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Rho kinase inhibitor
The Therapeutic Effects of Rho-ROCK Inhibitors on CNS Disorders
Investigating Small Molecule Therapeutics to Improve Regeneration and Functional Recovery Following Peripheral Nerve Damage
Rhoa-ROCK Signaling As a Therapeutic Target in Traumatic Brain Injury
Ripasudil in a Model of Pigmentary Glaucoma
Dose-Dependent Effects of Netarsudil, a Rho-Kinase Inhibitor, on the Distal Outflow Tract
Antifibrotic Activity of a Rho-Kinase Inhibitor Restores Outflow Function and Intraocular Pressure Homeostasis
Role of Rho-Associated Protein Kinase Inhibition As Therapeutic Strategy for Parkinson's Disease: Dopaminergic Survival and En
Graftless Corneal Endothelial Replacement
FIG. 10B Dition
Rho-Kinase Inhibitors for the Treatment of Refractory Diabetic Macular Oedema
Additive Intraocular Pressure-Lowering Effects of The
(ROCK) Inhibitor, Y-27632, Inhibits the Dissociation-Induced Cell Death of Salivary Gland Stem Cells
Short-Term Effects of Y-27632, a Rho-Associated Protein Kinase
RKI-1447, a Rho-Kinase Inhibitor Causes Ocular Hypotension, Actin Stress Fiber Disruption and Increased Phagocytosis
Safety and Efficacy of Ripasudil in Japanese Patients with Glaucoma
The Rho Kinase Inhibitor Fasudil Inhibits Tumor Progression in Human and Rat Tumor Models
Rho Kinases and Cardiac Remodeling Toru Shimizu, MD, Phd; James K
The Rho-Associated Kinase Inhibitor Fasudil Can Replace Y-27632 for Use in Human Pluripotent Stem Cell Research
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Ocular Hypotension, Actin Stress Fiber Disruption and Phagocytosis Increase by RKI-1447, a Rho-Kinase Inhibitor
Ripasudil in a Model of Pigmentary Glaucoma