An Osteopathic Approach to Raynaud's Phenomenon
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The Male Reproductive System
Management of Men’s Reproductive 3 Health Problems Men’s Reproductive Health Curriculum Management of Men’s Reproductive 3 Health Problems © 2003 EngenderHealth. All rights reserved. 440 Ninth Avenue New York, NY 10001 U.S.A. Telephone: 212-561-8000 Fax: 212-561-8067 e-mail: [email protected] www.engenderhealth.org This publication was made possible, in part, through support provided by the Office of Population, U.S. Agency for International Development (USAID), under the terms of cooperative agreement HRN-A-00-98-00042-00. The opinions expressed herein are those of the publisher and do not necessarily reflect the views of USAID. Cover design: Virginia Taddoni ISBN 1-885063-45-8 Printed in the United States of America. Printed on recycled paper. Library of Congress Cataloging-in-Publication Data Men’s reproductive health curriculum : management of men’s reproductive health problems. p. ; cm. Companion v. to: Introduction to men’s reproductive health services, and: Counseling and communicating with men. Includes bibliographical references. ISBN 1-885063-45-8 1. Andrology. 2. Human reproduction. 3. Generative organs, Male--Diseases--Treatment. I. EngenderHealth (Firm) II. Counseling and communicating with men. III. Title: Introduction to men’s reproductive health services. [DNLM: 1. Genital Diseases, Male. 2. Physical Examination--methods. 3. Reproductive Health Services. WJ 700 M5483 2003] QP253.M465 2003 616.6’5--dc22 2003063056 Contents Acknowledgments v Introduction vii 1 Disorders of the Male Reproductive System 1.1 The Male -
Neonatal Dermatology Review
NEONATAL Advanced Desert DERMATOLOGY Dermatology Jennifer Peterson Kevin Svancara Jonathan Bellew DISCLOSURES No relevant financial relationships to disclose Off-label use of acitretin in ichthyoses will be discussed PHYSIOLOGIC Vernix caseosa . Creamy biofilm . Present at birth . Opsonizing, antibacterial, antifungal, antiparasitic activity Cutis marmorata . Reticular, blanchable vascular mottling on extremities > trunk/face . Response to cold . Disappears on re-warming . Associations (if persistent) . Down syndrome . Trisomy 18 . Cornelia de Lange syndrome PHYSIOLOGIC Milia . Hard palate – Bohn’s nodules . Oral mucosa – Epstein pearls . Associations . Bazex-Dupre-Christol syndrome (XLD) . BCCs, follicular atrophoderma, hypohidrosis, hypotrichosis . Rombo syndrome . BCCs, vermiculate atrophoderma, trichoepitheliomas . Oro-facial-digital syndrome (type 1, XLD) . Basal cell nevus (Gorlin) syndrome . Brooke-Spiegler syndrome . Pachyonychia congenita type II (Jackson-Lawler) . Atrichia with papular lesions . Down syndrome . Secondary . Porphyria cutanea tarda . Epidermolysis bullosa TRANSIENT, NON-INFECTIOUS Transient neonatal pustular melanosis . Birth . Pustules hyperpigmented macules with collarette of scale . Resolve within 4 weeks . Neutrophils Erythema toxicum neonatorum . Full term . 24-48 hours . Erythematous macules, papules, pustules, wheals . Eosinophils Neonatal acne (neonatal cephalic pustulosis) . First 30 days . Malassezia globosa & sympoidalis overgrowth TRANSIENT, NON-INFECTIOUS Miliaria . First weeks . Eccrine -
5 the Da Vinci Code Dan Brown
The Da Vinci Code By: Dan Brown ISBN: 0767905342 See detail of this book on Amazon.com Book served by AMAZON NOIR (www.amazon-noir.com) project by: PAOLO CIRIO paolocirio.net UBERMORGEN.COM ubermorgen.com ALESSANDRO LUDOVICO neural.it Page 1 CONTENTS Preface to the Paperback Edition vii Introduction xi PART I THE GREAT WAVES OF AMERICAN WEALTH ONE The Eighteenth and Nineteenth Centuries: From Privateersmen to Robber Barons TWO Serious Money: The Three Twentieth-Century Wealth Explosions THREE Millennial Plutographics: American Fortunes 3 47 and Misfortunes at the Turn of the Century zoART II THE ORIGINS, EVOLUTIONS, AND ENGINES OF WEALTH: Government, Global Leadership, and Technology FOUR The World Is Our Oyster: The Transformation of Leading World Economic Powers 171 FIVE Friends in High Places: Government, Political Influence, and Wealth 201 six Technology and the Uncertain Foundations of Anglo-American Wealth 249 0 ix Page 2 Page 3 CHAPTER ONE THE EIGHTEENTH AND NINETEENTH CENTURIES: FROM PRIVATEERSMEN TO ROBBER BARONS The people who own the country ought to govern it. John Jay, first chief justice of the United States, 1787 Many of our rich men have not been content with equal protection and equal benefits , but have besought us to make them richer by act of Congress. -Andrew Jackson, veto of Second Bank charter extension, 1832 Corruption dominates the ballot-box, the Legislatures, the Congress and touches even the ermine of the bench. The fruits of the toil of millions are boldly stolen to build up colossal fortunes for a few, unprecedented in the history of mankind; and the possessors of these, in turn, despise the Republic and endanger liberty. -
Diagnosis and Treatment of Multiple System Atrophy: an Update
ReviewSection Article Diagnosis and Treatment of Multiple System Atrophy: an Update Abstract the common parkinsonian variant (MSA-P) from PD. In his review provides an update on the diagnosis a clinicopathologic study1, primary neurologists (who Tand therapy of multiple system atrophy (MSA), a followed up the patients clinically) identified only 25% of sporadic neurodegenerative disorder characterised MSA patients at the first visit (42 months after disease clinically by any combination of parkinsonian, auto- onset) and even at their last neurological follow-up (74 nomic, cerebellar or pyramidal symptoms and signs months after disease onset), half of the patients were still and pathologically by cell loss, gliosis and glial cyto- misdiagnosed with the correct diagnosis in the other half plasmic inclusions in several brain and spinal cord being established on average 4 years after disease onset. structures. The term MSA was introduced in 1969 Mean rater sensitivity for movement disorder specialists although prior to this cases of MSA were reported was higher but still suboptimal at the first (56%) and last Gregor Wenning obtained an MD at the under the rubrics of striatonigral degeneration, olivo- (69%) visit. In 1998 an International Consensus University of Münster pontocerebellar atrophy, Shy-Drager syndrome and Conference promoted by the American Academy of (Germany) in 1991 and idiopathic orthostatic hypotension. In the late Neurology was convened to develop new and optimised a PhD at the University nineties, |-synuclein immunostaining was recognised criteria for a clinical diagnosis of MSA2, which are now of London in 1996. He received his neurology as the most sensitive marker of inclusion pathology in widely used by neurologists. -
Hypertrichosis Terminalis
97297 Med Genet 1996;33:972-974 An autosomal dominant syndrome of acromegaloid facial appearance and generalised J Med Genet: first published as 10.1136/jmg.33.11.972 on 1 November 1996. Downloaded from Department of hypertrichosis terminalis Medical Genetics, Belfast City Hospital, Lisburn Road, Belfast BT9 7AB, Northern Ireland Alan D Irvine, Olivia M Dolan, David R Hadden, Fiona J Stewart, E Ann Bingham, A D Irvine Norman C Nevin F J Stewart N C Nevin Department of Dermatology, Royal Abstract A family with four affected subjects in three Group of Hospitals, Belfast BT12 6BA, We report a family in which a phenotype generations is reported. Affected subjects show Northern Ireland of acromegaloid facial appearance (AFA) a characteristic acromegaloid facial appearance 0 M Dolan and generalised hypertrichosis terminalis (AFA) and generalised hypertrichosis ter- E A Bingham segregates through three generations. minalis. The syndrome is inherited as an auto- Sir George E Clark Congenital hypertrichosis terminalis and somal dominant trait and appears to be fully Metabolic Unit, Royal AFA have been previously reported as in- penetrant. Acromegaloid facial appearance and Group of Hospitals, dependent autosomal dominant traits. hypertrichosis terminalis have been described Belfast BT12 6BA, Northern Ireland This is the first report to delineate an auto- in association with thickened oral mucosa and D R Hadden somal dominant transmission of the com- gingival hyperplasia.'"2 Affected patients in our Correspondence to: bined phenotype. family did not have intraoral lesions. The Dr Irvine. (JtMed Genet 1996;33:972-974) syndrome in our family appears to be discrete Received 14 May 1996 from the acromegaloid facial appearance syn- Revised version accepted for Key words: terminal hypertrichosis; acromegaloid drome,3 Gorlin-Chaudhry-Moss syndrome,4 publication 28 June 1996 facies; autosomal dominant. -
Vicks Vaporub Shows Its Speed
Vicks VapoRub Professor Ron Eccles shows its speed Dr David Hull VICKS VAPORUB SHOWS ITS SPEED Vicks VapoRub (VVR) has been commercially available for over 100 years, as a remedy for congested nasal passages. A study led by Professor Ron Eccles and Dr David Hull has now demonstrated the speed of its effect in common cold sufferers. To begin, what attracted you to this area Dr David Hull: We continue to explore the in the treatment of upper respiratory tract of research? attributes of all products, young and old. infections such as the common cold and Diseases in collaboration with researchers As ideas, and sometimes new methods flu. Since science came to understand the MEASURING HOW RAPIDLY at the Common Cold and Nasal Research Professor Ron Eccles: During my modular emerge, we strive to bring this to bear by receptor biochemistry of these substances, Centre at Cardiff University, focussed on zoology undergraduate course at Liverpool gathering an improved understanding of the exploration of their effects has been the speed of action of VVR, compared to University, I chose to do a module in their effects. We knew that VapoRub was more easily explained. Also as we now VICKS VAPORUB EXERTS ITS a petrolatum control using a group of 50 pharmacology. I found the investigation fast-acting (just open the jar and you can feel have a receptor-based pharmacology for common-cold sufferers. Cold and flu sufferers of how drugs work in humans an amazing an effect), but we had not tried to quantify aromatic oils to work with, we can better PHARMACOLOGY report that one of the main desires for any and exciting area of study and decided that before and we did not know if the feeling plan experiments such as this one in the medication is a feeling of rapid relief from to switch my undergraduate studies to extended from just cooling to an actual expectation of an interesting and valuable Researchers at the Common Cold and Nasal Research Centre at Cardiff nasal congestion, as this symptom interferes pharmacology. -
Red, Weeping and Oozing P.51 6
DERM CASE Test your knowledge with multiple-choice cases This month–9 cases: 1. Red, Weeping and Oozing p.51 6. A Chronic Condition p.56 2. A Rough Forehead p.52 7. “Why am I losing hair?” p.57 3. A Flat Papule p.53 8. Bothersome Bites p.58 4. Itchy Arms p.54 9. Ring-like Rashes p.59 5. A Patchy Neck p.55 on © buti t ri , h ist oad rig D wnl Case 1 y al n do p ci ca use o er sers nal C m d u rso m rise r pe o utho y fo C d. A cop or bite ngle Red, Weleepirnohig a sind Oozing a se p rint r S ed u nd p o oris w a t f uth , vie o Una lay AN12-year-old boy dpriesspents with a generalized, itchy rash over his body. The rash has been present for two years. Initially, the lesions were red, weeping and oozing. In the past year, the lesions became thickened, dry and scaly. What is your diagnosis? a. Psoriasis b. Pityriasis rosea c. Seborrheic dermatitis d. Atopic dermatitis (eczema) Answer Atopic dermatitis (eczema) (answer d) is a chroni - cally relapsing dermatosis characterized by pruritus, later by a widespread, symmetrical eruption in erythema, vesiculation, papulation, oozing, crust - which the long axes of the rash extend along skin ing, scaling and, in chronic cases, lichenification. tension lines and give rise to a “Christmas tree” Associated findings can include xerosis, hyperlin - appearance. Seborrheic dermatitis is characterized earity of the palms, double skin creases under the by a greasy, scaly, non-itchy, erythematous rash, lower eyelids (Dennie-Morgan folds), keratosis which might be patchy and focal and might spread pilaris and pityriasis alba. -
Digital Clubbing
Review Article Digital clubbing Malay Sarkar, D. M. Mahesh1, Irappa Madabhavi2 Department of Pulmonary Medicine, Indira Gandhi Medical College, Shimla, 1Department of Endocrinology, Jawaharlal Institute of Postgraduate Medical Education and Research, Pondicherry, 2Post Graduate Student (Medicine), Indira Gandhi Medical College, Shimla, India ABSTRACT Digital clubbing is an ancient and important clinical signs in medicine. Although clubbed fingers are mostly asymptomatic, it often predicts the presence of some dreaded underlying diseases. Its exact pathogenesis is not known, but platelet‑derived growth factor and vascular endothelial growth factor are recently incriminated in its causation. The association of digital clubbing with various disease processes and its clinical implications are discussed in this review. KEY WORDS: Cancer, digital clubbing, hypertrophic osteoarthropathy, megakaryocytes, vascular endothelial growth factor Address for correspondence: Dr. Malay Sarkar, Department of Pulmonary Medicine, Indira Gandhi Medical College, Shimla ‑ 171 001, India. E‑mail: [email protected] INTRODUCTION long bones and occasional painful joint enlargement. It was initially known as hypertrophic pulmonary Digital clubbing is characterized by a focal bulbous osteoarthropathy (HPOA) based on the fact that majority enlargement of the terminal segments of the fingers of cases of HOA are due to malignant thoracic tumors. and/or toes due to proliferation of connective tissue The term “pulmonary” was later abandoned as it was between nail matrix and the distal phalanx. It results in realized that the skeletal syndrome may occur in several increase in both anteroposterior and lateral diameter of non‑pulmonary diseases and even may occur without the nails.[1] Clubbed fingers are also known as watch‑glass any underlying illness. -
Primary Follicular Mucinosis
International Journal of Scientific & Engineering Research Volume 8, Issue 6, June-2017 1303 ISSN 2229-5518 Case Report Primary Follicular Mucinosis: A Case Report From Saudi Arabia With Successful Treatment And Literature Review SalaimanAlsaiari1 AwadhAlAmri2 AmerAlmuqati Ibrahim Allihibi ABSTRACT: Background:Follicular mucinosis is an uncommon inflammatory disorder that characteristically presents as clearly defined, erythematous plaques or papules, with follicular projections, superficial scaling, and alopecia in terminal hair bearing areas, characterized histologically by mucin accumulation in pilosebaceous units (follicular epithelium and sebaceous glands) . The condition is generally divided into primary (idiopathic) and secondary forms in association with several conditions including benign and malignant diseases. There are many local and systemic treatments. Main observations: We report a case of 15 years old male with primary follicular mucinosis treated effectively by intralesional steroid injections. Conclusions: This is a new case of Primary follicular mucinosis from Saudi Arabia was treated successfully with intralesional corticosteroids without relapse. KEYWORDS:follicular mucinosis, intralesional corticosteroids, treatment. —————————— —————————— INTRODUCTIONIJSER Follicular mucinosis is a rare condition, of unknown cause, which affects all races, ages and both sexes.1,2It is defined as the accumulation of mucin in the follicular epithelium and sebaceous glands.3,5 It was initially described in 1957 by Pinkus who named it -
Dermatology Volume 58 Issue 2 March-April 2013 Indian Journal Of
Indian Journal of ISSN: 0019-5154 Dermatology Volume 58 Issue 2 March-April 2013 Indian Journal of Highlights of the issue Dermatology • Update on cutaneous calciphylaxis • Macrophage migration inhibitory factor in • V Dermatology olume • Fixed duration therapy in leprosy 58 • Issue • Environmental dermatoses in Ladakh • Demodex folliculorum as a risk factor in 2 • Diagnosing rosacea March-April • Annular lesions in Dermatology 2013 • Pages Clinical and photomicrograph of Mycosis fungoides, PET-CT for staging and response assessment IJD® Symposium: Integrative Dermatology 87-**** Guest Editor: S R Narahari IJD® www.e-ijd.org E‑Case Report Angiolymphoid Hyperplasia with Eosinophilia with Follicular Mucinosis Rameshwar Gutte, Bhavana Doshi, Uday Khopkar From the Department of Dermatology, Seth G. S. Medical College and King Edward Memorial Hospital, Mumbai, India Abstract Follicular mucinosis occurring along with angiolymphoid hyperplasia with eosinophils (ALHE) has been described in a 49-year-old male. The patient presented with pruritic hyperpigmented papules and nodules on the vertex and right parietal scalp. There was no any other complaint. Histopathological examination from one of the papule showed prominent blood vessels in the dermis lined by plump histiocytoid endothelial cells that were surrounded by a dense lymphoid infiltrate with numerous eosinophils; these findings are typical of angiolymphoid hyperplasia with eosinophilia. Features of follicular mucinosis were observed in the same section with 3 hyperplastic follicular infundibula containing pools of mucin in the infundibular epithelium. The concurrent occurrence of these 2 distinct histopathological patterns in the same biopsy specimen has been reported rarely. Key Words: Angiolymphoid hyperplasia, eosinophilia, follicular mucinosis, scalp What was known? perivascular area and other parts of the dermis. -
Prevalence of Developmental Oral Mucosal Lesions Among a Sample of Denture Wearing Patients Attending College of Dentistry Clinics in Aljouf University
European Scientific Journal August 2016 edition vol.12, No.24 ISSN: 1857 – 7881 (Print) e - ISSN 1857- 7431 Prevalence Of Developmental Oral Mucosal Lesions Among A Sample Of Denture Wearing Patients Attending College Of Dentistry Clinics In Aljouf University Abdalwhab M.A .Zwiri Assistant professor of oral medicine, Aljouf University, Sakaka, Aljouf , Saudi Arabia Santosh Patil Assistant professor of Radiology, Aljouf University, Sakaka, Aljouf , Saudi Arabia Fadi AL- Omair Intern dentist, Aljouf University, Sakaka, Aljouf , Saudi Arabia Mohammed Assayed Mousa Lecturer of prosthodontics, Aljouf University, Sakaka, Aljouf , Saudi Arabia Ibrahim Ali Ahmad Department of Dentistry, AlWakra Hospital, Hamad Medical Corporation, AlWakra, Qatar doi: 10.19044/esj.2016.v12n24p352 URL:http://dx.doi.org/10.19044/esj.2016.v12n24p352 Abstract Introduction: developmental oral lesions represent a group of normal lesions that can be found at birth or evident in later life. These lesions include fissured and geographic tongue, Fordyce’s granules and leukoedema. Study aims: to investigate the prevalence of some developmental oral mucosal lesions among dental patients wearing dentures who were attending college of dentistry clinics in Aljouf University, and specialized dental center of ministry of health. Methods and subjects: a retrospective design was conducted to collect data from 344 wearing denture dental patients who were attending college of dentistry clinics in Aljouf University, and specialized dental center of ministry of health. A working excel sheet was created for patients and included data related to personal information such as age and gender; and oral developmental lesions. The software SPSS version 20 was used to analyze data. Statistical tests including frequency, percentages, and One way Anova were used to describe data. -
Alopecia, Particularly: Alopecia Areata Androgenetic Alopecia Telogen Effluvium Anagen Effluvium
432 Teams Dermatology Hair disorders Color Code: Original, Team’s note, Important, Doctor’s note, Not important, Old teamwork Done by: Shaikha Aldossari Reviewer: Lama AlTawil 8 Team Leader: Basil Al Suwaine&Lama Al Tawil 432 Dermatology Team Lecture 8: Hair Disorders Objectives 1- Normal anatomy of hair follicle and hair cycle. 2- Causes, features and management of non scarring alopecia, particularly: Alopecia areata Androgenetic alopecia Telogen effluvium Anagen effluvium 3- Causes and features of scarring alopecia. 4- Causes and features of Excessive hair growth. hair disorder Excessive hair Alopecia growth non scarring Hirsutism Hypertrichosis scarring Anagen Telogen Androgenetic Alopecia effluvium effluvium Alopecia Areata P a g e | 1 432 Dermatology Team Lecture 8: Hair Disorders Anatomy of hair follicle: The Arrector piliResponsible for piloerection (goose bumps ) that happens when one is cold (produces energy and therefor warmth) . hair follicle becomes vertical instead of oblique Cuticle is the last layer here . what we can see outside . it has 7 layers of keratinocytes How many hairs in the body? 5 millions hairs in the body, 100,000 in the scalp. Growth rate: 0.3mm/day for scalp hair i.e.1cm/month Hair follicle bulge: -Very important part since it has stem cells .its the inertion of the arrector pili Hair follicle on vertical section: -So any pathological process affecting any part other Initially the shaft and the follicle are one than this, hair would still be able to regrow. organ then when you reach 1/3 the follicle -If we want to destroy a hair follicle, we’d target the bulge.