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Erythroferrone
The Role of Erythroferrone Hormone As Erythroid Regulator of Hepcidin and Iron Metabolism During Thalassemia and in Iron Deficiency Anemia- a Short Review
Ineffective Erythropoiesis: Associated Factors and Their Potential As Therapeutic Targets in Beta-Thalassaemia Major
Metabolism, Renal Insufficiency and Life Xpectancy
Why Does Iron Overload Associated with Insulin
The Erythroblastic Island As an Emerging Paradigm in the Anemia of Inflammation
Medium Cut-Off Dialyzer Improves Erythropoiesis Stimulating Agent
The Bridge Between Bone Marrow Adipocytes and Hematopoietic Cells Ziru Li and Ormond A
Iron Deficiency in Obesity and After Bariatric Surgery
Recent Trends in Treatment of Thalassemia T ⁎ Amal El-Beshlawy, Mona El-Ghamrawy
Codon-Optimized Fam132b Prevents Diet-Induced Obesity by Modulating Adrenergic Response and Insulin Action
Dysregulated Iron Metabolism in Polycythemia Vera: Etiology and Consequences
Hepcidin Is Regulated by Promoter-Associated Histone Acetylation and HDAC3
Iron Metabolism in Obesity and Metabolic Syndrome
Iron Metabolism and Iron Disorders Revisited in the Hepcidin
Hypoxia Pathway Proteins Are Master Regulators of Erythropoiesis
Original Article Erythroferrone Level in Β-Thalassemia Patients
Myokines and Heart Failure: Challenging Role in Adverse Cardiac Remodeling, Myopathy, and Clinical Outcomes
The Role of Hepcidin in Systemic Iron Regulation
Top View
The Interplay Between Drivers of Erythropoiesis and Iron Homeostasis in Rare Hereditary Anemias: Tipping the Balance
The Hepcidin Regulator Erythroferrone Is a New Member of the Erythropoiesis-Iron
Hepcidin: SNP-Like Polymorphisms Present in Iron Metabolism and Clinical Complications of Iron Accumulation and Deficiency
Influence of Mitochondrial and Systemic Iron Levels in Heart Failure Pathology
New Potential Players in Hepcidin Regulation
A New Insight Into the Treatment of Renal Anemia with HIF Stabilizer Satoru Kuriyama1,2*, Yukio Maruyama3 and Hirokazu Honda4
Biological Role of Myonectin and Its Correlation with Exercise
Evaluation of Erythroferrone, Hepcidin, and Iron Overload Status in Iraqi Transfusion
Intrinsic Erythroferrone IETM ELISA
Change One Thing Change Everything
Basis of Iron Regulation for Erythropoiesis
Erythroid Overproduction of Erythroferrone Causes Iron Overload and Developmental Abnormalities in Mice
C1q Complement/Tumor Necrosis Factor-Associated Proteins in Cardiovascular Disease and COVID-19
ELISA Kits Focusing on Emerging Research Fields: Biomarkers
Cystathionine Β-Synthase (CBS) Deficiency Suppresses
Transcriptional and Post-Transcriptional Regulation of Hepcidin and Iron Metabolism by Lipid Signaling in the Liver
Etters to the Ditor
Induction of Erythroferrone in Healthy Humans by Micro-Dose Recombinant
Dissertation
Characterization of Putative Erythroid Regulators of Hepcidin in Mouse Models of Anemia
Erythroferrone (Human) ELISA Kit the Only Specific & Sensitive Assay on the Market!
Erythroferrone Contributes to Hepcidin Repression in a Mouse Model of Malarial Anemia
Biochemical and Physiological Characterization of Secreted Proteins Regulating Whole-Body Energy Homeostasis
Serum Erythroferrone Levels Associate with Mortality and Cardiovascular Events in Hemodialysis and in CKD Patients: a Two Cohorts Study
Erythroid Overproduction of Erythroferrone Causes Iron Overload and Developmental Abnormalities in Mice
Clinical Study Anti-TNF-Mediated Modulation of Prohepcidin Improves Iron Availability in Inflammatory Bowel Disease, in an IL-6-Mediated Fashion
Relationship Between Intermittent Hypoxia and Type 2 Diabetes in Sleep Apnea Syndrome
The Relationship Between Systemic Iron Homeostasis and Erythropoiesis
Effect of Erythropoietin on the Expression of Murine Transferrin Receptor 2
Molecular Pathways Involved in the Development of Congenital Erythrocytosis
Next-Generation Nutritional Biomarkers to Guide Better Health Care Nestlé Nutrition Institute Workshop Series
Iron Metabolism and Related Ge
6Th Meeting of the International Bioiron Society September 6-10, 2015 Zhejiang University Hangzhou, China
Marathon Run-Induced Changes in the Erythropoietin-Erythroferrone-Hepcidin Axis Are Iron Dependent
Inherited Disorders of Iron Overload