Cellular senescence
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- Transcription Factor AP-1 Activity Is Required for Initiation of DNA
- Cell Receptor Senescence Response by an Endosomal NK Activated Kinase 1 Control Signals for A
- Cell Senescence, Multiple Organelle Dysfunction and Atherosclerosis
- Evidence of the Cellular Senescence Stress Response in Mitotically Active Brain Cells—Implications for Cancer and Neurodegeneration
- Progerin and Telomere Dysfunction Collaborate to Trigger Cellular Senescence in Normal Human Fibroblasts Kan Cao,1,2 Cecilia D
- Role of P53 in the Regulation of Cellular Senescence
- The Consequences of Ageing, Progeroid Syndromes and Cellular Senescence on Mechanotransduction and the Nucleus
- The Senescence-Associated Secretory Phenotype (SASP) in the Challenging Future of Cancer Therapy and Age-Related Diseases
- Various Modes of Cell Death Induced by DNA Damage
- Impact of Progerin Expression on Adipogenesis in Hutchinson—Gilford Progeria Skin-Derived Precursor Cells
- An Overview of Treatment Strategies for Hutchinson-Gilford Progeria Syndrome
- Oncogenes and Senescence: Breaking Down in the Fast Lane
- The Impact of Senescence-Associated T Cells on Immunosenescence and Age-Related Disorders Yuji Fukushima1, Nagahiro Minato2 and Masakazu Hattori1*
- Hallmarks of Progeroid Syndromes: Lessons from Mice and Reprogrammed Cells Dido Carrero, Clara Soria-Valles and Carlos López-Otıń*
- Progerin Expression Induces Inflammation, Oxidative Stress and Senescence in Human Coronary Endothelial Cells
- The Histone Code of Senescence
- Cellular Senescence Controlled by P53 Is a Barrier to Environmental Carcinogenesis
- Four Faces of Cellular Senescence