Cereblon
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- Cereblon Modulator Iberdomide Induces Degradation of the Transcription Factors Ikaros and Aiolos
- IKZF1/3 and CRL4CRBN E3 Ubiquitin Ligase Mutations and Resistance to Immunomodulatory Drugs in Multiple Myeloma
- Mutations in CRBN and Other Cereblon Pathway Genes Are Infrequently Associated with Acquired Resistance to Immunomodulatory Drugs
- Cereblon Modulators: Low Molecular Weight Inducers of Protein
- Imids Induce FAM83F Degradation Via an Interaction with Ck1α to Attenuate Wnt Signalling
- Investigating the Mechanism and Species- Specificity of Thalidomide Derivatives
- Cereblon Attenuates DNA Damage-Induced Apoptosis by Regulating the Transcription-Independent Function Of
- Regulation of AMPK Activity by CRBN Is Independent of the Thalidomide-CRL4CRBN Protein Degradation Axis
- Thalidomide and Its Metabolite 5‐Hydroxythalidomide Induce Teratogenicity Via the Cereblon Neosubstrate PLZF
- December 19, 2019 Thalidomide Embryopathy
- CRL4-Cereblon Complex in Thalidomide Embryopathy
- Thalidomide Mimics Uridine Binding to an Aromatic Cage in Cereblon
- AG Polymorphism in the CRBN Gene Acts As a Biomarker of Response to Treatment with Lenalidomide in Low&Sol
- An Enhanced Genetic Model of Relapsed IGH-Translocated Multiple
- The CRBN, CUL4A and DDB1 Expression Predicts the Response to Immunomodulatory Drugs and Survival of Multiple Myeloma Patients
- Role of Aiolos and Ikaros in the Antitumor and Immunomodulatory Activity of Imids in Multiple Myeloma: Better to Lose Than to Find Them
- CRBN), a Primary Direct Target of Thalidomide, Using Ferrite Glycidyl Methacrylate (FG) Beads
- Cereblon: a Protein Crucial to the Multiple Functions of Immunomodulatory Drugs As Well As Cell Metabolism and Disease Generation