GI-Procedure-Order-Updated.Pdf

Total Page:16

File Type:pdf, Size:1020Kb

GI-Procedure-Order-Updated.Pdf UF Health GI / Endoscopy Scheduling phone: 352.265.3636 GI Procedure Order (External Provider) Scheduling fax: 352.627.4074 Please complete patient information below, or attach patient demographic information prior to fasting. Patient’s Name – Last: __________________________________ First: _____________________________ MI: _______ DOB: ______/______/_________ Home #: ____________________________ Work #: _________________________ Cell #: _________________________ Referring Provider (Print Name): ________________________________________ Office Phone #: _________________ Priority Level: h STAT (Within 5 days) h Urgent (1-2 weeks) h Routine COLONOSCOPY PATIENT SAFETY h Screening: 50 years or older average age risk For patient safety reasons, please include the following • No personal/family history of polyps or cancer information on your patient: • Should be 10 years from last colo, or 4 years from last flex h List of medications sig unless mitigating factors per Medicare guidelines h Surgical and medical history h Diagnostic (state indication below) EGD (UPPER ENDOSCOPY) SPECIFIC INDICATIONS h Upper abdominal distress/dyspepsia h Personal history of polyps h 50 year old h Failure after test/treatment Type: ____________________________________________ h Dysphagia / Odynophagia (circle one) Colonoscopy date: __________________________________ h Gastrointestinal bleeding / iron deficiency with h Personal history of colorectal cancer suspected upper GI source Last colonoscopy date: ______________________________ h Barrett’s esophagus surveillance h Personal history of inflammatory bowel disease Date of last EGD: ___________________________________ h Colon cancer surveillance h Diagnosis h Other, please be specific: _____________________________ h Family history of colorectal cancer or polyps Relation: ________________________ Age at dx: ________ HEPATOLOGY / LIVER RELATED Relation: ________________________ Age at dx: ________ h Paracentesis* h Fecal occult blood positive h Liver biopsy* h Iron deficiency: If colonoscopy does not reveal bleeding source *Request will be reviewed by Hepatology Attending (melena or IDA), do you want an EGD done at the same time? h Yes h No ADVANCED PROCEDURE h Hematochezia (rectal bleeding) (To be reviewed by an Advanced Endoscopist prior to scheduling) h Evaluation of abnormality on barium enema or other pertinent EUS*: Upper EUS h Rectal EUS h test, describe: _____________________________________ h ERCP* h ERCP with Cholangioscopy* (SpyGlass) ________________________________________________ h Per-oral endoscopic myotomy (POEM)* ________________________________________________ h Endoscopic submucosal dissection (ESD)* h Other, describe: ____________________________________ h Small bowel Enteroscopy*: Push h (does not require review) ________________________________________________ Spirus antegrade h Spirus retrograde h ________________________________________________ h Capsule* h Capsule with EGD placement* ________________________________________________ *Please include all notes pertaining to diagnosis along h The referring physician approves the appropriate bowel cleansing with radiology reports and disks. solution and preparation per GI protocol if required for procedure h Indication: ________________________________________ Ordering physician’s signature (required) MD# Date Time Patient Name: Patient Identification #: *RX0001* RX0001 Revised 4/19/17 GI Procedure Order (External Provider) PS120633.
Recommended publications
  • Laparoscopy As a Diagnostic Tool in Ascites of Unknown Origin: a Retrospective Study Conducted at Kasturba Hospital, Manipal
    Research Article Open Access J Surg Volume 8 Issue 3 - March 2018 Copyright © All rights are reserved by Chetan R Kulkarni DOI: 10.19080/OAJS.2018.08.555740 Laparoscopy as a Diagnostic Tool in Ascites of Unknown Origin: A Retrospective Study Conducted at Kasturba Hospital, Manipal Chetan R Kulkarni1*, Badareesh Laxminarayan2 and Annappa Kudva3 1Assistant Professor, Department of Surgery, Kasturba Hospital, Manipal 2Associate Professor, Department of Surgery, Kasturba Hospital, Manipal 3Professor, Department of Surgery, Kasturba Hospital, Manipal Received: February 16, 2018; Published: March 01, 2018 *Corresponding author: Chetan R Kulkarni, Assistant Professor, Department of Surgery, Kasturba Hospital, Manipal, India. Tel: 9535974395;Email: Abstract Background: Laparoscopy as a minimally invasive technique has long played an important role in the evaluation of ascites. Methods: A retrospective analysis was carried out on the record of 80patients who underwent laparoscopy after appropriate investigations had failed to reveal the cause of ascites. Results: Tuberculous peritonitis was reported in 46(57%), malignancies in 18(25%), cirrhosis in 4(5%) and peritonitis of unknown etiology in 8(10%)Conclusion: of patients. Two (2.5%) patients had complications, an Ileal perforation and in other Incisional hernia. Keywords: Ascites;Laparoscopy Diagnostic was Laparoscopy able to diagnose the pathology in 72 (90%) patients with ascites of unknown origin. Introduction Laparoscopy, as a minimally invasive technique has developed rapidly in recent years. Endoscopic examination of conventional laboratory examinations (including ascitic fluid cell count, albumin level, total protein level, Gram stain, culture who termed it as “Celioscopy” [1,2]. The term ‘ascites’ refers to and cytology ) as well as after imaging investigations (including peritoneal cavity was first attempted in 1901 by George Kelling Materialultrasound andand CT Methods scan).
    [Show full text]
  • Recurrent Pneumothorax Following Abdominal Paracentesis
    Postgrad Med J (1990) 66, 319 - 320 © The Fellowship of Postgraduate Medicine, 1990 Postgrad Med J: first published as 10.1136/pgmj.66.774.319 on 1 April 1990. Downloaded from Recurrent pneumothorax following abdominal paracentesis P.J. Stafford Department ofMedicine, Newham General Hospital, Glen Road, Plaistow, London E13, UK. Summary: A 62 year old man presented with abdominal ascites, without pleural effusion, due to peritoneal mesothelioma. He had chronic obstructive airways disease and a past history of right upper lobectomy for tuberculosis. On two occasions abdominal paracentesis was followed within 72 hours by pneumothorax. This previously unreported complication of abdominal paracentesis may be due to increased diaphragmatic excursion following the procedure and should be considered in patients with preexisting lung disease. Introduction Abdominal paracentesis is a widely used palliative right iliac fossa. Approximately 4 litres of turbid therapy for malignant ascites and is generally fluid was drained over 72 hours. The protein accepted as a procedure with few adverse effects: concentration was 46 g/l and microbiology and pneumothorax is not a recognized complication. I cytology were not diagnostic. Laparoscopy re- report a patient with recurrent pneumothorax vealed matted loops of bowel with widespread apparently precipitated by abdominal paracen- peritoneal seedlings. Histological examination of copyright. tesis. these lesions showed malignant mesothelioma of the epithelioid type. The patient suffered a left pneumothorax within Case report 48 hours ofparacentesis (before laparoscopy). This was treated with intercostal underwater drainage A 62 year old civil servant presented with a 6-week but recurred on two attempts to remove the drain, history ofworsening abdominal distension.
    [Show full text]
  • Intestinal and Multiple Organ Transplantation 1679
    1678 TRANSPLANT ATION euglycemia and survive longer than 200 islets in allogeneic and xenogeneic diabetic hosts. Transplant Proc 1993; 25:953-954. 67. Gotoh M, Maki T, Satomi S, et al: Immunological characteristics of purified islet grafts. Transplantation 1986; 42j387. 68. Klima G, Konigsrainer A, Schmid T, et al: Is the pancreas reo jected independently of the kidney after combined pancreatic­ renal transplantation? Transplant Proc 1988; 20:665. 69. Prowse 5J, Bellgrau D, Lafferty KJ: Islet allografts are destroyed by disease recurrence in the spontaneously diabetic BB rat. Di­ abetes 1986; 35:110. 70. Markmann JF, Posselt AM, Bassiri H, et al: Major-histocompat­ ibility-complex restricted and nonrestricted autoil1'\mune effec­ tor mechanisms in BB rats. Transplantation 1991; 52:662-667. 71. Navarro X, Kennedy WR, Loewenson RB, et al: Influence of pancreas transplantation on cardiorespiratory reflexes, nerve conduction, and mortality in diabetes mellitus. Diabetes 1990; 39:802. 72. Weber q, Silva FG, Hardy MA, et al: Effect of islet transplan­ tation on renal function and morphology of short- and long­ term diabetic rats. Transplant Proc 1979; 11:549. 73. Gotzche 0, Gunderson HJ, Osterby R: Irreversibility of glomer­ ular basement membrane accumulation despite reversibility of renal hypertrophy with islet transplantation in early diabetes. Diabetes 1981; 30:481. 74. Fung H, Alessini M, Abu-Elmagd K, et al: Adverse effects asso­ ciated with the use ofFK 506. Transplant Proc 1991; 23:3105. 75. Tzakis AG: Personal communication, 1991. I CHAPTER 185 Figure 185-1. Cluster allograft (shaded portion), including the liver, pancreas, and duodenal segment of small intestine. (From Starzl TE, Todo S.
    [Show full text]
  • Septicaemia After Colonoscopy in Patients With
    450 Gut, 1991,32,450-451 Septicaemia after colonoscopy in patients with cirrhosis Gut: first published as 10.1136/gut.32.4.450 on 1 April 1991. Downloaded from j R Thornton, M S Losowsky Abstract PATIENT 2 Two patients with ulcerative colitis and In 1987, a 34 year old man underwent routine chronic active hepatitis with cirrhosis, who colonoscopy because ofhis ulcerative colitis of 12 developed Gram negative septicaemia after years' duration. Twenty three years earlier a colonoscopy are described. These and two liver biopsy had shown that he had chronic similar reported cases indicate that giving active hepatitis and cirrhosis. Hepatitis B prophylactic antibiotics to patients with cir- markers were negative. In 1983 he developed rhosis undergoing colonoscopy should be con- ascites and had remained on spironolactone since sidered, particularly when the cirrhosis is then. advanced. At the time ofhis colonoscopy he claimed that he felt reasonably well and was continuing to work. However, he had a moderate amount of Prophylactic antibiotics have been advised for ascites. His medication was: prednisolone 5 mg patients undergoing colonoscopy who have daily, spironolactone 200 mg daily, and sul- valvular heart disease, cardiac prostheses, severe phasalazine 1 g twice daily. Preoperative blood immunodepression, or hepatic cirrhosis with tests were: bilirubin 53 ,umol/1, alanine amino- ascites.' The last of these recommendations is transferase 38 IU/, alkaline phosphatase 206 IU/ based on a single case report in which it was not 1, albumin 28 g/l, prothrombin time 16 seconds certain that colonoscopy was responsible for the (control 14 seconds). infection, as hepatic angiography was performed After bowel preparation with three litres of the day before peritonitis developed.' We were Golytely, colonoscopy to the caecum was per- unaware of this case but our recent experience formed.
    [Show full text]
  • Guidelines on the Management of Ascites in Cirrhosis
    Downloaded from gut.bmjjournals.com on 25 September 2006 Guidelines on the management of ascites in cirrhosis K P Moore and G P Aithal Gut 2006;55;1-12 doi:10.1136/gut.2006.099580 Updated information and services can be found at: http://gut.bmjjournals.com/cgi/content/full/55/suppl_6/vi1 These include: References This article cites 148 articles, 21 of which can be accessed free at: http://gut.bmjjournals.com/cgi/content/full/55/suppl_6/vi1#BIBL Email alerting Receive free email alerts when new articles cite this article - sign up in the box at the service top right corner of the article Topic collections Articles on similar topics can be found in the following collections Liver, including hepatitis (945 articles) Notes To order reprints of this article go to: http://www.bmjjournals.com/cgi/reprintform To subscribe to Gut go to: http://www.bmjjournals.com/subscriptions/ Downloaded from gut.bmjjournals.com on 25 September 2006 vi1 GUIDELINES Guidelines on the management of ascites in cirrhosis K P Moore, G P Aithal ............................................................................................................................... Gut 2006;55(Suppl VI):vi1–vi12. doi: 10.1136/gut.2006.099580 1.0 INTRODUCTION N Grade 1 (mild). Ascites is only detectable by ultrasound examination. Ascites is a major complication of cirrhosis,1 occurring in 50% of patients over 10 years of N Grade 2 (moderate). Ascites causing moderate follow up.2 The development of ascites is an symmetrical distension of the abdomen. important landmark in the natural history of N Grade 3 (large). Ascites causing marked cirrhosis as it is associated with a 50% mortality abdominal distension.
    [Show full text]
  • Procedure Coding in ICD-9-CM and ICD- 10-PCS
    Procedure Coding in ICD-9-CM and ICD- 10-PCS ICD-9-CM Volume 3 Procedures are classified in volume 3 of ICD-9-CM, and this section includes both an Alphabetic Index and a Tabular List. This volume follows the same format, organization and conventions as the classification of diseases in volumes 1 and 2. ICD-10-PCS ICD-10-PCS will replace volume 3 of ICD-9-CM. Unlike ICD-10-CM for diagnoses, which is similar in structure and format as the ICD-9-CM volumes 1 and 2, ICD-10-PCS is a completely different system. ICD-10-PCS has a multiaxial seven-character alphanumeric code structure providing unique codes for procedures. The table below gives a brief side-by-side comparison of ICD-9-CM and ICD-10-PCS. ICD-9-CM Volume3 ICD-10-PCS Follows ICD structure (designed for diagnosis Designed and developed to meet healthcare coding) needs for a procedure code system Codes available as a fixed or finite set in list form Codes constructed from flexible code components (values) using tables Codes are numeric Codes are alphanumeric Codes are 3-4 digits long All codes are seven characters long ICD-9-CM and ICD-10-PCS are used to code only hospital inpatient procedures. Hospital outpatient departments, other ambulatory facilities, and physician practices are required to use CPT and HCPCS to report procedures. ICD-9-CM Conventions in Volume 3 Code Also In volume 3, the phrase “code also” is a reminder to code additional procedures only when they have actually been performed.
    [Show full text]
  • Ultra-Sound Guided Liver Biopsy
    Ultra-Sound Guided Liver Biopsy What is a liver biopsy? A liver biopsy is a procedure used for making the diagnosis of abnormal liver conditions. A small piece of liver tissue is removed using a special needle for examination under a microscope. The liver tissue allows the doctor to see if your liver is healthy or to better understand why you have liver damage or disease and how severe any damage is. The most common method of liver biopsy is percutaneously (“through the skin”). This procedure is often performed as an outpatient and does not routinely require hospital admission. A qualified gastroenterologist does the liver biopsy. This is a doctor who specializes in diseases of the digestive system and liver. Does the liver biopsy hurt? You may feel minor discomfort during the biopsy. Some people do have some discomfort at the site of the biopsy for the first 24 to 48 hours after the procedure but this is often relieved by simple painkillers such as Tylenol. Why do I need a liver biopsy? Your doctor will have discussed this with you or written to you about the need for a liver biopsy. If you have any questions, please ask. This test may be carried out for a number of reasons. Common indications include: Your symptoms, blood tests and scans (ultrasound, CT or MRI scans) suggest you have liver disease. However, sometimes it is not possible to tell what the cause is on the basis of these tests alone. There appears to be a lump in your liver which has been seen on previous scans and a sample of tissue is needed to identify what it is.
    [Show full text]
  • AASLD Position Paper : Liver Biopsy
    AASLD POSITION PAPER Liver Biopsy Don C. Rockey,1 Stephen H. Caldwell,2 Zachary D. Goodman,3 Rendon C. Nelson,4 and Alastair D. Smith5 This position paper has been approved by the AASLD and College of Cardiology and the American Heart Associa- represents the position of the association. tion Practice Guidelines3).4 Introduction Preamble Histological assessment of the liver, and thus, liver bi- These recommendations provide a data-supported ap- opsy, is a cornerstone in the evaluation and management proach. They are based on the following: (1) formal re- of patients with liver disease and has long been considered view and analysis of the recently published world to be an integral component of the clinician’s diagnostic literature on the topic; (2) American College of Physi- armamentarium. Although sensitive and relatively accu- cians Manual for Assessing Health Practices and De- rate blood tests used to detect and diagnose liver disease signing Practice Guidelines1; (3) guideline policies, have now become widely available, it is likely that liver including the AASLD Policy on the Development and biopsy will remain a valuable diagnostic tool. Although Use of Practice Guidelines and the American Gastro- histological evaluation of the liver has become important enterological Association Policy Statement on Guide- in assessing prognosis and in tailoring treatment, nonin- lines2; and (4) the experience of the authors in the vasive techniques (i.e., imaging, blood tests) may replace specified topic. use of liver histology in this setting, particularly with re- Intended for use by physicians, these recommenda- gard to assessment of the severity of liver fibrosis.5,6 Sev- tions suggest preferred approaches to the diagnostic, ther- eral techniques may be used to obtain liver tissue; a table apeutic, and preventive aspects of care.
    [Show full text]
  • Detailed Categories 2016-Update
    KNHSS Kuwait National Healthcare-associated Infections Surveillance System 5. Bile duct, liver or pancreatic surgery Excision of bile ducts or operative procedures on the biliary tract, liver or pancreas (does not include operations only on gallbladder) 50.0 Hepatotomy Incision of abscess of liver Removal of gallstones from liver Stromeyer-Little operation 50.12 Open biopsy of liver Wedge biopsy 50.14 Laparoscopic liver biopsy Excludes: Closed (percutaneous)[needle] biopsy of liver (50.11) Transjugular liver biopsy (50.13) 50.21 Marsupialization of lesion of liver 50.22 Partial hepatectomy Wedge resection of liver Excludes: Closed (percutaneous)[needle] biopsy of liver (50.11) 50.23 Open ablation of liver lesion or tissue 50.25 Laparoscopic ablation of liver lesion or tissue 50.26 Other and unspecified ablation of liver lesion or tissue 50.29 Other destruction of lesion of liver Cauterization of hepatic lesion Enucleation of hepatic lesion Evacuation of hepatic lesion Excludes: Percutaneous ablation of liver lesion or tissue (50.24) Percutaneous aspiration of lesion (50.91) Laser interstitial thermal therapy [LITT] of lesion or tissue of liver under guidance(17.63) 1 KNHSS Kuwait National Healthcare-associated Infections Surveillance System 50.3 Lobectomy of liver 50.4 Total hepatectomy 50.61 Closure of laceration of liver 50.69 Other repair of liver Hepatopexy 51.31 Anastomosis of gallbladder to hepatic ducts 51.32 Anastomosis of gallbladder to intestine 51.33 Anastomosis of gallbladder to pancreas 51.34 Anastomosis of
    [Show full text]
  • Liver Biopsy
    William F. Erber, M.D., P.C. Gastroenterology and Endoscopy Diseases of the Digestive Tract, Liver and Pancreas Board Certified William F. Erber, M.D., F.A.C.P., F.A.C.G., A.G.A.F. 591 Ocean Parkway Jonathan A. Erber, M.D. Brooklyn, N.Y. 11218 Tel (718) 972-8500 Fax (718) 972-0064 www.drerber.com Liver Biopsy In a liver biopsy (BYE-op-see), the physician examines a small piece of tissue from your liver for signs of damage or disease. A special needle is used to remove the tissue from the liver. The physician decides to do a liver biopsy after tests suggest that the liver does not work properly. For example, a blood test might show that your blood contains higher than normal levels of liver enzymes or too much iron or copper. An x ray could suggest that the liver is swollen. Looking at liver tissue itself is the best way to determine whether the liver is healthy or what is causing it to be damaged. Preparation Before scheduling your biopsy, the physician will take blood samples to make sure your blood clots properly. Be sure to mention any medications you take, especially those that affect blood clotting, like blood thinners. One week before the procedure, you will have to stop taking aspirin, ibuprofen, and anticoagulants. The digestive system You must not eat or drink anything for 8 hours before the biopsy, and you should plan to arrive at the hospital about an hour before the scheduled time of the procedure. Your physician will tell you whether to take your regular medications during the fasting period and may give you other special instructions.
    [Show full text]
  • Chyle Leak Post Laparoscopic Cholecystectomy: a Case Report, Literature Review and Management Options
    7 Case Report Page 1 of 7 Chyle leak post laparoscopic cholecystectomy: a case report, literature review and management options Ferdinand Ong1, Amitabha Das1, Kheman Rajkomar2 1Department of Upper Gastrointestinal Surgery, Liverpool Hospital, Sydney, NSW, Australia; 2Department of Upper Gastrointestinal Surgery, Bankstown-Lidcombe Hospital, Sydney, NSW, Australia Correspondence to: Dr. Kheman Rajkomar. Eldridge Road, Bankstown-Lidcombe Hospital, Bankstown NSW 2200, Sydney, Australia. Email: [email protected]. Abstract: Chyle leak after a laparoscopic cholecystectomy (LC) is very rarely reported. However, it is needs to be recognised promptly and managed as otherwise it can lead to further metabolic and infective complications. We present the case of a 48 years old man who was admitted with ultrasound proven acute calculous cholecystitis. His vital signs were within normal range but his murphy’s sign was positive. His white cell count (WCC) and liver function tests were within normal limit. He underwent an uneventful standard LC with cholangiography during the same admission with no anomalous biliary or hepatic arterial anatomy noted during the procedure. Post operatively he was noted to have 125 mL of white fluid in his drain. The fluid triglyceride was 23.2 mmol/L, cholesterol level was 2.8 mmol/L, and drain/serum triglyceride of 15.5, hence confirming it to be chyle. He was clinically otherwise very well. He was managed conservatively as a low volume chyle leak with a fat free diet. The triglyceride content in the drain effluent decreased to 1.3mmol/L by day 6 and the fluid turned straw coloured in that interval. The drain was removed and the patient discharged home without any further issues.
    [Show full text]
  • Perforated Duodenal Ulcer an Alternative Therapeutic Plan
    SPECIAL ARTICLE Perforated Duodenal Ulcer An Alternative Therapeutic Plan Arthur J. Donovan, MD; Thomas V. Berne, MD; John A. Donovan, MD n alternative plan for the treatment of a perforated duodenal ulcer is proposed. We will focus on the now-recognized role of Helicobacter pylori in the genesis of the ma- jority of duodenal ulcers and on the high rate of success of therapy with a combina- tion of antibiotics and a proton-pump inhibitor or histamine2 blocker in treatment of suchA ulcers. Knowledge that half the cases of perforated duodenal ulcer may have securely sealed spontaneously at the time of presentation is incorporated in the therapeutic plan. Patients with a perforated duodenal ulcer who have already been evaluated for H pylori and are not infected or, if infected, have received appropriate therapy should undergo an ulcer-definitive operation if they are suitable surgical candidates. Most authorities recommend surgical closure of the perforation and a parietal cell vagotomy. The remaining patients should have a gastroduodenogram with water- soluble contrast medium. If the perforation is sealed, the patient can be treated nonsurgically. If the perforation is leaking, secure surgical closure of the perforation is necessary. Following recov- ery from the immediate consequences of the perforation, evaluation for H pylori should be con- ducted. If the patient is infected, combined medical therapy is recommended. If the patient is not infected, Zollinger-Ellison syndrome should be ruled out and medical therapy is recommended if the ulcer has not been treated previously. Elective ulcer-definitive surgery should be considered for the occasional uninfected patient who has already received appropriate medical therapy for the ulcer.
    [Show full text]