended Users Primary health care providers, for example: family physicians, osteopathic physicians, chiro- practors, physical therapists, occupational therapists, nurses, pharmacists, psychologists. Purpose To help Alberta clinicians make evidence-informed decisions about care of patients with non- specific low back pain. Objectives • To increase the use of evidence-informed conservative approaches to the prevention, assessment, diagnosis, and treatment in primary care patients with low back pain • To promote appropriate specialist referrals and use of diagnostic tests in patients with low back pain • To encourage patients to engage in appropriate self-care activities Target Population Adult patients 18 years or older in primary care settings. Exclusions: pregnant women; patients under the age of 18 years; diagnosis or treatment of specific causes of low back pain such as: inpatient treatments (surgical treatments); referred pain (from abdomen, kidney, ovary, pelvis, -
Inflammatory Back Pain in Patients Treated with Isotretinoin Although 3 NSAID Were Administered, Her Complaints Did Not Improve
Inflammatory Back Pain in Patients Treated with Isotretinoin Although 3 NSAID were administered, her complaints did not improve. She discontinued isotretinoin in the third month. Over 20 days her com- To the Editor: plaints gradually resolved. Despite the positive effects of isotretinoin on a number of cancers and In the literature, there are reports of different mechanisms and path- severe skin conditions, several disorders of the musculoskeletal system ways indicating that isotretinoin causes immune dysfunction and leads to have been reported in patients who are treated with it. Reactive seronega- arthritis and vasculitis. Because of its detergent-like effects, isotretinoin tive arthritis and sacroiliitis are very rare side effects1,2,3. We describe 4 induces some alterations in the lysosomal membrane structure of the cells, cases of inflammatory back pain without sacroiliitis after a month of and this predisposes to a degeneration process in the synovial cells. It is isotretinoin therapy. We observed that after termination of the isotretinoin thought that isotretinoin treatment may render cells vulnerable to mild trau- therapy, patients’ complaints completely resolved. mas that normally would not cause injury4. Musculoskeletal system side effects reported from isotretinoin treat- Activation of an infection trigger by isotretinoin therapy is complicat- ment include skeletal hyperostosis, calcification of tendons and ligaments, ed5. According to the Naranjo Probability Scale, there is a potential rela- premature epiphyseal closure, decreases in bone mineral density, back tionship between isotretinoin therapy and bilateral sacroiliitis6. It is thought pain, myalgia and arthralgia, transient pain in the chest, arthritis, tendonitis, that patients who are HLA-B27-positive could be more prone to develop- other types of bone abnormalities, elevations of creatine phosphokinase, ing sacroiliitis and back pain after treatment with isotretinoin, or that and rare reports of rhabdomyolysis. -
Sciatica and Chronic Pain
Sciatica and Chronic Pain Past, Present and Future Robert W. Baloh 123 Sciatica and Chronic Pain Robert W. Baloh Sciatica and Chronic Pain Past, Present and Future Robert W. Baloh, MD Department of Neurology University of California, Los Angeles Los Angeles, CA, USA ISBN 978-3-319-93903-2 ISBN 978-3-319-93904-9 (eBook) https://doi.org/10.1007/978-3-319-93904-9 Library of Congress Control Number: 2018952076 © Springer International Publishing AG, part of Springer Nature 2019 This work is subject to copyright. All rights are reserved by the Publisher, whether the whole or part of the material is concerned, specifically the rights of translation, reprinting, reuse of illustrations, recitation, broadcasting, reproduction on microfilms or in any other physical way, and transmission or information storage and retrieval, electronic adaptation, computer software, or by similar or dissimilar methodology now known or hereafter developed. The use of general descriptive names, registered names, trademarks, service marks, etc. in this publication does not imply, even in the absence of a specific statement, that such names are exempt from the relevant protective laws and regulations and therefore free for general use. The publisher, the authors, and the editors are safe to assume that the advice and information in this book are believed to be true and accurate at the date of publication. Neither the publisher nor the authors or the editors give a warranty, express or implied, with respect to the material contained herein or for any errors or omissions that may have been made. The publisher remains neutral with regard to jurisdictional claims in published maps and institutional affiliations. -
ICD9 & ICD10 Neuromuscular Codes
ICD-9-CM and ICD-10-CM NEUROMUSCULAR DIAGNOSIS CODES ICD-9-CM ICD-10-CM Focal Neuropathy Mononeuropathy G56.00 Carpal tunnel syndrome, unspecified Carpal tunnel syndrome 354.00 G56.00 upper limb Other lesions of median nerve, Other median nerve lesion 354.10 G56.10 unspecified upper limb Lesion of ulnar nerve, unspecified Lesion of ulnar nerve 354.20 G56.20 upper limb Lesion of radial nerve, unspecified Lesion of radial nerve 354.30 G56.30 upper limb Lesion of sciatic nerve, unspecified Sciatic nerve lesion (Piriformis syndrome) 355.00 G57.00 lower limb Meralgia paresthetica, unspecified Meralgia paresthetica 355.10 G57.10 lower limb Lesion of lateral popiteal nerve, Peroneal nerve (lesion of lateral popiteal nerve) 355.30 G57.30 unspecified lower limb Tarsal tunnel syndrome, unspecified Tarsal tunnel syndrome 355.50 G57.50 lower limb Plexus Brachial plexus lesion 353.00 Brachial plexus disorders G54.0 Brachial neuralgia (or radiculitis NOS) 723.40 Radiculopathy, cervical region M54.12 Radiculopathy, cervicothoracic region M54.13 Thoracic outlet syndrome (Thoracic root Thoracic root disorders, not elsewhere 353.00 G54.3 lesions, not elsewhere classified) classified Lumbosacral plexus lesion 353.10 Lumbosacral plexus disorders G54.1 Neuralgic amyotrophy 353.50 Neuralgic amyotrophy G54.5 Root Cervical radiculopathy (Intervertebral disc Cervical disc disorder with myelopathy, 722.71 M50.00 disorder with myelopathy, cervical region) unspecified cervical region Lumbosacral root lesions (Degeneration of Other intervertebral disc degeneration, -
Diagnosing and Treating Trigeminal Neuralgia in General Dentistry
general practice feature Chasing Pain Diagnosing and Treating Trigeminal Neuralgia in General Dentistry by Steven Olmos, DDS, DABCP, DABCDSM, DABDSM, DAAPM, FAAOP, FAACP, FICCMO, FADI, FIAO As dentists, we know quite a bit about tooth and gum pain, but when it comes to chronic facial pain and neuropathic pain, our dental school education leaves us unprepared. The objective of this article is to explain the differences between men and women with chronic orofacial pain and the relationship to proper functional breathing, using a case study as demonstration. 34 JANUARY 2016 // dentaltown.com general practice feature the United States, nearly half research published in Chest 2015 demonstrates that of all adults lived with chronic respiratory-effort-related arousal may be the most pain in 2011. Of 353,000 adults likely cause (nasal obstruction or mouth breath- 11 aged 18 years or older who were ing). Rising C02 (hypercapnia) in a patient with a surveyed by Gallup-Health- sleep-breathing disorder (including mouth breath- ways, 47 percent reported having at least one of ing) specifically stimulates the superficial masseter three types of chronic pain: neck or back pain, muscles to contract.12 knee or leg pain, or recurring pain.2 Identifying the structural area of obstruction A study published in The Journal of the Amer- (Four Points of Obstruction; Fig. 1) of the air- ican Dental Association October 2015 stated: way will insure the most effective treatment for a “One in six patients visiting a general dentist had sleep-breathing disorder and effectively reduce the experienced orofacial pain during the last year. -
Occipital Neuralgia: a Literature Review of Current Treatments from Traditional Medicine to CAM Treatments
Occipital Neuralgia: A Literature Review of Current Treatments from Traditional Medicine to CAM Treatments By Nikole Benavides Faculty Advisor: Dr. Patrick Montgomery Graduation: April 2011 1 Abstract Objective. This article provides an overview of the current and upcoming treatments for people who suffer from the signs and symptoms of greater occipital neuralgia. Types of treatments will be analyzed and discussed, varying from traditional Western medicine to treatments from complementary and alternative health care. Methods. A PubMed search was performed using the key words listed in this abstract. Results. Twenty-nine references were used in this literature review. The current literature reveals abundant peer reviewed research on medications used to treat this malady, but relatively little on the CAM approach. Conclusion. Occipital Neuralgia has become one of the more complicated headaches to diagnose. The symptoms often mimic those of other headaches and can occur post-trauma or due to other contributing factors. There are a variety of treatments that involve surgery or blocking of the greater occipital nerve. As people continue to seek more natural treatments, the need for alternative treatments is on the rise. Key Words. Occipital Neuralgia; Headache; Alternative Treatments; Acupuncture; Chiropractic; Nutrition 2 Introduction Occipital neuralgia is a type of headache that describes the irritation of the greater occipital nerve and the signs and symptoms associated with it. It is a difficult headache to diagnose due to the variety of signs and symptoms it presents with. It can be due to a post-traumatic event, degenerative changes, congenital anomalies, or other factors (10). The patterns of occipital neuralgia mimic those of other headaches. -
Occipital Neuralgia - Types of Headache/Migraine | American Migraine
Occipital Neuralgia - Types of Headache/Migraine | American Migraine ... http://www.americanmigrainefoundation.org/occipital-neuralgia/?print=y Occipital Neuralgia - Symptoms, Diagnosis, and Treatment Key Points: 1. Occipital neuralgia may be a cause of head pain originating in the occipital region (back of the head). 2. Pain is episodic, brief, severe, and shock-like. It originates from the occipital region and radiates along the course of the occipital nerves. 3. Attacks may be triggered by routine activities such as brushing the hair, moving the neck, or resting the head on a pillow. 4. Antiepileptic medications, tricyclic anti-depressants, and nerve blocks may be used for treatment. Introduction: Occipital neuralgia (ON) is a relatively rare primary headache disorder (primary headache disorders are not symptoms of or caused by another condition) affecting around 3.2/100,000 people per year.1 The term “neuralgia” refers to pain in the distribution of a nerve, in this case the occipital nerves. The greater, lesser, and third occipital nerves originate from the upper cervical nerve roots, course up the neck muscles, and exit near the base of the skull. These pure sensory nerves provide sensation to the back of the head, up to the top of the head, and behind the ears. The cause of ON is unknown; however, entrapment and irritation of the nerves have been proposed. Pain secondary to trauma such as whiplash injuries, inflammation, and compression of the occipital nerves by arteries or tumors have all been hypothesized, but no consensus has been reached. 1,2 ON may be provoked (triggered) simply by touching the affected region. -
Neuropathic Orofacial Pain the Brochure Is Provided Compliments Of
Neuropathic_Pain_Brochure_Neuropathic_Pain_Brochure 6/9/2010 11:41 AM Page 1 Neuropathic orofacial paiN The brochure is provided compliments of This brochure in intended for informational purposes only and should be considered a replacement for a professional treatment for a health care professional. To locate knowledgeable and experienced expert in orofacial pain, contact: The American Academy of Orofacial Pain 174 S. New York Ave. POB 478 Oceanville, NJ 08231 P: 609-504-1311 E: [email protected] W: www.aaop.org To locate knowledgeable and experienced expert in Trigeminal Neuralgia, contact: Trigeminal Neuralgia Association 2801 SW Archer Road Gainesville, FL 32608 P: 352-376-9955 E: [email protected] W: www.tna-support.org Neuropathic_Pain_Brochure_Neuropathic_Pain_Brochure 6/9/2010 11:41 AM Page 3 coNteNts 1-Neuropathic orofacial paiN 4-GettiNG help/What to expect 6-commoN Neuropathic orofacial paiN DisorDers aND their treatmeNt 6 - triGem iNal NeuralGia 8 - pre-triGem iNal NeuralGia 9 - atypical oDoNtalGia (phaNtom tooth paiN) 10 - chroNic reGioNal paiN syNDrome 12 - iN coNclusioN Neuropathic_Pain_Brochure_Neuropathic_Pain_Brochure 6/9/2010 11:41 AM Page 4 Neuropathic orofacial paiN Of the many pains that can effect the head and neck, perhaps the most confusing and difficult to diagnose are a group of maladies called Neuropathic Orofacial Pain Disorders. These neuropathic pain disorders are often chronic and arise from the brain and nerves of the head, face and neck. If you have experienced the frustration of having a toothache or face pain and, after seeing many doctors, still don't know where the pain is coming from, Brain you may be suffering from a neuropathic pain Spinal Cord disorder. -
Mechanical Low Back Pain Joshua Scott Will, DO; David C
Mechanical Low Back Pain Joshua Scott Will, DO; David C. Bury, DO; and John A. Miller, DPT Martin Army Community Hospital, Fort Benning, Georgia Low back pain is usually nonspecific or mechanical. Mechanical low back pain arises intrinsically from the spine, interverte- bral disks, or surrounding soft tissues. Clinical clues, or red flags, may help identify cases of nonmechanical low back pain and prompt further evaluation or imaging. Red flags include progressive motor or sensory loss, new urinary retention or overflow incontinence, history of cancer, recent invasive spinal procedure, and significant trauma relative to age. Imaging on initial presentation should be reserved for when there is suspicion for cauda equina syndrome, malignancy, fracture, or infection. Plain radiography of the lumbar spine is appropriate to assess for fracture and bony abnormality, whereas magnetic resonance imaging is better for identifying the source of neurologic or soft tissue abnormalities. There are multiple treatment modalities for mechanical low back pain, but strong evidence of benefit is often lacking. Moderate evidence supports the use of nonsteroidal anti-inflammatory drugs, opioids, and topiramate in the short-term treatment of mechanical low back pain. There is little or no evidence of benefit for acetamin- ophen, antidepressants (except duloxetine), skeletal muscle relaxants, lidocaine patches, and transcutaneous electrical nerve stimulation in the treatment of chronic low back pain. There is strong evidence for short-term effectiveness and moderate-quality evidence for long-term effectiveness of yoga in the treatment of chronic low back pain. Various spinal manipulative techniques (osteopathic manipulative treatment, spinal manipulative therapy) have shown mixed benefits in the acute and chronic setting. -
Diagnosis and Treatment of Lumbar Disc Herniation with Radiculopathy
Y Lumbar Disc Herniation with Radiculopathy | NASS Clinical Guidelines 1 G Evidence-Based Clinical Guidelines for Multidisciplinary ETHODOLO Spine Care M NE I DEL I U /G ON Diagnosis and Treatment of I NTRODUCT Lumbar Disc I Herniation with Radiculopathy NASS Evidence-Based Clinical Guidelines Committee D. Scott Kreiner, MD Paul Dougherty, II, DC Committee Chair, Natural History Chair Robert Fernand, MD Gary Ghiselli, MD Steven Hwang, MD Amgad S. Hanna, MD Diagnosis/Imaging Chair Tim Lamer, MD Anthony J. Lisi, DC John Easa, MD Daniel J. Mazanec, MD Medical/Interventional Treatment Chair Richard J. Meagher, MD Robert C. Nucci, MD Daniel K .Resnick, MD Rakesh D. Patel, MD Surgical Treatment Chair Jonathan N. Sembrano, MD Anil K. Sharma, MD Jamie Baisden, MD Jeffrey T. Summers, MD Shay Bess, MD Christopher K. Taleghani, MD Charles H. Cho, MD, MBA William L. Tontz, Jr., MD Michael J. DePalma, MD John F. Toton, MD This clinical guideline should not be construed as including all proper methods of care or excluding or other acceptable methods of care reason- ably directed to obtaining the same results. The ultimate judgment regarding any specific procedure or treatment is to be made by the physi- cian and patient in light of all circumstances presented by the patient and the needs and resources particular to the locality or institution. I NTRODUCT 2 Lumbar Disc Herniation with Radiculopathy | NASS Clinical Guidelines I ON Financial Statement This clinical guideline was developed and funded in its entirety by the North American Spine Society (NASS). All participating /G authors have disclosed potential conflicts of interest consistent with NASS’ disclosure policy. -
Treating Lower-Back Pain How Much Bed Rest Is Too Much?
® Treating lower-back pain How much bed rest is too much? ack pain is one of the most common reasons why people visit the doctor. The good news is that the pain often goes Baway on its own, and people usually recover in a week or two. Many people want to stay in bed when their back hurts. For many years, getting bed rest was the normal advice. However, newer data have shown that there is little to no role for bed rest in the treatment of low back pain. Here’s why: Staying in bed won’t help you get better faster. If you’re in terrible pain, bed rest may not actually ease the pain, but increase it. Research suggests that if you find comfortable positions and move around sometimes, you may not need bed rest at all. Research shows that: • More than 48 hours of bed rest may actually increase pain, by increasing the stiffness of the • The sooner you start physical therapy or return spine and the muscles. to activities such as walking, the faster you are • Many people recover just as quickly without any likely to recover. Longer bed rest can lead to bed rest. slower recovery. Longer bed rest can lead to slower recovery. What can I do for the pain? When you don’t move and bend, you lose Most people with lower-back pain should apply muscle strength and flexibility. With bed rest, heat or ice. Some people can get pain relief you lose about 1 percent of your muscle from an over-the-counter anti-inflammatory strength each day.