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- A Link Between Replicative Stress, Lamin Proteins, and Inflammation
- Progerin and Telomere Dysfunction Collaborate to Trigger Cellular Senescence in Normal Human Fibroblasts Kan Cao,1,2 Cecilia D
- Antisense-Based Progerin Downregulation in HGPS-Like Patients’ Cells
- Is Rapamycin an Effective Anti-Aging Drug?
- A New Genetic Drug for Hutchinsongilford Progeria Syndrome
- Tipping the Balance of Double-Strand Break Repair in Cells Modeling Hutchinson-Gilford Progeria Syndrome Andrew Robert Patrick University of South Carolina
- Temsirolimus Partially Rescues the Hutchinson-Gilford Progeria Cellular Phenotype
- Progerin-Expressing Endothelial Cells Are Unable to Adapt to Shear Stress
- 1-S2.0-S0022202X19315660-Main-2
- Metformin Decreases Progerin Expression and Alleviates Pathological Defects of Hutchinson–Gilford Progeria Syndrome Cells
- Hutchinson-Gilford Progeria Syndrome: a Premature Aging Disease Caused by LMNA Gene Mutations
- Mechanisms Controlling the Smooth Muscle Cell Death in Progeria Via
- Impact of Progerin Expression on Adipogenesis in Hutchinson—Gilford Progeria Skin-Derived Precursor Cells
- Gone with the Wnt/Notch: Stem Cells in Laminopathies, Progeria, and Aging
- Longwave Ultraviolet Light Induces the Aging-Associated Progerin In
- Naıve Adult Stem Cells from Patients with Hutchinson-Gilford Progeria
- Progerin (13A4D4): Sc-81611
- Hallmarks of Progeroid Syndromes: Lessons from Mice and Reprogrammed Cells Dido Carrero, Clara Soria-Valles and Carlos López-Otıń*
- Progerin Expression Disrupts Critical Adult Stem Cell Functions Involved in Tissue Repair
- Progerin Expression Induces Inflammation, Oxidative Stress and Senescence in Human Coronary Endothelial Cells
- Progeria Fact Sheet
- Human Ipsc-Based Modeling of Late-Onset Disease Via Progerin
- Expression of Progerin Does Not Result in an Increased Mutation Rate
- UVA-Induced Upregulation of Progerin Suppresses 53BP1-Mediated
- Progerin Reduces Lap2α-Telomere Association in Hutchinson-Gilford
- Treatment Strategies for Toxic Protein Progerin: Hutchinson- Gilford Progeria Syndrome Sadaf Shamim* Department of Biotechnology, University of Gujrat, Pakistan
- Gilford Progeria Syndrome
- Development of a CRISPR/Cas9-Based Therapy for Hutchinson–Gilford Progeria Syndrome
- Temsirolimus Partially Rescues the Hutchinson-Gilford Progeria Cellular Phenotype
- Rapamycin Activates Autophagy in Hutchinson-Gilford Progeria Syndrome Implications for Normal Aging and Age-Dependent Neurodegenerative Disorders
- 1 Commentary Adenine Base Editing to Treat Progeria Syndrome And
- Putting a Stop to Premature Ageing
- MG132 Induces Progerin Clearance and Improves Disease Phenotypes in Fibroblasts of Patients Affected with Hutchinson-Gilford Progeria-Like Syndromes