Heat Related Illness

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Heat Related Illness With the return of warmer weather, Alberta Health Services EMS would like to remind citizens to stay safe in the heat and sun this summer. While children and the elderly can be more susceptible to the effects of heat, basic prevention measures should be taken by all to avoid a heat related illness during periods of hot and humid weather. Heat exhaustion First aid • Heat exhaustion can occur due to • First aid for all heat related excessive fluid loss during periods illness begins with removing or of prolonged sweating in a hot and / sheltering the patient from the or humid environment (indoors or hot environment. outdoors). • Remove excess or tight fitting • Patients may suffer headaches, clothing and allow them to rest in weakness, fatigue, nausea / a cool environment. vomiting, thirst, chills, and profuse • If the patient is conscious and sweating. alert, provide suitable fluids such as water, juice, or a sports drink. • The patient is usually cold and damp to the touch and the skin may • If you are concerned, seek appear pale or dusky gray. medical attention or call 9-1-1. Heat stroke Prevention • Stay well-hydrated by drinking • Heat stroke is a medical emergency plenty of water. that requires prompt treatment. It can • be fatal. Limit alcohol consumption as alcohol dehydrates you. • It occurs when the body can’t cool • Always wear a broad brimmed itself naturally (e.g. perspiration). The hat to keep the sun off your face body’s temperature will continue to and neck. rise to dangerous levels. • Apply waterproof sunscreen with • Due to severe dehydration and the an SPF of 50+, especially for inability to sweat the patient may children. The sun’s UV rays appear flushed and skin may be hot peak between 10am-4pm, even and dry to the touch. on cloudy days. .

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Metabolomic Profiling Identifies a Novel Mechanism for Heat Stroke‑Related Acute Kidney Injury
MOLECULAR MEDICINE REPORTS 23: 241, 2021 Metabolomic profiling identifies a novel mechanism for heat stroke‑related acute kidney injury LING XUE1*, WENLI GUO2*, LI LI2, SANTAO OU2, TINGTING ZHU2, LIANG CAI3, WENFEI DING2 and WEIHUA WU2 Departments of 1Urology and 2Nephrology, Sichuan Clinical Research Center for Nephropathy, The Affiliated Hospital of Southwest Medical University;3 Department of Nuclear Medicine, The Affiliated Hospital of Southwest Medical University, Luzhou, Sichuan 646000, P.R. China Received April 13, 2020; Accepted August 20, 2020 DOI: 10.3892/mmr.2021.11880 Abstract. Heat stroke can induce a systemic inflammatory factor mitochondria‑associated 2 (Aifm2), high‑mobility group response, which may lead to multi‑organ dysfunction including box 1 (HMGB1) and receptor for advanced glycosylation end acute kidney injury (AKI) and electrolyte disturbances. To products (RAGE). Liquid chromatography‑mass spectrometry investigate the pathogenesis of heat stroke (HS)‑related AKI, analysis was conducted to find differential metabolites and a mouse model of HS was induced by increasing the animal's signaling pathways. The HS mouse model was built success‑ core temperature to 41˚C. Blood samples obtained from the fully, with significantly increased creatinine levels detected tail vein were used to measure plasma glucose and creati‑ in the serum of HS mice compared with controls, whereas nine levels. Micro‑positron emission tomography‑computed micro‑PET/CT revealed active metabolism in the whole tomography (micro‑PET/CT), H&E staining and trans‑ body of HS mice. H&E and TUNEL staining revealed that mission electron microscopy were conducted to examine the kidneys of HS mice exhibited signs of hemorrhage and metabolic and morphological changes in the mouse kidneys.
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Heat Illness Flyer
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