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KCNQ channels
Regulation of Ion Channels by Muscarinic Receptors
Characterization of Cardiac Iks Channel Gating Using Voltage Clamp Fluorometry Jeremiah D. Osteen Submitted in Partial Fulfillme
The KCNQ5 Potassium Channel Mediates a Component of the Afterhyperpolarization Current in Mouse Hippocampus
The KCNQ4-Mediated M-Current Regulates the Circadian Rhythm in Mesopontine Cholinergic Neurons
Structure and Function Studies on the Herg Potassium Channel Including the Development and Characteriza Tion of Membrane Mimetics
KV7 Potassium Channels: a Focus on Human Intra-Pulmonary Arteries
Roles of Key Ion Channels and Transport Proteins in Age-Related Hearing Loss
Complex Mechanisms of KCNQ Channel Activation by State-Dependent Modulators
Pulmonary Vasoconstrictor Action of KCNQ Potassium Channel Blockers Shreena Joshi1,2, Prabhu Balan1 and Alison M Gurney*1,2
I N a U G U R a L D I S S E R T a T I O N
Pulmonary Vasoconstrictor Action of KCNQ Potassium Channel Blockers
KCNQ2 Localization in the Brainstem
Single-Channel Analysis of KCNQ K Channels Reveals the Mechanism of Augmentation by a Cysteine-Modifying Reagent
Dynamic Subunit Stoichiometry Confers a Progressive Continuum of Pharmacological Sensitivity by KCNQ Potassium Channels
Role of Kv7.2Kv7.3 and M 1 Muscarinic Receptors in The
KCNQ Potassium Channels in Sensory System and Neural Circuits
KCNQ1/KCNE1) Channels Regulation
Reconstitution of Muscarinic Modulation of the KCNQ2/KCNQ3 K؉ Channels That Underlie the Neuronal M Current
Top View
Centipedes Subdue Giant Prey by Blocking KCNQ Channels
Small Molecule Investigation of KCNQ Potassium Channels: a Dissertation
Neuronal Kcnq Potassium Channels: Physiology and Role in Disease
Activation of KCNQ4 As a Therapeutic Strategy to Treat Hearing Loss
Activation of Kv7 Potassium Channels Inhibits Intracellular Ca 2 Increases
Potassium Channels in C. Elegans* 1,2,§ 1 1 1 1
Functional Expression of KCNQ (Kv7) Channels in Guinea-Pig Bladder Smooth Muscle and Their Contribution to Spontaneous Activity
Activation of Expressed KCNQ Potassium Currents and Native Neuronal M-Type Potassium Currents by the Anti-Convulsant Drug Retigabine
Kv7.5 Potassium Channel Subunits Are the Primary Targets for PKA-Dependent Enhancement of Vascular Smooth Muscle Kv7 Currents