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Junying Yuan
MINI-REVIEW Programmed Cell Death Regulation
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The Caenorhabditis Elegans Cell-Death Protein CED-3 Is a C.Y.Ste!Ne P.Rotease with Substrate Specl[Icltles S M Lar to Those of the Human CPP32 Protease
Mechanisms of Cell Death: Molecular Insights and Therapeutic Perspectives
Caspases: a Decade of Death Research
Connections Between Various Trigger Factors and the RIP1/RIP3 Signaling Pathway Involved in Necroptosis
Interacting Protein Kinase 1 (RIPK1) As a Therapeutic Target
Mediated Activation of Caspase-1 and Caspase-3 in a Mouse Model of Amyotrophic Lateral Sclerosis
H. Robert Horvitz
Solution Structure of Apaf-1 CARD and Its Interaction with Caspase-9 CARD: a Structural Basis for Specific Adaptor͞caspase Interaction
At the Frontiers of Neuroscience 36 May - June 2002 by PATRICIA THOMAS Reprinted from Harvard Magazine
G-Protein-Coupled Receptors Regulate Autophagy by ZBTB16-Mediated Ubiquitination and Proteasomal Degradation of Atg14l
Honoring Contributions of Richard a Lockshin to the Field of Cell Death
A RIPK1-Regulated Inflammatory Microglial State in Amyotrophic Lateral Sclerosis Chemokines and the Immune Response to Cancer Ti
Regulation of a Distinct Activated RIPK1 Intermediate Bridging Complex I and Complex II in Tnfα-Mediated Apoptosis
Activation of Caspase-2 in Apoptosis
View Was Recently Challenged Given the Curring in Proximal Tubules, We Have Unexpectedly Detected Discovery of a Regulated Necrosis Pathway, Necroptosis
Caspase Cascades in Human Immunodeficiency Virus-Associated Neurodegeneration
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Conference Program
Profile of Junying Yuan PROFILE Jennifer Viegas, Science Writer
Profile of Junying Yuan Jennifer Viegas, Science Writer
Renxiao Wang, Ph
Necrostatin-1 Blocks Both RIPK1 and IDO: Consequences for the Study of Cell Death in Experimental Disease Models
Symposium on Aging June 25, 2018 the Paul F
Autophagy in Neurodegenerative Diseases: from Mechanism to Therapeutic Approach
SPATA2 Regulates the Activation of RIPK1 by Modulating Linear Ubiquitination
A Decade of Caspases
PELI1 Functions As a Dual Modulator of Necroptosis and Apoptosis by Regulating Ubiquitination of RIPK1 and Mrna Levels of C-FLIP
RIPK1 Promotes Inflammation and Beta-Amyloid Accumulation in Alzheimer’S Disease
VDR-RIPK1 Interaction and Its Implications in Cell Death and Cancer Intervention Waise Quarni University of South Florida,
[email protected]
RIPK1 Inhibitors
Death-Domain Dimerization-Mediated Activation of RIPK1 Controls Necroptosis and RIPK1-Dependent Apoptosis
Gentamicin-Induced Acute Kidney Injury in an Animal Model Involves Programmed Necrosis of the Collecting Duct