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Double minute
Genome-Wide Purification of Extrachromosomal Circular DNA from Eukaryotic Cells
Double Minute Chromosomes in Glioblastoma Multiforme Are Revealed by Precise Reconstruction of Oncogenic Amplicons
Single Cell Multi-Omics Analysis of Chromothriptic Medulloblastoma Highlights Genomic and Transcriptomic Consequences of Genome Instability
A Acentric Fragment Linear One Telomere Radiation One Broken End Endonuclease Break-Inducing Mutagen
Hyper Telomere Recombination Accelerates Replicative Senescence and May Promote Premature Aging
Discordant Inheritance of Chromosomal and Extrachromosomal DNA Elements 2 Contributes to Dynamic Disease Evolution in Glioblastoma
Homogeneously Staining Regions Or Double Minute Chromosomes in Leukemia Abolfazl Movafagh Phd, Associate Professor
Gene Amplification As Double Minutes Or Homogeneously Staining Regions in Solid Tumors: Origin and Structure
Hydroxyurea Accelerates Loss of Extrachromosomally Amplified Genes from Tumor Cells1
Extrachromosomal Circular DNA: a New Potential Role in Cancer Progression Tianyi Wang1,2, Haijian Zhang3, Youlang Zhou3 and Jiahai Shi1,2*
DNA Replication-Dependent Intranuclear Relocation of Double Minute Chromatin
Mathematical Models of Gene Amplification with Applications To
Extraordinarily High Copy Numbers of the Plasmid THOMAS J
Circularization of Genes and Chromosome by CRISPR
Extrachromosomal Circular DNA-Based Amplification and Transmission of Herbicide Resistance in Crop Weed Amaranthus Palmeri
Ultrastructural Features of Minute Chromosomes Ina Methotrexate
Extrachromosomal Circular Dnas: Origin, Formation and Emerging Function in Cancer Man Wang, Xinzhe Chen, Fei Yu, Han Ding, Yuan Zhang, Kun Wang
Comprehensive Analysis of Chromothripsis in 2,658 Human Cancers Using Whole-Genome Sequencing
Top View
Current Understanding of Extrachromosomal Circular DNA In
Double Minute Chromosomes in Glioblastoma Multiforme Are Revealed by Precise Reconstruction of Oncogenic Amplicons
Novel Insights Into Extrachromosomal DNA: Redefining the Onco-Drivers Of
Chromothripsis and Beyond: Rapid Genome Evolution from Complex Chromosomal Rearrangements
Model for the Formation of Double Minutes from Prematurely
Double Minute Chromosomes Harboring MDM2 Amplification in A
Amplified Dihydrofolate Reductase Genes in Unstably Methotrexate
Double Minute Chromosomes Associated with Philadelphia Chromosome in Meyloid Leukemia
Mutations and Copy Number Alterations in IDH Wild-Type Glioblastomas Are Shaped by Different Oncogenic Mechanisms
Double Minutes Arise from Circular Extrachromosomal DNA Intermediates Which Integrate Into Chromosomal Sites in Human HL-60 Leukemia Cells
Extrachromosomal Oncogene Amplification Drives Tumor Evolution and Genetic Heterogeneity