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Polyglutamine tract
Polyq Disease: Misfiring of a Developmental Cell Death Program?
Datasheet: VMA00301KT Product Details
Modifiers and Mechanisms of Multi-System Polyglutamine
Molecular Pathogenesis of Spinocerebellar Ataxia Type 6
Polyglutamine Tract Binding Protein-1 Is an Intrinsically Unstructured Protein
Evolving Notch Polyq Tracts Reveal Possible Solenoid Interference Elements
Ataxin the Brain
Highlights of Kennedy's Disease
Decreased Interactions Between Calmodulin and a Mutant Huntingtin Model Might Reduce the Cytotoxic Level of Intracellular Ca2+: a Molecular Dynamics Study
Polyglutamine Neurodegenerative Diseases and Regulation of Transcription: Assembling the Puzzle
Polyglutamine Tracts As Modulators of Transcriptional Activation from Yeast to Mammals
TATA Binding Protein Antibody (1TBP18)
The Case of Toxic Soluble Dimers of Truncated PQBP-1 Mutants in X-Linked Intellectual Disability
Mechanisms of the CAG Repeat Mutation in Spinocerebellar Ataxia Type 1 Cara Kraus-Perrotta1 and Sarita Lagalwar2*
Schizophrenia Genetics: Expansion of Knowledge?
Intrinsic Disorder in Proteins with Pathogenic Repeat Expansions
Detection of Polyglutamine Expansion in a New Acidic Protein
The Polyglutamine Expansion in Spinocerebellar Ataxia Type 6 Causes a  Subunit-Specific Enhanced Activation of P/Q-Type Calcium Channels in Xenopus Oocytes
Top View
TATA Binding Protein (TBP) (NM 001172085) Human Tagged ORF Clone Product Data
CMGS Best Practice Guidelines for the Molecular Genetic Testing of Huntington Disease
Androgen Insensitivity Syndrome: Clinical Features and Molecular Defects
Huntingtin's Spherical Solenoid Structure Enables Polyglutamine
Newmechanismfor Neurodegenerative Diseases
Datasheet: VMA00301 Product Details
The Polyq Expansion Modulates the Configuration and Phosphorylation of Huntingtin
NEUROGENETICS: NOW and the FUTURE This Paper
The Molecular Pathology of Huntington's Disease
Polyglutamine Domain Flexibility Mediates the Proximity Between
An Update on the Neurological Short Tandem Repeat Expansion Disorders and the Emergence of Long-Read Sequencing Diagnostics
Expanding Our Understanding Minireview of Polyglutamine Diseases Through Mouse Models
Emerging Roles of Exosomes in Huntington's Disease
Clinical and Molecular Advances in Autosomal Dominant Cerebellar Ataxias: from Genotype to Phenotype and Physiopathology
Instability of Expanded CAG/CAA Repeats in Spinocerebellar Ataxia Type 17
Androgen Insensitivity
Polyglutamine Diseases and Transport Problems Deadly Traffic Jams on Neuronal Highways
Polyglutamine Domain Flexibility Mediates the Proximity Between
Length of Uninterrupted CAG Repeats, Independent of Polyglutamine Size, Results in Increased Somatic Instability and Hastened Age of Onset in Huntington Disease
Huntingtin and Its Role in Mechanisms of RNA-Mediated Toxicity
Polyglutamine Ataxias: from Clinical and Molecular Features to Current
Purine Nucleotides Metabolism and Signaling in Huntington's
Androgen Receptor Transactivation Domain and Control of Spermatogenesis
Polyglutamine Domain Flexibility Mediates the Proximity Between