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Kataegis
Intratumor Heterogeneity: Novel Approaches for Resolving Genomic Architecture and Clonal Evolution Ravi G
Chromothripsis in Human Breast Cancer
Breast Cancer's Somatic Mutation Landscape Points to DNA Damage
Mutator Catalogues and That the Proportion of CG Muta- Types
Chromoanagenesis: Cataclysms Behind Complex Chromosomal Rearrangements Franck Pellestor1,2
Somatic Mutation Distributions in Cancer Genomes Vary with Three-Dimensional Chromatin Structure
APOBEC-Induced Mutations in Human Cancers Are Strongly Enriched on the Lagging DNA Strand During Replication
Learning Mutational Signatures and Their Multidimensional Genomic
Exploring the Mechanisms of Gastrointestinal Cancer Development Using Deep Sequencing Analysis
The Ins and Outs of Telomere Crisis in Cancer Patrick Von Morgen and John Maciejowski*
Article Reference
Mutational Mechanisms Shaping the Coding and Noncoding Genome of Germinal Center Derived B-Cell Lymphomas
APOBEC3B-Dependent Kataegis and TREX1-Driven Chromothripsis In
Stressed DNA Replication Generates Stressed DNA Hannah L
Strand-Biased Cytosine Deamination at the Replication Fork Causes Cytosine to Thymine Mutations in Escherichia Coli
APOBEC3B Expression in Breast Cancer Reflects Cellular Proliferation, While a Deletion Polymorphism Is Associated with Immune Activation
Chromosome Segregation Errors Generate a Diverse Spectrum of Simple and Complex Genomic Rearrangements
Comprehensive Analysis of Chromothripsis in 2,658 Human Cancers Using Whole-Genome Sequencing
Top View
Kataegis: an R Package for Identification and Visualization Of
The Origins and Consequences of Localized and Global Somatic Hypermutation
Trends in Molecular Medicine
Strand-Biased Cytosine Deamination at the Replication Fork Causes Cytosine to Thymine Mutations in Escherichia Coli
Signatures of Mutational Processes in Human Cancer
Chromothripsis and Beyond: Rapid Genome Evolution from Complex Chromosomal Rearrangements
Molecular Mechanism and Clinical Impact of APOBEC3B-Catalyzed Mutagenesis in Breast Cancer Reuben S Harris
DNA Deaminases Induce Break-Associated Mutation Showers
* K-Means and Cluster Models for Cancer Signatures
Endogenous APOBEC3B Overexpression Constitutively
The Deaminase APOBEC3B Triggers the Death of Cells Lacking Uracil DNA Glycosylase
An Investigation Into the Non-Coding Genomic Landscape And
DNA Replication Stress Mediates APOBEC3 Family Mutagenesis In
Activation of DNA Damage Tolerance Pathways May Improve Immunotherapy of Mesothelioma